Please Scroll Down to See Forums Below needtogetaas
New member
THis is what I would call it. Bridge cruise what ever you call it its the same thing. Cruising gives it more of that "yes your still on" feel in the name.
Bridging
- Thread starter ryno9000
- Start date
THis is what I would call it. Bridge cruise what ever you call it its the same thing. Cruising gives it more of that "yes your still on" feel in the name.
I completely agree with your blast and cruise thread, but this is not bridging.
Bridging is a myth. No matter what dose you take of aas, you are on. Low, high, moderate, you are on.
Calling it bridging and thinking that it does not suppress you, IMO is an illogical statement and does not show much of an understanding of aas. That is all I'm saying.
I think you would agree that the term bridging is incorrect.
Cruise just means you're on, but at low dosages. I have nothing against this word, because you will still be supressed and will still get sides, even though you are "cruising"
But claiming that you can 'bridge" and it won't supress you, because bridging is something you do in between cycles is not a very good thing to do.
needtogetaas
New member
Yes this is true. I mean at the heart of it I truly feel that if you are going to bridge or cruise you should understand that (never fully recovering) becomes more and more of a real possibility. Can you still recover if you did a long blast and cruise cycle. Yes you can but the risk of never recovering goes up a lot.
The thought of staying on for life should not scare you if you plan on doing this. Because its a real possibility.
No one claimed that bridging will allow FULL HPTA recovery. No one stated that bridging will not suppress you.
If you read my article, I state that bridging is only to be used by advanced bodybuilders, where maintaining muscle mass is more important than a full HPTA recovery at THAT time.
Run 200-300mg of Primo with 50mg Proviron and BOOM; you are bridging EFFECTIVELY. Such a bridge will allow you to remain in a highly anabolic state while having a minimal influence on the HPTA. Of course, full HPTA recovery while still on any compound is virtually impossible.
All of the compounds I listed have a minimal influence on the HPTA at the dosages I prescribed. Even Dianabol, a strong androgenic steroid with moderate estrogenic properties, BARELY even reduced testosterone levels at 100mg ED!
"In this study, done in the early 80´s, a very high dose of Dbol (100mgs/day for 6 weeks) decreased plasma testosterone to about 40% of it´s normal value, plasma GH went up about a third, LH dropped to about 80% of it´s original value, and FSH went down about a third also"
Take this study on Proviron, another androgenic steroid:
Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links
Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.
Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.
We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS
iceman7230
New member
Ross hit the spot. No one ever said bridging was not supressive. Call it what you want bridging or crusing. It is used between cycles in order to stay on while not being as supressed as on a higher dosed cycle.
I can't beleive people get so uspet about this topic LOL!
Shit I am starting to get angry HA HA, better lower the masteron dose!
I can't beleive people get so uspet about this topic LOL!
Shit I am starting to get angry HA HA, better lower the masteron dose!
Okay, I can buy that ...BUT...there are many variables. For example, if you do a very suppressive cycle, bridging will keep you suppressed longer and possibly shut you down irreparably. But if you're still producing some T and go on low dose Primo, you might be able to thread in the same spot for a while. Of course, this is all speculation, which leads us to the same point -- either recover, or stay on.
