liver values and gear!!??

[GearedChupaCabra]

Ok heres my situation...i`ve been off any gear for almost 5 months and i was on accutane for about 3 months before my doctor took me off cuz of high liver values, so i`ve been off accutane almost a month and my last blood work a week ago showed that it was still high, normal is 40 and they were at 61,keep in mind that they were 3 times the normal before my last blood work, now i want to start a cycle test/EQ/inj. winstrol??? that been said i know that test in a high amount could increase liver values and EQ has no effect what so ever on your liver...so whats a high amount of test?? i was planning on using 750mgs of sustunon 400mg EQ and maybe 50mg winstrol inj eod...and milk thistle also...what are you thoughts on this?

Thanks in advanced,

GCC

 

[GearedChupaCabra]

bump

 

[GearedChupaCabra]

where the gurus at ?

 

[jboldman]

I am no guru but test at 750 will have little or no effect on your liver.

jb

 

[Ulter]

When I used winny and accutane together the first time I had high values as well. My doctor told me to use Tyler Liver Detox. Then Fonz came up with the liver research done with ALA. Ever since I started using Tyler Liver Detox and ALA I have had no liver problems no matter what I take. Even at 100mg/day of winny and accutane on top of it.
You can get Tyler at the AF Store if you're interested.


AF Store

 

[GearedChupaCabra]

Thanks for the replies fellas...i`m going to get some Tyler liver detox...in the mean while is it ok to start this cycle??

GCC

 

[nandi12]

Don't make the same mistake doctors often do and blame steroids when exercise induced tissue breakdown is the real reason for elevated enzymes.

J Am Osteopath Assoc 2001 Jul;101(7):391-4

Evaluation of aminotransferase elevations in a bodybuilder using anabolic steroids: hepatitis or rhabdomyolysis?

Pertusi R, Dickerman RD, McConathy WJ.

Department of Medicine, University of North Texas Health Science Center, 3500 Camp Bowie Blvd, Fort Worth, TX 76107-2699, USA.

The use of anabolic steroids among competitive athletes, particularly bodybuilders, is widespread. Numerous reports have noted "hepatic" dysfunction secondary to anabolic steroid use based on elevated serum aminotransferase levels. The authors' objective was to assess whether primary care physicians accurately distinguish between anabolic steroid-induced hepatotoxicity and serum aminotransferase elevations that are secondary to acute rhabdomyolysis resulting from intense resistance training. Surveys were sent to physicians listed as practicing family medicine or sports medicine in the yellow pages of seven metropolitan areas. Physicians were asked to provide a differential diagnosis for a 28-year-old, anabolic steroid-using male bodybuilder with an abnormal serum chemistry profile. The blood chemistries showed elevated aspartate aminotransferase (AST), alanine aminotransferase (ALT), and creatine kinase (CK) levels, and normal gamma-glutamyltransferase (GGT) levels. In the physician survey (n = 84 responses), 56% failed to mention muscle damage or muscle disease as a potential diagnosis, despite the markedly elevated CK level of the patient. Sixty-three percent indicated liver disease as their primary diagnosis despite normal GGT levels. Prior reports of anabolic steroid-induced hepatotoxicity that were based on aminotransferase elevations may have overstated the role of anabolic steroids. Correspondingly, the medical community may have been led to emphasize anabolic steroid-induced hepatotoxicity and disregard muscle damage when interpreting elevated aminotransferase levels. Therefore, when evaluating enzyme elevations in patients who use anabolic steroids, physicians should consider the CK and GGT levels as essential elements in distinguishing muscle damage from liver damage.