The 25 MOST ASKED (and misunderstood) Questions On EF. Don't Ask Again!

[Ross]

Well, pal, you can use lots of smiley faces and call me bro all day long but you tend to be a little disparaging in a passive aggressive way and frankly you're out of your league. You may have done a lot of gear but knowing what's best in all applications is a different story.

I don't know why you are taking my criticisms of your article personally, but if you want we can post up pictures.

Seriously though, I actually like you Nelson and find most of your posts not only accurate and informative but also entertaining. Sorry if I offended you in any way, that's just my personality my friend. On the internet, I am often misunderstood...

 

[the_alcatraz]

The Bridge
By Ross



Post-cycle regimens containing Aromatase Inhibitors and SERM's are simply not enough for the SERIOUS bodybuilder to maintain his muscular gains post-cycle. Once a bodybuilders reaches a certain point of muscular development, the continued use of a mild anabolic becomes justified..

The steroid user has TWO options:

1.) A Bridge
2.) A Cruise

In this chapter, we discuss the purpose of the BRIDGE.

The Bridge allows you to remain in an anabolic state while simultaneously having a MINIMAL intereference with HPTA function. Once you are FULLY RECOVERED and your PCT is complete, you can begin bridging while awaiting your FULL CYCLE. This will allow you to make GREATER THAN NATURAL GAINS, while still maintaining normal testosterone levels.

Bridging can ONLY be accomplished using a very specific and limited number of compounds. The selected compound must first be MINIMALLY supressive to the HPTA, and secondly, must still be healthy and effective in small dosages.

The following steroid combinations can be used effectively for Bridging.

Anavar/Proviron= 20mgs/25mgs
Anavar/Masteron= 20mgs/300mgs
Primobolan/Masteron= 200mgs/200mgs
Turinabol/Proviron= 30mgs/25mgs
Turinabol/Masteron= 30mgs/300mgs
Winstrol/Masteron= 25mgs/200mgs
Dianabol/Proviron= 15mgs/25mgs
Dianabol/Masteron= 15mgs/200mgs

**ADD AndroGenerator to COMPLETELY minimize HPTA inhibition!


NOT ALL ANDROGENS CAUSE SHUTDOWN*

"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.

SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol, masteron, proviron, halo, primo)

Very Androgenic/Progestenic/Estrogenic steroids(Tren, Deca, Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.

The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.
-------------------------------------------------------------------------

Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links

Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.

Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.

We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS



[R]

Dude, a bridge between cycles is an illusion. You're either "on" or "off"

 

[needtogetaas]

I just love threads like this. Lets keep it clean.

 

[needtogetaas]

Briging is a bunch of bullshit. You are ether on for life or you are not.

 

[Ross]

Briging is a bunch of bullshit. You are ether on for life or you are not.

The notion that you are either "ON OR OFF" is based on the FALSE ASSUMPTION that all steroids cause HPTA shutdown, when this is NOT TRUE.

I have successfully bridged with MANY COMPOUNDS, meaning, I was able to maintain an AVERAGE testosterone level while still receiving additional anabolic support, allowing me to make MUCH GREATER THAN NATURAL GAINS.

Have I convinced you my bro?

 

[Tatyana]

The Bridge
By Ross



Post-cycle regimens containing Aromatase Inhibitors and SERM's are simply not enough for the SERIOUS bodybuilder to maintain his muscular gains post-cycle. Once a bodybuilders reaches a certain point of muscular development, the continued use of a mild anabolic becomes justified..

The steroid user has TWO options:

1.) A Bridge
2.) A Cruise

In this chapter, we discuss the purpose of the BRIDGE.

The Bridge allows you to remain in an anabolic state while simultaneously having a MINIMAL intereference with HPTA function. Once you are FULLY RECOVERED and your PCT is complete, you can begin bridging while awaiting your FULL CYCLE. This will allow you to make GREATER THAN NATURAL GAINS, while still maintaining normal testosterone levels.

Bridging can ONLY be accomplished using a very specific and limited number of compounds. The selected compound must first be MINIMALLY supressive to the HPTA, and secondly, must still be healthy and effective in small dosages.

The following steroid combinations can be used effectively for Bridging.

Anavar/Proviron= 20mgs/25mgs
Anavar/Masteron= 20mgs/300mgs
Primobolan/Masteron= 200mgs/200mgs
Turinabol/Proviron= 30mgs/25mgs
Turinabol/Masteron= 30mgs/300mgs
Winstrol/Masteron= 25mgs/200mgs
Dianabol/Proviron= 15mgs/25mgs
Dianabol/Masteron= 15mgs/200mgs

**ADD AndroGenerator to COMPLETELY minimize HPTA inhibition!


NOT ALL ANDROGENS CAUSE SHUTDOWN*

"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.

SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol, masteron, proviron, halo, primo)

Very Androgenic/Progestenic/Estrogenic steroids(Tren, Deca, Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.

The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.
-------------------------------------------------------------------------

Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links

Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.

Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.

We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS



[R]

You are referencing a 24 year old paper, when I know more recent work has been done with regards to men who have complete HPTA shutdown due to things like prostate cancer therapy.

There are old papers that are useful, with research that will never be reproduced, however, I would find some newer data.

 

[splperformance]

Briging is a bunch of bullshit. You are ether on for life or you are not.

+1...certain people calls it bridging but in reality it is a very low dosed aas cycle with a few compounds that you take in between very high dosed cycles with a bunch of mixed compounds...

off aas = NO aas at all...NONE
on aas = taking any aas no matter what it is or what dose it's being taken at

so yes, needto is right...you're either on for life (or a long ass time) or you're off...there is no grey space when it comes to gear

 

[the_alcatraz]

Guys let's stick to debating and attacking ideas and opinions presented and not turn this into a drama thread.

The members reading this thread care about which ideas are best and can help them grow, not who's dick is bigger.

The personal stuff is for PM's.

Agreed. Good post before things get out of control and people start bashing each other. LOL

 

[Tatyana]

The notion that you are either "ON OR OFF" is based on the FALSE ASSUMPTION that all steroids cause HPTA shutdown, when this is NOT TRUE.

I have successfully bridged with MANY COMPOUNDS, meaning, I was able to maintain an AVERAGE testosterone level while still receiving additional anabolic support, allowing me to make MUCH GREATER THAN NATURAL GAINS.

Have I convinced you my bro?

One person's experience is not evidence that steroids do not cause HPTA shutdown.

If you want to prove this as an individual, you need some data, like posting your blood results when on, bridging and then off (if you do come off).

 

[Nelson Montana]

One person's experience is not evidence that steroids do not cause HPTA shutdown.

If you want to prove this as an individual, you need some data, like posting your blood results when on, bridging and then off (if you do come off).

Exactly. And how can anyone say they maintained normal T levels while bridging? If you're bridging, you're ON!

As for the winstrol/drol debate, I still think there's a misconception here. I'm speaking in terms of building muscle tissue. Not temporary strength gains, or size or water. 100 mgs of winstrol a day will build more muscle than 100 mgs of drol a day.