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*The Perpetual Muscle Mass EXPLOSION: Pre-PCT(Active Recovery), Bridging & Cruising*

  • Thread starter Thread starter Ross
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Re: *The Perpetual Muscle Mass EXPLOSION: Pre-PCT(Active Recovery), Bridging & Cruisi

Primordial Performance said:
Ross, have you ever had your LDL & HDL checked?

-Pp
LOL

Are you serious?
 
- Ross - said:
You will NOT get depressed if you run a Dianaviron(Dianabol/Proviron) bridge.

Dianabol is far more Dopaminergic than testosterone. Proviron also induces a PROFOUND feeling of well-being. TESTOSTERONE is not actually what makes you feel good, it is the ANDROGENIC component of testosterone, which is derived from DHT. Proviron is DHT with a methyl-1 group added.

I COMPLETELY understand your conern, this is the EXACT reason I never wanted to come off of test...it makes you feel GRRRREEEAAT! Like fuckin TONY THE TIGER!

BUT--so does Dianabol and Proviron. Give it a shot. Do you EVER wanna have CHILDREN?

If you do NOT care about ever restoring your HPTA, then Cruising is indeed for you. Although, in most cases I am avidly AGAINST cruising if the user is still in is 20's or early 30's.

Having said that, it IS a very effective means to sustaining and producing muscle mass, and I COMPLETELY support your decision to do so, if you have REALLY CONSIDERED all of the consequences.
No, I don't want to have children in the least. Going at a super low dosage for awhile and then ramping it up again....will that give the effect of on-cycle and then off-cycle? What I mean is, will I make good gains again after being at the low dosage for awhile and then increasing like I did when I first went on cycle? Hope that is understandable.
 
Sam5 said:
No, I don't want to have children in the least. Going at a super low dosage for awhile and then ramping it up again....will that give the effect of on-cycle and then off-cycle? What I mean is, will I make good gains again after being at the low dosage for awhile and then increasing like I did when I first went on cycle? Hope that is understandable.

Here is a sample Cruise Protocol:


Weeks 1-10: Testosterone Enanthate, 200mgs (Cruise)
Weeks 10-20: Testosterone Enanthate, 500mgs(Cycle)
Weeks 10-16: Dianabol, 30mgs ED
Weeks 18-22: Anavar, 50mgs ED
Weeks 20-30: Testosterone Enanthate, 200mgs (Cruise)
Weeks 30-40: Testosterone Enanthate, 500mgs (Cycle)
Weeks 30-36: Anadrol, 50mgs ED
Weeks 38-42: Winstrol, 75mgs ED
Weeks 40-50: Testosterone Enanthate, 200mgs (Cruise)



My gift to you brother.
 
Hi mate, quick question.

Whats your BW like? Everything, not just T. Estrogen, SHBG etc...

Also a question was raised on another board (promuscle.com) about some AS acting directly on the pituitary and not at the hypothalamus. Directly reducing LH synthesis in the pituitary. What are your thoughts?
 
Swifto said:
Hi mate, quick question.

Whats your BW like? Everything, not just T. Estrogen, SHBG etc...

Also a question was raised on another board (promuscle.com) about some AS acting directly on the pituitary and not at the hypothalamus. Directly reducing LH synthesis in the pituitary. What are your thoughts?

Hey Swifto!

I am a healthy young man. Cholesterol, liver, brain, kidneys...:)The only time my liver values were considerably elevated was when I was using 80mgs of Turinabol ED.

I use VERY low dosages, as you know Swifto. I am sure this helps.

As for your question...

Pituitary effects of steroid hormones on secretion of follicle-stimulating hormone and luteinizing hormone

T. M. Nett, , a, A. M. Turzillob, M. Barattac and L. A. Rispolia
a Animal Reproduction & Biotechnology Laboratory, Colorado State University, Fort Collins, CO 80523, USA
b Department of Physiology, University of Arizona, Tucson, AZ, USA
c Department of Veterinary Morphophysiology, Via Leonardo da Vinci 44, I-10095 Grugliasco, Torino, Italy

Available online 4 June 2002.





Abstract

Steroid hormones have a profound influence on the secretion of the gonadotropins, follicle-stimulating hormone (FSH) and luteinizing hormone (LH). These effects can occur as a result of steroid hormones modifying the secretion of gonadotropin-releasing hormone (GnRH) from the hypothalamus, or a direct effect of steroid hormones on gonadotropin secreting cells in the anterior pituitary gland. With respect to the latter, we have shown that estradiol increases pituitary sensitivity to GnRH by stimulating an increase in expression of the gene encoding the GnRH receptor. Since an estrogen response element (ERE) has not been identified in the GnRH receptor gene, this effect appears to be mediated by estradiol stimulating production of a yet to be identified factor that in turn enhances expression of the GnRH receptor gene. However, the importance of estradiol for enhancing pituitary sensitivity to GnRH during the periovulatory period is questioned because an increase in mRNA for the GnRH receptor precedes the pre-ovulatory rise in circulating concentrations of estradiol. In fact, it appears that the enhanced pituitary sensitivity during the periovulatory period may occur as a result of a decrease in concentrations of progesterone rather than due to an increase in concentrations of estradiol. Estradiol also is capable of altering secretion of FSH and LH in the absence of GnRH. In a recent study utilizing cultured pituitary cells from anestrous ewes, we demonstrated that estradiol induced a dose-dependent increase in secretion of LH, but resulted in a dose-dependent decrease in the secretion of FSH. We hypothesized that the discordant effects on secretion of LH and FSH might arise from estradiol altering the production of some of the intrapituitary factors involved in synthesis and secretion of FSH. To examine this hypothesis, we measured amounts of mRNA for activin B (a factor known to stimulate synthesis of FSH) and follistatin (an activin-binding protein). We found no change in the mRNA for follistatin after treatment of pituitary cells with estradiol, but noted a decrease in the amount of mRNA for activin B. Thus, the inhibitory effect of estradiol on secretion of FSH appears to be mediated by its ability to suppress the expression of the gene encoding activin.


Corresponding author. Tel.:+1-970-491-1307; fax: +1-970-491-3557


Domestic Animal Endocrinology
Volume 23, Issues 1-2, July 2002, Pages 33-42
Fourth International Conference on Farm Animal Endocrinology
 
- Ross - said:
Here is a sample Cruise Protocol:


Weeks 1-10: Testosterone Enanthate, 200mgs (Cruise)
Weeks 10-20: Testosterone Enanthate, 500mgs(Cycle)
Weeks 10-16: Dianabol, 30mgs ED
Weeks 18-22: Anavar, 50mgs ED
Weeks 20-30: Testosterone Enanthate, 200mgs (Cruise)
Weeks 30-40: Testosterone Enanthate, 500mgs (Cycle)
Weeks 30-36: Anadrol, 50mgs ED
Weeks 38-42: Winstrol, 75mgs ED
Weeks 40-50: Testosterone Enanthate, 200mgs (Cruise)



My gift to you brother.

Thank you very much man. You exhibit the exact kind of behavior that a really good salesman exhibits. You are friendly, non-argumentative, knowledgeable but not arrogant, and willing to spend time with the customer. Did you say your book is out yet. You may be friends with him, but this is exactly what Nelson does not do and because of that I wouldn't buy anything from him. Keep me updated on your work. And will your book cater to people like me who never come off and even different cases that have not been mentioned above or in any of the threads here at EF? Thanks again. :) :) :)
 
Sam5 said:
Thank you very much man. You exhibit the exact kind of behavior that a really good salesman exhibits. You are friendly, non-argumentative, knowledgeable but not arrogant, and willing to spend time with the customer. Did you say your book is out yet. You may be friends with him, but this is exactly what Nelson does not do and because of that I wouldn't buy anything from him. Keep me updated on your work. And will your book cater to people like me who never come off and even different cases that have not been mentioned above or in any of the threads here at EF? Thanks again. :) :) :)

No problem my man, hearing you say that just made my day.

Feels GREAT to help! This is exactly what these boards were FOUNDED UPON; helping out our fellow brothers in iron.

I will keep you updated my friend. I appreciate your enthusiasm. Thanks again for the feedback! :) :) :)
 
- Ross - said:
No problem my man, hearing you say that just made my day.

Feels GREAT to help! This is exactly what these boards were FOUNDED UPON; helping out our fellow brothers in iron.

I will keep you updated my friend. I appreciate your enthusiasm. Thanks again for the feedback! :) :) :)
What I'm saying is I would like to purchase your book if possible. I guess I didn't make myself clear on that one. You know, if guys that had products to sell on this board or any board took your approach they would have much more success. You've consistently demonstrated with facts, experience, and good communication that you are a credible person and that is hard to do with someone like myself.

I'm one of those guys that thinks most of these people on board that claim to be anything are quacks. And I know Anthony is not a quack, he just has not demonstrated his knowledge to me as you have. Like he said earlier in another thread though, the forums are not where the money is at, but I disagree.

As a salesman, if someone comes along that has ragged clothes on or just doesn't seem rich, and I choose to ignore that person then I am passing up not only a potential sale, but the word of mouth advertising that that person most likely will give me. If you want to increase your net profits you must look at every forum, avenue, or person as a customer and not pass it off because they produce such a low percentage of net profit. Cents makes dollars.

And I don't agree with constantly soliciting either, but put a small word out and then just treat people right and you will have success.
 
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