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*****deca More Anabolic Than Testosterone?*****
- Thread starter winstrol69
- Start date
jc21
New member
well by plumbing i guess you mean my sex drive...it is great, i get laid atleast twice a day
my hairline is ok...i think it is starting to thin a little. but i am sure i am gonna go bald someday soon anyway, my father was before he was 30.
nope i am not taking ru-486, if you wanna send me some i will take it
, i have never really had a problem with gyno, if i do feel it then i will drop the dosage down to something i know i can handle.
This isnt some spur of the moment cycle. i have worked my way up gradually on all the dosages.
my hairline is ok...i think it is starting to thin a little. but i am sure i am gonna go bald someday soon anyway, my father was before he was 30.
nope i am not taking ru-486, if you wanna send me some i will take it
, i have never really had a problem with gyno, if i do feel it then i will drop the dosage down to something i know i can handle.This isnt some spur of the moment cycle. i have worked my way up gradually on all the dosages.
J
johnboy
Guest
here it is from the chemical mastermind himself
by Bill Roberts - This drug is unique (so far as I know) in that 5a -reductase, the enzyme which converts testosterone to the more-potent DHT, actually converts nandrolone to a less-potent compound. Therefore this AAS is somewhat deactivated in the skin, scalp, and prostate, and these tissues experience an effectively-lower androgen level than the rest of the body. Therefore, for the same amount of activity as another drug at the androgen receptors (ARs) in muscle tissue, Deca gives less activity in the scalp, skin, and prostate. Thus, it is the best choice for those particularly concerned with these things.
Its effectiveness at the androgen receptor of muscle tissue is superior to that of testosterone: it binds better. Yet, it gives only about half the muscle-building results per milligram. This I think is a result of its being less effective or entirely ineffective in non-AR-mediated mechanisms for muscle growth.
It also appears less effective or entirely ineffective in activity on nerve cells, certainly on the nerve cells responsible for erectile function. Use of Deca as the sole AAS often results in complete inability to perform sexually.
These problems can be solved by combining with a drug that does supply the missing activity: e.g. testosterone.
Nandrolone is proven to be a progestin. This fact is of clear importance in bodybuilding, because while moderate Deca-only use actually lowers estrogen levels as a consequence of reducing natural testosterone levels and thus allowing the aromatase enzyme less substrate to work with, Deca nonetheless can cause gyno in some individuals. Furthermore, just as progesterone will to a point increase sex drive in women, and then often decrease it as levels get too high, high levels of progestogenic steroids can kill sex drive in male bodybuilders, though there is a great deal of individual variability as to what is too much.
Incidentally, this progestogenic activity also inhibits LH production, and contrary to common belief, even small amounts of Deca are quite inhibitory, approximately as much so as the same amount of testosterone.
To some extent, nandrolone aromatizes to estrogen, and it does not appear that this can be entirely blocked by use of aromatase inhibitors – indeed, aromatase may not be involved at all in this process (there is no evidence in humans that such occurs) with the enzyme CYP 2C11 being in my opinion the more likely candidate for this activity. In any case, Cytadren, an aromatase inhibitor, has not been found effective in avoiding aromatization of nandrolone.
The drug is moderately effective at doses of 400 mg/week. The long half-life of Deca-Durabolin makes it unsuited to short alternating cycles, but suitable for more traditional cycles, with a built-in self-tapering effect in the weeks following the last injection.
by Bill Roberts - This drug is unique (so far as I know) in that 5a -reductase, the enzyme which converts testosterone to the more-potent DHT, actually converts nandrolone to a less-potent compound. Therefore this AAS is somewhat deactivated in the skin, scalp, and prostate, and these tissues experience an effectively-lower androgen level than the rest of the body. Therefore, for the same amount of activity as another drug at the androgen receptors (ARs) in muscle tissue, Deca gives less activity in the scalp, skin, and prostate. Thus, it is the best choice for those particularly concerned with these things.
Its effectiveness at the androgen receptor of muscle tissue is superior to that of testosterone: it binds better. Yet, it gives only about half the muscle-building results per milligram. This I think is a result of its being less effective or entirely ineffective in non-AR-mediated mechanisms for muscle growth.
It also appears less effective or entirely ineffective in activity on nerve cells, certainly on the nerve cells responsible for erectile function. Use of Deca as the sole AAS often results in complete inability to perform sexually.
These problems can be solved by combining with a drug that does supply the missing activity: e.g. testosterone.
Nandrolone is proven to be a progestin. This fact is of clear importance in bodybuilding, because while moderate Deca-only use actually lowers estrogen levels as a consequence of reducing natural testosterone levels and thus allowing the aromatase enzyme less substrate to work with, Deca nonetheless can cause gyno in some individuals. Furthermore, just as progesterone will to a point increase sex drive in women, and then often decrease it as levels get too high, high levels of progestogenic steroids can kill sex drive in male bodybuilders, though there is a great deal of individual variability as to what is too much.
Incidentally, this progestogenic activity also inhibits LH production, and contrary to common belief, even small amounts of Deca are quite inhibitory, approximately as much so as the same amount of testosterone.
To some extent, nandrolone aromatizes to estrogen, and it does not appear that this can be entirely blocked by use of aromatase inhibitors – indeed, aromatase may not be involved at all in this process (there is no evidence in humans that such occurs) with the enzyme CYP 2C11 being in my opinion the more likely candidate for this activity. In any case, Cytadren, an aromatase inhibitor, has not been found effective in avoiding aromatization of nandrolone.
The drug is moderately effective at doses of 400 mg/week. The long half-life of Deca-Durabolin makes it unsuited to short alternating cycles, but suitable for more traditional cycles, with a built-in self-tapering effect in the weeks following the last injection.
ajc
New member
When you hear of a steroid referred to as a highly androgenic compound, it means that the substance (i.e. Testosterone) is more androgenic than it is anabolic. This is not to say that it is not highly anabolic...it's just saying that the androgenic properties outweigh the anabolic properties.
Deca, is the opposite. It has more anabolic properties than it does androgenic properties, therefore it is considered a highly anabolic compound.
But, I'm sure we all know that test and deca work best when we use both....so shoot 'em up.
Deca, is the opposite. It has more anabolic properties than it does androgenic properties, therefore it is considered a highly anabolic compound.
But, I'm sure we all know that test and deca work best when we use both....so shoot 'em up.
firstenrgy
New member
Hugh Gellatts said:
The simplistic genuis of that statement earns you some karma.
firstenrgy
winstrol69 said:
Someone brought up a great point here.
How are they measuring "anabolism?" Are they basing it totally on its affinity for the androgen receptor. If so, this might be true. But saying something is more "anabolic" because it binds to the AR more is foolhardy. Anabolism is measured by NET gains, not merely the affinity of binding.
Are they measuring anabolism on gains in net body mass? If so, I would really LOVE to see the data that shows people gain more lean mass using the same diet and program on 200-400 mg of deca than 200-400 mg of testosterone.
Are they measuring anabolism on ability to keep gains after administration. Once again, this is foolhardy. Deca would be superior in this, but i would get the NET gains kept are still lower.
NFG
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