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How to Use the Medicinal effect and Power of DECA: Its Tea Time with OMEGA
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tinytim-nor
Banned
how about you show me evidence that what i said is incorrect?
i dare you
if you dont like this forum dont visit it. simple as
dont come on here with a bad attitude and insult us just because we dont agree with your crazy, super high dose oral only cycles and our educated, logical thinking regarding progestins.
Hah! Burden of proof lies on the one claiming his opinion "is based on fact!"
What fact??
Where did you hear this "fact"?
Your the one who repeatedly disputes the relevance of animal studies, even though virtually ALL steroid profiles are based on ONE very old and questionable study done on rats with regards to the different steroids anabolic and androgenic ratings.
Where does your 60% figure come from?
I never said I didn't like the forum, only that there's just as much unfounded bro-knowledge being presented as facts here as on most other steroid-boards.
My crazy high dose oral only cycles?
My crazy cycle at the moment consists of test E and tren E. But read up on oxymetholone at pub med if you want evidence that anadrol will not kill a healthy liver in six weeks.
My bad attitude is the same as not being a good "bro", right? Shame on me for questioning the "facts", asking for evidence and being insulting by not nodding my head in agreement of the good bros!
If you DO have any sort of evidence regarding tren actually being a progestin, I am prepared to change my belief.
But you don't have it do you?
Sorry for being so "difficult", I will go back to lurking and refrain from pointing out the emperors new clothes.
And let's disregard bloodwork when it doesn't back up what we already "know"!
Maybe you should ask RADAR why he hasn't dropped dead from liver failure after his 4 month dbol run? orals for more than six weeks? Instant death!
(ok, a bit cranky, I'm on a diet, hehe!)
TinyTim your talking to some of the most open minded people around so pretty please no need to get negative in the way you were ( not your last posts but the ones before.
with this particular subject there is a stale mate and should be left at that.
one side says- ok here are these 2 studies
the other side says - those studies are not applicable to humans on cycle either because we dont have utters, or because we are not euogonadal ( static T levels)
its really not going to change.
What me as Ozzie are saying is that that when your ON Test whether on Cycle or HRT, you will already have high E levels and when you introduce things like Tren, or Deca and like compounds you can bet your ass there will be an issue based on the practical reality we have presented.
Why do you think Cabergoline became popular overnight? Or Dostinex? hmm?
anyway if you disagree with me fine, I disagree with you and I will leave it at that.
PS Welcome to elite dont let out butting of penises scare you away
no ones asking you to censor yourself...... its just the nature of this particular convo that I said what i said
with this particular subject there is a stale mate and should be left at that.
one side says- ok here are these 2 studies
the other side says - those studies are not applicable to humans on cycle either because we dont have utters, or because we are not euogonadal ( static T levels)
its really not going to change.
What me as Ozzie are saying is that that when your ON Test whether on Cycle or HRT, you will already have high E levels and when you introduce things like Tren, or Deca and like compounds you can bet your ass there will be an issue based on the practical reality we have presented.
Why do you think Cabergoline became popular overnight? Or Dostinex? hmm?
anyway if you disagree with me fine, I disagree with you and I will leave it at that.
PS Welcome to elite
dont let out butting of penises scare you awayno ones asking you to censor yourself...... its just the nature of this particular convo that I said what i said
RADAR
Well-known member
A sheep's pituitary gland more closely represents the size and function of the human pituitary than any other animal, even in relation to the female pituitary. I think it's safe to say that me using this study to represent my theory is a whole lot better than you trying to argue your point with NO study to back your claims.
We react the same way as animals with a pituitary to injecting gonadotropic hormones which makes the pituitary produce LH and send it to the leydig cells. It doesn't sound like the SCIENTIST that decided to use MALE sheep to simulate what would happen in humans were off base at all. All I'm saying is, if you want to make a point then use research to back yourself.
If thy had wanted a more accurate test subject then Why not do tests on our closest cousin?.......... Apes.
endojuicedr
Banned
This study looked at ER and PgR expression in normal breast tissue (i.e. not cancer tissue) in tamoxifen treated women. They found that tamoxifen "shows no stimulatory activity on either PgR levels, a well known oestrogen regulated protein... or the important parameter of cell proliferation (Figure 2)." "In conclusion, the data presented do not show any adverse effects of tamoxifen on normal breast tissue."
This finding was confirmed in the most extensive study that I've seen looking at the effects of tamoxifen in normal breast tissue, which was published in 2003. This quote couldn't be any more relevant or explicit. Read it and reread it:
Quote:
The observation that PR status is different in response to tamoxifen depending on the normal and tumour breast tissue is highly important. In normal epithelial breast tissue, tamoxifen downregulates PR, while in breast cancer tissue it upregulates the receptor, which indicates that tamoxifen plays a greater agonistic effect in tumour tissue than in normal tissue.
In the present study, ERa and PR expressions decreased significantly on the normal breast tissue (epithelium) of patients receiving 5, 10 or 20 mg/day of tamoxifen for 50 days compared with the placebo group. The important finding was that low doses of tamoxifen decreased ERa and PR expression to levels observed with the standard dose of tamoxifen.
Here are images showing the effect of different doses of tamoxifen on the level of estrogen receptors (ER, on the left) and progesterone receptors (PR, on the right) in normal breast tissue:
These results in normal breast tissue are in perfect accordance with my statement that "There is no evidence showing that tamoxifen upregulates the progesterone receptor in the breast (which is what the worry is all about). It shows it does the opposite."
This finding was confirmed in the most extensive study that I've seen looking at the effects of tamoxifen in normal breast tissue, which was published in 2003. This quote couldn't be any more relevant or explicit. Read it and reread it:
Quote:
The observation that PR status is different in response to tamoxifen depending on the normal and tumour breast tissue is highly important. In normal epithelial breast tissue, tamoxifen downregulates PR, while in breast cancer tissue it upregulates the receptor, which indicates that tamoxifen plays a greater agonistic effect in tumour tissue than in normal tissue.
In the present study, ERa and PR expressions decreased significantly on the normal breast tissue (epithelium) of patients receiving 5, 10 or 20 mg/day of tamoxifen for 50 days compared with the placebo group. The important finding was that low doses of tamoxifen decreased ERa and PR expression to levels observed with the standard dose of tamoxifen.
Here are images showing the effect of different doses of tamoxifen on the level of estrogen receptors (ER, on the left) and progesterone receptors (PR, on the right) in normal breast tissue:
These results in normal breast tissue are in perfect accordance with my statement that "There is no evidence showing that tamoxifen upregulates the progesterone receptor in the breast (which is what the worry is all about). It shows it does the opposite."
Female Breast tissue
the male body is different has Test as it main hormone which Aromatizes this hormonal back drop effects the way things operate endocrinology in regard to P's expression.
I am siting here grinning because its so easy to ignore advice handed to you on a silver platter to help you.
We are males on cycle! the hormonal back drop is what creates the vulnerability/sides. Yes there are blood tests as I remember from HucckleBerry Finnaplex and 2Thick, but that was years ago.
Not to mention the body of threads talking about how Deca has a horrible relationship with Nolva and Tren.
the male body is different has Test as it main hormone which Aromatizes this hormonal back drop effects the way things operate endocrinology in regard to P's expression.
I am siting here grinning because its so easy to ignore advice handed to you on a silver platter to help you.
We are males on cycle! the hormonal back drop is what creates the vulnerability/sides. Yes there are blood tests as I remember from HucckleBerry Finnaplex and 2Thick, but that was years ago.
Not to mention the body of threads talking about how Deca has a horrible relationship with Nolva and Tren.
Last edited:
cgar1228
New member
Cgar Im sorry it was directed to you, not mocking you
I was back lashing at the Nolva Study
Ill edit my last post for you
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