Hemocrit levels?

[MASSIVEMONSTER]

A friend of mine says the reason my BP boots up from 115-75 to bloody 140-90 while on ONLY TEST is cos my blood thickens and he says take aspirin to sort it.
My last cycle i took hawthorn, loads vit c, magnesium, potassium, did cv, ate low fat. you name it, i did it and my bp was still borderline. i have now come off all this stuff and off gear and my bp has dropped to 115-75 again. how can it go up this much, it makes me angry and confused. will taking aspirin do the job? any other ideas. I know this has been posted before. dont scream at me but just wondering about upcoming summer cycle... dont want high bp all summer.

MM

 

[MASSIVEMONSTER]

oh and yes i will be adding adex this time out but did add some towards end last cycle and didnt bring bp down much. Can your blood get thicker?

 

[todoveritas]

oh and yes i will be adding adex this time out but did add some towards end last cycle and didnt bring bp down much. Can your blood get thicker?

Your well-meaning friend is steering you down the wrong road. Although AAS do increase erythropoiesis (RBC production) via androgen receptors located in the kidney, and while increased RBC mass may decrease blood viscosity, this does not necessarily have anything to do with your blood pressure. Two separate mechanisms.

Yes, if you are concerned about blood viscosity, then one aspirin tablet a day will do the trick. Careful though, evidence is showing that AAS may already inhibit various clotting factors. (Incidentally, blood thickness induced by Erythropoetin was thought to be instrumental in causing the death of Andreas Muntzer.)

The HTN attributed to AAS use is thought to be induced by the salt and water retention. Because the body is overloaded with water, the heart and blood vessels must transport more fluid than normal through the body, thus resulting in an elevated blood pressure. It is here where Adex exerts its effect.

 

[ur2insecure]

Why not see a Dr....

Your BP is mentioned in all your posts, so it must be of real concern to you, he will be the best person to advise/reassure you.

 

[MASSIVEMONSTER]

my doc like all the ones i have had have no idea about gear. Theyll just say dont take it without giving out decent advice

 

[itlnstln]

Why not see a Dr....

Your BP is mentioned in all your posts, so it must be of real concern to you, he will be the best person to advise/reassure you.

Or you could just post it 30 different ways in the next 4min and see what happens

 

[maldorf]

Your well-meaning friend is steering you down the wrong road. Although AAS do increase erythropoiesis (RBC production) via androgen receptors located in the kidney, and while increased RBC mass may decrease blood viscosity, this does not necessarily have anything to do with your blood pressure. Two separate mechanisms.

Yes, if you are concerned about blood viscosity, then one aspirin tablet a day will do the trick. Careful though, evidence is showing that AAS may already inhibit various clotting factors. (Incidentally, blood thickness induced by Erythropoetin was thought to be instrumental in causing the death of Andreas Muntzer.)

The HTN attributed to AAS use is thought to be induced by the salt and water retention. Because the body is overloaded with water, the heart and blood vessels must transport more fluid than normal through the body, thus resulting in an elevated blood pressure. It is here where Adex exerts its effect.

Yeah, there is an increase in blood volume because of all the retained water. Need to take an Anti-E like adex at sufficient dosage.

 

[USHP]

Or you could just post it 30 different ways in the next 4min and see what happens

Here we go, I just read this. Note what I highlighted.



Factors influencing blood pressure
The physics of the circulatory system, as of any fluid system, are very complex. That said, there are many physical factors that influence blood pressure. Each of these may in turn be influenced by physiological factors, such as diet, exercise, disease, drugs, etc.

Some physical factors are:

Rate of pumping. In the circulatory system, this rate is called heart rate, the rate at which blood (the fluid) is pumped by the heart. The higher the heart rate, the higher (potentially, assuming no change in stroke volume) the blood pressure.
Volume of fluid. In the case of the circulatory system, this is blood volume, the amount of blood present in the body. The more blood present in the body, the higher the rate of blood return to the heart and the resulting cardiac output. There is some relationship between dietary salt intake and increased blood volume, potentially resulting in higher blood pressure, though this varies with the individual and is highly dependent on autonomic nervous system response.
In cardiac physiology, the rate and volume of flow are accounted for in a combined fashion by cardiac output. Cardiac output is the product of the heart rate, or the rate of contraction, multiplied by the stroke volume, the amount of blood pumped out from the heart with each contraction. Basically, it represents the efficiency with which the heart circulates the blood throughout the body.
Resistance. In the circulatory system, this is the resistance of the blood vessels. The higher the resistance, the higher the blood pressure. Resistance is related to size (The larger the blood vessel, the lower the resistance), as well as the smoothness of the blood vessel walls. Smoothness is reduced by the buildup of fatty deposits on the arterial walls. Substances called vasoconstrictors can reduce the size of blood vessels, thereby increasing blood pressure. Vasodilators (such as nitroglycerin) increase the size of blood vessels, thereby decreasing blood pressure.
Viscosity, or thickness of the fluid. If the blood gets thicker, the result is an increase in blood pressure. Certain medical conditions can change the viscosity of the blood. For instance, low red blood cell concentration, anemia, reduces viscosity, whereas increased red blood cell concentration increases viscosity. (The effect of so-called blood thinners are not on viscosity but on ability of the blood to clot, thus a misnomer.)
In practice, each individual's autonomic nervous system responds to and regulates all these interacting factors so that, although the above issues are important, the actual blood pressure response of a given individual varies widely because of both split-second and slow-moving responses of the nervous system and end organs. These responses are very effective in changing the variables and resulting blood pressure from moment to moment.



I just read that at Wikipedia the online encyclopedia. I'm not saying it's correct but it didn't take me long to find a completely opposite statement then what todoveritas posted and I wasn't trying to refute his claim. The world of info on the internet is very strange.

 

[todoveritas]

Here we go, I just read this. Note what I highlighted.



Viscosity, or thickness of the fluid. If the blood gets thicker, the result is an increase in blood pressure. Certain medical conditions can change the viscosity of the blood. For instance, low red blood cell concentration, anemia, reduces viscosity, whereas increased red blood cell concentration increases viscosity.
I just read that at Wikipedia the online encyclopedia. I'm not saying it's correct but it didn't take me long to find a completely opposite statement then what todoveritas posted and I wasn't trying to refute his claim. The world of info on the internet is very strange.

Good job! We all need to question what is told to us as "fact" and I applaud anyone smart enough to do their own research.
However, my information comes from a recent article in a respected medical journal (Hypertension. 2005;45:625.) which actually followed blood viscosity levels in 700 patients. Following anectodal evidence that abnormalities in whole blood viscosity (WBV) were implicated in hypertension. "In apparent contrast with previous reports, the present study found that arterial hypertension was not associated with WBV estimated using a regression equation ...or hematocrit." WBV was negatively related to pulse pressure (ß=–0.13; P<0.001) and systolic pressure (ß=–0.09; P<0.02), mainly because of negative relations with hematocrit (ß=–0.11 and –0.10).

It is thought that the prrevious studies used mainly diastolic pressure to diagnose hypertension (ie, a measure mostly related to increased peripheral resistance and therefore expected to be related to higher WBV and hematocrit); this study used a more accurate estimate of blood pressure.

Thanks for keeping me on my toes!

 

[USHP]

Good job! We all need to question what is told to us as "fact" and I applaud anyone smart enough to do their own research.
However, my information comes from a recent article in a respected medical journal (Hypertension. 2005;45:625.) which actually followed blood viscosity levels in 700 patients. Following anectodal evidence that abnormalities in whole blood viscosity (WBV) were implicated in hypertension. "In apparent contrast with previous reports, the present study found that arterial hypertension was not associated with WBV estimated using a regression equation ...or hematocrit." WBV was negatively related to pulse pressure (ß=–0.13; P<0.001) and systolic pressure (ß=–0.09; P<0.02), mainly because of negative relations with hematocrit (ß=–0.11 and –0.10).

It is thought that the prrevious studies used mainly diastolic pressure to diagnose hypertension (ie, a measure mostly related to increased peripheral resistance and therefore expected to be related to higher WBV and hematocrit); this study used a more accurate estimate of blood pressure.

Thanks for keeping me on my toes!

Yea, I wasn't taking it too seriously because of the source but goes to show how much conflicting info is out there. I'm doing all kinds of research on PCT so you can just imagine how confused that is getting. I began the research based on my gut feeling that HCG is being used in dosages that are too high and sometimes it's being used too frequently generally speaking.