Studies are great and can lead us to experiment with new stratagies however, a lot of studies fail to transfer to real world result and experiences.
Of course they dont directly transfer to real world experience, BUT they do tell us how drugs affect our systems and then we can make the correlations between these interactions, draw some conclusions and act accordingly. The real problem arises from people who do not understand the inner workings and can not or will not read abstracts and studies and apply what is learned to the real world. Im not saying I know it all, hell I hardly know anything, but I am always trying to expand my knowledge and when something is questioned I try not to immediatly denounce those claims becuase it has been regurgitated here by a couple dozen people in the past. Studies, while never encompassing all the aspects we need, they do contain pieces to the puzzle which we need to put together.
IGF-1 has been shown to cause gyno, so has estrogen and progesterone. Now, can any of these cause it alone in the absence of one of the other elements?
thx for the info Zyg...hey Zyg,in all your experiences w/ tren,have you experienced decrease in libido or bitch tits...?by using ONLY tren...w/o combining it w/ another substance
On 75mg/eod tren and 50mg/ed winny I got oily and very itchy skin no loss in labido at all. Keep in mind that increased progesterone has proven to increase sex drive to a certain point and then it drastically falls off. No sign of gyno in the least.
On 75mg/ed tren, 600mg/wk enth and 600mg/wk EQ and .5mg anastrozole ed I have far fewer sides than I did on the fina at half the dose and winny. I dont have the oily skin and itchy scalp or achy joints. Not even the slightest lump nor itch in my nipples. Now, if tren not only causes an increase in IGF-1 but bind to the PR receptor as well, it is in essence increasing 2 factor that are largely responsible for causing gyno. We know that nolva not only reduces IGF-1 but also blocks estrogen and if tren shuts you down hard and you have virtually no natural test, then there would be very little estrogen to begin with which means you have only 1 available peice(ie PR) of the gyno puzzle in place. The real question should be how much and which pieces of the puzzle need to be in place for the onset of gyno?