AAS and the heart

[satchboogie]

SO WHAT??

another few studies that need to be studied.

flaws in the first study:

1) the steroid using group.. no detalis on the duration of each users steroid history. have they been using heavy doses for 12 years? have they been using light doses for 15 years. surely with abuse of steroids, left ventricle enlargement is inevitable. but i doubt a few moderate cycles a year would cause any significant enlargening.

2) the fraternal twins.....
you mentioned the following: They were described in the study as "extremely large with profound muscular hypertrophy"

if they were both EXTREMEMELY large, its pretty safe to assume that BOTH used gear in the past. besides, deca stays in the system for no more than 18 months, and the doctors wouldnt be able to detect steroid use prior to that.

NELSON MADE A GREAT POINT.. STUDIES NEED TO BE STUDIED!

 

[Krazykat]

Oh I quite agree Satchboogie - what`s with that name!?

I never said that these studies were correct or conclusive, but nevertheless they are interesting.

 

[Lift Chief]

^

 

[C-Rock455]

If this started to happen could one not tell by their blood pressure? What I mean is if your left ventrical was becoming larger wouldn't your systolic and diastolic numbers get closer together?

 

[Juice Authority]

SO WHAT??

another few studies that need to be studied.

flaws in the first study:

1) the steroid using group.. no detalis on the duration of each users steroid history. have they been using heavy doses for 12 years? have they been using light doses for 15 years. surely with abuse of steroids, left ventricle enlargement is inevitable. but i doubt a few moderate cycles a year would cause any significant enlargening.

2) the fraternal twins.....
you mentioned the following: They were described in the study as "extremely large with profound muscular hypertrophy"

if they were both EXTREMEMELY large, its pretty safe to assume that BOTH used gear in the past. besides, deca stays in the system for no more than 18 months, and the doctors wouldnt be able to detect steroid use prior to that.

NELSON MADE A GREAT POINT.. STUDIES NEED TO BE STUDIED!

I can only hope and pray that Nelson doesn't chime in on this thread. With the right participants this thread could be a very informative discussion.

 

[Counterstrike]

hey J.A i know your in love with Nelson lol
As far as enlarged hearts, I do recall a study that had said athletes have larger hearts than the average guy that included runners, swimmers etc.. I guess gear could effect the heart but if it does I would thing it would be more severe from harsher drugs. Like fina, test etc.. whatever gear makes you grow like Arnold is going to make the heart grow with it I would assume if it smild gear like EQ,var,winny etc. the heart won't effected as much as the harsher roids

 

[HighIntensity]

hey J.A i know your in love with Nelson lol
As far as enlarged hearts, I do recall a study that had said athletes have larger hearts than the average guy that included runners, swimmers etc.. I guess gear could effect the heart but if it does I would thing it would be more severe from harsher drugs. Like fina, test etc.. whatever gear makes you grow like Arnold is going to make the heart grow with it I would assume if it smild gear like EQ,var,winny etc. the heart won't effected as much as the harsher roids

since when has winny become a mild steriod?

 

[Counterstrike]

well winny is not liek other harsher drugs its not mild like primo or eq but i don't really think is that much more harsher than var its maybe a little tiny bit more than var ~~~~~~~

 

[satchboogie]

COUNTERSTRIKE....

actually, the highly anabolic steroids are probably more likely to cause an increase in muscle size then the androgens.

HIGHINTENSITY...

winny IS a mild steroid.
thats why you done see winny only cycles. on the other hand, primo/deca/test only cycles are very common.

JA.....

lmfao

 

[Rio 2001]

Although AAS use is not directly linked to cardiovascular disease, it is fair to consider it a risk factor for developing it. Structural alterations are among the effects to consider, but one should be aware of other implications that if neglected, can lead to CV disease in the long run:
1)atherogenesis due to AASs effects on lipoprotein concentrations
2) a thrombosis model involving the effects of AASs on clotting factors and platelets (" thickening "of the blood)
3) AAS effect on blood pressure

Since structural alteration are the issue here, there are some things to consider.
First, heavy resistance training alone can thicken the left ventricle wall beyond 11mm ( and commonly beyond 13mm, which is abnormally high), although in AAS users this is much more common. However, cardiac FUNCTION remain preserved.In other words,AAS may POTENTIATE concentric left ventricular hypertrophy with decreasing ventricular compliance without affecting cardiac function. Androgenic receptors in the cardiac muscle are not to blame, at least until now. A much more plausible explanation would be the load increase that follows the strenght increase due to AAS use concomitantly with a higher blood pressure, all that leading to structural ADAPTATIVE changes.
This is nothing to be freaked out about, just another reason why heavy lifters/AAS users like ourselves should have medical follow-up always.