Please Scroll Down to See Forums Below Finished my Test/Tren/Dbol cycle about a month ago, we had a health fair at work today, they had a machine that they put a little blood in that told blood sugar and cholesterol, blood sugar was 88 but cholestrol was 387, I thought the nurse was going to rip my chest open and do a triple by-pass right there she was tripping so hard! Anyway guys how long before it goes back down, I'm going to have it checked again just need to know how long to give it....
just had my cholestrerol checked...
- Thread starter jokerswild
- Start date
O
odoyal rulez
Guest
holy shit man...you really need to see your pcp...that number isnt a joke...
justinjones1963
New member
That is a high number (and I had to laugh at the nurses reaction).
It will take a while to get back down but you need to start running some supps now to get it back to normal. We are all different so I can't say exactly how long it will take. It took me about 5-6 months to pump my HDL from 20 to 40.
Start taking this below, also your doc may give you a script for having levels that high.
Start taking this ED and keep taking it, especially during a cycle:
20-40mg of Policosanol (you have high levels so I would hit 40mg)
Red Yeast Rice 1200mg
CoQ12 30mg
I also take:
Fish Oil & Flax 3g each ED (good for Essential Fatty Acids)
Garlic 400mg x 3 ED
Also take
Sesapure (dosage varies)
NOW makes a great Red Yeast Rice Supplement - dirt cheap and it has CoQ12 in it.
AFStore.com has Sesapure.
The rest you can get anywhere.
Also start eating some leafy green veggies at each meal and make one meal fish every day. That will help.
It will take a while to get back down but you need to start running some supps now to get it back to normal. We are all different so I can't say exactly how long it will take. It took me about 5-6 months to pump my HDL from 20 to 40.
Start taking this below, also your doc may give you a script for having levels that high.
Start taking this ED and keep taking it, especially during a cycle:
20-40mg of Policosanol (you have high levels so I would hit 40mg)
Red Yeast Rice 1200mg
CoQ12 30mg
I also take:
Fish Oil & Flax 3g each ED (good for Essential Fatty Acids)
Garlic 400mg x 3 ED
Also take
Sesapure (dosage varies)
NOW makes a great Red Yeast Rice Supplement - dirt cheap and it has CoQ12 in it.
AFStore.com has Sesapure.
The rest you can get anywhere.
Also start eating some leafy green veggies at each meal and make one meal fish every day. That will help.
Mavafanculo
New member
is coq12 2 better than coq10?
Mavafanculo
New member
with total that high, you may have had pre-existing hypercholest -
in any event, high dose garlic to prevent plaqing, policosanol, inositol hexanicotinate (non-flush niacin IN THE IH FORM ^ ONLY), omega-3s, olive oil, psyllium seed husk, red yeast extract and/or statins along with co-q10. search on the above for plenty of good stuff.
also obviously low animal fat diet, walk/run 2 or 3 days a week, lots of water.
in any event, high dose garlic to prevent plaqing, policosanol, inositol hexanicotinate (non-flush niacin IN THE IH FORM ^ ONLY), omega-3s, olive oil, psyllium seed husk, red yeast extract and/or statins along with co-q10. search on the above for plenty of good stuff.
also obviously low animal fat diet, walk/run 2 or 3 days a week, lots of water.
macrophage69alpha
New member
such numbers are not entirely abnormal during and after a cycle. Those are on the high end.
above listed are all good suggestions, as well as watching saturated fats. Adding fish oil and other polyunsaturates to diet and cardio. if you were doing any decent amount (30-40min per day- even brisk walking) cardio those numbers are exceptionally high
above listed are all good suggestions, as well as watching saturated fats. Adding fish oil and other polyunsaturates to diet and cardio. if you were doing any decent amount (30-40min per day- even brisk walking) cardio those numbers are exceptionally high
DieselGunz
New member
damn that is high I was checked after a cycle and was in the 200s and that was thought to be bad
black sheep
Banned
were you fasting for 12 hours before the test? If not - the test is not nearly accurate. Go to your doc, fast properly then get a test - that is the only way to tell correctly.
I'm still taking letro and AIFM, could either of these be the cause of the high number?
macrophage69alpha
New member
letro can because of its near total suppression. Why not just use AIFM since you are off cycle?
Also, what is your family history like? There is a chance you may need to be on a cholesterol drug all the time. Lipitor does cause liver stress so be aware if doc wants to put you on that. Really clean up your diet and do the supps as others suggested.
Quad
Quad
policosanol, no flush niacin, and gugglelipids. immediately.
i've had terrible cholesterol, 0 hdl, 187 ldl and 191 total. yours trumps that.
go to your doc immediately and set up blood work for now, 8 weeks from now, 16 weeks, and 24 weeks. that should be ample time to drop those numbers. if after 16 weeks your still in terrible shape you may need to be on cholesterol meds.
i've had terrible cholesterol, 0 hdl, 187 ldl and 191 total. yours trumps that.
go to your doc immediately and set up blood work for now, 8 weeks from now, 16 weeks, and 24 weeks. that should be ample time to drop those numbers. if after 16 weeks your still in terrible shape you may need to be on cholesterol meds.
Quadsweep said:
No family history that I know of, I going to drop the letro and run the above listed supplements for a month then have it checked, by then all the AAS and anti E's should be flushed out, hopefully it will come down ALOT.....
macrophage69alpha
New member
guggulsterones do nothing for cholesterol
Guggulipid for the treatment of hypercholesterolemia: a randomized controlled trial.Szapary PO, Wolfe ML, Bloedon LT, Cucchiara AJ, DerMarderosian AH, Cirigliano MD, Rader DJ.
Department of Medicine, University of Pennsylvania School of Medicine, Philadelphia 19104-6021, USA. [email protected]
CONTEXT: Herbal extracts from Commiphora mukul (guggul) have been widely used in Asia as cholesterol-lowering agents, and their popularity is increasing in the United States. Recently, guggulsterones, the purported bioactive compounds of guggul, have been shown to be potent antagonists of 2 nuclear hormone receptors involved in cholesterol metabolism, establishing a plausible mechanism of action for the hypolipidemic effects of these extracts. However, there are currently no published safety or efficacy data on the use of guggul extracts in Western populations. OBJECTIVE: To study the short-term safety and efficacy of 2 doses of a standardized guggul extract (guggulipid, containing 2.5% guggulsterones) in healthy adults with hyperlipidemia eating a typical Western diet. DESIGN: Double-blind, randomized, placebo-controlled trial using a parallel design, conducted March 2000-August 2001. PARTICIPANTS AND SETTING: A total of 103 ambulatory, community-dwelling, healthy adults with hypercholesterolemia in the Philadelphia, Pa, metropolitan area. INTERVENTION: Oral, 3 times daily doses of standard-dose guggulipid (1000 mg), high-dose guggulipid (2000 mg), or matching placebo. MAIN OUTCOME MEASURES: Percentage change in levels of directly measured low-density lipoprotein cholesterol (LDL-C) after 8 weeks of therapy. Secondary outcome measures included levels of total cholesterol, high-density lipoprotein cholesterol (HDL-C), triglycerides, and directly measured very low-density lipoprotein cholesterol (VLDL-C), as well as adverse events reports and laboratory safety measures including electrolyte levels and hepatic and renal function. RESULTS: Compared with participants randomized to placebo (n = 36), in whom levels of LDL-C decreased by 5%, both standard-dose guggulipid (n = 33) and high-dose guggulipid (n = 34) raised levels of LDL-C by 4% (P =.01 vs placebo) and 5% (P =.006 vs placebo), respectively, at 8 weeks, for a net positive change of 9% to 10%. There were no significant changes in levels of total cholesterol, HDL-C, triglycerides, or VLDL-C in response to treatment with guggulipid in the intention-to-treat analysis. While guggulipid was generally well tolerated, 6 participants treated with guggulipid developed a hypersensitivity rash compared with none in the placebo group. CONCLUSIONS: Despite plausible mechanisms of action, guggulipid did not appear to improve levels of serum cholesterol over the short term in this population of adults with hypercholesterolemia, and might in fact raise levels of LDL-C. Guggulipid also appeared to cause a dermatologic hypersensitivity reaction in some patients.
Guggulipid for the treatment of hypercholesterolemia: a randomized controlled trial.Szapary PO, Wolfe ML, Bloedon LT, Cucchiara AJ, DerMarderosian AH, Cirigliano MD, Rader DJ.
Department of Medicine, University of Pennsylvania School of Medicine, Philadelphia 19104-6021, USA. [email protected]
CONTEXT: Herbal extracts from Commiphora mukul (guggul) have been widely used in Asia as cholesterol-lowering agents, and their popularity is increasing in the United States. Recently, guggulsterones, the purported bioactive compounds of guggul, have been shown to be potent antagonists of 2 nuclear hormone receptors involved in cholesterol metabolism, establishing a plausible mechanism of action for the hypolipidemic effects of these extracts. However, there are currently no published safety or efficacy data on the use of guggul extracts in Western populations. OBJECTIVE: To study the short-term safety and efficacy of 2 doses of a standardized guggul extract (guggulipid, containing 2.5% guggulsterones) in healthy adults with hyperlipidemia eating a typical Western diet. DESIGN: Double-blind, randomized, placebo-controlled trial using a parallel design, conducted March 2000-August 2001. PARTICIPANTS AND SETTING: A total of 103 ambulatory, community-dwelling, healthy adults with hypercholesterolemia in the Philadelphia, Pa, metropolitan area. INTERVENTION: Oral, 3 times daily doses of standard-dose guggulipid (1000 mg), high-dose guggulipid (2000 mg), or matching placebo. MAIN OUTCOME MEASURES: Percentage change in levels of directly measured low-density lipoprotein cholesterol (LDL-C) after 8 weeks of therapy. Secondary outcome measures included levels of total cholesterol, high-density lipoprotein cholesterol (HDL-C), triglycerides, and directly measured very low-density lipoprotein cholesterol (VLDL-C), as well as adverse events reports and laboratory safety measures including electrolyte levels and hepatic and renal function. RESULTS: Compared with participants randomized to placebo (n = 36), in whom levels of LDL-C decreased by 5%, both standard-dose guggulipid (n = 33) and high-dose guggulipid (n = 34) raised levels of LDL-C by 4% (P =.01 vs placebo) and 5% (P =.006 vs placebo), respectively, at 8 weeks, for a net positive change of 9% to 10%. There were no significant changes in levels of total cholesterol, HDL-C, triglycerides, or VLDL-C in response to treatment with guggulipid in the intention-to-treat analysis. While guggulipid was generally well tolerated, 6 participants treated with guggulipid developed a hypersensitivity rash compared with none in the placebo group. CONCLUSIONS: Despite plausible mechanisms of action, guggulipid did not appear to improve levels of serum cholesterol over the short term in this population of adults with hypercholesterolemia, and might in fact raise levels of LDL-C. Guggulipid also appeared to cause a dermatologic hypersensitivity reaction in some patients.
macrophage69alpha
New member
well, its important to clarify.
Either guggulsterones do nothing or they only work in certain populations. Now it is possible that India has a population with a differing response to guggulsterones (ethnic genetic differences) OR (IMHO more likely) the studies in india were flawed and the research either fabricated or skewed to reach their results (since india is the primary exporter of guggul and guggulsterones there is a significant financial interest in research proving that it works)
Either guggulsterones do nothing or they only work in certain populations. Now it is possible that India has a population with a differing response to guggulsterones (ethnic genetic differences) OR (IMHO more likely) the studies in india were flawed and the research either fabricated or skewed to reach their results (since india is the primary exporter of guggul and guggulsterones there is a significant financial interest in research proving that it works)
macrophage69alpha
New member
1: JAMA. 2003 Aug 13;290(6):765-72. Links
Comment in:
JAMA. 2003 Dec 3;290(21):2800-1; author reply 2801.
JAMA. 2003 Dec 3;290(21):2800; author reply 2801.
Guggulipid for the treatment of hypercholesterolemia: a randomized controlled trial.Szapary PO, Wolfe ML, Bloedon LT, Cucchiara AJ, DerMarderosian AH, Cirigliano MD, Rader DJ.
Department of Medicine, University of Pennsylvania School of Medicine, Philadelphia 19104-6021, USA. [email protected]
CONTEXT: Herbal extracts from Commiphora mukul (guggul) have been widely used in Asia as cholesterol-lowering agents, and their popularity is increasing in the United States. Recently, guggulsterones, the purported bioactive compounds of guggul, have been shown to be potent antagonists of 2 nuclear hormone receptors involved in cholesterol metabolism, establishing a plausible mechanism of action for the hypolipidemic effects of these extracts. However, there are currently no published safety or efficacy data on the use of guggul extracts in Western populations. OBJECTIVE: To study the short-term safety and efficacy of 2 doses of a standardized guggul extract (guggulipid, containing 2.5% guggulsterones) in healthy adults with hyperlipidemia eating a typical Western diet. DESIGN: Double-blind, randomized, placebo-controlled trial using a parallel design, conducted March 2000-August 2001. PARTICIPANTS AND SETTING: A total of 103 ambulatory, community-dwelling, healthy adults with hypercholesterolemia in the Philadelphia, Pa, metropolitan area. INTERVENTION: Oral, 3 times daily doses of standard-dose guggulipid (1000 mg), high-dose guggulipid (2000 mg), or matching placebo. MAIN OUTCOME MEASURES: Percentage change in levels of directly measured low-density lipoprotein cholesterol (LDL-C) after 8 weeks of therapy. Secondary outcome measures included levels of total cholesterol, high-density lipoprotein cholesterol (HDL-C), triglycerides, and directly measured very low-density lipoprotein cholesterol (VLDL-C), as well as adverse events reports and laboratory safety measures including electrolyte levels and hepatic and renal function. RESULTS: Compared with participants randomized to placebo (n = 36), in whom levels of LDL-C decreased by 5%, both standard-dose guggulipid (n = 33) and high-dose guggulipid (n = 34) raised levels of LDL-C by 4% (P =.01 vs placebo) and 5% (P =.006 vs placebo), respectively, at 8 weeks, for a net positive change of 9% to 10%. There were no significant changes in levels of total cholesterol, HDL-C, triglycerides, or VLDL-C in response to treatment with guggulipid in the intention-to-treat analysis. While guggulipid was generally well tolerated, 6 participants treated with guggulipid developed a hypersensitivity rash compared with none in the placebo group. CONCLUSIONS: Despite plausible mechanisms of action, guggulipid did not appear to improve levels of serum cholesterol over the short term in this population of adults with hypercholesterolemia, and might in fact raise levels of LDL-C. Guggulipid also appeared to cause a dermatologic hypersensitivity reaction in some patients.
Comment in:
JAMA. 2003 Dec 3;290(21):2800-1; author reply 2801.
JAMA. 2003 Dec 3;290(21):2800; author reply 2801.
Guggulipid for the treatment of hypercholesterolemia: a randomized controlled trial.Szapary PO, Wolfe ML, Bloedon LT, Cucchiara AJ, DerMarderosian AH, Cirigliano MD, Rader DJ.
Department of Medicine, University of Pennsylvania School of Medicine, Philadelphia 19104-6021, USA. [email protected]
CONTEXT: Herbal extracts from Commiphora mukul (guggul) have been widely used in Asia as cholesterol-lowering agents, and their popularity is increasing in the United States. Recently, guggulsterones, the purported bioactive compounds of guggul, have been shown to be potent antagonists of 2 nuclear hormone receptors involved in cholesterol metabolism, establishing a plausible mechanism of action for the hypolipidemic effects of these extracts. However, there are currently no published safety or efficacy data on the use of guggul extracts in Western populations. OBJECTIVE: To study the short-term safety and efficacy of 2 doses of a standardized guggul extract (guggulipid, containing 2.5% guggulsterones) in healthy adults with hyperlipidemia eating a typical Western diet. DESIGN: Double-blind, randomized, placebo-controlled trial using a parallel design, conducted March 2000-August 2001. PARTICIPANTS AND SETTING: A total of 103 ambulatory, community-dwelling, healthy adults with hypercholesterolemia in the Philadelphia, Pa, metropolitan area. INTERVENTION: Oral, 3 times daily doses of standard-dose guggulipid (1000 mg), high-dose guggulipid (2000 mg), or matching placebo. MAIN OUTCOME MEASURES: Percentage change in levels of directly measured low-density lipoprotein cholesterol (LDL-C) after 8 weeks of therapy. Secondary outcome measures included levels of total cholesterol, high-density lipoprotein cholesterol (HDL-C), triglycerides, and directly measured very low-density lipoprotein cholesterol (VLDL-C), as well as adverse events reports and laboratory safety measures including electrolyte levels and hepatic and renal function. RESULTS: Compared with participants randomized to placebo (n = 36), in whom levels of LDL-C decreased by 5%, both standard-dose guggulipid (n = 33) and high-dose guggulipid (n = 34) raised levels of LDL-C by 4% (P =.01 vs placebo) and 5% (P =.006 vs placebo), respectively, at 8 weeks, for a net positive change of 9% to 10%. There were no significant changes in levels of total cholesterol, HDL-C, triglycerides, or VLDL-C in response to treatment with guggulipid in the intention-to-treat analysis. While guggulipid was generally well tolerated, 6 participants treated with guggulipid developed a hypersensitivity rash compared with none in the placebo group. CONCLUSIONS: Despite plausible mechanisms of action, guggulipid did not appear to improve levels of serum cholesterol over the short term in this population of adults with hypercholesterolemia, and might in fact raise levels of LDL-C. Guggulipid also appeared to cause a dermatologic hypersensitivity reaction in some patients.
solidspine
New member
Have it checked again, simply sounds like a mistake.
solidspine said:
no mistake, they checked it twice, along with 23 of my co-workers whose counts were between 80-170....
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