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The doctor told me I will be Dead in a year...

I'm sorry to hear this news.... However, don't get all depressed and have a pity party for yourself daily! You will be able to control this with meds and a proper diet and some form of training. It's not the end of the world, unless you do not follow a new healthy lifestyle.

Good luck and I hope that you love yourself enough to change. :)
 
Better than having a stroke and needing to be spoon fed I suppose.

Seriously get your bp meds in check and cut out the sodium.

And I assume you came to www.elitefitness.com because you have an interest in the gym. Start going
 
mountain muscle said:
Renal artery stenosis doesn't sound like fun.
It's easily treated with a stent. It can cause renal failure if not detected early, and it can cause severe hypertension if it isn't identified and treated. That severe hypertension will lead to cardiac failure, stroke, etc. as the hypertension from RAS is often uncontrollable without stents to open up the renal artery again.

I never mentioned this before, but if you do not have RAS, I would ask them to check your cortisol/aldosterone levels and to check your urine catecholamines. RAS is the most common cause of malignant hypertension, but if you have episodes of headaches, sweating, chest pain, etc., then the urine catecholamines should definitely be checked to make sure you do not have a pheochromocytoma.

Blood pressure that high is more often from an identified and treatable cause.

While I was in residency, my cousin called me at work one day asking that I speak to a physician who was discharging her husband. He had been admitted with chest pain and found to have an elevated CPK (about 6,000, normal is <400) and severe hypertension (220/110) on presentation. They were discharging him on three blood pressure medicines, one of which they started in the hospital and made his blood pressure worse (it was metoprolol for what it's worth, which has a paradoxical effect in pheochromocyoma and actually makes your blood pressure increase in the majority of patients with a pheo). When I talked to the physician and told him to check his urine catecholamines, he laughed and said I was a resident who had "Ivy league syndrome" and said that it was so rare it's not worth checking. My cousin and her husband insisted on it. Guess what? He had it. After surgery to remove it, he's now doing well without any medicines at all.

I would insist that your physician check for RAS first, and if that is negative, then to check for a pheo (urinary catecholamines) and hyperaldosteronism or other endocrine disorders (aldosterone, cortisol, thyroid, etc.).
 
swatdoc said:
It's easily treated with a stent. It can cause renal failure if not detected early, and it can cause severe hypertension if it isn't identified and treated. That severe hypertension will lead to cardiac failure, stroke, etc. as the hypertension from RAS is often uncontrollable without stents to open up the renal artery again.

Hey Swat, do the ace inhibitors make you dehydrated? I have been drinking almost two gallons a day and keep water by my bed and still have constant dry mouth and am thirsty.

Only other thing I am taking is amoxicilan.

BP seems to be going down too.

It was 165/100 an hour ago. Might go up during the Avs game though.
 
mountain muscle said:
Hey Swat, do the ace inhibitors make you dehydrated? I have been drinking almost two gallons a day and keep water by my bed and still have constant dry mouth and am thirsty.

Only other thing I am taking is amoxicilan.

BP seems to be going down too.

It was 165/100 an hour ago. Might go up during the Avs game though.

Any Sympathy Poon yet?
 
mountain muscle said:
Hey Swat, do the ace inhibitors make you dehydrated? I have been drinking almost two gallons a day and keep water by my bed and still have constant dry mouth and am thirsty.

Only other thing I am taking is amoxicilan.

BP seems to be going down too.

It was 165/100 an hour ago. Might go up during the Avs game though.
You quoted my message as I was editing it to add some more info.

When you're dehydrated, your renal juxtoglomerular cells produce renin. Renin stimulates angiotensin II production through a complex pathway (by way of angiotensin I), which is dependent on angiotensin-converting enzyme (ACE). This is how ACE-inhibitors work. They prevent the production of angiotensin II.

Angiotensin II had effects on heart, lungs, and kidneys. It causes the kidneys to produce aldosterone, which causes sodium retention (this would cause water retention). Angiotensin II also has a direct effect on sodium reabsorption in the renal proximal tubules. ACE-I's ultimately block sodium retention by decreased production of aldosterone by inhibiting production of angiotensin II.

So in effect, ACE-I's will block sodium reabsorption, which might lead to increased urination and predispose you to dehydration. When you're dehydrated, you won't be able to mount as great a response to dehydration as someone who is not on ACE-I's.

It's been a while since I learned all this crap (ahem, "stuff") in medical school, so some of the finer details of the above may have been lost by my post-med school dementia. The basic concept applies though.
 
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