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Quickest dose of HcG to plump my balls
- Thread starter ryno9000
- Start date
ryno9000
New member
I've never heard of it until now. Anyone vouch for it? What is the dosage protocol? On cycle, or PCT?
j_p
Newbie
that is a big dose and not the best way to dose the hcg. I rec 500iu 2X a week (about 3-4 days apart) also Triporellin may help you with your recovery.
j_p
Newbie
some info on it:
GnRH (Triptorelin) 100mcg
Dosing and side effects
Like many chemicals, we want to really pay attention to our dosing. GnRH makes a great jumpstart, probably now the most effective jumpstart chem, because unlike hcg, it stimulates both lh - leutenizing hormone - and FSH - follicle stimulating hormone - to a higher extent and has a much more lasting effect. But much like hcg, dihydrotestosterone, HMB, ect ect, we need to be very careful with our pituitary and avoid hyper-stimulation. We need to pulse it once, at a small dose, simulating the pulse that is normally sent from our brain, and then let our bodies do the rest of the work.
GnRH is so powerful that large doses (around 4mg), repeated once a month, is being used as a chemical form of castration. This dose is so intense on the pituitary, that it hyper-stimulates, resulting in castration-like levels of testosterone serum in the body. Much like hcg, dosing is delicate, and too much is not a good thing. We need to use GnRH as a restart, one-and-done, and not over-do things because it may have a much more opposite and negative effect.
Without any further talk, here is my recommendation for use. One single 100mcg dose per cycle, after all esters have cleared the body and you are 100% ready for recovery. hcg should still be used on-cycle, but in my opinion this full-stimulation should be saved for the PCT and recovery phase. Use hcg on cycle to continue simulating lh - leutenizing hormone - , and then GnRH in the post cycle. Studies I have read have seen results from even 600mcg used in a three-day period, and still hpta - hypothalamic-pituitary-testicular axis - function was completely restored, and his hormone levels remained within the normal range during three checkups within the following year. This suggests that the restart will not have the “flare” effect if used at reasonable doses. Another study showed the same effect, with a dose of only one 100mcg injection into a bodybuilder who had been shutdown for 13 years. That said, no more then 100mcg per 4 months. Do not exceed 1mg within a year to avoid the castration-like shutdown of your system. That even gives you room to do it after an 8-week cycle, take the appropriate time off, and then begin another. And for oral-only cycles that are under 8 weeks, save your money, as Triptorelin is not cheap stuff. Better yet, don’t do oral only cycles, as they are a waste of time, but that’s a whole nother fish to fry, which I will do later. __________________
GnRH (Triptorelin) 100mcg
Dosing and side effects
Like many chemicals, we want to really pay attention to our dosing. GnRH makes a great jumpstart, probably now the most effective jumpstart chem, because unlike hcg, it stimulates both lh - leutenizing hormone - and FSH - follicle stimulating hormone - to a higher extent and has a much more lasting effect. But much like hcg, dihydrotestosterone, HMB, ect ect, we need to be very careful with our pituitary and avoid hyper-stimulation. We need to pulse it once, at a small dose, simulating the pulse that is normally sent from our brain, and then let our bodies do the rest of the work.
GnRH is so powerful that large doses (around 4mg), repeated once a month, is being used as a chemical form of castration. This dose is so intense on the pituitary, that it hyper-stimulates, resulting in castration-like levels of testosterone serum in the body. Much like hcg, dosing is delicate, and too much is not a good thing. We need to use GnRH as a restart, one-and-done, and not over-do things because it may have a much more opposite and negative effect.
Without any further talk, here is my recommendation for use. One single 100mcg dose per cycle, after all esters have cleared the body and you are 100% ready for recovery. hcg should still be used on-cycle, but in my opinion this full-stimulation should be saved for the PCT and recovery phase. Use hcg on cycle to continue simulating lh - leutenizing hormone - , and then GnRH in the post cycle. Studies I have read have seen results from even 600mcg used in a three-day period, and still hpta - hypothalamic-pituitary-testicular axis - function was completely restored, and his hormone levels remained within the normal range during three checkups within the following year. This suggests that the restart will not have the “flare” effect if used at reasonable doses. Another study showed the same effect, with a dose of only one 100mcg injection into a bodybuilder who had been shutdown for 13 years. That said, no more then 100mcg per 4 months. Do not exceed 1mg within a year to avoid the castration-like shutdown of your system. That even gives you room to do it after an 8-week cycle, take the appropriate time off, and then begin another. And for oral-only cycles that are under 8 weeks, save your money, as Triptorelin is not cheap stuff. Better yet, don’t do oral only cycles, as they are a waste of time, but that’s a whole nother fish to fry, which I will do later. __________________
j_p
Newbie
encyclopedia
Triptorelin
Systematic (IUPAC) name 5-oxo-D-prolyl-L-histidyl-Ltryptophyl-L-seryl-Ltyrosyl-3-(1H-indol-2-yl)-L-alanylleucyl-L-arginyl-L-prolylglycinamide Identifiers CAS number 57773-63-4 ATC code L02AE04 ChemSpider 13835459 Chemical data Formula C64H82N18O13 Mol. mass 1311.5 g/mol SMILES eMolecules & PubChem Pharmacokinetic data Excretion Renal Therapeutic considerations Pregnancy cat. D Legal status ℞-only Routes Implant Triptorelin (acetate or pamoate), a decapeptide (pGlu-His-Trp-Ser-Tyr-D-Trp-Leu-Arg-Pro-Gly-NH2), is a gonadotropin-releasing hormone agonist (GnRH agonist). By causing constant stimulation of the pituitary, it decreases pituitary secretion of gonadotropins luteinizing hormone (LH) and follicle stimulating hormone (FSH). Like other GnRH agonists, triptorelin may be used in the treatment of hormone-responsive cancers such as prostate cancer or breast cancer, precocious puberty, estrogen-dependent conditions (such as endometriosis or uterine fibroids), and in assisted reproduction. Triptorelin is marketed under the brand names Decapeptyl (Ipsen) and Diphereline and Gonapeptyl (Ferring Pharmaceuticals). In the United States, it is sold by Watson as Trelstar.
During the treatment of prostate cancer it does cause a surge of testosterone (an initial uplevel of testosterone levels), known as a flare effect. In men a reduction of serum testosterone levels into the range normally seen after surgical castration occurs approximately two to four weeks after initiation of therapy. In contrast, gonadotropin-releasing hormone antagonists do not cause a surge, but a sudden reduction of testosterone levels.
Systematic IUPAC Name: [d-Trp6]GnRH
Triptorelin
Systematic (IUPAC) name 5-oxo-D-prolyl-L-histidyl-Ltryptophyl-L-seryl-Ltyrosyl-3-(1H-indol-2-yl)-L-alanylleucyl-L-arginyl-L-prolylglycinamide Identifiers CAS number 57773-63-4 ATC code L02AE04 ChemSpider 13835459 Chemical data Formula C64H82N18O13 Mol. mass 1311.5 g/mol SMILES eMolecules & PubChem Pharmacokinetic data Excretion Renal Therapeutic considerations Pregnancy cat. D Legal status ℞-only Routes Implant Triptorelin (acetate or pamoate), a decapeptide (pGlu-His-Trp-Ser-Tyr-D-Trp-Leu-Arg-Pro-Gly-NH2), is a gonadotropin-releasing hormone agonist (GnRH agonist). By causing constant stimulation of the pituitary, it decreases pituitary secretion of gonadotropins luteinizing hormone (LH) and follicle stimulating hormone (FSH). Like other GnRH agonists, triptorelin may be used in the treatment of hormone-responsive cancers such as prostate cancer or breast cancer, precocious puberty, estrogen-dependent conditions (such as endometriosis or uterine fibroids), and in assisted reproduction. Triptorelin is marketed under the brand names Decapeptyl (Ipsen) and Diphereline and Gonapeptyl (Ferring Pharmaceuticals). In the United States, it is sold by Watson as Trelstar.During the treatment of prostate cancer it does cause a surge of testosterone (an initial uplevel of testosterone levels), known as a flare effect. In men a reduction of serum testosterone levels into the range normally seen after surgical castration occurs approximately two to four weeks after initiation of therapy. In contrast, gonadotropin-releasing hormone antagonists do not cause a surge, but a sudden reduction of testosterone levels.
Systematic IUPAC Name: [d-Trp6]GnRH
j_p
Newbie
- Objective
Design
Case report.
Setting
Endocrinology unit of the University of Brescia.
Patient(s)
A 34-year-old man.
Intervention(s)
A single dose (100 μg) of triptorelin (triptorelin test).
Main Outcome Measure(s)
Clinical symptoms, androgen normalization, levels of serum testosterone, follicle-stimulating hormone, and luteinizing hormone.
Result(s)
Within 1 month, the patient's serum testosterone was in the normal range, and he reported a return to normal energy and libido.
Case report
A 34-year-old man presented to our department in September 2008 for loss of libido and energy and for mild depression. He was a computer programmer and a nonprofessional bodybuilder with an unremarkable personal medical history. He admitted to having used doping drugs since he was 21 years old. More specifically, he would perform cycles of intramuscular injections of nandrolone (25 mg) and stanazol (25 mg) daily for 8 weeks, followed by mesterolone (50 mg/day) for 15 days. Then he would then take clomiphene citrate (50 mg/day) for 1 week, followed by an injection of human chorionic gonadotropin (2,000 IU) three times in 1 week. He had repeated these cycles from 1995 to 2005. From 2005 to August 2008, to his nandrolone and stanazol cycle he added an intramuscular injection of boldenone (50 mg) daily for 3 weeks. He said he had bought all the drugs on the Internet.
The patient was 175 cm tall and 80 kg, and he appeared very muscular and toned. His blood pressure and pulse rate were normal. Examination of his heart, lungs, and abdomen were likewise unremarkable. The physical examination showed normal secondary sexual characteristics, but the genital examination revealed bilateral testicular atrophy (volume 2.9 mL and weak consistence). Despite his testicular atrophy, the semen analysis revealed a normal count (79 × x106spermatozoa/mlmL) and mild morphology derangements (between 46% and 58%). The blood count and chemistry were normal, but his level of creatine kinase was 454 IU/L (normal range: 20–170 IU/L), alanine aminotransferase 61 IU/L (normal range: 5–50 IU/L), and aspartate aminotransferase 23 IU/L (normal range: 5–50 IU/L).
In February 2009, the patient continued to report loss of libido and great tiredness. A second physical examination was performed. His levels of alanine transferase and creatine kinase were all within the normal range, but the endocrinologic investigations were still abnormal with the exception of sex hormone-binding globulin level. *The patients testosterone measured 0.3 ng/mL - normal range is between 2.0 ng/mL and 12 ng/ML. Because the situation had persisted for months after ASS withdrawal, we administered a single dose (100 μg) of triptorelin (triptorelin test), which showed a normal response (Fig. 1). Ten days after the triptorelin test, the patient reported a great amelioration of energy, and his serum testosterone was 7.0 ng/mL. One month later, his serum testosterone was within the normal range, and he reported a return to normal libido and energy.
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- Figure 1.
Triptorelin test showing a normal response.
