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How monitoring Glucose gets you the Olympic Body

three words. You're wrong

Animal said:
Well, there is not point going any further when this kinda comments are made.


'Insulin helps systemic anabolic functions. '

No it does NOT! Insulins only job is to transport glucose across the cell membrane. It does NOTHING else and can do NOTHING else except attach to the insulin receptor.

And not just those related to glucose metabolism and regulation.

Wrong from above

Insulin also helps intracellular transport, utilization of amino acids,

NO IT does NOT. AA's have their OWN transport system as does HGH.


fatty acids,

NO.

proteins,

WTF do you people want to believe? Insulin helps protein transport and not one person catches this bunch a bunk?

and enzymes.

Where is the insulin enzyme activator and transporter.

It also stimulates cellular metabolism.

It does?
 
This was super informative. Thanks for sharing the knowledge. I have been trying to find literature about the relationship between insulin and GH. So far the only thing I found was that the pancreas will choose to generate insulin- before (stimulating the pituitary) generating GH.

I definately am reading everywhere of the anabolic power of insulin. It seems to be a real miracle drug if you can get the balance and get it to work to its maximum potential. I dont know how to do that. Your insight has helped.

The third paragraph 'the does and doesnt of Insulin and protein conversion' That is where I realized it was pancreatic health that would establish the proper balance. Trying to separate 'meat' and 'protein' and categorizing meat as a food that gets broken down as other foods--meat is broken down more efficiently by the pancreas putting less stress on it, and letting it balance its functions more easily. I assume that would be the goal: to balance how the hormones in the body are utilized.

Juice Authority said:


For one thing, it is well established that GH administration causes insulin resistance. This is improved dramatically if IGF-1 is administered along with GH. This is evidently due to the insulin like action of IGF-1 in skeletal muscle. Insulin seems to be one of, if not the most anabolic hormones in the body. By blunting its action, GH impairs the anabolic effect of insulin.

Insulin is superior to ALA in their ability to shuttle glucose into the cell, and even more importantly they upregulate glycogen synthase.

AAS stimulate protein synthesis, but do not seem to have an effect on protein breakdown. Insulin on the other hand can both promote protein synthesis and inhibit protein breakdown. This, and its ability to promote amino acid transport into tissue are the advantages of insulin over AS as I see it. Obviously both used together would be best
 
I read Fonzs' article on glucose monitoring. He would be a good person to ask. You want levels that are specifically for body builders.

SpeedFreak5 said:
How do you monitor the levels. Do I get one of thoughs diabetic machines and prick my finger or what.
 
'For one thing, it is well established that GH administration causes insulin resistance. '


Yea, so what? Eating lotsa sugar makes you insulin resistant over time, too. HGH increases insulin secretion and that prevelance of more insulin is what makes you insulin resistant.

Still, nobody can find one single study or anything where an insulin receptor does anything but shuttle glucose. Nothing else fits in that receptor either.
 
tia373 said:
three words. You're wrong


im not stcking up for animal cause i really cant stand the way he goes about his debates.

with that being said you cant just say hes wrong. why is he wrong? explain
 
Animal said:
'For one thing, it is well established that GH administration causes insulin resistance. '


Yea, so what? Eating lotsa sugar makes you insulin resistant over time, too. HGH increases insulin secretion and that prevelance of more insulin is what makes you insulin resistant.

Still, nobody can find one single study or anything where an insulin receptor does anything but shuttle glucose. Nothing else fits in that receptor either.

Really??? ONE of the most important actions of insulin is to control amino acid transport in fat, liver, and muscle cells.

http://endo.endojournals.org/cgi/content/full/139/3/832

Studies of Avandia and other drugs in its class show that the more insulin a person has, the better the drugs work. Insulin shifts the cellular metabolism from the former to the latter by lowering triglyceride levels in muscle and elevating glycogen levels, by virtue of stimulating glycogen synthase.

Here is an intresting link to the avandia web site. It includes a little cartoon that is like "insulin for dummies", as well as some other intresting facts for the layman. You should find this most interesting.

http://diabeteslife.com/insulin_resistance.html
 
GH is primarily a diabetogenic hormone. It, simply put, works (partially anyways) in an opposing function to insulin. If Exo GH causes glycogen release from fat cells then wouldnt it make sense that the higher blood suger levels that GH can cause will trigger insulin release by the body?
 
Great read and thread.

The primary way to lower insulin is to lower caloric intake. Elimination of sugar's from diet and increasing protein is very helpful, as is exercise.
 
Oh, so then anybody on a ketogenic diet isn't getting any AA's into their body and there's no way they can gain muscle, right?

Insulin may ALLOW something to take place, but that's a BIG difference than insulin HAS TO BE there and insulin IS THE REASON something happens.

Of course being in a fed state is going to allow other things to happen, but insulin does not attach to anything else and directly transport it into the cell. System A is part of a sodium regulated transport system with similar results being garnered using sodium and passive glucose uptake.

Even so, when insulin is present 40% of the studies show anabolic actions, 40% show no anabolic actions and 20% or so show a negative affect.

It would be nice to see the rest of those two studies which this paper so claims to be set in concrete as there are thousands of studies on this subject and there are a entire 2 on 'system A'.
 
Animal said:
It would be nice to see the rest of those two studies which this paper so claims to be set in concrete as there are thousands of studies on this subject and there are a entire 2 on 'system A'.

You claimed no studies existed that showed the insulin receptor did anything but shuttle glucose. Well, you're wrong since I just posted one. What more do you want? Do you argue just for the sake of arguing even with irrefutable facts to the contrary?
 
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