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Hemocrit levels?

g mac said:
Yea, I wasn't taking it too seriously because of the source but goes to show how much conflicting info is out there. I'm doing all kinds of research on PCT so you can just imagine how confused that is getting. I began the research based on my gut feeling that HCG is being used in dosages that are too high and sometimes it's being used too frequently generally speaking.

I totally agree with you g mac. I try to keep as close as possible to legitimate medical journals. By the way, I think you're right on in the hCG department, and far too many people overuse it in their cycles. It's not as harmless as most people think and you're in a world of hurt if you oversensitize those Leydig cells and develop androgen resistance syndrome. Personally, I think 1000-1500 units Q wk is fine for PCT. Here's a link to an article you might find interesting in your research.

http://jcem.endojournals.org/cgi/content/abstract/90/5/2595
 
todoveritas said:
I totally agree with you g mac. I try to keep as close as possible to legitimate medical journals. By the way, I think you're right on in the hCG department, and far too many people overuse it in their cycles. It's not as harmless as most people think and you're in a world of hurt if you oversensitize those Leydig cells and develop androgen resistance syndrome. Personally, I think 1000-1500 units Q wk is fine for PCT. Here's a link to an article you might find interesting in your research.

http://jcem.endojournals.org/cgi/content/abstract/90/5/2595

Saved it. Thanks
 
todoveritas said:
Your well-meaning friend is steering you down the wrong road. Although AAS do increase erythropoiesis (RBC production) via androgen receptors located in the kidney, and while increased RBC mass may decrease blood viscosity, this does not necessarily have anything to do with your blood pressure. Two separate mechanisms.

Yes, if you are concerned about blood viscosity, then one aspirin tablet a day will do the trick. Careful though, evidence is showing that AAS may already inhibit various clotting factors. (Incidentally, blood thickness induced by Erythropoetin was thought to be instrumental in causing the death of Andreas Muntzer.)

The HTN attributed to AAS use is thought to be induced by the salt and water retention. Because the body is overloaded with water, the heart and blood vessels must transport more fluid than normal through the body, thus resulting in an elevated blood pressure. It is here where Adex exerts its effect.

It is also due to the extra amount of blood being carried in the body while on AAS.
 
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