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People need to stop coming in this thread and trying to scam others. WTF
Taking Anabolic Steroids 101!
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lee24yorkshire
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Hi there ive been training for 5 years on n off, 18 month contstant up to press. Ive been reading up on the forum as im considerring doin a test e cycle for my first time which will be as follows any thourghts/comments would be appreciated and any advice on pct i.e substute products/cheaper/safer products than im thinking of using. Im 5'11" 175lbs 11% BF.
Cycle:
Weeks:
1 to 10 Enanthate 500mg/week - 2 injects Mon, Thurs
1 to 12 Nolvadex 10mg everyday
1 to 12 arimidex 0.25 everyday
PCT to start 2 weeks after last test injection as follows:
day 1 300mg Clomid, 20mg Nolva, 0.25 A-dex
day 2 to 30 100mg Clomid, 20mg Nolva, 0.25 A-dex
day 31 to 37 20mg Nolva, 0.25mg A-dex
I have also read somewhere it can be an idea to start the Nolva and A-dex 2 week prior to cycle do you think this is necessary?
Thanks in advance
Lee
Btw great site loads better than the other few ive have recently found
Cycle:
Weeks:
1 to 10 Enanthate 500mg/week - 2 injects Mon, Thurs
1 to 12 Nolvadex 10mg everyday
1 to 12 arimidex 0.25 everyday
PCT to start 2 weeks after last test injection as follows:
day 1 300mg Clomid, 20mg Nolva, 0.25 A-dex
day 2 to 30 100mg Clomid, 20mg Nolva, 0.25 A-dex
day 31 to 37 20mg Nolva, 0.25mg A-dex
I have also read somewhere it can be an idea to start the Nolva and A-dex 2 week prior to cycle do you think this is necessary?
Thanks in advance
Lee
Btw great site loads better than the other few ive have recently found
I have a question
wont go with the stats no time sorry!
Finishing a 12 week Cypionate (500mg week) and deca(400mg week)
Wondering if i can go on winny now, wait 2 weeks OR
do my pct (hcg +nolva) now and get on winny later
( im trying to lose bloating )
im not looking up for a complete cutting cycle...wich i will do later trough summer (winny+anavar or clen)
thanks
wont go with the stats no time sorry!
Finishing a 12 week Cypionate (500mg week) and deca(400mg week)
Wondering if i can go on winny now, wait 2 weeks OR
do my pct (hcg +nolva) now and get on winny later
( im trying to lose bloating )
im not looking up for a complete cutting cycle...wich i will do later trough summer (winny+anavar or clen)
thanks
needtogetaas
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lee24yorkshire
New member
u did pal n im very grateful for your input top lad still reading n researching am a few month off doin it yet but when i do hopefully i'll get it right thanks to people like you n others that contribute to this superb forum top marks
Lee
Lee
needtogetaas
New member
Lets spread this thread to the whole AAS world
Okay I read the introduction, and I don't have all that info. But, I do have a few questions.
I am 39. I used to be a calendar model. I had a great body, and did not really even work at it. I have had 2 kids in the last 2 years. I was 5'2" 100'...Now I am 5'2" 130'. I eat terrible. I have no energy, and I am getting ready to start a fitness program.
Now my questions are:
1. I have heard that steroids can help me shed the fat quicker. Is this true?
2. If it is true are there any safe steroids for women?
3. What is the fastest way for me to shed fat?
Please help I can't even look at myself in the mirror any more!
I am 39. I used to be a calendar model. I had a great body, and did not really even work at it. I have had 2 kids in the last 2 years. I was 5'2" 100'...Now I am 5'2" 130'. I eat terrible. I have no energy, and I am getting ready to start a fitness program.
Now my questions are:
1. I have heard that steroids can help me shed the fat quicker. Is this true?
2. If it is true are there any safe steroids for women?
3. What is the fastest way for me to shed fat?
Please help I can't even look at myself in the mirror any more!
needtogetaas
New member
Okay I read the introduction, and I don't have all that info. But, I do have a few questions.
I am 39. I used to be a calendar model. I had a great body, and did not really even work at it. I have had 2 kids in the last 2 years. I was 5'2" 100'...Now I am 5'2" 130'. I eat terrible. I have no energy, and I am getting ready to start a fitness program.
Now my questions are:
1. I have heard that steroids can help me shed the fat quicker. Is this true?
Yes it can but!!!
2. If it is true are there any safe steroids for women?
var, winny epi-strong but!!!
3. What is the fastest way for me to shed fat?
Starting out with a good diet and training is he best place
Please help I can't even look at myself in the mirror any more!
I know it is real hard not to want to reach for the easy way out first. We all want results and we want them now. However please do not jump right into steroids or any kind of drugs right now. I am more then willing to help you get good results rather fast right now with out using drugs. I will send you a pm girl.
needtogetaas
New member
will keep talking with you in pm's , just give me time ok
About_Blank
New member
Great post man!!! Props to you. Very informative to us Newbies. Thanks!
About_Blank
New member
By far, one of the best posts i ever read!! Every time i read it i get something new that i must have missed the first 3 or 4 times. Something every one should read even if your a veteran to the game. MAD PROPS to needto!!!! Keep doin wat u do.
im 37 had health issues that have my t levels way below normal..when im on t (enenthate) 400mg im normal..on 40 mg of lisinopril and wellubtin 350mg..off for over a year and cant get t levels up..im 6 feet was 290 now 255 but no luck..watching my diet and working out 45 a day and cardio was over an hour till now...now im back to 45 mintutes weights and 40cardio still need to lose 25 pounds but no luck in gettin my libido back up ..any advice,,,
needtogetaas
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ghdfans2010
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I am a ghd hair straightener lover! Would you like to share your story about ghd with us?
needtogetaas
New member
lol thanks guys
Hi Everyone. Forgive me to post this question here... (i've used the search function but did not find any)...
Would it be more effective to use Primo than Sustanon?
I have been on a dbol-susta-deca-eq for the last 3 cycles and got some good and consistent gains...
I was given a free-supply of primo so i was thinking of using it instead of susta...
or can i add it on my regular stack?
Appreciate your inputs.
Would it be more effective to use Primo than Sustanon?
I have been on a dbol-susta-deca-eq for the last 3 cycles and got some good and consistent gains...
I was given a free-supply of primo so i was thinking of using it instead of susta...
or can i add it on my regular stack?
Appreciate your inputs.
needtogetaas
New member
I sent you a pm bro
Essie
New member
Whoa! NeedtogetAAS ... Bro, hell of a lot of respect to you! You must have spent years researching to get hold of all of this knowledge. I have used bog standard cycles with only 500iu hcg 5 d/on 5 d/off and Nolva as pct ... never knew what all the stuff really, I mean REALLY does ... just had a general scoop-over idea. Very useful info on here. Once again, thanks man!
needtogetaas
New member
Its not easy gathering all kinds of info and bringing it all together in one place, but my elitefitness members are well worth it
Essie
New member
Its not easy gathering all kinds of info and bringing it all together in one place, but my elitefitness members are well worth it
Well, in that case, I sure am glad to be part of EF
needtogetaas
New member
bshaw12682
New member
I'm 27 male. Wiegh 187, I eat around 2300 cal a day and around 225 grams of protein and work out three to four days a week for a hour. What would be the best thing to use.... I got Deca, ses 250, and some boldenon, I dont know anything so just bought what i've heard of. How much and how often should i use them
needtogetaas
New member
just to add size to your from. Not even talking about the kind of cals you need to get steroids working at peak. You need to eat your body weight times 16. This is a basic bottom of the line amount of cals to add muscle. So you would need 2992 cals every single day. You are now eating damn close to 700 cals a day less then that.
So before you do anything else. Try adding 700 cals a day to your diet. OOOOOO sorry my man.
AX HOMEBOY
New member
This is just one sick post, know where I have to go the day I need some help 
mad mad maaaaad props needto!
mad mad maaaaad props needto!needtogetaas
New member
I am going to come back to this post later when I have more time. I have come to realise that there is no official Post Cycle Therapy or cookie cutter once size fits all pct for all cycles. Pct,pree pct,bridging for all different cycles is way more then something one cookie cutter official Post Cycle Therapy can cover.
So I will cover many different cycles,length of cycles,compounds used,what can be used on cycle,what can be used during cycle,what can be used for pct, methods of pre pct, methods of bridging and more. I think the members of ef diserve much more then a cookie cutter pct.
In the mean time if you need pct advice just send me a pm and I would be more then happy to help you.
First off I would like to say if you cant sit through something like this and read the whole thing. Then you really have no business taking these kinds od drugs in the first place. You also have no business making a comment on this thread ether. Thanks.
What Is Nolvadex/Tamoxifen?
Tamoxifen is considered as the antagonist of the estrogen receptor which again is primarily present in the breast tissue of the human body. It is interesting to note that certain breast cancer cells require that the estrogen levels need to grow with passing time. Ideally, Tamoxifen has been used as the standard endocrine for the treatment of early breast cancer patients. It is therefore used as an anti estrogen therapy and it is mainly given to postmenopausal women. The role of an estrogen is to bind as well as activate the estrogen receptors that are present in the breast cells of a human body. The role of Tamoxifen is to stop estrogen to bind with the receptor. Although it is metabolized into compounds that aid in the binding of estrogen receptors, Tamoxifen does not allow the estrogen receptors to get activated in the breast cells of the human body. Hence, the growth of breast cancer cells can be stopped by making use of this compound. Nonetheless, results vary from person to person and the use of Tamoxifen cannot be deduced as a permanent cure for breast cancer patients.
It is ideally a drug which is taken orally in the form of an edible tablet and it is known to interfere with the activity of the estrogen levels present in the breast tissue. It has been studied that unless the estrogen levels in the human body are kept under strict control, they can lead to breast cancer. Tamoxifen has primarily been used for the past 30 years for treating patients suffering from breast cancer. It has also been administered to patients who are in their early stages of breast cancer. Even patients whose breast cancer has spread to various parts of the body have been known to use Tamoxifen on a regular basis. It has been stated that this drug has the ability to stop cancer cells from spreading within the human body but ironically there is no substantial study which clearly backs this statement with the help of substantial proof. Nonetheless, owing to the hype that it has received via media, people who are having breast cancer or those women who run the risk of developing breast cancer have been known to take this medicine on a regular basis. Interestingly, it has also been seen that women who are suffering from ductul carcinoma in stu, which in turn is similar to invasive breast cancer, have also been known to administer this medicine on a regular basis.
In the past 20 years steroid users have been using nolvadex for a number of reasons. To ether help reduce bloat or gyno problems during a cycle or after a cycle to help recovery natural test production. In men, tamoxifen "nolvaldex" is sometimes used by steroid-taking, weight-training athletes.An alternative and highly similar compound is clomiphene citrate "clomid". These drugs are used as anti-estrogen therapy. In this regard, the drug is used for three purposes. The first purpose, is to reduce the effect of circulating estrogens even if Tamoxifen itself increase the circulating level of estrogens since they are not bound to the estrogen receptors. Abnormally high levels of estrogen in men, can be caused by taking highly aromatizing anabolic steroids e.g. Dianabol, Anadrol or Testosterone. In dosing with a dosing with 20 mg of Novaldex (Tamoxifen) for the duration of a steroid cycle, a reduction in water retention can be achieved. This prevents large fluctuations in water weight within the muscle.
Using Tamoxifen for the duration of a steroid cycle may or may not promote a preferable outcome for a weight training athlete, as the temporary increase in water weight within the muscle increases strength and allows larger weights to be used for the duration of the steroid cycle. Said water will dissipate once usage of steroids has ceased, and a dramatic loss in weight can be observed. Tamoxifen is also used to prevent estrogen related gynecomastia, resulting from elevated estrogenic levels. It can be taken as a preventative measure in small doses, or used at the onset of any symptoms e.g. nipple soreness/sensitivity. In the latter case, dosing reverses the affliction
However it Is now well known that well taking nolvadex serum level estrogen raises and yet another drug must be taken with it during cycle,during pct,or after pct to prevent estrogen rebound. (how retarded). Studies have of course shown the its use can cause a rise in lh and test production but at what cost? Many other factors must be taken into account.
All this is happening in complete ignorance as they are not aware that this medicine has certain side effects that can prove fatal in the longer run. At the same time robbing ones self of a better pct and cycle from using drugs like this.
Though I do feel its "ok" to use them "if you must" but use as little as you can and use support/pct sups to help alleviate the side effects and bad feelings one gets from these harsh drugs.
Where Was This drug Discovered?
Interestingly, this drug was discovered by AstraZeneca Pharmaceuticals which were earliest known as ICI pharmaceuticals. It is now sold under various trade names such as Nolvadex, Valodex and Istubal. Although it is sold under various names, it is primarily known and popularly termed as Tamoxifen. Although this drug is widely used in treating breast cancer patients, it also has adverse side effects which very few people are actually aware off.
Once praised for its benefits in preventing breast cancer recurrence, the lucrative pharmaceutical drug tamoxifen is now implicated in causing dangerous side-effects, including other types of cancers.
In the early 1970's, a shameful chapter closed on the widespread use of a known carcinogenic and endocrine-disrupting drug called DES (diethylstilboestrol), the first synthetic, non-steroidal estrogen drug. Against the advice of its creator, Sir Charles Dodd, between four and six million American and European women and 10,000 Australian women innocently used DES for the prevention of miscarriage and pregnancy complications.
In addition, DES became a popular though unproven drug for a variety of other conditions. It was used for the suppression of lactation, the treatment of acne, the treatment of certain types of breast and prostatic cancer, and as an inhibitor of growth in young girls, an estrogen replacement in menopause and a "morning after" pill.
It would take 30 years to accept what laboratory tests had indicated as early as 1938 — that DES was a highly dangerous and harmful drug. It was reported that, 20 years after taking DES, mothers had a 40 to 50 per cent greater risk of breast cancer than non-exposed mothers. In addition, the children of DES mothers showed a high incidence of reproductive abnormalities, miscarriages, vaginal cancer, testicular cancer, sterility and immune dysfunction. In fact, it is feared that repercussions of this drug will be felt for generations to come.
The irony of this entire debacle is that the medical establishment finally acknowledged that DES was useless in preventing miscarriages. Thus, DES, another disastrous experiment on women, was added to the long list of major medical blunders.
Out of this early research, a new drug appeared on the horizon which would be soon be heralded as a shining star in the war against the growing epidemic of breast cancer. In the late 1960's the pharmaceutical industry developed a drug called "tamoxifen". As a synthetic, non-steroidal compound with hormone-like effects (many of which are poorly understood), tamoxifen has a similar structure to DES. In fact, it was observed that tamoxifen caused the same abnormal changes seen in cells of women taking estradiol and DES. This similarity raised alarm bells for some.
Pierre Blais, well known as a drug researcher who was ejected from Canada's health protection bureaucracy when he spoke out about silicone breast implants, describes the story of tamoxifen as "the story of modern drug design which produces garbage drugs". He says, "Good drug design ceased, unfortunately, in the 1930s." Tamoxifen, Blais asserts, "...is a garbage drug that made it to the top of the scrap heap. It is a DES in the making."
Blais's dire predictions were ignored with the promise of a potential drug treatment for breast cancer. Tamoxifen was first approved by the US Food and Drug Administration (FDA) for use as a birth-control pill; however, it proved to induce rather than inhibit ovulation.(just goes to show how retarded they truly are) Although tamoxifen didn't work as a contraceptive, it was found to lower mammary cancer rates in animals. Animal studies showed that tamoxifen prevented estrogen from binding to receptor sites on breast tissue cells. Tamoxifen also reduced the incidence of breast cancer in rodents after administration of a breast-carcinogenic substance. This discovery provided the impetus to study its effects in treating human breast cancer.
Estrogen is the common link between most breast cancer risk factors, i.e., genetic, reproductive, dietary, lifestyle and environmental. It both stimulates the division of breast cells (healthy as well as cancerous) and, especially in its 'bad' form, increases the risk of breast cancer. Thus, hormonal drugs such as tamoxifen that block the effects of estrogen on the breast were expected to reduce the risk of breast cancer recurring in women treated for breast cancer.
Tamoxifen acts as a weak estrogen by competing for estrogen receptors much as phyto-estrogens do(I want you to keep this word PHYTO ESTROGENS IN MIND WE WILL COVER IT AGAIN LATER). Like phyto-estrogens, tamoxifen has mild estrogenic properties but is considered an anti-estrogen since it inhibits the activity of regular estrogens. More accurately, tamoxifen is an estrogen-blocker(Not a estrogen reducer)
HORMONAL EFFECTS OF TAMOXIFEN IN OLIGOSPERMIC MEN -- WILLIS et al. 73 (1): 171 -- Journal of Endocrinology
Yes the test shows over time that both lh and androgins were raised, but at the same time (serum level estrogen was tripled)and thus the reason many experence rebound gyno after its use.
Tamoxifen fights breast cancer by competing with estrogen for space on estrogen receptors in the tumor tissue. Every tamoxifen molecule that hooks onto an estrogen receptor prevents an estrogen molecule from linking up at the same site. Without a steady supply of estrogen, cells in an estrogen-receptor-positive (ER+) tumor do not thrive and the tumor's ability to spread is reduced.
However, tamoxifen exhibited two conflicting characteristics. It could act either as an anti-estrogen or as an estrogen. Therefore, while tamoxifen is anti-estrogenic to the breast, it also acts as an estrogen to the uterus and, to a lesser extent, the heart, blood vessels and bone. Moreover tamoxifen also acts as an estrogen in the liver thus causing the lowering of IGF-1
In This Issue -- 82 (21): 1661 -- JNCI Journal of the National Cancer Institute
http://cancerres.aacrjournals.org/cgi/reprint/49/7/1882.pdf
Effect of low dose tamoxifen on the insulin-like growth factor system in healthy women
Comparison of Tamoxifen and Testosterone Propionate in Male Rats: Differential Prevention of Orchidectomy Effects on Sex Organs, Bone Mass, Growth, and the Growth Hormone-IGF-I Axis -- Fitts et al. 25 (4): 523 -- Journal of Andrology
For people suffering from breast cancer I guess this would be a good thing. Since Lowering IGF would reduce the growth of everything. However this is not one any of the people using nolva for pct or on cycle use want now is it?
So, although it initially showed the tendency to counter breast cancer recurrence, it would soon be revealed that it also promoted particularly aggressive uterine and liver cancers, caused fatal blood clots and interfered with many other functions.
Doctors, however, were quick to jump on the tamoxifen bandwagon, turning a blind eye to its more injurious tendencies. Starting in the 1970's oncologists began using tamoxifen to treat women with cancer, often in combination with other drugs, radiation or surgery such as lumpectomy and mastectomy, with modest success. Like DES, tamoxifen's benefits were then extended for use as a preventive against osteoporosis and heart disease.
Today, doctors are treating about one million American breast cancer patients with tamoxifen, about 20 per cent of them for more than five years. As studies published in the New England Journal of Medicine in 1989 and the Journal of the National Cancer Institute in 1992 showed, women with breast cancer who took tamoxifen reduced their chances of developing cancer in the other breast (contralateral cancer) by about 30 to 50 per cent. These findings would later be challenged.
Tamoxifen is now recommended for all pre-menopausal women with hormone-positive cancers, as well as for most postmenopausal women with breast cancer and/or a growing number of women with hormone-negative cancers. Tamoxifen is currently used by more women with breast cancer than any other drug.
Tamoxifen (brand name Nolvadex) is now the most widely prescribed cancer medication in the world. It generated revenues of US $265 million in 1992. By 1995, worldwide sales of Nolvadex reached $400 million. (7) And at AUD $90 for one month's supply, it doesn't come cheap (the Australian Pharmaceutical Benefits Scheme covers $70).
Global sales of tamoxifen in 2001 were $1,024 million.[54] Since the expiration of the patent in 2002, it is now widely available as a generic drug around the world. Barr Labs Inc had challenged the patent (which in 1992 was ruled unenforcable) but later came to an agreement with Zeneca to licence the patent and sell tamoxifen at close to Zeneca's price.[55] As of 2004, tamoxifen was the world's largest selling hormonal drug on record and off record may be the number 1 selling drug in word of all time to date. So we are truly talking about billions in revenue world wide for drug companies,sources,ug's and more. Money is at the root of this drug and why its so heavily pushed on all forums by everyone. Its cheap to make and it brings in billions plain and simple.
These numbers are nothing compared to what this drug now makes for the drug companies,sources.ug's selling it. So you can bet your life they will make sure every test and study in the world is published to make sure its seen in a good light. This not even including its "off label use" Ie all us men using it for on cycle and pct. The use of the drug for this reason triples its sales and you can just emagen the amount of money its making. You do the math my friends!. At this very moment 500000000 sources and people with monitary ties to this drug are out there pushing like crazy to make sure you and everyone else keeps its use for pct alive. This is the #1 reason why we have not given up on this years ago.
Tamoxifen was developed by UK-based Imperial Chemical Industries (ICI), one of the world's largest multinational chemical corporations. Zeneca, an ICI subsidiary, is responsible for manufacturing and marketing the hormone and is now the world's largest cancer-drug company.
CARCINOGENENIC EFFECTS
It wasn't long before laboratory studies showed that tamoxifen acted as a carcinogen. It has been found that tamoxifen binds tightly and irreversibly to DNA, the genetic blueprint of a cell, causing a cancerous mutation to take place. Even Australia's conservative National Health and Medical Research Council (NHMRC) warned that no amount of tamoxifen is safe when it comes to carcinogenic effects.
In California there is a law called "Proposition 65" that requires the state to publish and maintain a list of all known carcinogens. In May 1995, the state's Carcinogen Identification Committee voted unanimously to add tamoxifen to its list.
When research is done on anti-cancer drugs (such as SERMs), the aim is to find a drug that prolongs life, with the least amount of acute side-effects. In other words, the goal isn’t so much about finding a cure, as it is finding something that can alleviate the symptoms and/or prolong life.
When it comes to steroid users so many are willing to forgo any and everything to get the one simple effect they desire (recovery). The popularity of these drugs stems from the popular advice to use these drugs for everything from testosterone recovery.bitch tits,make your dick grow bigger, increase the amount of jiz you drop on a girls face, and everything in between. Advice on its use is handed out like candy and everyones got a sweat tooth for quick advice. Of course many "vets and so called know it alls" defend it to the death and it can do no wrong. Mainly do to not wanting to be wrong,habit,they got money involved with it, or just for the sake of argument.
“Its FDA approved for cancer treatment. It must be safe!”
It’s wrong to assume that an “FDA approved” drug has a proven safety profile. The FDA has continually issued stronger health warnings for tamoxifen over the years. For instance, in 1994 the FDA demanded that the tamoxifen manufacturer Zeneca (an ICI sub-division), issue warning letters to health care practitioners about the increased risk of endometrial and gastro-intestinal cancers with tamoxifen use. Zeneca also reported adverse effects similar to those seen with DES, such as reproductive abnormalities in the animals whose mothers received tamoxifen. (remember, DES was the original synthetic estrogen, and also an analog to tamoxifen)
A number of cancer researchers have pointed out the health risks too, such as Elwood et al (6) -
“[Tamoxifen], therefore, is not appropriate for use in the general population because of the known increased risk of endometrial cancer”
What Are Side Effects Of Temoxifen
You Can Get Blood Clots!
Have you any idea that a regular dosage of Tamoxifen can actually increase the chances of blood clots? Well, this is a true fact and can be fatal for those who are using this drug to get rid or avoid the chance of getting Gyno on cycle and or for pct. According to recent medical studies, it has been noticed that people who have been using Tamoxifen on a regular basis have had a substantial increase in terms of their blood clots. Hence, as compared to those people who are not using this drug, their chances of getting blood clots is relatively higher.
A blood clot can be defined as an internal body mechanism by which the cut can be stopped from bleeding excessively. The proteins present in your blood work along with the platelets and in a bid to form a clot. This is also termed as coagulation. In the event of an injury, this can prove to be really very effective as it would stop the flow of blood from your wound and thus save your life. Nonetheless, if the blood clots while it is moving through your body, it can prove fatal. This is also termed as hyper coagulation and it can prove very dangerous for the concerned individual. Tamoxifen has been known to cause hyper coagulation and hence, it needs to be taken under strict medical supervision.
When the study was conducted, it was ascertained that a relatively large number of people developed this conditions and although not many people using this drug were actually studied, those that were using it regularly, were in a shock to find out that it also led to blood clots.
Hence, although this drug is helpful to a certain extent, we need to also see that the extent of damage it can do to our body in terms of hazardous blood clots are much more and hence, you as a steroid user need to exercise caution and spend some quality time researching on this so called ‘wonder-drug’ before making it an eminent part of your daily routine and or pct.
One of the main reasons why a blood clot is considered dangerous is because this drug causes a clot inside the blood vessel which in turn is known as thrombus. What happens is that at times this blood clot can travel through your blood streams and get pushed into your lungs. When this happens, you can be rest assured that your life is in acute danger as this condition is life threatening. This condition is also known as pulmonary embolus. Similarly, a clot this clot can also block the blood vessels in the brain and this in turn may lead to a stroke. When this blood clot clocks the blood vessels of your heart, it stops the blood from rushing to your heart area thereby reducing the oxygen supply to that area. This in turn leads to cardiac arrest.
All the above mentioned conditions arising from blood clots, which in turn are caused from a regular intake of Tamoxifen, can prove to be life threatening for the concerned individual. Hence, even before you decide to take this medication on a regular basis, you need to exercise caution and be prepared to face the ill effects of this so called ‘wonder-drug’.
Increased susceptibility to gyno -
Tamoxifen is often used to combat gyno during cycle when “flare ups” occur. While tamoxifen may provide immediate inhibition of proliferation, and serve as valuable tool, it can actually increase future susceptibility to gyno.
This is caused by tamoxifen’s ability to up-regulate the progesterone receptor. (54-56) This can dramatically increase the chances of developing gyno in future cycles when utilizing progestin based anabolics such as Nandrolone (Deca) or Trenbolone (or any pro-hormone acting upon the progesterone receptor).
It is interesting to speculate. Is tamoxifen use directly related to the increased gyno occurrences seen with modern day steroid users?
You Can Develop Cataract!
Cataract can be defined as a thin white layer of membrane which blocks the passing light to the retina thereby clouding your vision. Although it is relatively painless, it does cloud your vision and can even blind you if it is not removed through the means of a surgical procedure. The retina is ideally a nerve layer which is located at the back of the eye socket and its main purpose is to direct the light which is entering the eye via the means of electromagnetic signals to the brain. Once the brain receives these nerve signals, it is passed on to the nervous system, after which you can transform your vision into clear moving pictures. If this thin layer of membrane is blocked owing to any reason, you would have problems with your vision.
While aging is looked on as the major cause behind cataract, it has recently been noticed that patients using Tamoxifen have been identified as ones susceptible to cataract on a regular basis. people who are aging and using this drug on a regular basis are on a higher risk of contracting cataract as compared to those who are not using Tamoxifen. The other eye problems that can be faced by individuals include scarring of the corneal area and abrupt retinal changes.
In case you are using this drug regularly and you have a cloudy, fuzzy or foggy vision, you need to get your eyesight checked with immediate effect. In case you are unable to withstand the glare of lamps and are unable to catch a glimpse of the morning sun, then again you need to get your eyes checked. This is so because, Temoxifen has a natural tendency to obstruct the normal eye vision and if you do suffer from this symptom, you may not be able to drive at night as the headlamps of the opposing vehicle may blind you momentarily.
In order to get rid of cataract that has been developed owing to a continuous intake of Temoxifen, you may need to undergo a corrective surgery. In case you want to delay a surgical procedure, you may want to light up your room with plenty of tubes and bulbs and keep your eyeglass up to date with the latest prescription. Ideally, the only known cure for cataract that has been a resultant of Temoxifen is a surgical procedure.
If you would like to avoid this problem, you would have to seek an alternative to Temoxifen at the earliest given opportunity.
Libido reduction & erectile dysfunction
Erectile dysfunction ow libido, and general impotence are typical complaints from men recently discontinuing steroids or HRT therapy, which is often combated by Clomid or Nolvadex, paradoxically so.
Regardless of any positive effects on fertility or testosterone levels, Clomid and Nolvadex use is highly correlated with erectile dysfunction, libido suppression, and even emotional disorders Research with male breast cancer patients has also reported decreased libido, and thrombosis associated with tamoxifen use. he thrombotic effect (blood vessel clogging) could explain the mechanism by which SERMs may inhibit erectile function, by reducing circulation to erectile tissue (as discussed before)
Nolva/clomid both raise shbg.
This is something I do not see a lot of people disusing so I I wanted to make it well know. Just do a web search on TAMOXIFEN,clomid or nolva raises shbg or any variation and you will get all the studies and prof you need.
Trait Anxiety and Tamoxifen Effects on Bone Mineral Density and Sex Hormone- Binding Globulin -- Cameron et al. 64 (4): 612 -- Psychosomatic Medicine
iHOP - Information Hyperlinked over Proteins [ SHBG ]
Sex Hormone Binding Globulin in Clinical Perspective; Acta Obstetricia et Gynecologica Scandinavica - 66(3)ages 255-262 - Informa Healthcare
Wiley InterScience :: Session Cookies
2. Nolva lowers Igf-1 Again just a simple search on (TAMOXIFEN or nolva lowers IGF 1 and walla you got all the prof you need.
In This Issue -- 82 (21): 1661 -- JNCI Journal of the National Cancer Institute
http://cancerres.aacrjournals.org/cgi/reprint/49/7/1882.pdf
Effect of low dose tamoxifen on the insulin-like growth factor system in healthy women
Comparison of Tamoxifen and Testosterone Propionate in Male Rats: Differential Prevention of Orchidectomy Effects on Sex Organs, Bone Mass, Growth, and the Growth Hormone-IGF-I Axis -- Fitts et al. 25 (4): 523 -- Journal of Andrology
They can cause Major triglyceride and glucose problems and even to the point of Severe hypertriglyceridemia or also Pancreatitis
Severe hypertriglyceridemia caused by tamoxifen-tr... [Endocr J. 1997] - PubMed result
Tamoxifen-induced hypertriglyceridemia in association with diabetes mellitus - EM|consulte
SpringerLink - Journal Article
Capecitabine-Induced Severe Hypertriglyceridemia: Report of Two Cases -- Kurt et al. 40 (2): 328 -- The Annals of Pharmacotherapy
Elsevier: Article Locator
Estrogen and Triglycerides
http://annonc.oxfordjournals.org/cgi/reprint/11/8/1067.pdf
WikiGenes - Hypertriglyceridemia
A word on clomiphene (Clomid) –
Clomiphene (Clomid) consists of two stereoisomers which possess radically different pharmacodynamics. Zuclomiphene has predominantly estrogenic effects and slow clearance while the enclomiphene isomer has predominately anti-estrogenic effects and quick clearance. his creates a divergent effects between estrogen blockage and estrogen stimulation and an acute imbalance once Clomid administration is discontinued. Bodybuilders will often complain of “estrogenic rebound” after stopping Clomid, which could be attributed to the lingering estrogenic isomer zuclomiphene as the anti-estrogenic enclomiphene has long cleared the system. (Recently, enclomiphene has been isolated by the pharmaceutical company Repros, for use in Androxal™.)
For all intents and purposes, tamoxifen is a superior SERM, simply for the fact that tamoxifen provides a purely anti-estrogenic isomer, whereas Clomid provides a mix of anti and pro estrogenic effects.
In regards to the health consequences about to be listed, it can be safely assumed that Clomid will share similar detrimental effects as tamoxifen, since it shares the same triphenylethylene backbone and carcinogenic tendencies.
One of the main reasons why people make use of Clomid is for the purpose of recovering their bodies after a steroid cycle In simple words, this drug is mainly used in the form of post cycle therapy. Clomid has the actual potential to stimulate the production of hypothalamus which in turn would release a particular kind of hormone called gonadotrophic hormones. This hormone has the natural ability to allow the human testicles to secrete testosterone, which in turn would bring the depleting levels of testosterone in the body to its permissible levels. When this is achieved, the human body would stop losing its muscle mass in a natural way. Reacovery of test production is the gaols at any cost is the common thought.
Its a known fact that both clomid and nolvadex cause some really messed up mood swings.
Clomid/nolva have been known to cause severe mood swings in users and it has apparently been noticed that anyone who has been making use of Clomid/nolva have suffered from such side effects on a regular basis. Many users have categorically complained that the use of Clomid has been considered as the worst side effect that they have suffered so far. A few features of mood swings may include a change in the usual behaviour, tearful behaviour, excessive depression, anxiety and extremely sensitive in nature. Stop acting like you don't know what I am talking about. We all know its true.
Liver cancer -
Originally, tamoxifen was accepted as being non-toxic to the human liver upon finding that tamoxifen did not cause noticeable liver damage (DNA adducts) during short-term test tube studies with human liver cells.
However, it became apparent that test tube research was largely flawed due to the low rate of metabolism in such a superficial environment. It was soon discovered that the hepatotoxic effects from tamoxifen stem from the metabolism and buildup of the a-hydroxytamoxifen and N-desmethyltamoxifen metabolites, which would only appear in an in vivo environment. Surely enough, the results from the original rat studies showing dramatic carcinogenic effects on the liver, soon correlated with human data when researchers found the same type of liver DNA adducts in tamoxifen patients.
More recent human research has reported tamoxifen treated women to have 3x the risk of developing fatty liver disease, which occurs as soon as 3 months into therapy at only 20mg/day. In some cases, the disease lasts up to 3 years, despite cessation of tamoxifen therapy. Five and ten year follow-ups with patients on long term tamoxifen therapy show cases of deadly hepatocellular carcinoma.
In 2002, a bizarre study examined the use of tamoxifen for hepatocellular carcinoma treatment in humans. It was assumed that since tamoxifen could inhibit proliferation of breast cancer, it could offer the same benefit for liver cancer. The devastating results could not have been more indicative of tamoxifen’s hepatotoxic nature, as the tamoxifen treatment significantly increased the rate of death, compared to the group not receiving tamoxifen.
Finally, in a case study reviewing tamoxifen induced liver disease; D.F Moffat et al made a profound statement –
“Hepatocellular carcinoma in tamoxifen treated patients may be under-reported since there may be reluctance to biopsy liver tumours which are assumed to be secondary carcinoma of the breast.” In other words, it appears that liver carcinomas from a large number of breast cancer patients on tamoxifen therapy have been misdiagnosed as an infection from the breast cancer itself.
Although tamoxifen induced liver cancer may take years to manifest in a healthy male, its damaging effects could easily be exaggerated by other popular hepatotoxic drugs, such as 17aa oral steroids.
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Testosterone it’s What separates the men from the boys
Remember what it was like to be young and full of testosterone? The day you used to wake up with a massive rock hard morning wood and at that time in your life hardly even knew what to do with it. How about the good old days when you could down a package of Oreo’s with a gallon of milk without even gaining a single pound? What about that feeling of power and cockiness that was always present with you as a young man. Feeling like king of the world, no one could stand in your way and every woman on earth wanted to you because you were so perfect in every way. Oh yes those sure where the days that have long since passed, Or have they? Are these days gone forever or can we regain some of these feelings once again?
Testosterone is a lot more than just the pesky hormone that gets young boys into trouble. It’s responsible for much more than a teenage boys rebellion well going through the changes of life. Thankfully we have learned much more about testosterone. Immaturity is what gets a young man in trouble, but testosterone is what separates the men from the boys.
What is Testosterone?
Testosterone is a steroid hormone from the androgen group and is also the MAIN hormone in a male’s body. In mammals, testosterone is primarily secreted by the testes of males and by the ovaries of females, while a tiny amount is also secreted by the adrenal glands. It is the main male sex hormone and an anabolic steroid. Testosterone plays a key role in the development of male reproductive tissues which are the testis and prostate. Testosterone also promotes secondary sexual characteristics such as increased muscle and bone mass, hair growth, sexual behavior, development of the male genitals, increased glands, and sperm production. The adult human male body produces about ten times more testosterone than an adult human female body, but females are more sensitive to the testosterone. The brain and the bones are two important tissues in humans where the main effect of testosterone is carried out by way of aromatization to estradiol. Testosterone in the bones allows estradiol to accelerate maturation of the cartilage into bone, leading to closure of the epiphyses and end of growth. This in short explains why when young girls reach menarche, their growth starts to stunt. Estradiol serves as the most important feedback signal to the hypothalamus which triggers LH production. Testosterone is derived from cholesterol which is why people who suffer from cholesterol issues usually have low testosterone. Testosterone is a hormone that is triggered through the HPTA (hypothalamus). When the HPGA Axis is stimulated, the hypothalamus secretes gonadotropin-releasing hormone (GnRH), which on reaching the anterior pituitary, binds to the gonadotrophs and stimulates the release of both the luteinizing hormone (LH) and follicle stimulatinghormone (FSH) into the bloodstream. In the testes, testosterone is produced by the Leydig cells. The male generative glands additionally contain Sertoli cells which require testosterone for spermatogenesis. Like most hormones, testosterone is supplied to target tissues in the blood where much of it is transported bound to a specific plasma protein gondal, called the "sex hormone binding globulin" otherwise known as (SHBG). In males, LH binds to Leydig cells, stimulating production of the principal Leydig cell hormone, testosterone. Testosterone is secreted to the plasma and also carried to Sertoli cells by androgen binding protein (ABP). In Sertoli cells the 4 double bond of testosterone is reduced, producing dihydrotestosterone. A little more than 5% of testosterone is reduced to 5a-dihydrotestosterone (DHT) by the cytochrome through the enzyme 5a reductase. The conversion of testosterone to DHT leads to more sebaceous glands which in turn can leads to oily skin and acne. Less than 1% of testosterone is converted into estradiol by aromatase; also known as the CYP19A1 enzyme. Going back to the Sertoli Cells, in these cells it is regulated by FSH, again acting through a cAMP- and PKA-regulatory pathway. In addition, FSH stimulates Sertoli cells to secrete androgen-binding protein (ABP), which transports testosterone and DHT from Leydig cells to sites of spermatogenesis. There testosterone acts to stimulate protein synthesis and sperm development. Aromatase activity is also found in granulosa cells, but in these cells the activity is stimulated by FSH. Typically, then the cal-cell androgens produced in response to LH distribute to granulosa cells, whereas granulosa cell aromatase converts these androgens to estrogens. As granulosa cells mature they develop capable large numbers of LH receptors in the plasma membrane and become increasingly receptive to LH, increasing the amount of estrogen created from these cells. If not controlled it could lead to problems such as suppressed testosterone or gynecomastia due to the excess E2 levels. In a real short simplified expression; more SHBG leads to more Estrogen which leads to eventual negative effects.[2] Testosterone biosynthesis involves the cleavage of the sidechain of cholesterol by CYP11A, a mitochondrial cytochrome P450 oxidase with the loss of six carbon atoms to become pregnenolone ( the master precursor to all hormones). Next, two additional carbon atoms are removed by the CYP17A enzyme in the endoplasmic reticulum to give up a variety of carbon 19 steroide. In addition, the 3-hydroxyl group is oxidized by 3-ß-HSD to produce androstenedione. In the final and rate limiting step, the C-17 keto group androstenedione is condensed by 17-ß hydroxysteroid dehydrogenase to give way to testosterone.
From reading this explanation of how testosterone is produced even the untrained eye can see a few easy ways one can increase their testosterone production. By increasing the master hormone pregnenolone, by increasing Lutinizing hormone, and or by lowering shbg are just some of the many ways one can begin to raise natural test levels and gain its benefits once again.
Testosterone’s effect on blood Glucose Levels
Androgen (specifically testosterone) deficiency has in recent times come to the forefront of the medical literature after being overlooked for decades. The popularity of hypogonadism is greater than previously thought. Important links are being developed and established in the literature between androgen deficiency and metabolic disorders. There is an important health impact related to metabolic syndrome, insulin resistance, type 2 diabetes, and eventually vascular disease and ERECTILE DYSFUNCTION! Low concentrations of testosterone are associated with insulin resistance and mixed up in hyperglycemia, hypertension, dyslipidemia, and an increased risk of vascular disease. The increasing number of individuals with obesity and low testosterone continue to show the same continuous pattern. I came across a study that consisted of over 6000 men, the men with higher serum levels of testosterone were at much lower risk of type 2 diabetes than men with lower levels of testosterone. [6] Low testosterone demonstrated to have adverse effects on insulin levels and sudden spurts in blood glucose levels. From research lower total testosterone levels leads to less insulin resistance which equates to more body fat distribution. Research indicates insulin is capable of stimulating testosterone production in vivo and at the same time reducing SHBG concentrations in both normal-weight and obese men. Since testosterone SIGNIFIGANTLY boosts insulin sensitivity it also gives leeway for glycemic control which allow one to EAT MORE CARBOHYDRATES without any problem whatsoever! This explains why you hear people saying “when I was a young dude I was able to pound two quarter pounders with NO problem, but now if I eat one quarter pounder I get extremely bloated, those were the good old days.”
Aside from raining testosterone levels there are also powerful products that can have a outstanding positive effect on healthy insulin sensitivity. one such product of this nature would be Need2slin Need2slin and also
Testosterone is the Fountain of Youth?
Here is a small excerpt from a study that shows how testosterone declines within age which affects many organs and functions of the body. “Concentrations of sex steroids (especially testosterone) in serum decline progressively with age in men, as a result of complex alterations in reproductive physiology secondary causes of gonadal dysfunction and lifestyle factors and changes in the levels of binding proteins. Treatment of hypogonadism in younger and older men may result in an improvement in some relevant measures (e.g. osteopenia, sexual dysfunction, and muscle weakness). The average age of the study sample was 73 years of age and 389 (15%) were age 80 or older. Approximately 75% were Caucasian. Most reported themselves to be in excellent or good health compared with their peers. There were few current smokers, but a large proportion had smoked in the past. Alcohol consumption was four drinks per week on average. With the exception of race, distributions of these characteristics were not significantly different from those in the entire MrOS cohort. As men age; their SHBG levels rise and their total/free testosterone drops along their estradiol levels causing a loss in bone density. Higher BMI was related to lower testosterone and SHBG levels and higher estradiol concentrations. Total and free testosterone levels were slightly higher in men who rated their health status as excellent/good compared with those who rated it as fair/poor/very poor and were slightly higher in current smokers. Total testosterone levels were lower in Asian men and higher in African-American and Hispanic men. Free testosterone levels differed significantly by race (unadjusted P < 0.001; age- and BMI-adjusted P < 0.001) following the same trends as for total testosterone. No significant differences in total estradiol by race category were found. Unadjusted free estradiol differed slightly by race (P < 0.03) with whites having the lowest free estradiol concentration; however, after adjusting for age and BMI, the differences by race category diminished. SHBG concentrations differed by race category with Asian men having the lowest SHBG concentration and African-American and white men having the highest mean concentrations. Increasing levels of BMI positively, but slightly, influenced free estradiol. A larger proportion of free estradiol levels were related to free testosterone (positively) and SHBG (negatively) levels. Men with the highest free testosterone and lowest SHBG levels had free estradiol levels approxi-mately 3-fold higher than those with the lowest free testosterone and highest SHBG concentrations. The relationships between free testosterone and free estradiol, and between SHBG and estradiol, were linear. The concentrations of SHBG were slightly higher with greater age, were positively related to total testosterone levels, and were negatively associated with free estradiol levels. The rate of decline in free testosterone in older men is about 10% per decade. Higher SHBG levels were related to lower estradiol levels independent of free testosterone, suggesting that either SHBG has effects on estradiol levels over and above its testosterone-binding properties or that SHBG is actually a surrogate for other variables that may affect both SHBG and estradiol levels.” [7]
Another study showed total testosterone of elderly men were inversely associated with weight, BMI, waist to hip ratio, systolic and diastolic blood pressure, fasting plasma glucose and/or serum insulin, HOMA-IR, triglycerides, CRP and leptin levels and positively related to HDL cholesterol and adiponectin levels. Total testosterone was slightly lower among men who consumed at least one alcohol drink daily, compared with those who drank less or not at all. Those who maintained a healthy BMI/LBM index maintained higher levels of total testosterone. The risk of death was significantly elevated for men in the lowest quartile of the total and bioavailable(free) testosterone distributions. Low total and bioavailable testosterone were each significantly associated with elevated 20-years decline period risk of Cardio Vascular Disease mortality and death due to respiratory disease but not from cancer or death due to other fatal causes. Renal, liver disease, stroke and pulmonary disease have also been linked to low total and free testosterone in older men. [8]
Now both these articles conclude that it would be REALLY BENEFICIAL to take TRT (testosterone replacement therapy) if suffering from hypogonadism; which is primarily found in older men. Men who suffer from low testosterone do not get to reap the fruits of life as much as those who have higher levels of testosterone. Lots of older men use Testosterone replacement therapy to increase Rapid Eye Movement (REM) sleep in order to recover efficiently. There are studies that show that older men use testosterone replacement treatment for a better sense well of being. There are countless studies that show the distinct relationship between depression and testosterone levels. Depression and anxiety lead to lower levels of total and free testosterone, which would explain why certain SSRI’s have a noticeable effect on testosterone levels, serotonin along other neurotransmitters have been linked to effecting hormonal output. The one issue that some older men have with TRT is the higher increments in hemoglobin and hematocrit than young men after adjusting for testosterone levels. When older men take solid doses of testosterone studies show that they lose fat real quickly along with added muscle mass. These traits can be attributed to the ANABOLIC EFFECTS OF TESTOSTERONE! When men are receiving injections of testosterone; automatically nitrogen retention and protein synthesis capabilities rise much higher than the norm. This allows the individual the ability to consume more protein making anabolism even easier. Of course as age heightens; libido drops and old men lose the will or desire to engage in sexual activity. Testosterone also improves HDL which helps cholesterol in return providing proper blood flow to the corpus cavernosum allowing MAXIMUM erection strength. Testosterone replacement therapy has been touted to prevent osteoporosis through increasing the bone mineral density, when testosterone has a good ratio with estrogen it provides a nice sturdy foundation for bone structure, which again re-establishes the importance of testosterone to the male body. Older men as well as young men both deserve to enjoy the benefits of testosterone whether it is through endogenous or exogenous means.
Sleep really does Testosterone some Good!
Sleep really does Testosterone some Good!
Recent research suggests that testosterone plays a role in regulating the CNS during sleep and vice versa. When sleeping in particularly during Rapid Eye Movement; testosterone levels raise dramatically, however as one awakes and encounters the typical stressors, testosterone levels gradually fall throughout the waking day. Rapid Eye Movement occurs in intervals of 90 minutes per stage of sleep, so if one were to sleep 8 hours, the individual could go through REM about 6 times throughout their sleep. Sleep deprivation results in a collection of widespread symptoms leading to alterations in catecholamine, hormone levels, and behaviors. In particular, sleep loss has been connected with altered regulation of the hypothalamic-pituitary adrenal axis, and it impairs gonadal function by producing a marked reduction in testosterone concentration. Subnormal testosterone concentrations may contribute to sexual inadequacy in humans, which may affect established or desired sexual relations. Sleep deprivation really negatively impacts over trained athletes, so those who compete as an athlete NEED to sleep in order for their body to maximize its hormonal production. When sleep is interrupted, the rise of testosterone is also interrupted causing a sudden drop, again REM is EXTREMLY important for the rise of testosterone during sleep. The endocrine system (specifically TESTOSTERONE) has a responsibility to maintain the metabolic processes needed for tissue repair, regeneration, and recovery. The circulating testosterone levels also are play a role in erection frequency, as time goes erection during REM lessens, which would also would explain the correlation of testosterone with libido. Sleep deprivation can lead to many sleep disorders, one common one is linked to testosterone, and this disorder is Sleep Apnea. Sleep Apneic males with severe breathing issues exhibited delayed peak testosterone concentrations. Men with severe obstructive sleep apnea show significantly reduced serum concentrations of free and total testosterone and of sex hormone-binding globulin (SHBG), though their LH levels are normal. This endocrine defect was reversed after 3 months of continuous positive airway pressure (CPAP) therapy. Males who take artificial amounts of testosterone also may have issues with sleep disturbances. Testosterone raises the nocturnal metabolic rate which can negatively affect the way one sleeps. Again realize these are people on cycle and not people with normal amounts of testosterone. Basically low or TOO high levels of testosterone can negate REM sleep which is why one should get blood work done to see where they stand. Many studies show that sleep deprivation leads higher cortisol levels and lower total/free testosterone levels, so I tell you all SLEEP AND SLEEP WELL! Higher testosterone leads to better sleep and more frequencies of REM. [4]
For anyone needing help getting to sleep and staying a sleep at night Need2sleep is the perfect sleep aid Need 2 Sleep . Not only will it help get you to sleep faster but it helps get you into a deep sleep and keeps you there longer.
Testosterone good for the Brain!
Unlike what people may have thought due to the common “MeatHead” nickname given to muscular dumb guys, testosterone increases neurological function. An article I came across showed that as a result of decline in age, testosterone dropped, this led to increased risks for Alzheimer’s disease. Men with Alzheimer’s disease had lower levels of serum testosterone; this explains how both testosterone and estrogen have neuro-stimulative properties. Since Testosterone and Estrogen are neurosteroids this means they would also help prevent one from easily acquiring Addison’s disease. I even saw some research on PubMed that displayed testosterone’s importance in preventing Dementia, another “Slowing down of the Brain” disease. This is why dopamine in abundance has a positive effect on testosterone, although too much dopamine can cause over-stimulation leading to Schizophrenia. Some of you may be saying what he means by “neuro-stimulation”. When one ingests 200mg of caffeine, they are ingesting a bunch of stimulants which cause your neurotransmitters to rapidly fire, well certain sex hormones like testosterone act in this matter without the jittery feeling. This effect allows for better awareness, mental clarity, mental acuity, increased memory and so on. Caffeine has been proven to be effective in improving cognitive function, this goes to show you that when you compare caffeine to testosterone in the way they effect the brain, they both positive since they delay the effects of Dementia while allowing you to do more mental tasks
So far we have seen that testosterone is good for the heart, respiratory system, brain function, blood glucose levels, cholesterol, bone density, sense of well being, and now we see testosterone’s anabolic properties. Here is a full abstract from another article that shows the anabolic benefits of testosterone:
“Testosterone Therapy Prevents Gain in Visceral Adipose Tissue and Loss of Skeletal Muscle in Nonobese Aging Men
C. A. Allan, B. J. G. Strauss, H. G. Burger, E. A. Forbes and R. I. McLachlan
Prince Henry’s Institute (C.A.A., H.G.B., E.A.F., R.I.M.), Andrology Australia (C.A.A., R.I.M.), and Departments of Obstetrics and Gynecology (C.A.A., R.I.M.) and Medicine (B.J.G.S.), Monash University; and Clinical Nutrition and Metabolism Unit (B.J.G.S.), Monash Medical Centre, Monash University, Clayton, Victoria 3168, Australia
Address all correspondence and requests for reprints to: Professor R. I. McLachlan, Prince Henry’s Institute, P.O. Box 5152, Clayton, Victoria 3168, Australia. E-mail:[email protected] .
Background: Trials of testosterone therapy in aging men have demonstrated increases in fat-free mass (FFM) and skeletal muscle and decreases in fat mass (FM) but have not reported the impact of baseline body composition.
Objective: The objective of the study was to determine the effect, in nonobese aging men with symptoms of androgen deficiency and low-normal serum testosterone levels, of testosterone therapy on total and regional body composition and hormonal and metabolic indices.
Methods: Sixty healthy but symptomatic, nonobese men aged 55 yr or older with total testosterone (TT) levels less than 15 nM were randomized to transdermal testosterone patches or placebo for 52 wk. Body composition, by dual-energy x-ray absorptiometry (FM, FFM, skeletal muscle) and magnetic resonance imaging (abdominal sc and visceral adipose tissue, thigh skeletal muscle, and intermuscular fat) and hormonal and metabolic parameters were measured at wk 0 and 52.
Results: Serum TT increased by 30% (P = 0.01), and LH decreased by 50% (P < 0.001). Relative to placebo, total body FFM (P = 0.03) and skeletal muscle (P = 0.008) were increased and thigh skeletal muscle loss was prevented (P = 0.045) with testosterone therapy and visceral fat accumulation decreased (P = 0.001) without change in total body or abdominal sc FM; change in visceral fat was correlated with change in TT levels (r2 = 0.36; P = 0.014). There was a trend to increasing total and low-density lipoprotein cholesterol with placebo.
Conclusion: Testosterone therapy, relative to placebo, selectively lessened visceral fat accumulation without change in total body FM and increased total body FFM and total body and thigh skeletal muscle mass”.
PCT
now you guys can see that PCT (Post Cycle Therapy) is crucial to maintain gains and boost overall health. Many studies and blood panels have shown that PCT boosts liver cells, better ALT/LST levels, good cholesterol, proper testosterone to estrogen ratios, proper testosterone to cortisol ratios, solid IGF levels, proper hypothalamus function, proper brain function, good adrenals, good sense of well being, anti-oxidative, anti-cancerous, restoration of distorted blood vessels, proper maintenance of SHBG’s, bone and ligament/joint protection. As you can see there is plenty of reasons to have a PCT aligned besides maintaining gains. I have always been a firm believer of keeping testosterone and FREE testosterone levels up in order to keep vitality and overall quality of life. There are many way PCT protocols once could follow, I suggest do your own research and see what fits you best. Before/after a cycle and post PCT one should follow their body by doing hormonal/organ blood work to see where you stand.
Supplements to boost testosterone naturally!
For years people have tried many ways to boost testosterone naturally for their specific means of interest. We had the tribulus error which was proven not to do much for the human male. Studies recently suggest that tribulus does boost androgen receptors within the brain which causes the rise in libido associated with supplementation of it. Now AI’s have been proven to raise LH output, increase both total and FREE testosterone while lowering estrogen. Formestane, 6bromo, Exemestane, arimadex all do the job in boosting total/free testosterone while blocking estrogen. 6-bromo and Formestane also block prolactin which allows for even MORE TOTA/FREE TESTOSTERONE boost. It is able to block prolactin by blocking the progesterone receptors at the same time it blocks the estrogen receptors. This would make Formestane an IDEAL otc AI while on Deca to avoid the unwanted “deca-dick”. The benefits of Formestane are really undeniable and should be a part of everyone’s artillery. Formestane also boosts IGF levels which mean MORE MUSCLE GROWTH. Formestane can be used as a standalone, on cycle and during PCT! I did not mention ATD why, simple it has anti-androgen properties since it blocks the androgen receptor in the brain which leads to a lack of libido. It also does not boost testosterone levels as once thought, it appears that ATD provides false testosterone levels because ATD contains metabolites similar to Testosterone which cause the false high positives. Currently there are three reliable sources of Formestane:
1. Competitive Edge Labs Formestane
2. Formestane LV
3. https://www.mrsupps.com/Product-Forma-Stanzol_26.aspx
In this article: BCAAs raise T levels in bodybuilders
We get to see how effective BCAA’s are in boosting testosterone since they reduce cortisol levels which rise during intense training. People have tried to deny the effectiveness of BCAA’s but as you can see it boosts testosterone. It only took 6 grams of BCAA’s for 4 weeks to see a noticeable difference. Now imagine taking the effects of Gear (Bovine super plasma blood serum) which is 3x more potent than standard BCAA’s, this means MORE TESTOSTERONE boost and MORE MUSCLE PRESERVATION! More testosterone leads to gains in MUSCLE MASS, STRENGTH, RECOVERY, and a BETTER YOU!
HCGenerate brings a revolution to all herbal testosterone boosters because it contains a specific extract of Fadogia. Fadogia has shown to increase total testosterone levels at 6x more than that of some of the other popular herbal raws. It helps preserve and create new leydig cells which allows for more conversion of cholesterol to testosterone. Its ability to boost LH and testosterone dramatically makes it very comparable to HCG, which is why HCGenerate can be used on cycle to minimize shutdown; allowing PCT to be a breeze. HCGenerate also contains testofen which has been shown to DOUBLE FREE testosterone levels in HUMAN males. Free testosterone means that one is able to achieve the anabolic and androgenic effects of testosterone. When testosterone is not free; its USELESS because it cannot be used for any masculine purpose. So if one has really high testosterone but low free testosterone; they will not really reap the benefits that someone on a cycle of testosterone will receive. HCGenrate also contains Divanil which boosts FREE testosterone, lowers SHBG levels and boosts Nitric Oxide levels. HCGenerate is a MUST if you plan on using a SERM( Selective Estrogen Receptor Modulator) because SERM’s have been proven to raise SHBG’s which will drop free (useable) testosterone. Eventually high SHBG’s leads to not only low Free testosterone but Low total testosterone as seen with older individuals.
7,8 Benzoflavone has always intrigued me because of its effectiveness. There are studies that show boosts testosterone by increasing GrRH release in which it stimulates GABAergic modulation and at the same time blocks estrogen due to its AI properties. It has an IC50 value of 70nm which is RIDICULOUS for an herbal raw, this explains why individuals who use this raw have dramatic boosts in testosterone with low LH levels. This raw converts LH to testosterone at a high rate while keep estrogen to a norm; giving it a nice 1-2 punch. I have seen my testosterone levels with bloodwork boost over 50% which is definitely impressive and lets me know it’s a must to add in my PCT with HCGenerate. Forma-stanzol again has what you need and contains 7,8 benzoflavone.
https://www.mrsupps.com/Product-Forma-Stanzol_26.aspx
Forged Steel has really caught my eye as of recently since it boosts libido DRAMATICALLY and boosts testosterone at the same time! Let’s start off with the pumpkin seed powder that Forged Steel contains. Pumpkin seed powder has an abundance of Zinc which is crucial for maintaining testosterone efficiency, along bone density strength. Zinc protects the prostate from prostate enlargement which can be fatal. Zinc also is important for fertility in providing more counts of semen and semen mobility. Solid levels of Zinc also prevent testosterone from converting to DHT at a high rate which is what you want. Pumpkin Powder Seed has also been touted to raise FEMALE’S LIBIDO through her olfactory system. In essence, the scent of pumpkin makes a woman wetter in her “special place” which is what any testosterone filled dude wants! Companies are now starting to make colognes with Pumpkin seed powder extract to entice men to purchase their product. So it’s real simple, ingesting pumpkin seed leads to a concentration of pumpkin seed in the body, which leads to pore concentration, finally translates to women appeal and a good chance of GETTING LAID! Forged Steel also contains Muira Puama which decreases prolactin leading to increased TOTAL testosterone levels. It also boosts dopamine levels which lead to HARDER ERECTIONS and LASTING LONGER in the bedroom! Forged Steel is the REAL DEAL if you are looking for a quick boost prior to sexual activity.
FORGED STEEL - OrbitNutrition
One more raw I want you all to witness is mytosterone, this bad boy has been proven to boost testosterone by 60% while blocking estrogen by 9% and blocking DHT conversion by 20 plus %. Here is an excerpt to the study that showcases mytosterone:
“BACKGROUND: Maintaining endogenous testosterone (T) levels as men age may slow the symptoms of sarcopenia, andropause and decline in physical performance. Drugs inhibiting the enzyme 5alpha-reductase (5AR) produce increased blood levels of T and decreased levels of dihydrotestosterone (DHT). However, symptoms of gynecomastia have been reported due to the aromatase (AER) enzyme converting excess T to estradiol (ES). The carotenoid astaxanthin (AX) from Haematococcus pluvialis, Saw Palmetto berry lipid extract (SPLE) from Serenoa repens and the precise combination of these dietary supplements, Alphastat(R) (Mytosterone(trade mark)), have been reported to have inhibitory effects on both 5AR and AER in-vitro. Concomitant regulation of both enzymes in-vivo would cause DHT and ES blood levels to decrease and T levels to increase. The purpose of this clinical study was to determine if patented Alphastat(R) (Mytosterone(trade mark)) could produce these effects in a dose dependent manner. METHODS: To investigate this clinically, 42 healthy males ages 37 to 70 years were divided into two groups of twenty-one and dosed with either 800 mg/day or 2000 mg/day of Alphastat(R) (Mytosterone(trade mark)) for fourteen days. Blood samples were collected on days 0, 3, 7 and 14 and assayed for T, DHT and ES. Body weight and blood pressure data were collected prior to blood collection. One-way, repeated measures analysis of variance (ANOVA-RM) was performed at a significance level of alpha = 0.05 to determine differences from baseline within each group. Two-way analysis of variance (ANOVA-2) was performed after baseline subtraction, at a significance level of alpha = 0.05 to determine differences between dose groups. Results are expressed as means +/- SEM. RESULTS: ANOVA-RM showed significant within group increases in serum total T and significant decreases in serum DHT from baseline in both dose groups at a significance level of alpha = 0.05. Significant decreases in serum ES are reported for the 2000 mg/day dose group and not the 800 mg/day dose group. Significant within group effects were confirmed using ANOVA-2 analyses after baseline subtraction. ANOVA-2 analyses also showed no significant difference between dose groups with regard to the increase of T or the decrease of DHT. It did show a significant dose dependant decrease in serum ES levels. CONCLUSION: Both dose groups showed significant (p = 0.05) increases in T and decreases in DHT within three days of treatment with Alphastat(R) (Mytosterone(trade mark)). Between group statistical analysis showed no significant (p = 0.05) difference, indicating the effect was not dose dependent and that 800 mg/per day is equally effective as 2000 mg/day for increasing T and lowering DHT. Blood levels of ES however, decreased significantly (p = 0.05) in the 2000 mg/day dose group but not in the 800 mg/day dose group indicating a dose dependant decrease in E levels.”
Myodrol 120 caps, Axis Labs - OrbitNutrition
Pretty impressive huh, I think a stack of Myodrol, HCGenerate and forma-stanzol would be INSANE providing that one will SIGNIFIGANTLY boost total/free testosterone, lower conversion of testosterone to DHT and estradiol, and preventing high aromatization. The only supplement that has a legit dose of mytosterone is myodrol by Axis Labs: Myodrol 120 caps, Axis Labs - OrbitNutrition
Be it just to feel naturally more like a man or to help feel less like a woman during pct, Testosterone is what separates the men from the boys my friends and now you know how to get you some.
Sources:
1.Journal of Clinical Endocrinology & Metabolism Vol. 37, No. 1 148-151
doi:10.1210/jcem-37-1-148
2. Michael R. Waterman, Genes Involved in Androgen Biosynthesis and the Male PhenotypeDiane S. KeeneyDepartment of Biochemistry, Vanderbilt University School of Medicine, Nashville, Tenn., USA Vol. 38, No. 5-6, 1992
3.Steroid Hormones
4. Monica Levy Andersen*, Sergio Tufik. The effects of testosteroneonsleep and sleepdisorderedbreathing in men:Its bidirectionalinteraction with erectile function. Sleep Medicine Reviews (2008) 12, 365e379 (http://www.sono.org.br/pdf/2008_Ande...ep_Med_Rev.pdf)
5. ABDULAMAGED M. TRAISH, ANDRE GUAY, FARID SAAD. The Dark Side of Testosterone Deficiency: II. Type 2 Diabetes and Insulin Resistance. Journal of Andrology, Vol. 30, No. 1, January/February 2009. (The Dark Side of Testosterone Deficiency: II. Type 2 Diabetes and Insulin Resistance -- Traish et al. 30 (1): 23 -- Journal of Andrology)
6. Ding EL, Song Y, Malik VS, Liu S. Sex differences of endogenous sex hormones and risk of type 2 diabetes: a systematic review and meta-analysis. JAMA. 2006;295: 1288 –1299.[
7. Testosterone and Estradiol among Older Men. The Journal of Clinical Endocrinology & Metabolism Vol. 91, No. 4 1336-1344
Testosterone and Estradiol among Older Men -- Orwoll et al. 91 (4): 1336 -- Journal of Clinical Endocrinology & Metabolism
8. Low Serum Testosterone and Mortality in Older Men. The Journal of Clinical Endocrinology & Metabolism Vol. 93, No. 1 68-75(2008)
Low Serum Testosterone and Mortality in Older Men -- Laughlin et al. 93 (1): 68 -- Journal of Clinical Endocrinology & Metabolism
9. Older Men Are as Responsive as Young Men to the Anabolic Effects of Graded Doses of Testosterone on the Skeletal Muscle. The Journal of Clinical Endocrinology & Metabolism Vol. 90, No. 2 678-688 (2005)
10. Jones, T.H. (Barnsley/Sheffield) (eds): Advances in the Management of Testosterone Deficiency. Testosterone, Bone and Osteoporosis Front Horm Res. Basel, Karger, 2009, vol 37, pp 123-132 (2010)
11. NEJM -- The Effects of Supraphysiologic Doses of Testosterone on Muscle Size and Strength in Normal Men
12.Age-Related Testosterone Depletion and the Development of Alzheimer Disease. Vol.292 Nov.12, Sept. 22-29, 2004
13. Behav Brain Res. 2010 Jan 20; 206(2): 216-22.
14.Inhibition of human estrogen synthetase (aromatase) by flavones JT Kellis, Jr et al.Science, Sep 1984; 225: 1032 - 1034.
15. Pumpkin Seeds Shown to Boost Sex Drive
16. An open label, dose response study to determine the effect of a dietary supplement on dihydrotestosterone, testosterone and estradiol levels in healthy males. Journal of the International Society of Sports Nutrition 2008, 5:12 (2008)
17. orbitnutrition.com
18. mrsupps.com
I'm not going to lie. I didn't read the whole thread. I did real the first several pages, and have been lurking here for some time. There's one thing I don't understand....
Do 'cutting' AAS's increase fat loss, or do they just help you retain muscle? It seems like a simple question to me, but I can't seem to find any info on it.
Do 'cutting' AAS's increase fat loss, or do they just help you retain muscle? It seems like a simple question to me, but I can't seem to find any info on it.
knocker1980
New member
hi, i am fairly new to steroids and was just wondering if you could tell me the best one to use, ime 29 i weigh 17st 8, i am big set and 6"2 inches tall, my BMI is 32, i have lost 2 stone from dieting but seem to have come to a stand still, i have been told to use decca as it is supposed to help lose body fat and also give good gains, i go to the gym 4 times a week and i eat on average 1500 calories a day as well as having 3 pure protein shakes a day, also is it better to use a few different kinds at the same time to get maximum effect, i dont want to use oral steroids i want to inject as i have been told this is the best way, thank you
needtogetaas
New member
Any steroid can help fat lose due to speeding up nutrition retention. Some steroids like proverone do lock onto the receptors in fat and help the body brake it down better. This is called lipalasis <--ya spelled wrong o well.
Most steroids that are "cutting" steroids just cause a hell of a lot less bloat and can also lower estrogen and progesterone which will also cut fat a bit more and also drop water weight. For the most part it is about diet.. Yes there are steroids like primo and perabolan that have been proven to work on low cal and thus preserve muscle well cutting as well. Helladrol and katanadrol are also well known for this too.
Hope this helps a bit my friend.
needtogetaas
New member
When it comes to source info and wanting to get something off your chest this is will be the place to be. At the moment we are staffing the forum so we are looking for mods. Please stop by and join up. Please help spread the word.
Also unlike all the other source forums. This forum is based out of a steroid friendly country. THe forums are 100% encrypted. The site has a security force and a 24 hour tech team of 15 people watching over the site day and night to make sure no one gets in to the system.
You want a source forum that is "SAFE", and has a real team running the place. A net work of pros that have been in the game for years. Not only is this source forum better in every way but its FREE!!!!
Im actually new to using forums so please direct me if this is in an incorrect area. I have a question regarding first time use of AAS
I am 29years old, have been training hard for 10 years and am a qualified personal trainer. I am benching 100kg x 6 reps to give u an idea of strength and I train legs and back of course. I weigh 78KG and am fairly lean already and u can see my "4pack" but only when i flex and i have a fair bit of fat around the lower abs. I am very educated with diet as well and eat high protein meals 5-6 times a day and control my calorie intake. My goals for taking AAS is to get past my weight training plateau and more so to look good for my part time job as a topless waiter for hens nights. I want to put on about 4kg of muscle while remaining extremely lean. I have read a lot on the different type of AAS and I am thinking about taking sus 250 with winstrol, both intramuscular. 250 of sus per week and the winny EOD. This is my first time on AAS and would appreciate any guidance. Thanks in advance,
Heshy
I am 29years old, have been training hard for 10 years and am a qualified personal trainer. I am benching 100kg x 6 reps to give u an idea of strength and I train legs and back of course. I weigh 78KG and am fairly lean already and u can see my "4pack" but only when i flex and i have a fair bit of fat around the lower abs. I am very educated with diet as well and eat high protein meals 5-6 times a day and control my calorie intake. My goals for taking AAS is to get past my weight training plateau and more so to look good for my part time job as a topless waiter for hens nights. I want to put on about 4kg of muscle while remaining extremely lean. I have read a lot on the different type of AAS and I am thinking about taking sus 250 with winstrol, both intramuscular. 250 of sus per week and the winny EOD. This is my first time on AAS and would appreciate any guidance. Thanks in advance,
Heshy
Last edited:
needtogetaas
New member
try a cycle that is more like this
1-14 test 250mg every week
1-14 400mg primo every week
1-14 hcg 500 ius twice a week
10-14 hcgenerate 5 caps every day
14-20 forma-stanzol 5 pumps am and pm
16-20 unleashed and post cycle 3 caps of each every day
10-16 need2slin 1 cap 3 times a day 30 mins before meals
keep some letro on hand for side effects but do not use it unless you have to.
You can send me a pm to get more inof on a cycle like this my friend.
needtogetaas
New member
SapperKicks
New member
best thread for newbs. thanks
I am looking to start my fist cycle. I dont know what to take. I heard its good to stack an oral like Dbol with an injectable like testosterone. IS this true? I weigh 176, im 5'10 and im average in body build.
And also, how long does it take before i get to see results
LAstly, i am terrible at weight lifting. Am i going to get better during and after my first cycle?
And also, how long does it take before i get to see results
LAstly, i am terrible at weight lifting. Am i going to get better during and after my first cycle?
needtogetaas
New member
Easy HCG Dosage Calculator Chart
do not buy anything from the site as they will scam you. But they got a cool little hcg chart to use for the newblits
do not buy anything from the site as they will scam you. But they got a cool little hcg chart to use for the newblits
Can anyone here give me guidance with taking steroids. I am 35 yrs old. 160 lbs. 5'10" and lean with good athletic frame. I have purchased Dianabol 10, Deca Durabolin 250, equipoise 250, Testosterone C, clomid 50, and nolvadex 20 for a stack. This is my first cycle ever. Can I please get some expert advice on how to go about taking these products and what doseages I should use.
thanks
thanks
Saintinistic
New member
1st cycle? ffs, don't take all of those. Bloody hell...
Start off with just ONE, which would be the test. Do some reading around here BEFORE you listen to the person you got them off.
See, there are many things to consider. I was 37 when I did my 1st cycle of test. That is all a beginner really needs. save the rest for cycles 2 and 3.
You have a good athletic frame. What does that mean? Do you weight train? Have you been weight training for a few years?
You don't use them to get big, before you have even taken your body to it's limits.
Diet. whats yours like?
Read this thread from the beginning.
cheers
needtogetaas
New member
just pm me and I will help you.
needtogetaas
New member
http://www.elitefitness.com/forum/g...gens-side-effects-preventing-gyno-720251.html
Over the last 4-5 years there has been one subject people have come to me for help with over ans over again. One subject and one problem that every man fears and no man wishes on even there worst enemy. Gyno is a horrible horrible thing to deal with. It can ruin your life, rob you of confidence and change the way you look, act, and feel about your self. Often it leaves people feeling hopeless if they can not get rid of gyno right away and then surgery seems to be the only option they have to turn to.
Gyno surgery is not a cheap option though. It can cost not only thousands of dollars but life long problems and side effects. A messed up surgery job can never be fixed and once its done you are stuck with it.
Thankfully we have advanced and many of us are starting to learn that in time almost any if not all gyno can be cured with out surgery. Some times it may only take a month or two but for others it can take up to a year. However all gyno can be cured with the right combination of drugs,products, and cycles.
The main problem is no one knows how, or is willing to trust another to show them. Well for years now I have been helping thousands of people with gyno and I am happy about what I do. I do not charge for it because I feel charging a man when he is down like that is immoral and wrong. So I offer my service free of charge to hundreds of people a day when I have the time ( I always find the time). My friends some of the info in this Article may seem jumbled or even hard to understand to many. Try and read everything and understand what you can. In the end just remember I am always here to help you and you can ask me anything you like, Send me a pm, a email, or even call me any time I am always here to help a fellow friend or brother in Iron.
Just because you think I am to busy, or because you do not know me very well yet is no reason not to contact me. I am here to serve the members of this forum and all people who need help. This is no laughing matter and some people out there need serous help. That is why I am here!!!! I assure you that your gyno ( any kind) can be prevented and or cured. Do not believe the hype that the only way is surgery
First before we can understand how to combat and deal with gyno we must first have a better understanding of the hormones involved with creating gyno. What they are, what they do, and even where they come from.
What is Estrogen?
Estrogen hormones are vital to the estrous cycle, and function as the primary female sex hormone. It also contains neuro-stimulative properties which is why too much estrogen can cause anxiety. Estrogen is synthesized in all vertebrate mammals as well as certain insects. The existence of these steroid hormones in both mammals and insects conveys that estrogenic sex hormones have an antique evolutionary account.
E1 (good estrogen plays a role in bone formation and bone preservation. Estrogen does also play a role in preservation of cholesterol through increasing High Density Lipoprotein levels and lowering Low Density Lipoprotein levels. Estrogen also allows for healthy looking skin since it increases the production rate of melanin which explains why women are so pretty! Just take a look at Marilyn Monroe the perfect example of a beautiful bottle shaped women with plenty of estradiol. Estrogen also keeps women in a better mood, specifically estradiol.
Unfortunately, Estrogen (E2) also plays a role in initiating prostate/breast cancer. Estrogen also aggravates blood platelet aggression which could lead to a severe blood clot, which leads to a stroke or heart attack/heart disease. Estrogen decreases fecal matter motility which leads to constipation. Estrogen also leads to loss of muscle mass and gains of fat deposits within the adipose tissues. Estrogen also leads to an increase in cortisol levels along with a rise in SHBG's. This quickly will lead to a DROP IN TESTOSTERONE! In theory, this could explain why steroid user's testosterone levels do not come back 100% percent with when usinga SERM alone for pct!! there is documentation showing that Selective Estrogen Modulators raise SHBG levels as well as studies that show they " raise not lower" estrogen levels.
There are other sources of estrogen as well; the other common types are xenoestrogens, mycoestrogens, and phytoestrogens.
These are outside sources of estrogen and we encounter them every day of our lives. often they are the cause for pubertal gyno a grossly growing problem around the world.
Xenoestrogens are chemically produced compounds that have estrogenic effects and differ chemically from naturally occurring estrogenic substances such as female estrogen hormones. As a heterogeneous group of chemicals that are hormonally living compounds. Xenoestrogens show similarity to other estrogens such as phytoestrogens and mycoestrogens. Xenoestrogens also have pharmacological estrogens (estrogenic action is an intended effect, as in the contraceptive pill), but of course other chemicals can too have estrogenic effects. Xenoestrogens have been presented to the environment by industrial, agricultural, chemical companies and consumers only in the last 70 years give or take. However; archiestrogens have been a omnipresent part of the environment even prior to the existence of the human race. There is evidence that shows xenoestrogens create oncogenes by overstimulating proto-oncogenes. When an oncogene is highly stimulated it becomes a tumor cell which we all know is bad news. There is significant evidence in a variety of recent studies linking xenoestrogens to the onset of breast cancer by an increase in breast cancer growth within in the tissue of the mammary. Xenoestrogen exposure has shown to be a reason why boys have delayed pubertal onrises, these xenoestrogens have also been linked to giving pubertal boys gynecomastia or also known as GYNO. Gyno does not look appealing can really lower a young man or adult's self esteem. Gyno makes the difference of someone not being able to take of the shirt at the beach because of the fear that someone may make them feel less of a man by pointing out the gyno on his chest. (Pediatrics. 2003 Jul;112(1 Pt 2):247-52.) Xenoestrogen exposure and consumption has also been linked to testicular atrophy and reduction of gondal size. This issue leads to a hault in spermatogenesis, reproductive problems, barely or no sperm motility, an increase in estrogen to testosterone ratio which leads to the cessation of testosterone production, all these issues become VERY problematic to the HPTA.
One well known Xenoestrogen is BPA; which is known to dramatically decrease DNA methylation by increasing hypomethylation. This causes a sudden rise in estrogens which causes problems to the male endocrine system. Bisphenol A functions as a xenoestrogen by binding STRONGLY to estrogen-related receptor γ (ERR-γ). This unidentified ligand behaves as a constitutive activator of transcription. BPA seems to bind strongly to ERR-γ (dissociation constant = 5.5 nM), but not to the estrogen receptor (ER). BPA binds to the ERR-γ to preserve its basal constitutive activity. (J Biochem. 2007 Oct;142(4):517-24. Epub 2007 Aug 30.) BPA has also been linked to an increase in prostate size and aggrevating prostate cancer. Another study displayed how BPA is an estrogen agonist and causes PERMANENT growth in the prostate, in other words its irreversible which is NOT GOOD! Exogenous estrogen was also shown to be the culprit in permanent growth of the prostate. Just a heads up, BPA is what a lot of companies use to make their plastic, many coming from plastic bottles. You might want to give your kids more Brita or filtered water instead of plastic bottles to avoid exposure to BPA. Another option would be to avoid canned foods with their notoriety of containing BPA. The scary fact is that it does not have to be much BPA exposure in order to cause all these affects, which something people should really analyze. BPA has also been linked to causing a drop in dopamine which leads to a LOSS IN LIBIDO and a drop in memory, in other words, an aging brain. BPA has also been shown to have negative effects on the thyroids which can be detrimental to fat loss and the homeostasis of the body. I have seen people have such drastic weight fluctuations due to their thyroid malfunction condition. (Journal Of Health Science. Vol. 55 (2009), No. 2. 147-160).
Nonylphenol is of the organic compounds which are subsets of the alkylphenols. Nonylphenol is a useful precursor to certain detergents/laundry detergents. They are even used in contraceptives and condoms, really scary stuff indeed. Nonylphenol is considered to be an endocrine disruptor due to frail ability to mimic estrogen and in turn; disrupt the HPTA of the male endocrine system. The effects of nonylphenol is not as potent as other Xenoestrogens because nonylphenols are not very close structural mimics of estradiol, but the levels of nonylphenol can be sufficiently high to cause damage to the male endocrine system. Nonylphenol has been commonly detected in waste water streams across the world, which is a problem since we wash our clothes with that same water. For example, nonylphenol has been detected both in the Great Lakes and in the region of New York City. Nonylphenol is persistent in the environment, therefore lingers with the potential to negatively affect the humans and of course males endocrine systems. Nonylphenol also accumulates overtime, which is dangerous to those who eat meat, another reason why people should very conscious of where they buy their meat from.
Parabens are found in lotions and also known to be xenoestrogens with pro-breast cancer activity. However it is one of the weaker forms of xenoestrogens.
There is some evidence suggesting that the food preservative BHA is also a xenoestrogen, California has already made it a policy to label BHA as a carcinogen.
DDT which is a WELL KNOWN insecticide has also a xenoestrogen has been touted as an endocrine distributor because of its negative effects on semen quality. It's a highly estrogenic component that causes a decline in testosterone. There is research that shows that DDE a metabolite of DDT acts as anti-androgen. This means that one will feel and act less like a man when exposed to it, ranging from a lack of libido to being a straight up wimp. DDT has also been linked to causing increased risks of diabetes and also provoking a lack of function within the thyroid hormones. They have also linked DDT to testicular cancer, which is more proof for how destructive this xenoestrogen is to the endocrine system. DDT is also linked to breast cancer but that is pretty obvious since it is a XENOESTROGEN.
As you can see Xenoestrogens range from sunscreen lotion to women's cosmetics, this lets you know that our endocrine system is threatened everyday just by our environment alone.
My friends the odds are stacked against us, and it does not end here.
Phytoestrogens
Phytoestrogens also known as "dietary estrogens", are a varied group of naturally occurring non-steroidal plant compounds that share of their similar structure with estradiol, and have the ability to cause estrogenic much more often than anti-estrogenic effects. Phytoestrogens primarily belong to a large group of substituted polyphenolic compounds; which comprise of the coumestans, prenylated flavonoids (the hobs you find in beer) and isoflavones (soybean are genistein and daidzein which are all bad for the endocrine system) are three of the most active in estrogenic effects in this class. The most researched and documented are the isoflavones, which are normally found in soy and red clover. Lignans have also been identified as phytoestrogens, although they are not flavonoids. Mycoestrogens have comparable structures and effects, but are not related to plants; they are mold metabolites of Fusarium. Phytoestrogens exert their effects mostly through binding to estrogen receptors (ER). There are two variants of the estrogen receptor, alpha (ER-α) and beta (ER-β) and numerous phytoestrogens display fairly higher affinity for ER-β compared to ER-α. Besides the interaction with estrogen receptors, phytoestrogens can also modulate the concentration of endogenous estrogen hormones by binding or ceasing efficiency of some enzymes, and this could affect the bioavailability of sex hormones by binding or stimulating the synthesis of sex hormone binding globuline. Foods with the highest relative phytoestrogen content were nuts and oilseeds, followed by soy products, cereals and breads, legumes, meat products, and other processed foods that may contain soy, vegetables, fruits, alcoholic, and nonalcoholic beverages. Flax seed and other oilseeds contained the highest total phytoestrogen content, followed by soybeans and tofu. The highest concentrations of isoflavones are found in soybeans and soybean products followed by legumes, whereas lignans are the primary source of phytoestrogens found in nuts and oilseeds (e.g. flax) and also found in cereals, legumes, fruits and vegetables.
Phytoestrogen concentration varies in diverse foods, and can contrast significantly within the same group of foods depending on processing mechanisms and the type of soybean extract used. Legumes, whole grain cereals, and several seeds are high in phytoestrogens. A more broad list of foods known to have phytoestrogens includes: soybeans, tofu, soy beverages, flax, sesame seeds, barley, dried beans, lentils, apples, carrots, pomegranates, wheatberries, oats, wheat germ, rice bran, soy linseed bread, ginseng, hops bourbon, beer, fennel yams, rice, alfalfa, mung beans and anise.
There has been an increase in reports about incidences of male reproductive abnormalities and falling sperm counts have driven interest into the nature of these threats to worldwide fertility. Xenoestrogens have been flagged as major culprits. These non-steroidal estrogens/oestrogens of plant derivation are potent endocrine disruptors that modulate normal physiological functions. Phytoestrogens have also become a major factor in the usual Western fast food diet over the last few decades. Soy formula milk is another widespread source of phytoestrogens, now used increasingly as an alternative to breast or cow's milk for infants with allergies. This use is of a HUGE concern since the most vulnerable periods for oestrogenic abuse are thought to be the pre- and neonatal periods when almost irreversible harm can be brought onto the developing child. Studies concerning the safety of phytoestrogens are now needed either to relieve fears or increase awareness of the effects of our current diet on potential fertility. (2005, Vol. 8, No. 3 , Pages 197-207 (doi:10.1080/14647270500030266)).
Phytoestrogens don't even provide benefit in women
A HEALTHY women post menopausal consumes less than 1mg of phytoestrogens, which is pretty much nothing. There was a study conducted with 964 postmenopausal Caucasian women who participated in the Framingham Offspring Study and completed the Willett food-frequency questionnaire (FFQ). By searching the agricultural and medical literature, they were able to identify food sources of phytoestrogens. The concentrations of the diverse isoflavones, coumestrol and lignans in each food in the FFQ were scored in seven categories, then multiplied by the serving size of the food, and the frequency of its expenditure. The estimated daily median intake of the isoflavone daidzein was 39 microg (24-57 microg); of genistein, 70 microg (28-120 microg); of formononetin, 31 microg (13-44 microg); and of biochanin A, 6 microg (2-11 microg). Median total intake of isoflavones was 154 microg (99-235 microg). The main sources of isoflavones were peas and soy beans. The estimated daily intake of coumestans was 0.6 microg (0.2-1.7 microg), with broccoli as the key source. The estimated daily median intake of matairesinol was 19 microg (12-28 microg) and of secoisolariciresinol 560 microg (399-778 microg). The median total intake of lignans was 578 microg (416-796 microg). The main source of the lignans was fruits. (J Nutr. 2001 Jun;131(6):1826-32.)
As you see from the information above, one serving alone of these phytoestrogens can be detrimental to women's health over a period of time. Even more so to a man!
Phytoestrogens have also been linked to immunosuppressive effects along a decrease in thyroid output function. These studies that linked phytoestrogens to immunodeficiency showed how consumption of SOY isoflavone and genistein lowered t-cells which we know is something that occurs usually in HIV/AIDS patients. HIV/AIDS is a diease which causes one's immune system to shut down completely allowing one to catch any disease.
Estrogen is all around us; it is well known that the estradiol level in 55-year old men, for example, is usually a bit higher than that of a 55-year old woman. Unfortunately after the age of the 30, men's testosterone levels continue to plummet 10 percent every 10 years. Many factors lead to estrogen dominance ranging from life stress to the xenoestrogens we consume. A man, however, does not develop breasts because he has a higher testosterone level than women do and a lower estrogen ( the world is changing this).
As men age, their estradiol levels gradually rise, whereas their progesterone and testosterone levels gradually fall. The hormone balance changes. These gradual changes lead to reduction in testosterone benefits and eventually to estrogen dominance.
That is, a mans estradiol effects emerge since his testosterone level is not sufficient to block or balance out the Estrogen. The Estrogen Dominance then stimulates breast cell and prostate hypertrophy. Estrogen Dominance is responsible for the majority of breast cancers and is the only known cause of endometrial cancer in women. Since the male prostate is pretty much the equivalent of the uterus, it should not be unexpected that estrogen dominance is also a major cause of prostate cancer as stated before.
In today's world estrogen dominance is accruing at a much younger age and at a much faster rate. Even for much younger when when estrogen is not completely dominant the much higher levels of estrogen is still causing life long problems.
Estrogen dominance is a growing health concern for Men all over the world. Although it is more common in older men. It is quickly becoming a epidemic in younger men causing such problems as infertility, erectile dysfunction, enlarged prostate, and certain types of cancer. Many of the symptoms can be seen in our youth today. Many of the symptoms you may even notice in your own life.
The symptoms of estrogen dominance in men include:
Low sex drive
Impotency/erectile dysfunction
Infertility
Male pattern baldness
Gynecomastia, or "man boobs"
Weight gain
Enlarged prostate
Prostate cancer
Testicular cancer
mood swings
and many many more.
Symptoms of the on set of gyno
puffy nips
Itchy and or sore nips
lactating or fluid from the nipples
Pain to the touch
Gynecomestia
In adolescent boys, the condition is often a source of distress, but for the large majority of boys whose pubescent gynecomastia is not caused by obesity, usually the breast enlargement shrinks or disappears within several years. The condition is usually caused by an imbalance of estrogenic to androgenic (usually estradiol to testosterone ratio) effects on the breast tissue, resulting in a surge of unconstrained estrogen action on breast tissue. Approximately only 4 to 10% cases of gynecomastia are due to drugs. The aromatization takes place in the cyto-chrome enzyme P-450. Both Digoxin and Furosemide are drugs reported to cause the gyno as well, however; anabolic androgenic steroids are the most common drugs in causing gynecomastia. Breast prominence can result from hypertrophy of the breast tissue, chest adipose tissue (fat) and skin, and is normally a combination of the two. Breast distinction due exclusively to too much adipose is often termed pseudogynecomastia aka psedogyno or sometimes lipomastia.
Types of Gynecomastia
Puffy Nips: is one of the more common forms of gynecomastia. This glandular tissue buildup is concentrated under and in general confined to the areola, however; it can be slightly extended outside the areola forming a dome shaped image to the areola.
Glandular: This form of gyno is common with bodybuilders as a result of the use of anabolic androgenic steroids. The undeniable overload testosterone levels from AAS (specifically any form of test) are converted to estrogen via aromatization. Bodybuilders and along other athletes are afflicted with gynecomastia in its purest form when suffering from AAS gyno. However; drug-induced gynecomastia can almost be resolves with the use of proper supplementation, if caught before permanent fibrosis develops and even after it can still be cured.
Gynecomastia in lean men is generally only a breast tissue gland with no added adipose tissue; heavier men will have sloppier looking gyno because of the adipose tissue covering the glandular tissue. Guaranteed treatment of pure gynecomastia can be done simply by removal of the breast tissue, which also rids of the gyno tissue. Most of the time the glandular tissue is benign but at times it can become malignant, so gyno is NO laughing matter and must be taken serious especially during or after a cycle. So good bro's make sure you keep an eye out on your chest during cycle and take good care of yourself during and post cycle. Again I must stress that any and almost all gyno can be treated with out surgery but in some cases it is better to take care of it sooner rather then later.
Adolescent/Pubertal Gynecomastia: The Congenital or Hereditary Gynecomastia is on average evident by the ages of 9 to 18 in young males. About thirty percent to sixty percent of young adolescent boys experience pubertal gynecomastia. UNFORTUNATELY, thirty percent possibly will live with enlarged male breasts for the rest of their lives, but in other cases the gynecomastia will go away with age. However, severe forms of adolescent gynecomastia may require an involvement, in conference with the patient, the parents, and child development professionals. Now of course there is hope for men who have lived with gyno all of there lives. Your parents may have neglected it but there is no reason you have to.
Adult or andropause gyno: This is the most common form of gynecomastia due to declining androgen levels with a rise in estrogen in elderly or post andropause men. Gynecomastia based off of post andropause in most adults is composed mainly of glandular tissue but it may contain varying amounts of adipose and tough tissue. Now you know why old men have those wrinkly chests that look like they never had the shape of pecs on them before.
Asymmetric/Unilateral: Unilateral gynecomastia occurs when only one breast is bigger due to a case of gynecomastia. The other breast is usually normal in size and shape. Bilateral Asymmetry occurs when gynecomastia is present in both breasts, of course; each being to a different degree. Most bodybuilders and boys during puberty acquire unilateral gyno, and by a pretty big margin I might add.
Severe gynecomastia: is described as an excess or loose skin, and severely enlarged breasts. This form of gyno is determined in part by age, reason being older people suffering from gynecomastia tend to have less skin elasticity and therefore will have a greater profusion of excess skin associated to gynecomastia. Experienced plastic surgeons will perform as much of the surgical treatment of severe gynecomastia as feasible through an aereolar incision so as to avoid widespread scarring. However, some scarring may be inevitable when treating severe cases of gynecomastia. The larger the gyno the more the scaring and this is why even if you are going to have surgery once should try to reduce gyno in size as much as possible beforehand
Pseudogynecomastia: is not composed of glandular tissue, but rather of adipose tissue (fat). It looks a lot like typical gynecomastia but requires different treatment. Exercise and diet can be very effective in preventing and fighting off pseudogynecomastia. Only if this regimen is unsuccessful should surgery be considered an option. This is generally the only type of gynecomastia which can be improved with liposuction, but removal may be used in some cases as well. This form of gyno is also known as "false Gynecomastia" and is often associated with obesity whereby insulin interacts with a surplus of sugars or certain carbohydrates, specifically those of which that have been processed.
Not just estrogen causes gyno!!
PROGESTERONE. IGF, GH, AND PROLACTIN INDUCED GYNECOMASTIA
Gynecomastia can be as a result of a number of agents including estrogens, progestins, GH, IGF-1, and prolactin may all be involved. Regarding prolactin, androgens decrease prolactin levels while estrogens increase prolactin. Non-aromatizing androgens such as DHT have by no means been shown to raise prolactin levels in humans, but testosterone has, due to its aromatization to estradiol. Prolactin secreting tumors also known as prolactinomas, are often linked with gyno. Of course in these cases the prolactin is believed to induce gyno by suppressing testosterone production. Prolactinomas that are adequately large to cause gynecomastia, do cause gyno as a consequence of the impairment of Gonadotropin secretion, which leads to secondary hypogonadism. Remember Prolactin only has a direct effect on breast tissue only when estrogen is present so they say ( how much is needed?). This is why they say lowering estrogen levels will lead to lower prolactin levels and then eventually less occupied progesterone receptors. Keep in mind just using something to lower estrogen will not cure gyno when progesteonr/prolactin is one of the culprits. Also take into consideration that as IGF and GH levels rise, they will also rise within the mammary glands which could cause a growth of glandular tissue if the rise in IGF and GH is too rapid. This in basic terms means you got the GYNO.
After awhile, excess levels of prolactin also lead to laction which is when the mammary gland secretes milk. I don't know about you my friend but milk coming from my nips just is not something I want to experience. And I have no plans to feed young children even my own. Secretion is not the only way of knowing your gyno is of progesterone/prolactin nature but it is one clear indicator.
Aromatizing AAS
There are AAS that are more likely to induce gynecomastia whether it be due to aromatization or aromatization with progesterone site binding. Here is a small/general list of the more common and moderate to high aromatizing compounds.
Any form of Testosterone
Dianabol
Deca Durabolin/nanodrolone (progesterone)
MethylTestosterone
Methyltrienolone
Any form of Trenbolone (progesterone)and androgenic
Anadrol 50 Receptor cross over
Nondrolone Laurate (progesterone)
Norethandrolone
M 1,4ad
13-ethyl-3-methoxy-gona-2,5(10)-dien-17-one (progesterone)
2a, 17a-dimethyl-etiocholan-3-one-17b-ol
13 ethyl 3 methoxy
4-ad
Many people have reported receiving gyno from Superdrol, people question those accusations since superdrol is a 5aReduced compound, however; Professor Filimanov the formulator of Need2Slin.
" Prolactin is normaly caused by progestins, but can also be caused by dht, how?
For example, it is currently understood that when testosterone enters the cell cytoplasm it is subsequently converted to the more "active" androgen, dihydrotestosterone, DHT, by reduction at the 5alpha position, this is normal. Dihydrotestosterone is then either bound to a cytoplasmic "receptor" protein Rc, or is further metabolized to either 5alpha-androstane-3alpha,17beta-diol or 5alpha-androstane-3beta,17beta-diol ,DIOL. The binding of DHT to its cytoplasmic receptor protein results in translocation of the steroid-receptor complex into the nucleus where presumably the complex dissociates and DHT exerts its androgenic effects. The transport of DHT to the nucleus can also result from the conversion of testosterone to DHT by nuclear membrane-bound 5alpha-reductase. Prolactin augmentation of DHT effects is envisioned as resulting from interaction of prolactin with its receptor, which due to the large size of the prolactin molecule is probably located in or on the plasma membrane.
Because superdrol is androgenic, but lacks the ability to show affinity via 5ar, it circulates, and this causes the large amounts of androgens to look for a transporter, so that it can bind to the androgen receptor, so it uses prolactin which has a high affinity to cytoplasmic receptor protein, allowing the androgens, testosterone, to be carried and allowing them to convert to dht, only problem is prolactin hormone or luteotropic hormone is synthesised and secreted by sex binding lactotrope cells in the adenohypophysis (anterior pituitary gland, And this gland now produces more prolactin to help deal with the large amount of testosterone circulating that hasnt bound to the estrogen of androgen receptor, Part of the reason why superdol is so anabolic, So instead of binding to the androgen receptors in the scalp and the prostrate it converts to dht through this unique process, using prolactin to enter the cytoplasmic receptror protein, and allowing it to convert to dht and then bind to the androgen receptors in the muscle, causing its distinct hardening effects, it still can't bind to the scalp or prostrate via 5ar as the form of dht it has converted too doesnt allow for that affinity.
So more prolactin is produced to allow for the superdol to find a receptor ,this excess prolactin triggers a process that fills the breast with milk via a process called lactogenesis, in men however it causes a distinct enlargment of the mammary gland and can even cause a man to lactate.
If superdrol had better binding to the androgen receptor via 5AR then this problem would be prevented, the other thing is that prolactin production can remain elevated for months after a cycle has finished, and once the androgen has been removed, ( the cycle is over) the cytoplasmic receptor proteins have nothing to do other than to allow the prolactin to proceed with its hormonal action within the body, causing the male mammary gland to enlarge ready to produce milk... Hence the REBOUND GYNO, this is why proper pct is needed for superdrol, and the use of something to prevent prolactin."
As you see Professor Filimanov reinstates the correlation between prolactin/progesterone and estrogen which could lead to gyno even with a compound such as Superdrol.
Receptor cross over can also occur. Hormones are keys and your receptors are like locks. What happens is a hormone is ether placed in the body (by an out side source) or it is produced with in the body. This hormone we will call a "key". The hormone then sets out to find a "lock" that it can fit into,turn, and sequentially open up the components inside. To go one step more and make it a little more simple I will explain it like this.
Imagine your receptors as little treasure chest. Inside is chest it a set of instructions. This set of instructions can be a number of things. It can be directions to another chest or a task that must now be carried out. With out something to open the chest the instructions can never be carried out or the next chest found and opened. Following me ?
Some hormones are more like "dummy keys" will call them. They will find a "lock" and set them self into the "Lock hole" but then that is it. They never turn the key and open up whats inside. so we see hormones often work like this
1. Key fits a lock. AKA a hormone found a lock and set its self into it. Pretty simple
2. Key was not a dummy key and when it set its self into the lock it turned and opened up the chest letting out the instructions inside that will now be carried out.
3 "master keys" We will call them. Or "Muti function keys"
A subject not much talked about on any of the steroid forums or even in many articles I have come across is the fact that. In respect to anabolic steroids (out side source of hormones) most of them have all been explained as just "lock" and "key" . However the truth is pro hormones,disginer steroids and steroids all have "Multi function" and some even "Master key" functions.
as you can see explained earlier.
By now I am sure you are confused but that is fine. The body is a extremely complex thing and everyone's body acts the same yet differently when it comes to hormones. This is why a lot of times you must work with some one to help find out what works for you. To help find out more about you and how your body works, as well as the basics of science and how all things "should" work.
This last part has thrown many for a loop. Leaving many wondering why the have gyno when they did everything right, and used all the right compounds. The sad truth is there is no Guarantee that you will not end up with a case of gyno.
However there is one Guarantee and that being that no matter what the odds are on your side that your gyno can be reversed with proper supplementation my friends. You just need to know how. Often it is best to work with some one who has experience and has worked with many others to prevent or reverse gyno of all kinds.
Believe me friends, I know it is NOT easy by any means to feel comfortable about your body when you have gyno whether it be from puberty or AAS usage. It can really make one depressed knowing that their chest is not masculine appealing to the people around them or themselves. I know people who LOW SELF ESTEEM for years because of their pubertal gyno. We as people struggle on a daily basis to fit in whether it be through appearance, social status, or self accomplishment. The people I witnessed getting rid of their gyno, achieved all those forms of greatness and more at least to themselves which is all that matters; since it's your body and your life.
Now Of course it is time for the good part. The part every one has been waiting for. If all gyno can be cured then how?
Well My friends I am sorry to be the one to tell you. Yes there are drugs and products out there that work to reduce estrogen. Yes there are drugs that reduce progesterone and or porlactin. Yes there are serms that block the progesterone receptors. Yes all of these drugs and or supplements can be used to reduce,prevent, and even cure existing gyno.
However anyone out there giving you a cookie cutter gyno removing program is simple trying to do one thing. SELL YOU A PRODUCT/GROUP OF PRODUCTS AND OR DRUGS. Or they are only speaking from experience for what worked for them NOT YOU!!!
How many of you have used a friends advice because it worked for them,only to be disappointed when it does not work for you?
How many of us have tried everything only to find nothing worked? Or have you tried everything?
The fact of the matter is I do not hand out cookie cutter programs because although they may work for some they do not work for everyone. Each person must find out what works for them. Each person must learn how they will react to different products,drugs, and combination thereof.
One person my use nolva and it worked great for them. Another may use it and there gyno gets worse or they have a rebound long after. You must learn to read your body, learn what it is telling you. You must learn how to change things exactly when they need to be changed for you and why.
You must start off by having everything you need before hand. This is the one thing that is the same for everyone. When people make the statement "aaah just get some letro" Or hey man all you need is some arimadex and dostinex you will be fine" These people hardly know what they are talking about most of the time. They know what worked for them, they know what might have worked for others. However the most importent fact they know nothing about is WHAT WORKS FOR YOU AND HOW YOU WILL REACT.
Everyone is always looking for the cheap way out. Or the easy way out. SO often we jump all over the quick easy and cheap advice that is so readily handed out on the aas forums today.
When you are ready to do it right, when you want results, when you are at the end of your rope. Then you will bite the bullet and spend the money to get "everything" you need. A gyno removing program can have side effects and the proper supplements should be taken to counteract them.
When it comes to some causes of gyno you have to understand that it took years of exposure to out side estrogen to cause the problem. It may not be resolved overnight but it can be in the end and I am always here to help you do this.
Feel free to pm me any time my friends I am always here on the forums.
preventing Gyno of course is another topic in its self. When it comes to on cycle gyno prevention one should always use as little as he can or non at all if he can get away with it.
If you do not need it then do not use it. However always have it on hand IMO.
Many of the drugs known today for gyno prevention all have there pitfalls and some are better then others for different reasons. If prevention is what you are looking for then forma-stanzol happens to be the best choice for many different reasons. You can read more about this compound here in this thread. http://www.elitefitness.com/forum/a...roid-best-pct-best-gyno-treatment-698487.html
5 to 10 pumps morning and night is all one would need depending on the dosing of the cycle you are on. Back up plans should always be in place and on hand. One should always have letro and dostinex on hand but never a need to use them unless nothing else works.
During pct often people use just nolva or clomid and neglect that fact that these compounds do nothing to lower estrogen nor do they do anything at all for raised levels of prolactin or progesterone. Forma-stanzol on the other hand does.
Lastly I would like to open then thread up for discussion and questions by all. If you have any feel free to ask here or send me a pm and I will get to them as I can. Thank you to all.
Over the last 4-5 years there has been one subject people have come to me for help with over ans over again. One subject and one problem that every man fears and no man wishes on even there worst enemy. Gyno is a horrible horrible thing to deal with. It can ruin your life, rob you of confidence and change the way you look, act, and feel about your self. Often it leaves people feeling hopeless if they can not get rid of gyno right away and then surgery seems to be the only option they have to turn to.
Gyno surgery is not a cheap option though. It can cost not only thousands of dollars but life long problems and side effects. A messed up surgery job can never be fixed and once its done you are stuck with it.
Thankfully we have advanced and many of us are starting to learn that in time almost any if not all gyno can be cured with out surgery. Some times it may only take a month or two but for others it can take up to a year. However all gyno can be cured with the right combination of drugs,products, and cycles.
The main problem is no one knows how, or is willing to trust another to show them. Well for years now I have been helping thousands of people with gyno and I am happy about what I do. I do not charge for it because I feel charging a man when he is down like that is immoral and wrong. So I offer my service free of charge to hundreds of people a day when I have the time ( I always find the time). My friends some of the info in this Article may seem jumbled or even hard to understand to many. Try and read everything and understand what you can. In the end just remember I am always here to help you and you can ask me anything you like, Send me a pm, a email, or even call me any time I am always here to help a fellow friend or brother in Iron.
Just because you think I am to busy, or because you do not know me very well yet is no reason not to contact me. I am here to serve the members of this forum and all people who need help. This is no laughing matter and some people out there need serous help. That is why I am here!!!!
I assure you that your gyno ( any kind) can be prevented and or cured. Do not believe the hype that the only way is surgery First before we can understand how to combat and deal with gyno we must first have a better understanding of the hormones involved with creating gyno. What they are, what they do, and even where they come from.
What is Estrogen?
Estrogen hormones are vital to the estrous cycle, and function as the primary female sex hormone. It also contains neuro-stimulative properties which is why too much estrogen can cause anxiety. Estrogen is synthesized in all vertebrate mammals as well as certain insects. The existence of these steroid hormones in both mammals and insects conveys that estrogenic sex hormones have an antique evolutionary account.
E1 (good estrogen plays a role in bone formation and bone preservation. Estrogen does also play a role in preservation of cholesterol through increasing High Density Lipoprotein levels and lowering Low Density Lipoprotein levels. Estrogen also allows for healthy looking skin since it increases the production rate of melanin which explains why women are so pretty! Just take a look at Marilyn Monroe the perfect example of a beautiful bottle shaped women with plenty of estradiol. Estrogen also keeps women in a better mood, specifically estradiol.
Unfortunately, Estrogen (E2) also plays a role in initiating prostate/breast cancer. Estrogen also aggravates blood platelet aggression which could lead to a severe blood clot, which leads to a stroke or heart attack/heart disease. Estrogen decreases fecal matter motility which leads to constipation. Estrogen also leads to loss of muscle mass and gains of fat deposits within the adipose tissues. Estrogen also leads to an increase in cortisol levels along with a rise in SHBG's. This quickly will lead to a DROP IN TESTOSTERONE! In theory, this could explain why steroid user's testosterone levels do not come back 100% percent with when usinga SERM alone for pct!! there is documentation showing that Selective Estrogen Modulators raise SHBG levels as well as studies that show they " raise not lower" estrogen levels.
There are other sources of estrogen as well; the other common types are xenoestrogens, mycoestrogens, and phytoestrogens.
These are outside sources of estrogen and we encounter them every day of our lives. often they are the cause for pubertal gyno a grossly growing problem around the world.
Xenoestrogens are chemically produced compounds that have estrogenic effects and differ chemically from naturally occurring estrogenic substances such as female estrogen hormones. As a heterogeneous group of chemicals that are hormonally living compounds. Xenoestrogens show similarity to other estrogens such as phytoestrogens and mycoestrogens. Xenoestrogens also have pharmacological estrogens (estrogenic action is an intended effect, as in the contraceptive pill), but of course other chemicals can too have estrogenic effects. Xenoestrogens have been presented to the environment by industrial, agricultural, chemical companies and consumers only in the last 70 years give or take. However; archiestrogens have been a omnipresent part of the environment even prior to the existence of the human race. There is evidence that shows xenoestrogens create oncogenes by overstimulating proto-oncogenes. When an oncogene is highly stimulated it becomes a tumor cell which we all know is bad news. There is significant evidence in a variety of recent studies linking xenoestrogens to the onset of breast cancer by an increase in breast cancer growth within in the tissue of the mammary. Xenoestrogen exposure has shown to be a reason why boys have delayed pubertal onrises, these xenoestrogens have also been linked to giving pubertal boys gynecomastia or also known as GYNO. Gyno does not look appealing can really lower a young man or adult's self esteem. Gyno makes the difference of someone not being able to take of the shirt at the beach because of the fear that someone may make them feel less of a man by pointing out the gyno on his chest. (Pediatrics. 2003 Jul;112(1 Pt 2):247-52.) Xenoestrogen exposure and consumption has also been linked to testicular atrophy and reduction of gondal size. This issue leads to a hault in spermatogenesis, reproductive problems, barely or no sperm motility, an increase in estrogen to testosterone ratio which leads to the cessation of testosterone production, all these issues become VERY problematic to the HPTA.
One well known Xenoestrogen is BPA; which is known to dramatically decrease DNA methylation by increasing hypomethylation. This causes a sudden rise in estrogens which causes problems to the male endocrine system. Bisphenol A functions as a xenoestrogen by binding STRONGLY to estrogen-related receptor γ (ERR-γ). This unidentified ligand behaves as a constitutive activator of transcription. BPA seems to bind strongly to ERR-γ (dissociation constant = 5.5 nM), but not to the estrogen receptor (ER). BPA binds to the ERR-γ to preserve its basal constitutive activity. (J Biochem. 2007 Oct;142(4):517-24. Epub 2007 Aug 30.) BPA has also been linked to an increase in prostate size and aggrevating prostate cancer. Another study displayed how BPA is an estrogen agonist and causes PERMANENT growth in the prostate, in other words its irreversible which is NOT GOOD! Exogenous estrogen was also shown to be the culprit in permanent growth of the prostate. Just a heads up, BPA is what a lot of companies use to make their plastic, many coming from plastic bottles. You might want to give your kids more Brita or filtered water instead of plastic bottles to avoid exposure to BPA. Another option would be to avoid canned foods with their notoriety of containing BPA. The scary fact is that it does not have to be much BPA exposure in order to cause all these affects, which something people should really analyze. BPA has also been linked to causing a drop in dopamine which leads to a LOSS IN LIBIDO and a drop in memory, in other words, an aging brain. BPA has also been shown to have negative effects on the thyroids which can be detrimental to fat loss and the homeostasis of the body. I have seen people have such drastic weight fluctuations due to their thyroid malfunction condition. (Journal Of Health Science. Vol. 55 (2009), No. 2. 147-160).
Nonylphenol is of the organic compounds which are subsets of the alkylphenols. Nonylphenol is a useful precursor to certain detergents/laundry detergents. They are even used in contraceptives and condoms, really scary stuff indeed. Nonylphenol is considered to be an endocrine disruptor due to frail ability to mimic estrogen and in turn; disrupt the HPTA of the male endocrine system. The effects of nonylphenol is not as potent as other Xenoestrogens because nonylphenols are not very close structural mimics of estradiol, but the levels of nonylphenol can be sufficiently high to cause damage to the male endocrine system. Nonylphenol has been commonly detected in waste water streams across the world, which is a problem since we wash our clothes with that same water. For example, nonylphenol has been detected both in the Great Lakes and in the region of New York City. Nonylphenol is persistent in the environment, therefore lingers with the potential to negatively affect the humans and of course males endocrine systems. Nonylphenol also accumulates overtime, which is dangerous to those who eat meat, another reason why people should very conscious of where they buy their meat from.
Parabens are found in lotions and also known to be xenoestrogens with pro-breast cancer activity. However it is one of the weaker forms of xenoestrogens.
There is some evidence suggesting that the food preservative BHA is also a xenoestrogen, California has already made it a policy to label BHA as a carcinogen.
DDT which is a WELL KNOWN insecticide has also a xenoestrogen has been touted as an endocrine distributor because of its negative effects on semen quality. It's a highly estrogenic component that causes a decline in testosterone. There is research that shows that DDE a metabolite of DDT acts as anti-androgen. This means that one will feel and act less like a man when exposed to it, ranging from a lack of libido to being a straight up wimp. DDT has also been linked to causing increased risks of diabetes and also provoking a lack of function within the thyroid hormones. They have also linked DDT to testicular cancer, which is more proof for how destructive this xenoestrogen is to the endocrine system. DDT is also linked to breast cancer but that is pretty obvious since it is a XENOESTROGEN.
As you can see Xenoestrogens range from sunscreen lotion to women's cosmetics, this lets you know that our endocrine system is threatened everyday just by our environment alone.
My friends the odds are stacked against us, and it does not end here.
Phytoestrogens
Phytoestrogens also known as "dietary estrogens", are a varied group of naturally occurring non-steroidal plant compounds that share of their similar structure with estradiol, and have the ability to cause estrogenic much more often than anti-estrogenic effects. Phytoestrogens primarily belong to a large group of substituted polyphenolic compounds; which comprise of the coumestans, prenylated flavonoids (the hobs you find in beer) and isoflavones (soybean are genistein and daidzein which are all bad for the endocrine system) are three of the most active in estrogenic effects in this class. The most researched and documented are the isoflavones, which are normally found in soy and red clover. Lignans have also been identified as phytoestrogens, although they are not flavonoids. Mycoestrogens have comparable structures and effects, but are not related to plants; they are mold metabolites of Fusarium. Phytoestrogens exert their effects mostly through binding to estrogen receptors (ER). There are two variants of the estrogen receptor, alpha (ER-α) and beta (ER-β) and numerous phytoestrogens display fairly higher affinity for ER-β compared to ER-α. Besides the interaction with estrogen receptors, phytoestrogens can also modulate the concentration of endogenous estrogen hormones by binding or ceasing efficiency of some enzymes, and this could affect the bioavailability of sex hormones by binding or stimulating the synthesis of sex hormone binding globuline. Foods with the highest relative phytoestrogen content were nuts and oilseeds, followed by soy products, cereals and breads, legumes, meat products, and other processed foods that may contain soy, vegetables, fruits, alcoholic, and nonalcoholic beverages. Flax seed and other oilseeds contained the highest total phytoestrogen content, followed by soybeans and tofu. The highest concentrations of isoflavones are found in soybeans and soybean products followed by legumes, whereas lignans are the primary source of phytoestrogens found in nuts and oilseeds (e.g. flax) and also found in cereals, legumes, fruits and vegetables.
Phytoestrogen concentration varies in diverse foods, and can contrast significantly within the same group of foods depending on processing mechanisms and the type of soybean extract used. Legumes, whole grain cereals, and several seeds are high in phytoestrogens. A more broad list of foods known to have phytoestrogens includes: soybeans, tofu, soy beverages, flax, sesame seeds, barley, dried beans, lentils, apples, carrots, pomegranates, wheatberries, oats, wheat germ, rice bran, soy linseed bread, ginseng, hops bourbon, beer, fennel yams, rice, alfalfa, mung beans and anise.
There has been an increase in reports about incidences of male reproductive abnormalities and falling sperm counts have driven interest into the nature of these threats to worldwide fertility. Xenoestrogens have been flagged as major culprits. These non-steroidal estrogens/oestrogens of plant derivation are potent endocrine disruptors that modulate normal physiological functions. Phytoestrogens have also become a major factor in the usual Western fast food diet over the last few decades. Soy formula milk is another widespread source of phytoestrogens, now used increasingly as an alternative to breast or cow's milk for infants with allergies. This use is of a HUGE concern since the most vulnerable periods for oestrogenic abuse are thought to be the pre- and neonatal periods when almost irreversible harm can be brought onto the developing child. Studies concerning the safety of phytoestrogens are now needed either to relieve fears or increase awareness of the effects of our current diet on potential fertility. (2005, Vol. 8, No. 3 , Pages 197-207 (doi:10.1080/14647270500030266)).
Phytoestrogens don't even provide benefit in women
A HEALTHY women post menopausal consumes less than 1mg of phytoestrogens, which is pretty much nothing. There was a study conducted with 964 postmenopausal Caucasian women who participated in the Framingham Offspring Study and completed the Willett food-frequency questionnaire (FFQ). By searching the agricultural and medical literature, they were able to identify food sources of phytoestrogens. The concentrations of the diverse isoflavones, coumestrol and lignans in each food in the FFQ were scored in seven categories, then multiplied by the serving size of the food, and the frequency of its expenditure. The estimated daily median intake of the isoflavone daidzein was 39 microg (24-57 microg); of genistein, 70 microg (28-120 microg); of formononetin, 31 microg (13-44 microg); and of biochanin A, 6 microg (2-11 microg). Median total intake of isoflavones was 154 microg (99-235 microg). The main sources of isoflavones were peas and soy beans. The estimated daily intake of coumestans was 0.6 microg (0.2-1.7 microg), with broccoli as the key source. The estimated daily median intake of matairesinol was 19 microg (12-28 microg) and of secoisolariciresinol 560 microg (399-778 microg). The median total intake of lignans was 578 microg (416-796 microg). The main source of the lignans was fruits. (J Nutr. 2001 Jun;131(6):1826-32.)
As you see from the information above, one serving alone of these phytoestrogens can be detrimental to women's health over a period of time. Even more so to a man!
Phytoestrogens have also been linked to immunosuppressive effects along a decrease in thyroid output function. These studies that linked phytoestrogens to immunodeficiency showed how consumption of SOY isoflavone and genistein lowered t-cells which we know is something that occurs usually in HIV/AIDS patients. HIV/AIDS is a diease which causes one's immune system to shut down completely allowing one to catch any disease.
Estrogen is all around us; it is well known that the estradiol level in 55-year old men, for example, is usually a bit higher than that of a 55-year old woman. Unfortunately after the age of the 30, men's testosterone levels continue to plummet 10 percent every 10 years. Many factors lead to estrogen dominance ranging from life stress to the xenoestrogens we consume. A man, however, does not develop breasts because he has a higher testosterone level than women do and a lower estrogen ( the world is changing this).
As men age, their estradiol levels gradually rise, whereas their progesterone and testosterone levels gradually fall. The hormone balance changes. These gradual changes lead to reduction in testosterone benefits and eventually to estrogen dominance.
That is, a mans estradiol effects emerge since his testosterone level is not sufficient to block or balance out the Estrogen. The Estrogen Dominance then stimulates breast cell and prostate hypertrophy. Estrogen Dominance is responsible for the majority of breast cancers and is the only known cause of endometrial cancer in women. Since the male prostate is pretty much the equivalent of the uterus, it should not be unexpected that estrogen dominance is also a major cause of prostate cancer as stated before.
In today's world estrogen dominance is accruing at a much younger age and at a much faster rate. Even for much younger when when estrogen is not completely dominant the much higher levels of estrogen is still causing life long problems.
Estrogen dominance is a growing health concern for Men all over the world. Although it is more common in older men. It is quickly becoming a epidemic in younger men causing such problems as infertility, erectile dysfunction, enlarged prostate, and certain types of cancer. Many of the symptoms can be seen in our youth today. Many of the symptoms you may even notice in your own life.
The symptoms of estrogen dominance in men include:
Low sex drive
Impotency/erectile dysfunction
Infertility
Male pattern baldness
Gynecomastia, or "man boobs"
Weight gain
Enlarged prostate
Prostate cancer
Testicular cancer
mood swings
and many many more.
Symptoms of the on set of gyno
puffy nips
Itchy and or sore nips
lactating or fluid from the nipples
Pain to the touch
Gynecomestia
In adolescent boys, the condition is often a source of distress, but for the large majority of boys whose pubescent gynecomastia is not caused by obesity, usually the breast enlargement shrinks or disappears within several years. The condition is usually caused by an imbalance of estrogenic to androgenic (usually estradiol to testosterone ratio) effects on the breast tissue, resulting in a surge of unconstrained estrogen action on breast tissue. Approximately only 4 to 10% cases of gynecomastia are due to drugs. The aromatization takes place in the cyto-chrome enzyme P-450. Both Digoxin and Furosemide are drugs reported to cause the gyno as well, however; anabolic androgenic steroids are the most common drugs in causing gynecomastia. Breast prominence can result from hypertrophy of the breast tissue, chest adipose tissue (fat) and skin, and is normally a combination of the two. Breast distinction due exclusively to too much adipose is often termed pseudogynecomastia aka psedogyno or sometimes lipomastia.
Types of Gynecomastia
Puffy Nips: is one of the more common forms of gynecomastia. This glandular tissue buildup is concentrated under and in general confined to the areola, however; it can be slightly extended outside the areola forming a dome shaped image to the areola.
Glandular: This form of gyno is common with bodybuilders as a result of the use of anabolic androgenic steroids. The undeniable overload testosterone levels from AAS (specifically any form of test) are converted to estrogen via aromatization. Bodybuilders and along other athletes are afflicted with gynecomastia in its purest form when suffering from AAS gyno. However; drug-induced gynecomastia can almost be resolves with the use of proper supplementation, if caught before permanent fibrosis develops and even after it can still be cured.
Gynecomastia in lean men is generally only a breast tissue gland with no added adipose tissue; heavier men will have sloppier looking gyno because of the adipose tissue covering the glandular tissue. Guaranteed treatment of pure gynecomastia can be done simply by removal of the breast tissue, which also rids of the gyno tissue. Most of the time the glandular tissue is benign but at times it can become malignant, so gyno is NO laughing matter and must be taken serious especially during or after a cycle. So good bro's make sure you keep an eye out on your chest during cycle and take good care of yourself during and post cycle. Again I must stress that any and almost all gyno can be treated with out surgery but in some cases it is better to take care of it sooner rather then later.
Adolescent/Pubertal Gynecomastia: The Congenital or Hereditary Gynecomastia is on average evident by the ages of 9 to 18 in young males. About thirty percent to sixty percent of young adolescent boys experience pubertal gynecomastia. UNFORTUNATELY, thirty percent possibly will live with enlarged male breasts for the rest of their lives, but in other cases the gynecomastia will go away with age. However, severe forms of adolescent gynecomastia may require an involvement, in conference with the patient, the parents, and child development professionals. Now of course there is hope for men who have lived with gyno all of there lives. Your parents may have neglected it but there is no reason you have to.
Adult or andropause gyno: This is the most common form of gynecomastia due to declining androgen levels with a rise in estrogen in elderly or post andropause men. Gynecomastia based off of post andropause in most adults is composed mainly of glandular tissue but it may contain varying amounts of adipose and tough tissue. Now you know why old men have those wrinkly chests that look like they never had the shape of pecs on them before.
Asymmetric/Unilateral: Unilateral gynecomastia occurs when only one breast is bigger due to a case of gynecomastia. The other breast is usually normal in size and shape. Bilateral Asymmetry occurs when gynecomastia is present in both breasts, of course; each being to a different degree. Most bodybuilders and boys during puberty acquire unilateral gyno, and by a pretty big margin I might add.
Severe gynecomastia: is described as an excess or loose skin, and severely enlarged breasts. This form of gyno is determined in part by age, reason being older people suffering from gynecomastia tend to have less skin elasticity and therefore will have a greater profusion of excess skin associated to gynecomastia. Experienced plastic surgeons will perform as much of the surgical treatment of severe gynecomastia as feasible through an aereolar incision so as to avoid widespread scarring. However, some scarring may be inevitable when treating severe cases of gynecomastia. The larger the gyno the more the scaring and this is why even if you are going to have surgery once should try to reduce gyno in size as much as possible beforehand
Pseudogynecomastia: is not composed of glandular tissue, but rather of adipose tissue (fat). It looks a lot like typical gynecomastia but requires different treatment. Exercise and diet can be very effective in preventing and fighting off pseudogynecomastia. Only if this regimen is unsuccessful should surgery be considered an option. This is generally the only type of gynecomastia which can be improved with liposuction, but removal may be used in some cases as well. This form of gyno is also known as "false Gynecomastia" and is often associated with obesity whereby insulin interacts with a surplus of sugars or certain carbohydrates, specifically those of which that have been processed.
Not just estrogen causes gyno!!
PROGESTERONE. IGF, GH, AND PROLACTIN INDUCED GYNECOMASTIA
Gynecomastia can be as a result of a number of agents including estrogens, progestins, GH, IGF-1, and prolactin may all be involved. Regarding prolactin, androgens decrease prolactin levels while estrogens increase prolactin. Non-aromatizing androgens such as DHT have by no means been shown to raise prolactin levels in humans, but testosterone has, due to its aromatization to estradiol. Prolactin secreting tumors also known as prolactinomas, are often linked with gyno. Of course in these cases the prolactin is believed to induce gyno by suppressing testosterone production. Prolactinomas that are adequately large to cause gynecomastia, do cause gyno as a consequence of the impairment of Gonadotropin secretion, which leads to secondary hypogonadism. Remember Prolactin only has a direct effect on breast tissue only when estrogen is present so they say ( how much is needed?). This is why they say lowering estrogen levels will lead to lower prolactin levels and then eventually less occupied progesterone receptors. Keep in mind just using something to lower estrogen will not cure gyno when progesteonr/prolactin is one of the culprits. Also take into consideration that as IGF and GH levels rise, they will also rise within the mammary glands which could cause a growth of glandular tissue if the rise in IGF and GH is too rapid. This in basic terms means you got the GYNO.
After awhile, excess levels of prolactin also lead to laction which is when the mammary gland secretes milk. I don't know about you my friend but milk coming from my nips just is not something I want to experience. And I have no plans to feed young children even my own. Secretion is not the only way of knowing your gyno is of progesterone/prolactin nature but it is one clear indicator.
Aromatizing AAS
There are AAS that are more likely to induce gynecomastia whether it be due to aromatization or aromatization with progesterone site binding. Here is a small/general list of the more common and moderate to high aromatizing compounds.
Any form of Testosterone
Dianabol
Deca Durabolin/nanodrolone (progesterone)
MethylTestosterone
Methyltrienolone
Any form of Trenbolone (progesterone)and androgenic
Anadrol 50 Receptor cross over
Nondrolone Laurate (progesterone)
Norethandrolone
M 1,4ad
13-ethyl-3-methoxy-gona-2,5(10)-dien-17-one (progesterone)
2a, 17a-dimethyl-etiocholan-3-one-17b-ol
13 ethyl 3 methoxy
4-ad
Many people have reported receiving gyno from Superdrol, people question those accusations since superdrol is a 5aReduced compound, however; Professor Filimanov the formulator of Need2Slin.
" Prolactin is normaly caused by progestins, but can also be caused by dht, how?
For example, it is currently understood that when testosterone enters the cell cytoplasm it is subsequently converted to the more "active" androgen, dihydrotestosterone, DHT, by reduction at the 5alpha position, this is normal. Dihydrotestosterone is then either bound to a cytoplasmic "receptor" protein Rc, or is further metabolized to either 5alpha-androstane-3alpha,17beta-diol or 5alpha-androstane-3beta,17beta-diol ,DIOL. The binding of DHT to its cytoplasmic receptor protein results in translocation of the steroid-receptor complex into the nucleus where presumably the complex dissociates and DHT exerts its androgenic effects. The transport of DHT to the nucleus can also result from the conversion of testosterone to DHT by nuclear membrane-bound 5alpha-reductase. Prolactin augmentation of DHT effects is envisioned as resulting from interaction of prolactin with its receptor, which due to the large size of the prolactin molecule is probably located in or on the plasma membrane.
Because superdrol is androgenic, but lacks the ability to show affinity via 5ar, it circulates, and this causes the large amounts of androgens to look for a transporter, so that it can bind to the androgen receptor, so it uses prolactin which has a high affinity to cytoplasmic receptor protein, allowing the androgens, testosterone, to be carried and allowing them to convert to dht, only problem is prolactin hormone or luteotropic hormone is synthesised and secreted by sex binding lactotrope cells in the adenohypophysis (anterior pituitary gland, And this gland now produces more prolactin to help deal with the large amount of testosterone circulating that hasnt bound to the estrogen of androgen receptor, Part of the reason why superdol is so anabolic, So instead of binding to the androgen receptors in the scalp and the prostrate it converts to dht through this unique process, using prolactin to enter the cytoplasmic receptror protein, and allowing it to convert to dht and then bind to the androgen receptors in the muscle, causing its distinct hardening effects, it still can't bind to the scalp or prostrate via 5ar as the form of dht it has converted too doesnt allow for that affinity.
So more prolactin is produced to allow for the superdol to find a receptor ,this excess prolactin triggers a process that fills the breast with milk via a process called lactogenesis, in men however it causes a distinct enlargment of the mammary gland and can even cause a man to lactate.
If superdrol had better binding to the androgen receptor via 5AR then this problem would be prevented, the other thing is that prolactin production can remain elevated for months after a cycle has finished, and once the androgen has been removed, ( the cycle is over) the cytoplasmic receptor proteins have nothing to do other than to allow the prolactin to proceed with its hormonal action within the body, causing the male mammary gland to enlarge ready to produce milk... Hence the REBOUND GYNO, this is why proper pct is needed for superdrol, and the use of something to prevent prolactin."
As you see Professor Filimanov reinstates the correlation between prolactin/progesterone and estrogen which could lead to gyno even with a compound such as Superdrol.
Receptor cross over can also occur. Hormones are keys and your receptors are like locks. What happens is a hormone is ether placed in the body (by an out side source) or it is produced with in the body. This hormone we will call a "key". The hormone then sets out to find a "lock" that it can fit into,turn, and sequentially open up the components inside. To go one step more and make it a little more simple I will explain it like this.
Imagine your receptors as little treasure chest. Inside is chest it a set of instructions. This set of instructions can be a number of things. It can be directions to another chest or a task that must now be carried out. With out something to open the chest the instructions can never be carried out or the next chest found and opened. Following me ?
Some hormones are more like "dummy keys" will call them. They will find a "lock" and set them self into the "Lock hole" but then that is it. They never turn the key and open up whats inside. so we see hormones often work like this
1. Key fits a lock. AKA a hormone found a lock and set its self into it. Pretty simple
2. Key was not a dummy key and when it set its self into the lock it turned and opened up the chest letting out the instructions inside that will now be carried out.
3 "master keys" We will call them. Or "Muti function keys"
A subject not much talked about on any of the steroid forums or even in many articles I have come across is the fact that. In respect to anabolic steroids (out side source of hormones) most of them have all been explained as just "lock" and "key" . However the truth is pro hormones,disginer steroids and steroids all have "Multi function" and some even "Master key" functions.
as you can see explained earlier.
By now I am sure you are confused but that is fine. The body is a extremely complex thing and everyone's body acts the same yet differently when it comes to hormones. This is why a lot of times you must work with some one to help find out what works for you. To help find out more about you and how your body works, as well as the basics of science and how all things "should" work.
This last part has thrown many for a loop. Leaving many wondering why the have gyno when they did everything right, and used all the right compounds. The sad truth is there is no Guarantee that you will not end up with a case of gyno.
However there is one Guarantee and that being that no matter what the odds are on your side that your gyno can be reversed with proper supplementation my friends. You just need to know how. Often it is best to work with some one who has experience and has worked with many others to prevent or reverse gyno of all kinds.
Believe me friends, I know it is NOT easy by any means to feel comfortable about your body when you have gyno whether it be from puberty or AAS usage. It can really make one depressed knowing that their chest is not masculine appealing to the people around them or themselves. I know people who LOW SELF ESTEEM for years because of their pubertal gyno. We as people struggle on a daily basis to fit in whether it be through appearance, social status, or self accomplishment. The people I witnessed getting rid of their gyno, achieved all those forms of greatness and more at least to themselves which is all that matters; since it's your body and your life.
Now Of course it is time for the good part. The part every one has been waiting for. If all gyno can be cured then how?
Well My friends I am sorry to be the one to tell you. Yes there are drugs and products out there that work to reduce estrogen. Yes there are drugs that reduce progesterone and or porlactin. Yes there are serms that block the progesterone receptors. Yes all of these drugs and or supplements can be used to reduce,prevent, and even cure existing gyno.
However anyone out there giving you a cookie cutter gyno removing program is simple trying to do one thing. SELL YOU A PRODUCT/GROUP OF PRODUCTS AND OR DRUGS. Or they are only speaking from experience for what worked for them NOT YOU!!!
How many of you have used a friends advice because it worked for them,only to be disappointed when it does not work for you?
How many of us have tried everything only to find nothing worked? Or have you tried everything?
The fact of the matter is I do not hand out cookie cutter programs because although they may work for some they do not work for everyone. Each person must find out what works for them. Each person must learn how they will react to different products,drugs, and combination thereof.
One person my use nolva and it worked great for them. Another may use it and there gyno gets worse or they have a rebound long after. You must learn to read your body, learn what it is telling you. You must learn how to change things exactly when they need to be changed for you and why.
You must start off by having everything you need before hand. This is the one thing that is the same for everyone. When people make the statement "aaah just get some letro" Or hey man all you need is some arimadex and dostinex you will be fine" These people hardly know what they are talking about most of the time. They know what worked for them, they know what might have worked for others. However the most importent fact they know nothing about is WHAT WORKS FOR YOU AND HOW YOU WILL REACT.
Everyone is always looking for the cheap way out. Or the easy way out. SO often we jump all over the quick easy and cheap advice that is so readily handed out on the aas forums today.
When you are ready to do it right, when you want results, when you are at the end of your rope. Then you will bite the bullet and spend the money to get "everything" you need. A gyno removing program can have side effects and the proper supplements should be taken to counteract them.
When it comes to some causes of gyno you have to understand that it took years of exposure to out side estrogen to cause the problem. It may not be resolved overnight but it can be in the end and I am always here to help you do this.
Feel free to pm me any time my friends I am always here on the forums.
preventing Gyno of course is another topic in its self. When it comes to on cycle gyno prevention one should always use as little as he can or non at all if he can get away with it.
If you do not need it then do not use it. However always have it on hand IMO.
Many of the drugs known today for gyno prevention all have there pitfalls and some are better then others for different reasons. If prevention is what you are looking for then forma-stanzol happens to be the best choice for many different reasons. You can read more about this compound here in this thread. http://www.elitefitness.com/forum/a...roid-best-pct-best-gyno-treatment-698487.html
5 to 10 pumps morning and night is all one would need depending on the dosing of the cycle you are on. Back up plans should always be in place and on hand. One should always have letro and dostinex on hand but never a need to use them unless nothing else works.
During pct often people use just nolva or clomid and neglect that fact that these compounds do nothing to lower estrogen nor do they do anything at all for raised levels of prolactin or progesterone. Forma-stanzol on the other hand does.
Lastly I would like to open then thread up for discussion and questions by all. If you have any feel free to ask here or send me a pm and I will get to them as I can. Thank you to all.
Cauliflower Ear
New member
great info Needto
macapple
New member
I would like to introduce myself as I am new to these forums, I would also welcome some advice on my first cycle. I will give you a quick history of my work out routine and my daily diet. I would like to thanks all of you in advance since you have already provided me with much information.
I am 35-year-old, 5’8” and weight 150lbs. I have been weight training and excising on and off for over 15 years. My best weight was while in my 20’s during a three-year consistent training schedule where I was 158lbs, at the time I was not to concerned with stats so I do not have them to compare to now.
For the last year and a half my workout routine varied but currently it is a 5-day a week rotation, working out M, T, W, F, and Sa. I usually wake up around 5 am to prep (food and pre-work out supplements) for a one-hour workout session from 6-7am. Time does vary, give or take 30 minutes this will also pertain to my eating habits.
Daily routine is as follows…
Monday – Chest and Triceps
Warm-Up: 35lbs bench press 10-12 reps.
Workout: Chest
Bench Press: 110lbs 3 set of 10
Incline Dumbbells: 45lbs 3 set of 10
Cable Crossovers: 50lbs 3 set of 10
Decline Press 100 3 set of 10
Workout: Triceps
Skull Crushers: 50lbs 3 set of 10
Cable Rope Extension: 35lbs 3 set of 10
Kick Backs: 20lbs 3 set of 10
Bench Dips: 50-100
Workout: Calves
Seated Calf Raises: 65lbs 100
Tuesday – Back and Biceps
Warm-Up: Stretch and Pulls Ups
Workout: Back
Seated Rows: 130lbs 3 set of 10
Laying T-Bar: 45lbs 3 set of 10
Pull Downs: 110lbs 3 set of 10
One Arm Row: 40lbs 3 set of 10
Workout: Biceps
Preacher Curls: 50lbs 3 set of 10
Concentration Curls: 20lbs 3 set of 10
Overhead Cable Curls: 40 3 set of 10
Dumbbell Curl: 20lbs 3 set of 10
Workout: Abs
Sit Ups: 50
Bicycle Kicks: 50
Leg Ups: 50
Wednesday – Legs
Workout: Quadriceps
Single Leg Press: 80lbs 3 set of 10
Leg Extensions: 120lbs 3 set of 10
Lunges: 100
Workout: Hamstrings
Lying Leg Curls: 60lbs 3 set of 10
Stiff Legs Dumbbells: 40lbs 3 set of 10
Standing Leg Curls 30lbs 3 set of 10
Workout: Calves
Seated Calf Raises: 65lbs 100
Workout: Stretch
Thursday – Off
Friday – Shoulders and Traps
Workout: Shoulders
Arnold Dumbbell Press: 45lbs 3 set of 10
Dumbbell Raises/Iron Crosses/Should Rotate 3 set of 10 each
Rocky Presses: 65lbs 3 set of 10
Workout: Traps
Barbell Front Shrugs: 125lbs 3 set of 10
Barbell Behind Shrugs: 125lbs 3 set of 10
Dumbbell Upright Rows: 25lbs 3 set of 10
Workout: Abs
Sit Ups: 50
Bicycle Kicks: 50
Leg Ups: 50
Saturday - Start Monday New Cycle Begins
Sunday - Off
Once a month I will do a burn out session for each exercise where I will do my absolute max for 8 reps and then drop the weight to about ¼ of the max and do 12-15 reps until spent.
I’m pretty good with my diet; I eat for the purpose of eating. I do not have any emotional attachment for flavor of food. This makes it pretty simple for me to not have any unnecessary desires. When I cheat and want something sweet I grab a box of raisins or have some unscheduled fruit. Once a week we go to sushi and I usually only order Tuna based items. Any other rare occasion what we eat out I stick with a chicken and vegetable type dish. My calorie intake ranges from 2400-2800 daily. This usually fluctuates on whether or not I have a protein shake after work/before dinner. I have a daily chart broken by grams per meal, if anyone is interested. Here is the break down…
Pre- Workout: 600 am
Whole Wheat Oatmeal
Brown Sugar
4 Egg Whites
Whey Protein Drink
Breakfast: 800am
2 Chicken Breasts (Workout Days)
Brown Rice
Vegetables
9 Egg White Vegetable Omelet (Non Workout Days)
Mid-Morning Snack: 1100am
Chicken Breast
Brown Rice
Vegetables
Lunch: 100pm
2 Chicken Breasts
Pinto Beans
White Rice
Vegetables
Mid Afternoon Snack: 300pm
Chicken Breast
Brown Rice
Vegetables
Late-Afternoon Snack: 500pm
Muscle Milk Protein Shake
Dinner: 630pm
Chicken Breast
Pasta/Rice
Vegetables
Bedtime: 900pm
Casin Protein Shake
Yes, I eat a lot of chicken, all day everyday. About once a week we will have steak but this gives you a very good idea of my eating habits. Here is a quick glance of what my daily intake totals are
Cals Fat Cholesterol Sodium Carbs Fiber Protein Sugars
250 30g 400-450mg 2100mg 250g 25g 200g 15g
As part of my routine I also take a few supplements that include Flax, Fish-Oil, Whey, Casin, Super Pump, Size-On. I am going to be curious on how this should/could fit into the cycle.
Ok, here is where I would like some help. What I have so far is a bottle of Dimethabolin, HCGenerate, Life Support, Unleashed and Post Cycle. I have access to Dostinex and Nolvedex at the local pharmacy. Should I include these into my cycle, if so, how? What my thoughts are on how to take them are as followed
1-4 weeks Dime/HCG
4-8 weeks Unleashed/PC
Is there something else I should add to this? Is this ok for a first timer? Also, how should I rotate in the supplements I have? Should I be taking a creatine while on a cycle? Many of these small questions cannot seem to find the answer too. My over all goal from the cycle is to gain about 8-12 lbs. of muscle while losing a 3-4 lbs. of fat to allow for visible definition of muscle.
I am very glad I cam across this forum and look forward to hearing the advice you guy have to offer.
I am 35-year-old, 5’8” and weight 150lbs. I have been weight training and excising on and off for over 15 years. My best weight was while in my 20’s during a three-year consistent training schedule where I was 158lbs, at the time I was not to concerned with stats so I do not have them to compare to now.
For the last year and a half my workout routine varied but currently it is a 5-day a week rotation, working out M, T, W, F, and Sa. I usually wake up around 5 am to prep (food and pre-work out supplements) for a one-hour workout session from 6-7am. Time does vary, give or take 30 minutes this will also pertain to my eating habits.
Daily routine is as follows…
Monday – Chest and Triceps
Warm-Up: 35lbs bench press 10-12 reps.
Workout: Chest
Bench Press: 110lbs 3 set of 10
Incline Dumbbells: 45lbs 3 set of 10
Cable Crossovers: 50lbs 3 set of 10
Decline Press 100 3 set of 10
Workout: Triceps
Skull Crushers: 50lbs 3 set of 10
Cable Rope Extension: 35lbs 3 set of 10
Kick Backs: 20lbs 3 set of 10
Bench Dips: 50-100
Workout: Calves
Seated Calf Raises: 65lbs 100
Tuesday – Back and Biceps
Warm-Up: Stretch and Pulls Ups
Workout: Back
Seated Rows: 130lbs 3 set of 10
Laying T-Bar: 45lbs 3 set of 10
Pull Downs: 110lbs 3 set of 10
One Arm Row: 40lbs 3 set of 10
Workout: Biceps
Preacher Curls: 50lbs 3 set of 10
Concentration Curls: 20lbs 3 set of 10
Overhead Cable Curls: 40 3 set of 10
Dumbbell Curl: 20lbs 3 set of 10
Workout: Abs
Sit Ups: 50
Bicycle Kicks: 50
Leg Ups: 50
Wednesday – Legs
Workout: Quadriceps
Single Leg Press: 80lbs 3 set of 10
Leg Extensions: 120lbs 3 set of 10
Lunges: 100
Workout: Hamstrings
Lying Leg Curls: 60lbs 3 set of 10
Stiff Legs Dumbbells: 40lbs 3 set of 10
Standing Leg Curls 30lbs 3 set of 10
Workout: Calves
Seated Calf Raises: 65lbs 100
Workout: Stretch
Thursday – Off
Friday – Shoulders and Traps
Workout: Shoulders
Arnold Dumbbell Press: 45lbs 3 set of 10
Dumbbell Raises/Iron Crosses/Should Rotate 3 set of 10 each
Rocky Presses: 65lbs 3 set of 10
Workout: Traps
Barbell Front Shrugs: 125lbs 3 set of 10
Barbell Behind Shrugs: 125lbs 3 set of 10
Dumbbell Upright Rows: 25lbs 3 set of 10
Workout: Abs
Sit Ups: 50
Bicycle Kicks: 50
Leg Ups: 50
Saturday - Start Monday New Cycle Begins
Sunday - Off
Once a month I will do a burn out session for each exercise where I will do my absolute max for 8 reps and then drop the weight to about ¼ of the max and do 12-15 reps until spent.
I’m pretty good with my diet; I eat for the purpose of eating. I do not have any emotional attachment for flavor of food. This makes it pretty simple for me to not have any unnecessary desires. When I cheat and want something sweet I grab a box of raisins or have some unscheduled fruit. Once a week we go to sushi and I usually only order Tuna based items. Any other rare occasion what we eat out I stick with a chicken and vegetable type dish. My calorie intake ranges from 2400-2800 daily. This usually fluctuates on whether or not I have a protein shake after work/before dinner. I have a daily chart broken by grams per meal, if anyone is interested. Here is the break down…
Pre- Workout: 600 am
Whole Wheat Oatmeal
Brown Sugar
4 Egg Whites
Whey Protein Drink
Breakfast: 800am
2 Chicken Breasts (Workout Days)
Brown Rice
Vegetables
9 Egg White Vegetable Omelet (Non Workout Days)
Mid-Morning Snack: 1100am
Chicken Breast
Brown Rice
Vegetables
Lunch: 100pm
2 Chicken Breasts
Pinto Beans
White Rice
Vegetables
Mid Afternoon Snack: 300pm
Chicken Breast
Brown Rice
Vegetables
Late-Afternoon Snack: 500pm
Muscle Milk Protein Shake
Dinner: 630pm
Chicken Breast
Pasta/Rice
Vegetables
Bedtime: 900pm
Casin Protein Shake
Yes, I eat a lot of chicken, all day everyday. About once a week we will have steak but this gives you a very good idea of my eating habits. Here is a quick glance of what my daily intake totals are
Cals Fat Cholesterol Sodium Carbs Fiber Protein Sugars
250 30g 400-450mg 2100mg 250g 25g 200g 15g
As part of my routine I also take a few supplements that include Flax, Fish-Oil, Whey, Casin, Super Pump, Size-On. I am going to be curious on how this should/could fit into the cycle.
Ok, here is where I would like some help. What I have so far is a bottle of Dimethabolin, HCGenerate, Life Support, Unleashed and Post Cycle. I have access to Dostinex and Nolvedex at the local pharmacy. Should I include these into my cycle, if so, how? What my thoughts are on how to take them are as followed
1-4 weeks Dime/HCG
4-8 weeks Unleashed/PC
Is there something else I should add to this? Is this ok for a first timer? Also, how should I rotate in the supplements I have? Should I be taking a creatine while on a cycle? Many of these small questions cannot seem to find the answer too. My over all goal from the cycle is to gain about 8-12 lbs. of muscle while losing a 3-4 lbs. of fat to allow for visible definition of muscle.
I am very glad I cam across this forum and look forward to hearing the advice you guy have to offer.
needtogetaas
New member
MacApple
you over work your muscles and you don't eat enough, Problem solved. Get on a 5x5 program and up your cals by about 2k and then you will add muscle.
you over work your muscles and you don't eat enough, Problem solved. Get on a 5x5 program and up your cals by about 2k and then you will add muscle.
Snaketounge
New member
I just started deca, one shot a week/200mgs. I also just started t400/one shot a week. I got a 2 month kit of somatropin I dident start yet.. Should I get good results??
mexico2509
New member
have u ever done a cycle? whats ur workout like? whats ur diet like?? if you arent on a decent diet and havent been working out for quite a while it doenst matter what ur weight is... but more info is needed for anyone on here to help u
riflemancho
New member
help with cycle
[FONT="]1-6 500mg propionate
1-6 winstrol 40mg daily
1-6 50mg proviron daily
1-10 vitamin complex
pct
7-10 liv52 3x3 tablets
2-5 pregnyl 250 ui every 3 days
7-8 carsil
7-10 vitamin e 1000 iu
7-10 clomid 100mg/50mg/50mg/50mg
6-10 – tamoxifen 30mg
10-12 tribulus
7-10 clenbuterol 6-8 tablets
8-10 ketotifen
7-10 pamaton
24 years old, 1.89, 95kg, lean, training 3 years and a half. Any advice? Want to add 7-8 to 10-11kg muscle with that cycle.
[/FONT]
[FONT="]1-6 500mg propionate
1-6 winstrol 40mg daily
1-6 50mg proviron daily
1-10 vitamin complex
pct
7-10 liv52 3x3 tablets
2-5 pregnyl 250 ui every 3 days
7-8 carsil
7-10 vitamin e 1000 iu
7-10 clomid 100mg/50mg/50mg/50mg
6-10 – tamoxifen 30mg
10-12 tribulus
7-10 clenbuterol 6-8 tablets
8-10 ketotifen
7-10 pamaton
24 years old, 1.89, 95kg, lean, training 3 years and a half. Any advice? Want to add 7-8 to 10-11kg muscle with that cycle.
[/FONT]
riflemancho
New member
Allidoiswin123
New member
22 years old 200 pounds been training for 3-4 years it's my second cycle
Running
Week 1-10 test prop 200mg eod
6-10 50 mg winstrol Ed
The real question I have is its qv test prop rated at 200 mg/ml and I've searched and searched and can't find anywhere where qv has test prop rated at 200mg/ml. I can post pics of it but I'm starting to wonder I've been on for 4 weeks Nd
and havent noticed much. Diet is on point so I'm sketching out
Running
Week 1-10 test prop 200mg eod
6-10 50 mg winstrol Ed
The real question I have is its qv test prop rated at 200 mg/ml and I've searched and searched and can't find anywhere where qv has test prop rated at 200mg/ml. I can post pics of it but I'm starting to wonder I've been on for 4 weeks Nd
and havent noticed much. Diet is on point so I'm sketching out
Revolutionjdub
New member
How would tren and anadrol work in a stack? I'm 24 in pretty good shape and this will be my third cycle but I don't know If I'm doing somthing wrong. It's a six week cycle.
Weeks 1-6 tren/ 1cc every other day
Weeks 3-6 anadrol 50 mg everyday
Should I start the anadrol earlier? I'm open to try different ways to use r better results.
Weeks 1-6 tren/ 1cc every other day
Weeks 3-6 anadrol 50 mg everyday
Should I start the anadrol earlier? I'm open to try different ways to use r better results.
androl 50 mg
need little help bro.
i took androl 50 mg in april.. only for 4 weeks
1 week 25 mg a day
2 week 50 mg a day
3 week 50 mg a day
4 week 50 mg a day
i want to start my second cycle
from when i can start and what it should be..
I am 1.85 M tall, 78 kg (kilogram)
my chest is 42.5"
my biceps 15.5"
and shoulders 20.5"
plz advice me
I am in India and its very hot here
need little help bro. i took androl 50 mg in april.. only for 4 weeks
1 week 25 mg a day
2 week 50 mg a day
3 week 50 mg a day
4 week 50 mg a day
i want to start my second cycle
from when i can start and what it should be..
I am 1.85 M tall, 78 kg (kilogram)
my chest is 42.5"
my biceps 15.5"
and shoulders 20.5"
plz advice me
I am in India and its very hot here
needtogetaas
New member
needtogetaas does Clomiphene, Clomid when he has to lol
I am not the biggest fan of clomid however it is a drug that has been around for years and it has its uses and its place among us. With the invention and discovery of newer herbs,supplements natural compounds and synergistic blend. Clomid is fast become a thing of the past and rarely used by anyone other then old school hard heads. Still My job has always been to educated the masses and who am I to leave out information one mite need. Besides what works best for one may not work at all for another and if you love clomid then all the power to you. In this text I will give you the blah blah blah run down of clomid. But I think you would be wise to also read this thread http://www.elitefitness.com/forum/anabolic-steroids/taking-anabolic-steroids-101-a-642856.html Post 5-6-7 .
Full Article on clomid here
needtogetaas does Clomiphene, Clomid when he has to lol. Need To Build Muscle Inc. Official Blog
I am not the biggest fan of clomid however it is a drug that has been around for years and it has its uses and its place among us. With the invention and discovery of newer herbs,supplements natural compounds and synergistic blend. Clomid is fast become a thing of the past and rarely used by anyone other then old school hard heads. Still My job has always been to educated the masses and who am I to leave out information one mite need. Besides what works best for one may not work at all for another and if you love clomid then all the power to you. In this text I will give you the blah blah blah run down of clomid. But I think you would be wise to also read this thread http://www.elitefitness.com/forum/anabolic-steroids/taking-anabolic-steroids-101-a-642856.html Post 5-6-7 .
Full Article on clomid here
needtogetaas does Clomiphene, Clomid when he has to lol. Need To Build Muscle Inc. Official Blog
Whats up, Im Danny and i have some questions for @needtogetaas.
I have been thinking of taking some winstrol to help slim down.
I am:
Height: 5'9
Weight: 177lbs
Age: 29
I have been working out hard for about 3 1/2 months. Attached is my current routine.
My current food intake is as follows:
Average:
CAL: 1700
Carbs: 130
Fat: 40
Protein: 200
Any suggestions will be greatly appreciated.
Thanks,
Danny
I have been thinking of taking some winstrol to help slim down.
I am:
Height: 5'9
Weight: 177lbs
Age: 29
I have been working out hard for about 3 1/2 months. Attached is my current routine.
My current food intake is as follows:
Average:
CAL: 1700
Carbs: 130
Fat: 40
Protein: 200
Any suggestions will be greatly appreciated.
Thanks,
Danny
norfolkman
New member
dear sir, just a quick message , my fist day on here so forgive me if i mess up, i am 55 years old takeing 500mg of testo e per 7 days i am 85kg i would say very little fat if any look 75kg am 5ft 9ins tall for the first time in my life i have time for me, bin takeing testo e for 3 mounths feel 40 yeares old wonderfull my age very low in testcan i keep takeing on weekly with no rest ? also can i up the dose i feel i could mybe do another 250mg,looking also trying to get bigger if you think i have not left it late,would like your help and cam post photos and more imformation, if you can help me , norfolkman thank you very much
Great thread I have spent hours & hours reading Q&As from guests (prior to me starting a cycle want as much information as possible to get the best results and least sides) trying to find out this & that and most of info is attached in this article. This should be an automatic link to any new guest at the site entry to prevent so many new threads asking very similar questions. In short thank you for the detail and for the time it must have taken to input..
gixxermaniak
New member
Getting ready to take d-drol first cycle what else should I take with it the Guy at the gym said take it alone and just take clomid at the end of week 6
needtogetaas
New member
The guy at the gym is a retard and I advice you punch him in the face next time you see him..
If you want to run a dbol cycle right it would look like this..
Weeks
1-4 Dbol 30-75mg every day spread out into 3 doses at the least
1-4 N2guard 7 caps a day spread out
1-4 Bridge 3 caps 3 times a day morning noon and night.
4-8 post cycle and unleashed 1 cap each 3 times a day
4-10 forma-stanzol 5 pumps rubbed on your chest 2 times a day morning and night.
For the first 4 weeks then drop down to 3 and 3 the last 2 weeks
this is a great way to run that cycle and if you like you can extend it to 6 weeks just push the weeks of the pct back and do not start the bridge till week 2. Use 5 caps a day of the N2gaurd rather then 7 a day and it will last the full 6 weeks.
Take more than 2500mls a week is a waste of money and if you cant grow using that much then your not lifting weights properly, not eating properly, not sleeping properly...just give up....Lee Priest never took more than 1250mls a week +gh+igf.......some wankers take 5000mls a week+ of test etc....start planning your funeral.....Best gear and uncomplicated cycle is Sustanon 250...1000mls a week is enough for most people add some dianabol if you want...probably wont be a pro but you'll still be huge.............lol
needtogetaas
New member
ESTER PROFILES
Sustanon: The "king" of testosterone blends.
The four different testosterone esters in this product certainly look appealing to the consumer, there is no denying that. But for the athlete I think it is all just a matter of marketing (Hell, why buy one ester when you can get four?). In clinical situations I can see some strong uses for it. If you were undergoing testosterone replacement therapy for example, you would probably find Sustanon a much more comfortable option than testosterone enanthate. You would need to visit the doctor less frequently for an injection, and blood levels should be more steadily maintained between treatments. But for the bodybuilder who is injecting 4 ampules of Sustanon per week, there is no advantage over other testosterone products. In fact, the high price tag for Sustanon usually makes it a very poor buy in the face of cheaper testosterone enanthate/cypionate. Bodybuilders should probably stop looking at the four ester issue, and stick with totals (Sustanon is just a 250mg testosterone ampule). Were enanthate to be available for say $10 per amp of 250mg, and Sustanon priced nearly double that, buying the Sustanon would be like throwing money away. If you could get nearly double the milligram amount for the same price with enanthate, this is the better product to go with hands down. Leave the high priced stuff for the guys who don't know any better.
Acetate: Chemical Structure C2H4O2.
Also referred to as Acetic Acid; Ethylic acid; Vinegar acid; vinegar; Methanecarboxylic acid. Acetate esters delay the release of a steroid for only a couple of days. Contrary to what you may have read, acetate esters do not increase the tendency for fat removal. Again, there is no known mechanism for it to do so. This ester is used on oral primobolan tablets (metenolone acetate), Finaplix (trenbolone acetate) implant pellets, and occasionally testosterone.
Propionate: Chemical Structure C3H6O2.
Also referred to as Carboxyethane; hydroacrylic acid; Methylacetic acid; Ethylformic acid; Ethanecarboxylic acid; metacetonic acid; pseudoacetic acid; Propionic Acid. Propionate esters will slow the release of a steroid for several days. To keep blood levels from fluctuating greatly, propionate compounds are usually injected two to three times weekly. Testosterone propionate and methandriol dipropionate (two separate propionate esters attached to the parent steroid methandriol) are popular items.
Phenylpropionate: Chemical Structure C9H10O2.
Also referred to as Propionic Acid Phenyl Ester. Phenylpropionate will extend the release of active steroid a few days longer than propionate. To keep blood levels even, injections are given at least twice weekly. Durabolin is the drug most commonly seen with a phenylpropionate ester (nandrolone phenylpropionate), although it is also used with testosterone in Sustanon and Omnadren.
Isocarpoate: Chemical Structure C6H12O2.
Also referred to as Isocaproic Acid; isohexanoate; 4-methylvaleric acid. Isocaproate begins to near enanthate in terms of release. The duration is still shorter, with a notable hormone level being sustained for approximately one week. This ester is used with testosterone in the blended products Sustanon and Omnadren.
Caproate: Chemical Structure C6H12O2.
Also referred to as Hexanoic acid; hexanoate; n-Caproic Acid; n-Hexoic acid; butylacetic acid; pentiformic acid; pentylformic acid; n-hexylic acid; 1-pentanecarboxylic acid; hexoic acid; 1-hexanoic acid; Hexylic acid; Caproic acid. This ester is identical to isocarpoate in terms of atom count and weight, but is laid out slightly different (Isocaproate has a split configuration, difficult to explain here but easy to see on paper). Release duration would be very similar to isocaproate (levels sustained for approximately one weak), perhaps coming slightly closer to enanthate due to its straight chain. Caproate is the slowest releasing ester used in Omnadren, which is why most athletes notice more water retention with this compound.
Enanthate: Chemical Structure C7H14O2.
Also referred to as heptanoic acid; enanthic acid; enanthylic acid; heptylic acid; heptoic acid; Oenanthylic acid; Oenanthic acid. Enanthate is one of the most prominent esters used in steroid manufacture (most commonly seen with testosterone but is also used in other compounds like Primobolan Depot). Enanthate will release a steady (yet fluctuating as all esters are) level of hormone for approximately 10-14 days. Although in medicine enanthate compounds are often injected on a bi-weekly or monthly basis, athletes will inject at least weekly to help maintain a uniform blood level.
Cypionate: Chemical Structure C8H14O2.
Also referred to as Cyclopentylpropionic acid, cyclopentylpropionate. Cypionate is a very popular ester here in the U.S., although it is scarcely found outside this region. Its release duration is almost identical to enanthate (10-14 days), and the two are likewise thought to be interchangeable in U.S. medicine. Althletes commonly hold the belief than cypionate is more powerful than enanthate, although realistically there is little difference between the two. The enanthate ester is in fact slightly smaller than cypionate, and it therefore releases a small (perhaps a few milligrams) amount of steroid more in comparison.
Decanoate: Chemical Structure C10H20O2.
Also referred to as decanoic acid; capric acid; caprinic acid; decylic acid, Nonanecarboxylic acid. The Decanoate ester is most commonly used with the hormone nandrolone (as in Deca-Durabolin) and is found in virtually all corners of the world. Testosterone decanoate is also the longest acting constituent in Sustanon, greatly extending its release duration. The release time with Decanoate compounds is listed to be as long as one month, although most recently we are finding that levels seem to drop significantly after two weeks. To keep blood levels more uniform, athletes (as they have always known to do) will follow a weekly injection schedule.
Undecylenate: Chemical Structure C11H20O2.
Also referred to as Undecylenic acid; Hendecenoic acid; Undecenoic acid. This ester is very similar to decanoate, containing only one carbon atom more. Its release duration is likewise very similar (approximately 2-3 weeks), perhaps extending a day or so past that seen with decanoate. Undecylenate seems to be exclusive to the veterinary preparation Equipoise (boldenone undecylenate), although there is no reason it would not work well in human-use preparations (Equipoise certainly works fine for athletes). Again, weekly injections are most common.
Undecanoate: Chemical Structure C11H22O2.
Also referred to as Undecanoic Acid; 1-Decanecarboxylic acid; Hendecanoic acid; Undecylic acid. Undecanoate is not a commonly found ester, and only appears to be used in the nandrolone preparation Dynabolan, and oral testosterone undecanoate (Andriol). Since this ester is chemically very similar to undecylenate (it is only 2 hydrogen atoms larger), it has a similar release duration (approximately 2-3 weeks). Although this ester is used in the oral preparation Andriol, there is no reason to believe it carries any properties unique of other esters. Andriol in fact works very poorly at delivering testosterone, bolstering the idea that oral administration is not the idea use of esterified androgens.
Laurate: Chemical structure C12H24O2.
Also referred to as Dodecanoic acid, laurostearic acid, duodecyclic acid, 1-undecanecarboxylic acid, and dodecoic acid. Laurate is the longest releasing ester used in commercial steroid production, although longer acting esters do exist. Its release duration would be closer to one month than the other esters listed above, although realistically we are probably to expect a notable drop in hormone level after the third week. Laurate is exclusively found in the veterinary nandrolone preparation Laurabolin, perhaps seen as slightly advantageous over a decanoate ester due to a less frequent injection schedule. Again athletes will most commonly inject this drug weekly, no doubt in part due to its low strength (25mg/ml or 50mg/ml).
New info that was added to a post in this thread but also adding to the end in case people who have already read most of this thread miss it
Sustanon: The "king" of testosterone blends.
The four different testosterone esters in this product certainly look appealing to the consumer, there is no denying that. But for the athlete I think it is all just a matter of marketing (Hell, why buy one ester when you can get four?). In clinical situations I can see some strong uses for it. If you were undergoing testosterone replacement therapy for example, you would probably find Sustanon a much more comfortable option than testosterone enanthate. You would need to visit the doctor less frequently for an injection, and blood levels should be more steadily maintained between treatments. But for the bodybuilder who is injecting 4 ampules of Sustanon per week, there is no advantage over other testosterone products. In fact, the high price tag for Sustanon usually makes it a very poor buy in the face of cheaper testosterone enanthate/cypionate. Bodybuilders should probably stop looking at the four ester issue, and stick with totals (Sustanon is just a 250mg testosterone ampule). Were enanthate to be available for say $10 per amp of 250mg, and Sustanon priced nearly double that, buying the Sustanon would be like throwing money away. If you could get nearly double the milligram amount for the same price with enanthate, this is the better product to go with hands down. Leave the high priced stuff for the guys who don't know any better.
Acetate: Chemical Structure C2H4O2.
Also referred to as Acetic Acid; Ethylic acid; Vinegar acid; vinegar; Methanecarboxylic acid. Acetate esters delay the release of a steroid for only a couple of days. Contrary to what you may have read, acetate esters do not increase the tendency for fat removal. Again, there is no known mechanism for it to do so. This ester is used on oral primobolan tablets (metenolone acetate), Finaplix (trenbolone acetate) implant pellets, and occasionally testosterone.
Propionate: Chemical Structure C3H6O2.
Also referred to as Carboxyethane; hydroacrylic acid; Methylacetic acid; Ethylformic acid; Ethanecarboxylic acid; metacetonic acid; pseudoacetic acid; Propionic Acid. Propionate esters will slow the release of a steroid for several days. To keep blood levels from fluctuating greatly, propionate compounds are usually injected two to three times weekly. Testosterone propionate and methandriol dipropionate (two separate propionate esters attached to the parent steroid methandriol) are popular items.
Phenylpropionate: Chemical Structure C9H10O2.
Also referred to as Propionic Acid Phenyl Ester. Phenylpropionate will extend the release of active steroid a few days longer than propionate. To keep blood levels even, injections are given at least twice weekly. Durabolin is the drug most commonly seen with a phenylpropionate ester (nandrolone phenylpropionate), although it is also used with testosterone in Sustanon and Omnadren.
Isocarpoate: Chemical Structure C6H12O2.
Also referred to as Isocaproic Acid; isohexanoate; 4-methylvaleric acid. Isocaproate begins to near enanthate in terms of release. The duration is still shorter, with a notable hormone level being sustained for approximately one week. This ester is used with testosterone in the blended products Sustanon and Omnadren.
Caproate: Chemical Structure C6H12O2.
Also referred to as Hexanoic acid; hexanoate; n-Caproic Acid; n-Hexoic acid; butylacetic acid; pentiformic acid; pentylformic acid; n-hexylic acid; 1-pentanecarboxylic acid; hexoic acid; 1-hexanoic acid; Hexylic acid; Caproic acid. This ester is identical to isocarpoate in terms of atom count and weight, but is laid out slightly different (Isocaproate has a split configuration, difficult to explain here but easy to see on paper). Release duration would be very similar to isocaproate (levels sustained for approximately one weak), perhaps coming slightly closer to enanthate due to its straight chain. Caproate is the slowest releasing ester used in Omnadren, which is why most athletes notice more water retention with this compound.
Enanthate: Chemical Structure C7H14O2.
Also referred to as heptanoic acid; enanthic acid; enanthylic acid; heptylic acid; heptoic acid; Oenanthylic acid; Oenanthic acid. Enanthate is one of the most prominent esters used in steroid manufacture (most commonly seen with testosterone but is also used in other compounds like Primobolan Depot). Enanthate will release a steady (yet fluctuating as all esters are) level of hormone for approximately 10-14 days. Although in medicine enanthate compounds are often injected on a bi-weekly or monthly basis, athletes will inject at least weekly to help maintain a uniform blood level.
Cypionate: Chemical Structure C8H14O2.
Also referred to as Cyclopentylpropionic acid, cyclopentylpropionate. Cypionate is a very popular ester here in the U.S., although it is scarcely found outside this region. Its release duration is almost identical to enanthate (10-14 days), and the two are likewise thought to be interchangeable in U.S. medicine. Althletes commonly hold the belief than cypionate is more powerful than enanthate, although realistically there is little difference between the two. The enanthate ester is in fact slightly smaller than cypionate, and it therefore releases a small (perhaps a few milligrams) amount of steroid more in comparison.
Decanoate: Chemical Structure C10H20O2.
Also referred to as decanoic acid; capric acid; caprinic acid; decylic acid, Nonanecarboxylic acid. The Decanoate ester is most commonly used with the hormone nandrolone (as in Deca-Durabolin) and is found in virtually all corners of the world. Testosterone decanoate is also the longest acting constituent in Sustanon, greatly extending its release duration. The release time with Decanoate compounds is listed to be as long as one month, although most recently we are finding that levels seem to drop significantly after two weeks. To keep blood levels more uniform, athletes (as they have always known to do) will follow a weekly injection schedule.
Undecylenate: Chemical Structure C11H20O2.
Also referred to as Undecylenic acid; Hendecenoic acid; Undecenoic acid. This ester is very similar to decanoate, containing only one carbon atom more. Its release duration is likewise very similar (approximately 2-3 weeks), perhaps extending a day or so past that seen with decanoate. Undecylenate seems to be exclusive to the veterinary preparation Equipoise (boldenone undecylenate), although there is no reason it would not work well in human-use preparations (Equipoise certainly works fine for athletes). Again, weekly injections are most common.
Undecanoate: Chemical Structure C11H22O2.
Also referred to as Undecanoic Acid; 1-Decanecarboxylic acid; Hendecanoic acid; Undecylic acid. Undecanoate is not a commonly found ester, and only appears to be used in the nandrolone preparation Dynabolan, and oral testosterone undecanoate (Andriol). Since this ester is chemically very similar to undecylenate (it is only 2 hydrogen atoms larger), it has a similar release duration (approximately 2-3 weeks). Although this ester is used in the oral preparation Andriol, there is no reason to believe it carries any properties unique of other esters. Andriol in fact works very poorly at delivering testosterone, bolstering the idea that oral administration is not the idea use of esterified androgens.
Laurate: Chemical structure C12H24O2.
Also referred to as Dodecanoic acid, laurostearic acid, duodecyclic acid, 1-undecanecarboxylic acid, and dodecoic acid. Laurate is the longest releasing ester used in commercial steroid production, although longer acting esters do exist. Its release duration would be closer to one month than the other esters listed above, although realistically we are probably to expect a notable drop in hormone level after the third week. Laurate is exclusively found in the veterinary nandrolone preparation Laurabolin, perhaps seen as slightly advantageous over a decanoate ester due to a less frequent injection schedule. Again athletes will most commonly inject this drug weekly, no doubt in part due to its low strength (25mg/ml or 50mg/ml).
New info that was added to a post in this thread but also adding to the end in case people who have already read most of this thread miss it
perryprada
New member
hey i need help with some thing ok i did 3 weeks of test cyp only and that was it i didnt feel a thing so i thought it was fake 4 days ago my prostate swole n i dun know what do do can some one tell me if this gose away or is this permanant
needtogetaas
New member
Take saw palmetto and cranberry extract for a few weeks stay away from drinking and or taking in lots of caffeine
fabbiones2273
New member
anabol
.bodypumplist.altervista.org/
.bodypumplist.altervista.org/
Im going to be doing my first cycle and could really use some helo so i dont screw my self up or do anything wrong my rmail is [email protected]. if some could please give me some help it would be grealty appriciated.
PLEASE HELP ANYONE!!! i am a speed athlete power explosion and recovery are mainly what i am focusd on can any one tell me what would be my best bet for a cycle ive done research on diffrent types of steroids and detectuion time but as far as if i need to run a testosterone or an estrogen blocker like arimadex durring or after, i am mainly looking for sumthing to gain sum strength a dense lean mass no water bloat or or anything to get real bulky (need to stay mobile to sprint) but in short it would b nice to get leaner train harder recover faster and gain sum size with some gear that is out of my system quickly so i can b clean for the season, any adivce helps thanks guys love the info u guys have and the knowlege u guys have gained over the years
needtogetaas
New member
http://www.elitefitness.com/forum/a...goes-hard-super-sarm-triple-stack-854783.html
this is the kind of cycle you want to do my friend.. Talk to this guy he will square you away with how to use and get it all too....
hello guys... im in my 2nd cycle of test soon... my first it was prop+e= 350 per week for 6 six weeks.....
for my 2nd i want to try using cypionate along with deca or primo... and using var for the 1st 3weeks before the ester kick in.... and add stanozlol at the end of cycle... i want run 8 weeks 200mg per week for cypionate and 100mg for deca... what do you think??? it shoul be ok on lean mass... and for the PCT its much enough just use clomid and tamo?
for my 2nd i want to try using cypionate along with deca or primo... and using var for the 1st 3weeks before the ester kick in.... and add stanozlol at the end of cycle... i want run 8 weeks 200mg per week for cypionate and 100mg for deca... what do you think??? it shoul be ok on lean mass... and for the PCT its much enough just use clomid and tamo?
Hi,
Thank you for your time. At the risk of sounding abrupt, I'll use as little of it as possible.
Having started my first cycle 7 days ago my eyes seem to have been opened and this has raised questions I can't seem to find the answer for in forums.
My stats:
Age 38
Height 6'4"
Weight 173lb (78.5kg)
Male
Been training most of the last 15 years, more that 8 of which was with weights the last 2 year training hard with weights.
Diet: I've tried it all at one time or another. I once ate 6 full meals a day and trained 4 days a week, I'd guess it was over 4000 or 5000 kCal/day of which about 1/3 by weight was protein. 7 months of that and I gained 10lbs of which I lost 60% within 6 months of eating a 'normal diet' while still training. My 2 training partners did the same everything and gained at least three times as much and retained multiples more mass.
At the moment I'm eating about 4 meals a day 25% protein and about 4000 kCal/day which I plan to increase tomorrow. I'll up the protein to 100g/day
Currently taking 1st course:
Dianabol 30mg/day (will start on 40mg/day from tomorrow)
Deca 400mg/week
Arimidex 1/2 tab/day
Body fat less than 10%
I'm not preparing for a specific event, rather just ran out of patients with being an extremely hard gainer. I'd like to gain 'permanent' muscle mass and ideally not gain so fast as to have stretch marks.
I gather I should add testosterone to this, but the more I read the more questions get raised. My main practical question is could I not use Tren rather than Testo?
And out of curiosity why does the Dbol not count as testo. in this situation according to most forums, I'm having quite the opposite of the Decca Dick reaction up to now?
And does the Arimidex reducing water retention affect or nullify the Dbol strength gains?
If you could shed some light it would be great,
thanks.
Thank you for your time. At the risk of sounding abrupt, I'll use as little of it as possible.
Having started my first cycle 7 days ago my eyes seem to have been opened and this has raised questions I can't seem to find the answer for in forums.
My stats:
Age 38
Height 6'4"
Weight 173lb (78.5kg)
Male
Been training most of the last 15 years, more that 8 of which was with weights the last 2 year training hard with weights.
Diet: I've tried it all at one time or another. I once ate 6 full meals a day and trained 4 days a week, I'd guess it was over 4000 or 5000 kCal/day of which about 1/3 by weight was protein. 7 months of that and I gained 10lbs of which I lost 60% within 6 months of eating a 'normal diet' while still training. My 2 training partners did the same everything and gained at least three times as much and retained multiples more mass.
At the moment I'm eating about 4 meals a day 25% protein and about 4000 kCal/day which I plan to increase tomorrow. I'll up the protein to 100g/day
Currently taking 1st course:
Dianabol 30mg/day (will start on 40mg/day from tomorrow)
Deca 400mg/week
Arimidex 1/2 tab/day
Body fat less than 10%
I'm not preparing for a specific event, rather just ran out of patients with being an extremely hard gainer. I'd like to gain 'permanent' muscle mass and ideally not gain so fast as to have stretch marks.
I gather I should add testosterone to this, but the more I read the more questions get raised. My main practical question is could I not use Tren rather than Testo?
And out of curiosity why does the Dbol not count as testo. in this situation according to most forums, I'm having quite the opposite of the Decca Dick reaction up to now?
And does the Arimidex reducing water retention affect or nullify the Dbol strength gains?
If you could shed some light it would be great,
thanks.
I just had to post in this shit. Every time I read post # 1 it cracks me the fuck up and I love this place. To the guy above me who hasnt posted since and probably wont. Probably a joke, I weighed more than you before ever touching a weight, and im about 5 inches shorter than you. I honestly dont see how its possible to weigh 173 and be 6'4''. I think its a joke.
ANYWAY. N2 is the shit and this post is f-in awesome. Keep this shit sticky
ANYWAY. N2 is the shit and this post is f-in awesome. Keep this shit sticky
Well thanks for the motivation Mr. O. Like I said hard gainer. Possibly an understatement, hey? Well I wish it was a joke, but any takers about the questions?
It's now been 2 weeks on my first cycle and I've gained about 5lb, *&^%en great. But cautious, not getting too excited, want to see what going to happen still. Going to add some Test and Tren soon I think.
It's now been 2 weeks on my first cycle and I've gained about 5lb, *&^%en great. But cautious, not getting too excited, want to see what going to happen still. Going to add some Test and Tren soon I think.
Hi I was hoping to get some advice esp regarding PCT recommendations. I'm25, 5'10-11ish approx. 182. My current diet is around 2000calories im trying to shred up, I take in on average 220g protein, between 50 and 80 carbs, and around 60g of fat. THis diet has worked for me as I have kept my strength and lost quite a bit of fat overall. I am looking to start my first cycle ever, and have decided 8wks with Test prop, anavar, and possible provion.
My workouts consist of Cardio in the am Mon-Fri. Alternating 20 mins of HIT and 40mins of a steady pace. I hit all body parts twice a week, once heavy and again lighter with slower reps. I also do abs daily except for sunday.
For PCT, I am somewhat at a loss, I am considering Unleashed and Post Cycle, as well as Formastanzol, as well as clomid. I am also leaning towards incorporating HCGenerate the last 4-5 weeks of my cycle.
Any Suggestions or advice would be a huge help and appreciated. Thank You.
My workouts consist of Cardio in the am Mon-Fri. Alternating 20 mins of HIT and 40mins of a steady pace. I hit all body parts twice a week, once heavy and again lighter with slower reps. I also do abs daily except for sunday.
For PCT, I am somewhat at a loss, I am considering Unleashed and Post Cycle, as well as Formastanzol, as well as clomid. I am also leaning towards incorporating HCGenerate the last 4-5 weeks of my cycle.
Any Suggestions or advice would be a huge help and appreciated. Thank You.
Start your own thread in the steroid section bro, you'll get a response much quicker there. I'm sure Needto will answer eventually but you could wait a long while for that.
thelastmrbigg15
Banned
i would suggest dropping the prop and picking up sustanon (lots less pinning)
Hi Every one i am Majid my age is 26. I worked out alot but it doesnt bulk me. As my weight is only 58kg. I am 6 feet tall. My friend told me to take a stack or deca durabolin and testosteron. Will anyone please guide me the right way i start with my first cycle to Bulk in no then less time. But do remember onething i am in Pakistan its quite Hot now a days as its summer season. Please help me and reply soon
Bro you'd be best off starting your own thread, you'll get replies quicker.
But at 6feet tall and only 58kg you shouldn't use steroids. You need to build a base first. The most important thing is diet, if you want to gain weight you must eat more.
thelastmrbigg15
Banned
he def. needs more food... oatmeal, pasta, chicken breast, tuna, beef if u can eat it, potatoes, the works bro within a year you can easily be 160-180 lbs or 70-80 kilos simple rule train like an animal, eat like a horse, and sleep like a baby
^^^ this +1
Majid I'm 1" taller than you and twice your weight and I didn't use anything but food and hard work to get there. You CAN do it.
Majid I'm 1" taller than you and twice your weight and I didn't use anything but food and hard work to get there. You CAN do it.
themondtster
New member
Bump. Great info!
Sent from my PG86100 using EliteFitness
Sent from my PG86100 using EliteFitness
im a bit overweight. nothin major.
ive just come back from an ichilles tendon injury so cardio is out of the question for a while. while im lifting weights woul a course of steroids not only build me muscle but cut down my body fat. im a little restricted as i cant do much in a standing position , almost everything is sitting or on s bench.
Sent from my GT-I9100 using EliteFitness
ive just come back from an ichilles tendon injury so cardio is out of the question for a while. while im lifting weights woul a course of steroids not only build me muscle but cut down my body fat. im a little restricted as i cant do much in a standing position , almost everything is sitting or on s bench.
Sent from my GT-I9100 using EliteFitness
Ezskankens
New member
wow,a little long,but good post.
tjrolltide
New member
Sus 250, anodrol 25, (anastrozole 1 for help) .... Good stack
Im trying to lean up.... Im dieting, working out hard, and have started the sus 250 once ( Advice I got was 2x week)... anastrozole once ( 1 x a week in future) and Ive taken the anadrol twice so far... For the lean look do I need the aDrol( heard it gives a lot of wAter weight and I'm looking for the hard, lean, more vascular look. I know the Sus with diet, workout, and cardio will. WHAT DO yALL THINK?
Im trying to lean up.... Im dieting, working out hard, and have started the sus 250 once ( Advice I got was 2x week)... anastrozole once ( 1 x a week in future) and Ive taken the anadrol twice so far... For the lean look do I need the aDrol( heard it gives a lot of wAter weight and I'm looking for the hard, lean, more vascular look. I know the Sus with diet, workout, and cardio will. WHAT DO yALL THINK?
needtogetaas
New member
[email protected]
New member
one of the most informative extensive threads that I've ever read on any forum. You come across like a lay man, a doctor, and a college professor. I've been reading for several hours and taking notes. I'm going to take a break, eat a little and get back to reading again. I don't need to look anywhere else, everything is right here! Thank you.
ssbadazzdeuce
New member
First off where can i get some online? Any legit sites? Pm me please anything is appreciated.
Sent from my N860 using EliteFitness
Sent from my N860 using EliteFitness
[email protected]
New member
I smell a big fat law enforcement rat here! You've been a Platinum Member for over a year and you want to know where you can buy some? Some of what?
Crack cocaine, heroin, or steroids online? Any legit sites?
Guy, are you trying to pull some damn bullshit on this forum!
PM you? No fucking way I'll tell you anything.
Your post is needs to be scrutinized by some moderator.
- Status
