macrophage69alpha said:
wow.
boldenone is a steroid. to which an ester can be added. just like testosterone is a steroid to which propionate, enanthate, etc. esters can be added.
this is a poorly thought out assumption and incorrect. its like saying anadrol is closer to oral primobolan. because they are both orals. or like saying oral dbol and injectable dbol are different
methylation CHANGES the structure and activity of a steroid. Esterification just delays its release into the system, the steroid must be cleaved from the ester.
Methyltestosterone is methytestosterone IT IS NOT testosterone. Its back to guru school for you.
Oral vs/ injectable dbol, as a matter of fact- will act differently in the body. By orally ingesting a product, you subject it to a first pass, and therefore more interaction with various enzymes in the liver. In fact, when you compare oral vs/ injectable versions of the EXACT same product, you find vastly different effects on many enzymes found in the liver, including aromatase, SHBG, etc...
Yeah....boldenone, as it clearly states in my book is the same thing as Dbol, but the boldenone we see on the market is estrified. Hence me saying "Boldenone" is estrified, because we have until this point ALWAYS seen it with the Undeclynate ester. Now, we're seeing it w/o the ester, and even with the propionate ester...but for most people, in common parlance (which was what I was speaking in): Boldenone = EQ (although we know this not to be 100% true)- people use "Boldenone" as the same thing as "EQ" and here, I was speaking to the average member of this board, not the people with an agenda to push their ineffective products- or alternately who take "inventor" credit for products they didn't originate (topical Yohimbe, Sesalean, etc...).
Anyway, where was I? ohh..ok...
Methylating a steroid allows it to survive the first pass through the liver, but by sublingual administration, it would appear to be a non-issue with this particular product.
Passing through the liver is the only reason that a product would be methylated. This product doesn't pass through the liver, so calling it D-bol w/o 17aa or EQ without an ester is the same thing...or calling it "boldenone" or even "Quinbolone" w/o an ester.
Passing through the liver is not done with Andriol, which is an oral testosterone that goes through the lymphatic system. There are ways to get a compound into the bloodstream without passing it first through the liver. typically, passing a steroid through the liver (when it is methylated) increases it's anabolic rating. We see this with Methyltestosterone, which is rated more highly than regular testosterone injectables (by Vida) as an anabolic agent.
This is most likely due to interaction with the aromatase enzyme in the liver, which is more pronounced with orals. D-Blade wouldn't go this route, so it would probably be less anabolic than Dbol, but also wouldn't effect liver enzymes as harshly either.
I think you're right...one of us has to go back to school...
You seem to have overlooked the liver as well as it's functions and it's importance in your absurd "AIFM" product, which avoids the liver in large part because of it's transdermal delivery method. Just as AIFM avoids the liver and therefore the source of the aromatase emzyme (making it useless as a shit flavored lollypop, for aromatase inhibition)- so does D-Blade.
The difference, is that D-Blade is TRYING to avoid the liver, and the aromatase enzyme, while AIFM "claims" to actually act on that enzyme.
This is also why Aromasin is far superior to AIFM- by virtue of oral delivery it interacts with the aromatase enzyme and SHBG much more strongly.
But please, keep going...your agenda is clear, and has been clear to the members here for a long time now.