JIN & T3-LOOKING STRICTLY FOR FAT LOSS?

[semi1979]

That is what I was saying, Low dose would be considered "safer" then ramping upto 100 or 125mcg ed. Maybe I misunderstood your original post but I thought you said it was better to ramp up to higher dose.

The best way to take the T3 is to ramp up high to 100 mcgs a day for 15 days the slowly taper off. The whole cycle shouldnt last more than 40 days max. This is the best way to take it . First day 25mcgs second 50 third 75 fourth 100. stay at 100 for 15 days the 1 week at 75 mcgs 1 week at 50 then 1 week at 25 then 3 days at 12.5 mcgs your thyroid wil not be affectef this way.

 

[genarr3]

You fellas are talking about two different things. We use T3 along with HGH to overcome GH induced hypothyroidism. That would be a dose of approximately 25mcg daily. It's also used by itself to accelerate thyroid function, that's when you would ramp up to a much higher dose then 25mcg/ED.
Choose what you want to do.

 

[Juice Authority]

You fellas are talking about two different things. We use T3 along with HGH to overcome GH induced hypothyroidism. That would be a dose of approximately 25mcg daily. It's also used by itself to accelerate thyroid function, that's when you would ramp up to a much higher dose then 25mcg/ED.
Choose what you want to do.

Exactly. More than 25mcgs ed of T3 will block IGF-1 in the liver nullifying the effect of GH.

T3 and GH use are incompatible. T3 elevates levels of IGF binding protein to the point that they render IGF-1 unbioavailable.

J Hepatol 1996 Mar;24(3):313-9

Effects of long-term growth hormone (GH) and triiodothyronine (T3) administration on functional hepatic nitrogen clearance in normal man.

Wolthers T, Grofte T, Moller N, Vilstrup H, Jorgensen JO.

Department of Medicine M (Endocrinology and Diabetes), Aarhus University Hospital, Denmark.

BACKGROUND/AIMS: A decline in urea excretion is seen following long-term growth hormone administration, reflecting overall protein anabolism. Conversely, hyperthyroidism is characterized by increased urea synthesis and negative nitrogen metabolism. These seemingly opposite effects are presumed to reflect different actions on peripheral protein metabolism. The extent to which these hormonal systems have different direct effects on hepatic urea genesis has not been fully characterized. METHODS: We measured urea nitrogen synthesis rates and blood alanine levels concomitantly before, during, and after a 4-h constant intravenous infusion of alanine (2 mmol.kg bw-1.h-1). Urea nitrogen synthesis rate was estimated hourly as urinary excretion corrected for gut hydrolysis and accumulation in body water. The slope of the linear relationship between urea nitrogen synthesis rate and alanine concentration represents the liver function as to conversion of amino-N, and is denoted the functional hepatic nitrogen clearance. Eight normal male subjects (age 21-27 years; body mass index 22.4-27.0 kg/m2) were randomly studied four times: 1) after 10 days of subcutaneous saline injections, 2) after 10 days of subcutaneous growth hormone injections (0.1 IU/kg per day), 3) after 10 days of triiodothyronine administration (40 micrograms on even dates, 20 micrograms on uneven dates) and 4) after 10 days given 2)+3). All injections were given at 20 00 h. RESULTS: Growth hormone decreased functional hepatic nitrogen clearance (l/h) by 30% (from 33.8 +/- 3.2 l/h (control) to 23.8 +/- 1.5 l/h (10 days growth hormone) (mean +/- SE) (ANOVA; p < 0.01)). Triiodothyronine did not change functional hepatic nitrogen clearance [36.7 +/- 3.2 l/h), but triiodothyronine given together with growth hormone abolished the effect of growth hormone functional hepatic nitrogen clearance [38.8 +/- 4.8 l/h). CONCLUSIONS: The results show that long-term growth hormone administration acts on liver by decreasing functional hepatic nitrogen clearance, thereby retaining amino-N in the body. Triiodothyronine has no effect on functional hepatic nitrogen clearance, but given together with growth hormone, it abolishes the effect of growth hormone on functional hepatic nitrogen clearance. A possible mechanism is the known effect of thyroid hormones in reducing the bioavailability of insulin-like growth factor-I. Thus, the effects of growth hormone and triiodothyronine on amino-N homeostasis are interdependent and to some extent exerted via interplay in their regulation of liver function as to amino-N conversion.

 

[serbyte]

Use the GH in the morning and afternoon, because doing it before bed will stop the bigest natural release of GH for the day, therefore supressing your system. It could have negative feedback if taken at night. Also, taking such high doses without pyramiding will give you bad side effects, such as sore. swolen joints, and even carpal tunnel.

 

[NJjuice22]

I have been using T3 and GH for 2 months now so let me share my opinion...all you need is 25mcg and you will notice right away the added fat loss and you wont have any of the worn down feeling from high dose t3. Also as far as diet...do low carb and the results will be great, i cant believe the difference in the last 3 weeks since i switched to low carb.