Please Scroll Down to See Forums Below 
NYBodyguard
New member
i mean where in the world do people pull this bullshit together about propecia with deca, crazy
The NEED to know basics on keeping my hair this cycle
- Thread starter GeneticFreak18
- Start date
NYBodyguard
New member
contiue taking propecia and use nioxin
Raptorex
New member
NYBodyguard said:
I highly suggest you read this thread if this discussion is of concern to you, otherwise you can just write it off as crazy bullshit.
http://www.elitefitness.com/forum/showthread.php?t=24651
I suffered the most hair loss doing Deca. I wasn't using propecia, just straight deca cycle. Compared to that, Sustanol, Ethanate, haven't had a noticeable effect. I know I didn't prepare right for my Deca cycle, but I also wasn't taking much.
I know my experience goes across commonly held beliefs and probably even studies. But I was surprised at how much hair loss I got on deca, so the next cycle, I thought I would do Sust and I didn't notice much if any effect on my hair line at all. Didn't do Propecia the second time either.
To summarize, I would drop the deca. For no other reason than, at least if you aren't running deca, you can take the propecia.
I know my experience goes across commonly held beliefs and probably even studies. But I was surprised at how much hair loss I got on deca, so the next cycle, I thought I would do Sust and I didn't notice much if any effect on my hair line at all. Didn't do Propecia the second time either.
To summarize, I would drop the deca. For no other reason than, at least if you aren't running deca, you can take the propecia.
Last edited:
mrplunkey
New member
This is my standard response to posts like this, so sorry if you've seen it before.
There are two types of hairloss:
1) MPB, which is slow and virtually permanent (Rogaine can re-stimulate a weakened follacle to produce a better hair).
2) Tellogen Effluvium which is fast and temporary (the hair grows back)
Sudden changes in your hormone levels can trigger Tellogen Effluviums and not necessarily accelerate MPB. The ongoing presence of strong androgens in people with the genetic susceptability to MPB will accelerate MPB.
IMO, the best way to fight MPB = propecia (finesteride) or deutestride (sp) -- unless you are using deca or tren. Furthermore, go light on the highly androgenic substances overall.
The best way to fight Tellogen Effluvium = work to keep blood levels constant. For example, go from 1 inject/week to 2/injects week... or from 2 injects/week to 3/injects week. I personally think one of the reasons people lose so much hair on Winny from Tellogen Effluviums is when they do something like 50 mg's IM EoD -- I did that and hair flew off my head. Considering winny's 8-hour half-life, my blood was a roller-coaster. Good news is, it grew back
There are two types of hairloss:
1) MPB, which is slow and virtually permanent (Rogaine can re-stimulate a weakened follacle to produce a better hair).
2) Tellogen Effluvium which is fast and temporary (the hair grows back)
Sudden changes in your hormone levels can trigger Tellogen Effluviums and not necessarily accelerate MPB. The ongoing presence of strong androgens in people with the genetic susceptability to MPB will accelerate MPB.
IMO, the best way to fight MPB = propecia (finesteride) or deutestride (sp) -- unless you are using deca or tren. Furthermore, go light on the highly androgenic substances overall.
The best way to fight Tellogen Effluvium = work to keep blood levels constant. For example, go from 1 inject/week to 2/injects week... or from 2 injects/week to 3/injects week. I personally think one of the reasons people lose so much hair on Winny from Tellogen Effluviums is when they do something like 50 mg's IM EoD -- I did that and hair flew off my head. Considering winny's 8-hour half-life, my blood was a roller-coaster. Good news is, it grew back
krishna
New member
The deca and tren can make you lose hair and there's pretty much nothing you can do about it internally. With test compounds, propecia will work. I wouldn't take propecia with deca or tren either. If I was you I'd start a topical arsenal. Nizoral shampoo, minoxidil, and spiro. I think you can even use emu oil to transport crushed propecia pills (although dutasteride will work better) directly into the scalp. This will avoid it circulating through your system too much.
Guvna
New member
view said:
No, that is a myth derived and propagated from the conjectures of shitty internet chemists.
there isnt one study to affirm direct this, it is pure speculation.
However, there are prostate studies that could be argued either way-so it is moot.
even so, if one has test in his cycle, and he is a finasteride user, there is NO REASON whatsoever to stop because you are taking deca...
Deca+ finasteride= unlikely increase in hairloss (though possible [maybe] in some predisposed people).
Deca + test + finasteride = a very good stack to keep hairloss to a minimum.
Taken from einstein, though he was a scammer...
"I heard you can't use finasteride with deca"
Without fail, you'll hear this statement at least once a month.
First off, let me preface this by saying that this is true, in certain contexts.
However, I'm an advocate of including testosterone in every cycle, so my explanation is with the assumption that one will be using nandrolone in addition to testosterone
For those that do not know, testosterone converts to DHT via the 5 alpha reductase enzyme. DHT is far more androgenic than testosterone and is the main contributor to MPB in those genetically predisposed.
Finasteride is a 5 alpha reductase inhibitor, and therefore limits the conversion of testosterone to DHT via competitive inhibition.
Nandrolone itself is a 5 alpha reductase inhibitor (although it yields DHN in the process). So less DHT is produced if nandrolone is present. Also, DHN is much less androgenic than nandrolone and much less androgenic than DHT. So, finasteride with deca (nandrolone) should both work to reduce DHT levels, and since finasteride will inhibit 5a reductase, it'll also keep levels of DHN lower too.
The misconception that deca with finasteride is a bad combination stems from the the days when deca only cycles were more common (often referred to as the days before jason updated the AR homepage cycles ) If one were running a deca only cycle (or any deca cycle without test), then finasteride would be a poor choice, because DHN is less androgenic than nandrolone. However, since we're assuming test to be included in a deca cycle, and both test and DHT are far more androgenic than nandrolone, if your goal is preventing the formation of the most androgenic compounds, then both deca and finasteride work together to reduce DHT formation.
To sum things up, if you're running a test + deca cycle and are worried about MPB (and you're susceptible), then including finasteride is a far better option than NOT including it, although the nandrolone will help to reduce DHT formation on its own to some extent.
This doesn't touch on the many positive effects of DHT (libido, muscle hardness, decreased SHBG and therefore increased bioavailable AAS).....this is only in the context of preventing hairloss.
Also,
DHN is accused of being the culprit....
Metabolism and receptor binding of nandrolone and testosterone under in vitro and in vivo conditions.
Bergink EW, Geelen JA, Turpijn EW.
The metabolism and receptor binding of nandrolone (N) and testosterone (T) were studied under in vitro and in vivo conditions. The results of both in vitro incubation studes with 3H-N and 3H-T in tissue homogenates from rats and in vivo infusion studies with 3H-N and 3H-T in conscious rats show the importance of the enzymes 5 alpha-reductase and 3 alpha/beta-hydroxysteroid-oxidoreductases in the prostate and the importance of the enzyme 17 beta-hydroxysteroid dehydrogenase in the kidney for the effects of N and T on these tissues. Following infusion of a combined dose of 3H-N and 3H-T there is a preferential retention at the receptor of 5 alpha-)dihydrotestosterone (DHT) over 5 alpha-dihydronandrolone (DHN), N and T (DHT much greater than DHN greater than N greater than T) in the prostate because T is a better substrate than N for 5 alpha-reductase and because DHT binds more strongly to the androgen receptor than DHN, N and T. In the kidney 5 alpha-reductase is not important; there is a preferential retention of N in T (DHN and DHT were only present in small amounts) because N is less susceptible than T for metabolic inactivation by the enzyme 17 beta-hydroxysteroid dehydrogenase and N binds strongly to the androgen receptor. Both in vitro and in vivo studies show that N and T were relatively stable in spleen, thymus and muscular tissue (only shown in vivo) and, as a result, the same amount of N and T was bound to the receptor in these tissues in the in vivo infusion experiment.(ABSTRACT TRUNCATED AT 250 WORDS)
Last edited:
