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Another 30 something heart failure
- Thread starter mwm5
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Who?
lol....
Mike matarazzo audio interview-must hear
Training is going terrible, but I have not given it up completely. If you listen to the audio link by pro Mike Matarazzo, you will hear that he doesnt lift at all anymore. I probably will end up going that route eventually. When I lift I feel like im going to puke and get light headed ect when I push it too hard. Lifting real light weights in the range of 16-20 reps and not going to failure. Squats are the worst. I get by, lets just say that. Here is the link I posted with the audio interview of Mike Matarazzo about his heart attack. He has a pacemaker/defibrillator just like me. His ejection fraction is the same as mine.
audio interview with Mike Matarazzo - Professional Muscle
Training is going terrible, but I have not given it up completely. If you listen to the audio link by pro Mike Matarazzo, you will hear that he doesnt lift at all anymore. I probably will end up going that route eventually. When I lift I feel like im going to puke and get light headed ect when I push it too hard. Lifting real light weights in the range of 16-20 reps and not going to failure. Squats are the worst. I get by, lets just say that. Here is the link I posted with the audio interview of Mike Matarazzo about his heart attack. He has a pacemaker/defibrillator just like me. His ejection fraction is the same as mine.
audio interview with Mike Matarazzo - Professional Muscle
Last edited:
doorhandle
Banned
Its quite simple actually,
Why does androgen use shorten lifespan. It is the same process that high blood pressure increases stroke and heart attack.
It is the thickening of the muscle of the arteries! Most practicioners know that what causes these maladies is a direct result of thickening of the muscle that involves the arteries of the cardiovascular system.
High blood pressure is a sheer mechanical force against the inner linings of the arterial system. Steroids in and of themselves increase the blood pressure through the angiotensin renal system.
What the double effect of these though, is not only does it increase the pressure in the system which increases risk of stroke and heart attack by a minimum of 3-fold. Is that it also aggravates the muscle building effects of the tension in the system.
The arteries are muscular, increase that muscle. Then you increase the risk of bursting and/or rupture of small plaques that build in there. The plaque in arteries has the consistency of toothpaaste. Those can rupture especially with undue stress. The thickening of the artery muscle as well makes it more brittle and prone to these issues.
If one were to be genetically incline to a heart attack, even if they controlled for blood pressure. The risks of 'building areterial muscle" would make it a reason not to engage in an experiment.
The disease process is fairly clear as far as the thickening of arterial muscle and heart attacks as in hypertensive heart disease. The idea one could escape it by simply controlling blood pressure is not well stateed, In fact, the evidence points to the fact that the thickening of arterial walls is more suggestive of stroke and heart attack than blood preessure alone.
That most steroid users fail to control their blood pressure causes the disease process of atherosclerosis to accelerate, but regardless if they do. The evidence is that it would be fruitless. As they tend to develop coronary arterial disease at an accelerated rate regardless if they 'control' for the side effects usually regarded as risk factors.
All evidence points to, if statistically taken into analysis, that steroid use is a strong indicator of eartly cardiac mortality, morbidity and disease regardless if risk factors are controlled. Genetic links if someone has had a heart attack prior to age 55 in the family will exponentiate that risk.
It really depends on if the person has a reason to use steroids, be it that they have low self-esteem or if they have a family whom they love. People will make that decision. But it is pretty clear that anabolic steroid will affect the anatomy of the arteriovascular system. More muscle in the artery system is directly correlated, directly correlate AGAIN with more stroke and heart attack..
Taking a muscle building agent, will decrease your life as an epidemiological study can prove
Why does androgen use shorten lifespan. It is the same process that high blood pressure increases stroke and heart attack.
It is the thickening of the muscle of the arteries! Most practicioners know that what causes these maladies is a direct result of thickening of the muscle that involves the arteries of the cardiovascular system.
High blood pressure is a sheer mechanical force against the inner linings of the arterial system. Steroids in and of themselves increase the blood pressure through the angiotensin renal system.
What the double effect of these though, is not only does it increase the pressure in the system which increases risk of stroke and heart attack by a minimum of 3-fold. Is that it also aggravates the muscle building effects of the tension in the system.
The arteries are muscular, increase that muscle. Then you increase the risk of bursting and/or rupture of small plaques that build in there. The plaque in arteries has the consistency of toothpaaste. Those can rupture especially with undue stress. The thickening of the artery muscle as well makes it more brittle and prone to these issues.
If one were to be genetically incline to a heart attack, even if they controlled for blood pressure. The risks of 'building areterial muscle" would make it a reason not to engage in an experiment.
The disease process is fairly clear as far as the thickening of arterial muscle and heart attacks as in hypertensive heart disease. The idea one could escape it by simply controlling blood pressure is not well stateed, In fact, the evidence points to the fact that the thickening of arterial walls is more suggestive of stroke and heart attack than blood preessure alone.
That most steroid users fail to control their blood pressure causes the disease process of atherosclerosis to accelerate, but regardless if they do. The evidence is that it would be fruitless. As they tend to develop coronary arterial disease at an accelerated rate regardless if they 'control' for the side effects usually regarded as risk factors.
All evidence points to, if statistically taken into analysis, that steroid use is a strong indicator of eartly cardiac mortality, morbidity and disease regardless if risk factors are controlled. Genetic links if someone has had a heart attack prior to age 55 in the family will exponentiate that risk.
It really depends on if the person has a reason to use steroids, be it that they have low self-esteem or if they have a family whom they love. People will make that decision. But it is pretty clear that anabolic steroid will affect the anatomy of the arteriovascular system. More muscle in the artery system is directly correlated, directly correlate AGAIN with more stroke and heart attack..
Taking a muscle building agent, will decrease your life as an epidemiological study can prove
Most of these people do not have chest pain. The only symptom they usually get is shortness of breath, primarily with exertion. They sometimes have palpitations. With cardiomegaly or IHSS (idiopathic hypertrophic subaortic stenosis), usually the first sign of illness is a sudden cardiac arrest where the person goes into ventricular tachycardia.I knew you would feel me on this. Damn chest pains I always dismiss because I am always being told it is anxiety. Watch one day I will be having a heart attack and I will be saying to myself "it's just anxiety".lol
That would be pro-arrhythmic if they cause arrhythmias.
chris302001
Banned
what do u think of IM/EM combined residency? Just a quick yay or nay?
doorhandle
Banned
What? The primary reason that people go to the ER is chest pain. SOB with exertion is the primary cause in women, the primary cause in men is angina. The greek work for strangulation. Women feel heart attacks differently than men. But c'mon, the instance of IHSS is so very rare in comparison to coronary artery disease, why bring it up?
IHSS and vtac is a screening test in athletes that one can hardly justify the cost! Now, sudden cardiac arrest, we would be looking more towards long-QT syndrome more than a bulging ventricular septum. The idea of chest pain being from angina due to high blood pressure due left ventricular hypertrophy which is caused by lack of blood flow through a thickened myocardium (i.e the inner wall of the heart gets starved of oxygen due to the distance blood has to travel as the myocardium-muscle squeezes, causing chest pain) again rare.
The reason why people go to the ER, is first gerd, second, chest wall pain. 3rd, heart disease.
IHSS is down the list to probably to nil, never, ever
Out of several thousand patients I can tell you how many have had IHSS, beyond a sigmoidal septal defect that affects many elderly. It is so rare, that I can count on my left hand.
Ridiculous
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