PCT Help

[halfaclue]

This will be one of my longest and most brutual cycles and i want a good recovery and keep as much as possible.

Week 1 50dbol ed;
Week 2 50dbol ed;
Week 3 50dbol ed; 100mg suspension ed
Week 4 50dbol ed; 100mg suspension ed
Week 5 50dbol ed; 100mg suspension ed
Week 6 250mg Enathate eod
Week 7 250mg Enathate eod
Week 8 250mg Enathate eod; DNP 200mg Day 1-2, 400mg Day 3-7
Week 9 250mg Enathate eod; DNP 400mg ed
Week 10 250mg Enathate eod
Week 11 250mg Enathate eod Currently in this week
Projected finish is un-clear, definitely doing 75mg fina/100mg prop ed
Week 12 250mg Enathate eod
Week 13 75mg fina/100mg prop ed
Week 14 75mg fina/100mg prop ed
Week 15 75mg fina/100mg prop ed

I also have a bottle of EQ (200mg/ml 10ml)
60 winny tabs at 50mg each.

Should I use the EQ or winny or just save it? I used novladex when taking D-bol with no chest issues. I have liquidex now that I am taking. How much and how long for the PCT and what would people recommend.

 

[halfaclue]

^

 

[halfaclue]

where are the BB'ers? Guys who do competitions? Mods?

 

[halfaclue]

Bueller Bueller???

 

[pumacorp]

Use that eq the last 5 weeks of the cycle..U will come off alot better...U need an anabolic on the end...Androgens are best used in the 1st half of a long cycle...

 

[halfaclue]

What about on the PCT side? Normally I am running 12 weeks of test with winny at the end so the PCT is that crazy..with cycle I think I will need a strong PCT but I'm not sure where to start other then clomid.

 

[RAZOR67]

i would run the prop a week or two past the tren..this will aid in recocery..

Pheedno's PCT

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My post cycle therapy consists of a three compound administration which is designed so that there is a primary and secondary LH stimulator which both are maximizing potential early in the duration; with the primary being phased out in extended protocol. With the addition of an Aromatase Inhibitor, which makes the above possible, the individual will also endure less of an increase in Sex Hormone Binding Globulin, which allows free testosterone levels to reach base line at a much quicker pace. The individual will also see less of a problem in most cases with sexual libido as the bounding SHBG is controlled(to an extent). Below you will find my suggested bare minimum, as well as a sample of an extended protocol. Extended PCT protcol is cycle length dependant so the below is not the standard for all cycles


PCT for cycles 8-16wks:
Day 1-30- .25mg L-dex + 100mg Clomid + 20mg Nolva

Extended protocol sample for a 12+ month cycle:
Day 1-15_ .25mg L-dex + 100mg Clomid + 20mg Nolva
Day 16-45_.25mg L-dex + 75mg Clomid + 20mg Nolva
Day 46-65_.25mg L-dex + 20mg Nolva
Day 66-80_.25mg L-dex

Now IMO, selective estrogen receptor modulators(SERMs) such as Clomiphine and Tamoxifen are selective to which tissues they bind too. Clomid being selective to the suprapituitary, while Tamox is selective to breast, bone, and liver ERs. I've come to this conclusion based on the comparison of studies on both SERMs. In every study showing benefit to HPTA from tamoxifin, the duration of the administration is 3-12months(This includes studies cited by William Llewellyn in his Nolva vs Clomid article). In studies showing levels of LH, FSH, and Testosterone checked after short durations of tamox, they were either insignificant, or their was an actual drop. I believe this is because tamox selectively works at the mammery(as well as bone and liver), thus taking longer for LH stimulation to occur.
With clomid, benefit to gonadotrophin concentrations, LH, FSH, and serum testosterone can be seen in short periods of 2-6wks. Because of the apparent selective nature of the two, and given our usual PCT duration, clomid is by far superior at LH stimulation than Nolva. Now both is the wise choice for a couple of reasons:

1. Nolva acts as the preventive measure to the estrogen flux
occured PC while clomid is the primary LH stimulator(Even more so in the case an AI is not used).
2. If your running a longer PCT, clomid needs to be discontinued after a while as it has been shown to desensitize GnRH, this due, IMO, to it's selective nature to the suprapituitary. In the longer forms of PCT, the clomid will be phased out, leaving Nolva and L-dex

Arimidex(or L-dex)
Estrogen is the main inhibitence of restoring HPTA, and AI administration has been shown to increase gonadotrophin concentrations and serum Testosterone by up to 50%. In addition, by adding L-dex, the inhibitence of excess estrogen allows Tamox to work greater at LH stimulation in the begining stages of PCT, since the need to prevent binding in the mammery is lessened by the reduction in estrogen biosynthesis
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[halfaclue]

thanks Bro!