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Anthony Roberts
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- Ross - said:
Which, according to the literature on it, would be equivalent to 1mg of the injectable version....
*The Perpetual Muscle Mass EXPLOSION: Pre-PCT(Active Recovery), Bridging & Cruising*
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Which, according to the literature on it, would be equivalent to 1mg of the injectable version....
R
Ross
Guest
Anthony Roberts said:
Unfortunately primobolan is highly underdosed, so most likely that is the underlying issue. Even considering the poor bioavailability of Acetate.
300-400mgs of SCHERINGS and I am GOLDEN..
R
Ross
Guest
jmead said:
Who's Primobolan matched the label claim? Most Primobolan is underdosed--SEVERELY.
Every single anabolic steroid has an androgenic component. This androgenicity causes a negative feedback loop in the hypothalamo-hypophysial testicular axis.
Thus, every anabolic steroid will negatively affect your HPTA. Every anabolic steroid will supress your endogeneous testosterone production to some extent.
The above premise and conclusion have been demonstrated beyond any resonable doubt by the medical and scientific communities from many decades of research and experiments and is widely published and accepted. It is extremely basic knowledge, and I would hope that anyone touting anabolic steroid use would be familar with it a priori. I can easily provide well over a hundred references from peer-reviewed studies and research published in areas of endocrinology and many other areas of medical science if you care to look into your errors in reasoning.
The fact that you seem to think otherwise shows a severe lack of knowledge -- and the internets do not count as knowledge when it comes to something as complex as medical science, pharmacodynamics the endocrine system. Extorting the lack of black-and-white differences between "complete shutdown" and it's opposite would be a far more credited choice than fallaciously stating that xyz steroids do not inhibit the HTHP testicular axis. However, I do not believe you even understand the impact of exogeneous androgens on the human organism if you state definitively that everyone is completely "shut down" after each and every cycle.
Please dig a bit deeper into some biochemical and medical literature before explicitly stating something that is blatently false. I would suggest a good text from Yesalis to begin with.
The amount of disinformation and lack of knowledge on today's steroid forums really scares me. People -- actual research is not done on internet discussion boards. A good starting point would be your local library.
Thus, every anabolic steroid will negatively affect your HPTA. Every anabolic steroid will supress your endogeneous testosterone production to some extent.
The above premise and conclusion have been demonstrated beyond any resonable doubt by the medical and scientific communities from many decades of research and experiments and is widely published and accepted. It is extremely basic knowledge, and I would hope that anyone touting anabolic steroid use would be familar with it a priori. I can easily provide well over a hundred references from peer-reviewed studies and research published in areas of endocrinology and many other areas of medical science if you care to look into your errors in reasoning.
The fact that you seem to think otherwise shows a severe lack of knowledge -- and the internets do not count as knowledge when it comes to something as complex as medical science, pharmacodynamics the endocrine system. Extorting the lack of black-and-white differences between "complete shutdown" and it's opposite would be a far more credited choice than fallaciously stating that xyz steroids do not inhibit the HTHP testicular axis. However, I do not believe you even understand the impact of exogeneous androgens on the human organism if you state definitively that everyone is completely "shut down" after each and every cycle.
Please dig a bit deeper into some biochemical and medical literature before explicitly stating something that is blatently false. I would suggest a good text from Yesalis to begin with.
The amount of disinformation and lack of knowledge on today's steroid forums really scares me. People -- actual research is not done on internet discussion boards. A good starting point would be your local library.
R
Ross
Guest
samoth said:Every single anabolic steroid has an androgenic component. This androgenicity causes a negative feedback loop in the hypothalamo-hypophysial testicular axis.
Thus, every anabolic steroid will negatively affect your HPTA. Every anabolic steroid will supress your endogeneous testosterone production to some extent.
The above premise and conclusion have been demonstrated beyond any resonable doubt by the medical and scientific communities from many decades of research and experiments and is widely published and accepted. It is extremely basic knowledge, and I would hope that anyone touting anabolic steroid use would be familar with it a priori. I can easily provide well over a hundred references from peer-reviewed studies and research published in areas of endocrinology and many other areas of medical science if you care to look into your errors in reasoning.
The fact that you seem to think otherwise shows a severe lack of knowledge -- and the internets do not count as knowledge when it comes to something as complex as medical science, pharmacodynamics the endocrine system. Extorting the lack of black-and-white differences between "complete shutdown" and it's opposite would be a far more credited choice than fallaciously stating that xyz steroids do not inhibit the HTHP testicular axis. However, I do not believe you even understand the impact of exogeneous androgens on the human organism if you state definitively that everyone is completely "shut down" after each and every cycle.
Please dig a bit deeper into some biochemical and medical literature before explicitly stating something that is blatently false. I would suggest a good text from Yesalis to begin with.
The amount of disinformation and lack of knowledge on today's steroid forums really scares me. People -- actual research is not done on internet discussion boards. A good starting point would be your local library.
EVERY anabolic steroid will AFFECT YOUR HPTA!
NOT EVERY ANABOLIC STEROID WILL CAUSE SHUTDOWN!
Primobolan will suppress your endogenous testosterone production, but it will NOT SHUTDOWN THE HPTA, even in fairly high dosages.
Not even Dianabol will SHUTDOWN THE HPTA in EXTREMELY HIGH DOSAGES OF 100mgs ED:
"In this study, done in the early 80´s, a very high dose of Dbol (100mgs/day for 6 weeks) decreased plasma testosterone to about 40% of it´s normal value, plasma GH went up about a third, LH dropped to about 80% of it´s original value, and FSH went down about a third also. "
DECA HOWEVER, will shutdown the HPTA with one single SHOT:
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- Ross - said:EVERY anabolic steroid will AFFECT YOUR HPTA!
NOT EVERY ANABOLIC STEROID WILL CAUSE SHUTDOWN!
Every anabolic steroid will cause a negative feedback loop affecting one's HPTA. By the same reasoning, every steroid can theoretically cause "absolute" shutdown, and, conversely, every steroid has the potential to not cause "absolute" shutdown. Dancing in circles leads nowhere and shows nothing.
Your flaw in reasoning is seeing this "shutdown" as an absolute black-and-white idea. This is incorrect.
Please provide publications demonstrating the latter.
Oh, wait, you're referring to this absolute shutdown concept again, aren't you?
I really don't think any anabolic steroid will cause this absolute complete shutdown you keep mentioning. The axis to which you're referring is not thought of in such terms, thus your argumentative foundation cannot be expounded.
Incorrect.
Umm... you know what that graph is of, right? Hint: Check the axes. It says absolutely nothing regarding endogeneous hormone production, merely the decay rate in time as a function of plasma concentrations for different esterifications of nandrolone administered at different sites.
I'm really starting to wonder if this is a joke account or something. You keep reiterating stuff that doesn't make sense while backing it up with either unrelated or unsubstantiated attempt at evidence.
Bruce
New member
not mine haha. 200/ml 50%eo in grapeseed oil, couldn't be smoother or more ovoerdosed lol- Ross - said:Unfortunately primobolan is highly underdosed, so most likely that is the underlying issue. Even considering the poor bioavailability of Acetate.
300-400mgs of SCHERINGS and I am GOLDEN..
85% of medical research is bunk. That's why there's a new movement in health sciences called EBM(Evidence Based Medicine). Before believing wholeheartedly in any study, I would first check to see if it follows the EBM process of research. Studies that have been proven have also been disproven based on invalid research methods. I'm not saying anyone's suggested studies are bunk, but there's a good chance some of it is based on the numbers.
Bruce
New member
he seems like a good bro but i agree seems like a computer or something sometimes. we went at it over dost opinions and he just keeps spewing the same advertisment looking responses. but maybe he just sticks by his point that firmly. either way he copy and pastes
samoth said:Every anabolic steroid will cause a negative feedback loop affecting one's HPTA. By the same reasoning, every steroid can theoretically cause "absolute" shutdown, and, conversely, every steroid has the potential to not cause "absolute" shutdown. Dancing in circles leads nowhere and shows nothing.
Your flaw in reasoning is seeing this "shutdown" as an absolute black-and-white idea. This is incorrect.
Please provide publications demonstrating the latter.
Oh, wait, you're referring to this absolute shutdown concept again, aren't you?
I really don't think any anabolic steroid will cause this absolute complete shutdown you keep mentioning. The axis to which you're referring is not thought of in such terms, thus your argumentative foundation cannot be expounded.
Incorrect.
Umm... you know what that graph is of, right? Hint: Check the axes. It says absolutely nothing regarding endogeneous hormone production, merely the decay rate in time as a function of plasma concentrations for different esterifications of nandrolone administered at different sites.
I'm really starting to wonder if this is a joke account or something. You keep reiterating stuff that doesn't make sense while backing it up with either unrelated or unsubstantiated attempt at evidence.
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