Please Scroll Down to See Forums Below needtogetaas
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anny one got a link.and fast cuz this shits getting moved in 3-2-1 
ephedra
- Thread starter needtogetaas
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in
needtogetaas
New member
you fuck.PuddleMonkey said:
needtogetaas
New member
bump before the move.
T
The Shadow
Guest
Its not illegal to buy...go a google search
T
The Shadow
Guest
needtogetaas
New member
I did but all I got was some sites to ephedrin.I dont want ephedrin I want ephedra.The Shadow said:
T
The Shadow
Guest
needtogetas said:I did but all I got was some sites to ephedrin.I dont want ephedrin I want ephedra.
er.......ok
YOu do know that ephedrine hcl is the active ingredient in "ephedra"....
needtogetaas
New member
so there is no diferace.The Shadow said:
T
The Shadow
Guest
needtogetas said:
no.....
Ripped Feul contained like 300 something mg od Ephedra...thats equiv to 24-25 mg Ephedrine hcl.
needtogetaas
New member
god I loved ripped feul.The Shadow said:
but what elce was in it.is there anny thing out there that will work like ripped foul.that shit was the bomb.
T
The Shadow
Guest
Make your own
Ripped Fuel was:
Ephedra - get ehpedrine hcl - 24-25 mg
Caffeine - but your own tabs - 200 mg
Asprin - buy your own - 300-325 mg
Ripped Fuel was:
Ephedra - get ehpedrine hcl - 24-25 mg
Caffeine - but your own tabs - 200 mg
Asprin - buy your own - 300-325 mg
needtogetaas
New member
how should I take it.I reamaber taking like 2-4 ripped fuels evry 6-8 houres.The Shadow said:
needtogetaas
New member
bump for shadow.
T
The Shadow
Guest
needtogetas said:
Depending on tolerance.....I would work UP to taking one each of the listed supps 3 times per day........that would be the same as TWO RIPPED FUEL per dose....
needtogetaas
New member
so all that is like taking 2 ripped fuels,sweet.I am all over this.thanks for youre help bro.
hidngod said:
yea you can buy pure ephedrine HCL here, its not illegal as long as it is sold in 8mg tabs, so you now have to take 3 tabs instead of one.
The fat burners themselves dont' have ephedrine or ephedra in them anymore unless you know the owners of supp stores who usually have them under the counter for well known customers.
sawastea
Well-known member
needtogetaas
New member
thank you bros.
DieselGunz
New member
Great thread!
needtogetaas
New member
ya I lernd some good shit from it.DieselGunz said:
DieselGunz
New member
needtogetas said:
deff, Shadow knows his shit for sure
sawastea
Well-known member
Mikus said:
Yohimbine HCL is not needed...
The addition of the 2-adrenolytic drug, yohimbine, to caffeine and ephedrine is an attractive support to obesity treatment. However, administration of three drugs influencing the cardiovascular system could have some undesirable effects, especially in obese subjects.
We tried to determine if the supporting pharmacological treatment of obese women composed of ephedrine, caffeine and yohimbine could modify the cardiovascular state at rest and during static (handgrip) or dynamic (cycloergometer) exercise. We found that loss of body mass did not differ between groups receiving only diet and groups with two or three drugs administration together with diet. We supposed that 10 days is too short a time to define real weight loss after treatment. Further study is needed to define the influence of these doses of drugs on metabolic effects and weight loss during a longer period of treatment.
However, the aim of our study was to determine if caffeine and ephedrine used together or these two drugs in combination with yohimbine were safe for the cardiovascular system. Ten days of observation is a sufficient period to detect some cardiovascular reactions. A very low caloric diet is usually associated with a decrease in adrenergic system activity. The aim of ephedrine, caffeine and yohimbine administration was to diminish this phenomenon and to increase the effect of a low caloric diet. We did not observe significant changes in most haemodynamic parameters after 10 days of diet only administration. However, the ejection fraction decreased. It is possible that this drop is one of the symptoms indicating this phenomenon.
We found that the therapy composed of ephedrine (2 25 mg) and caffeine (2 200 mg) did not modify the haemodynamic parameters at rest and during the handgrip exercise. Only during cycloergometer exercise was ejection fraction increased. This fact suggests that the combination of the drugs we used did not exert undesirable effects on the cardiovascular system. The increased in ejection fraction even has a profitable significance. It is also an indirect proof that ephedrine and caffeine in the doses we used could weakly activate the adrenergic system. For this reason the charge in the cardiovascular system in our obese subjects during static or dynamic exercise at the time of ephedrine and caffeine administration was not very large and seemed safe.We observed different reactions of the cardiovascular system when three drugs were used. Application of three drugs produced a depression of the cardiovascular system at rest, which was expressed by a decrease in stroke index and ejection fraction. This was probably the result of diastolic pressure and heart rate increase. In these circumstances, the systole of the heart is less efficient. We could speculate that these effects of yohimbine were due to an increase in noradrenaline concentration in the plasma ( Hedner et al., 1992; Tavernier et al., 1992). If this was the case, noradrenaline could increase the afterload, and thereby cardiac performance would be decreased. This could be an undesirable effect of yohimbine treatment. The same drug regimen during the handgrip test led to a decrease in ejection fraction only. Diastolic pressure, heart rate and ejection fraction tended in a similar direction during the handgrip test, but did not show significant changes between groups. We suppose that activation of the adrenergic system and following cardiovascular system during exercise is much stronger than after pharmacological stimulation. For this reason, the pharmacological influence of drugs is proportionally attenuated.
When cycloergometer exercise was performed, not only ejection fraction, but also cardiac load increased in the group receiving three drugs. The last effect was not observed during the handgrip test. However, cycloergometer exercise and handgrip are quite different types of effort. During dynamic exercise, heart rate reached higher values than during the handgrip test. Systolic pressure was inversely higher during the handgrip test. It could be speculated that, during the handgrip test, -activation predominated but during the cycloergometer exercise -activation predominated. End-diastolic index observed during dynamic exercise reached greater values than during the handgrip test. Central translocation of blood volume from the lower part of the body during exercise on the cycloergometer could have cardinal meaning. This centralization of circulation was probably in part due to the work of the leg muscles. Stroke index during cycloergometer exercise was also greater than during the handgrip test. Increased volume of heart and circulating blood, augmented heart rate together with the influence of the three drugs evoked an increase in cardiac load. The influence of yohimbine in these circumstances is very important, because we did not observe an increase in cardiac load when only two drugs were administered. This fact has significant clinical implications, because it suggests that yohimbine evokes some dangerous changes in the cardiovascular system. For this reason, this drug could only be used in some persons with caution.
Our results indicate that the pharmacological support of a low caloric diet by ephedrine and caffeine during obesity treatment induces only minimal changes in the cardiovascular system, but the addition of yohimbine to this regimen may lessen the cardiac performance at rest and during the handgrip test. Using three drugs during the cycloergometer exercise leads to an increase in cardiac work. Therefore, these findings demonstrate that chronic administration of ephedrine and caffeine has no undesirable effects on cardiovascular state in obese patients. The addition of yohimbine should be treated with caution and must be excluded in particular obese individuals with cardiovascular complications.
Waluga M, Janusz M, Karpel E, Hartleb M, Nowak A. Cardiovascular effects of ephedrine, caffeine and yohimbine measured by thoracic electrical bioimpedance in obese women. Clin Physiol. 1998 Jan;18(1):69-76
Int J Mol Med. 2001 Jul;8(1):103-9. Related Articles, Links
Divergent effects of an alpha2-adrenergic antagonist on lipolysis and thermogenesis: interactions with a beta3-adrenergic agonist in rats.
Gomez-Ambrosi J, Fruhbeck G, Aguado M, Milagro FI, Margareto J, Martinez AJ.
Department of Physiology and Nutrition, University of Navarra, 31008 Pamplona, Spain.
This study was undertaken in order to test the hypothesis that selective beta3-AR stimulation and simultaneous blockade of alpha2-AR would result in an increase of lipolysis and thermogenesis in rats. Incubation of isolated white adipocytes with the alpha2-AR antagonist yohimbine produced a concentration-dependent increase in glycerol release (P<0.001) for all assayed concentrations (10-12-10-6 M) and potentiated the lipolytic effect of the beta3-AR agonist Trecadrine. However, in vivo administration of yohimbine produced a marked decrease in body temperature (1.3-1.5 degrees C, P<0.001) and blocked the thermogenic effect of Trecadrine when simultaneously administered. A similar response was observed for whole body oxygen consumption. Furthermore, yohimbine did not modify brown adipose tissue oxygen consumption, but blocked the beta3-AR-mediated increase triggered by Trecadrine. Brown adipose tissue UCP-2 and -3 mRNA expression was not changed by yohimbine. In conclusion, the present work indicates that in vitro alpha2-AR blockade by yohimbine potentiates the beta3-AR-mediated stimulation of lipolysis. On the other hand, in vivo alpha2-AR antagonism blocks the thermogenic effects mediated by beta3-AR stimulation, suggesting a possible interplay between the receptors.
needtogetaas
New member
post up bros
needtogetaas
New member
well I ariganaly wanted a post for some ephedra.not ephedrin.still havent got one.I would like some ephedra.Lestat said:
Is there a reason you wanted ephedra over ephedrine HCL? I've never known anyone to like ephedra, really. All of my friends and I from back home felt that ephedra had (comparatively) significantly diminished effects compared to ephedrine.
And Shadow's "homemade" ECA stack is the best, and CHEAPEST!! And to argue whomever said aspirin isn't neccessary, do some more research. Aspirin's effects on the 'blood', so to speak, has an impact on more drugs than it doesn't have an impact on. Nothing to do with the whole 'synergism in ECA' aspect, mind you. The extent to which the sought-after effects of ephedrine are effected is debatable, but nonetheless...
HTH
And Shadow's "homemade" ECA stack is the best, and CHEAPEST!! And to argue whomever said aspirin isn't neccessary, do some more research. Aspirin's effects on the 'blood', so to speak, has an impact on more drugs than it doesn't have an impact on. Nothing to do with the whole 'synergism in ECA' aspect, mind you. The extent to which the sought-after effects of ephedrine are effected is debatable, but nonetheless...
HTH
needtogetaas
New member
I just started useing shadows eca stake.its only bin a week and I have lost 5lbs.its just I use to use ripped foel and that shit was fucking amazing.it even gave me a good pump in the gym.so I was thinking it was the ephedra that was doing it.samoth said:
needtogetas said:
Are you using ephedra in the stack? If so, have you ever tried ephedrine HCL for comparison?
Lots of the name brand ECA stacks like Ripped Fuel are great supps... but their price is outrageous. Shadow's "homemade" ECA stack is the exact same for a fraction the cost. It's like the whole name brand vs. off brand pharmaceutical debate... they're the exact same chemicals made under the same conditions... is there a reason to pay more for the name brand? Studies showed no.
sawastea
Well-known member
This is from an article written by Stephen Schmitz, MD, MPH nd Bruce Kneller, BSN-RN.
Aspirin
"Aspirin {salicyclic acid acetate} and it's naturally occurring methyl ester (methyl salicylate)-- found in the leaves of Gaultheria procumbrens and on the bark of Betula lenta-- have long been used as analgesics, anti-inflammatories, antipyretics and recently as anti-coagulants. The "A" portion of ECA, aspirin has been thought of potentiating the thermogenic and lipolytic properties of both ephedrine and caffeine. Recent research seems to contradict this however. At a study at The University of London's Department of Nutrition and Dietetics, 40 women (20 non-obese and 20 obese) were given either ephedrine and caffeine (30 mg and 100 mg) or ephedrine, caffeine and aspirin (30 mg, 100 mg, and 300 mg) post prandially (1050 kJ liquid meal). Using indirect calorimetry, observations were made in all groups every 30 minutes for 160 minutes. There was no significant differences between the groups that received aspirin and the groups that did not. We conclude that aspirin does not potentiate the acute thermic effect of ephedrine and caffeine.6
In another study conducted at King's College in London, it was demonstrated that aspirin does not potentiate the thermogenic response to ephedrine in lean women and only slightly so in obese women.7 The increase seen in this study represented a 1.2 kcal per hour increase in metabolism for lean women and a 2.4 kcal per hour increase in metabolism in obese women over use of ephedrine alone, which over an entire week, would represent less than one ounce of bodyweight. We feel that this is insignificant.7
Based on these two studies, we feel that aspirin and its analogs do not play a substantial role in inducing or increasing thermogenesis and/or lipolysis but they do have the potential to induce SAEs when consumed over long periods of time (e.g. ulceration of the stomach and increases in bleeding times). Therefore, we feel that aspirin and it's analogs should not be used in any thermogenic/lipolytic stack."
6) Horton TJ, Geissler CA. Post-prandial Thermogenesis with Ephedrine, Caffeine and Aspirin in Lean, Predisposed Obese and Non-obese women. Int J Obes Relat Metab Disorder, 1996 Feb;20(2):91-95.
7) Horton TJ, Geissler CA. Aspirin Potentiates the Effect of Ephedrine on the Thermogenic Response to a Meal in Obese but not Lean Women. Int J Obes, 1991 May;15(5):359-366.
Most of the arguements that people make for aspirin state that aspirin extends the thermogenic effects of ephedrine by inhibiting the enzyme cyclooxygenase and therefore decreasing prostaglandin synthesis (why aspirin is an anti-inflammatory). This helps combat one of ephedrine's effects. Ephedrine stimulates the release of norepinephrine, which stimulates the release of adenosine and the synthesis of prostaglandins by the activated tissue. Since aspirin inhibits the synthesis of prostaglandins (1), it is believed it enhances the effect of norepinephrine (the reason why caffeine is included in the stack to combat adenosine). By increasing the amount of norepinephrine one would expect to increase lipolysis. Caffeine achieves this, however in actual studies aspirin as yet to prove the same. One can only hypothesize on why aspirin's effect on prostaglandins does not effect lipolysis.
There have been no studies that I am aware of that show that an ECA stack is more effective than an EC stack. The only studies that have been proven to work are when an ECA stack has been done in a study against a placebo. Couple this information with the effects aspirin has on the stomach (nausea, heartburn, and sometimes bleeding ulcers) one could conclude that the most effective stack would be just Ephedrine and Caffeine.
1. Rawson ES, Clarkson PA. Ephedrine as an ergogenic aid. Performance-enhancing Substances in Sport and Exercise. Ed Bahrke MS, Yesalis CE. Human Kinetics. 2002.
Aspirin
"Aspirin {salicyclic acid acetate} and it's naturally occurring methyl ester (methyl salicylate)-- found in the leaves of Gaultheria procumbrens and on the bark of Betula lenta-- have long been used as analgesics, anti-inflammatories, antipyretics and recently as anti-coagulants. The "A" portion of ECA, aspirin has been thought of potentiating the thermogenic and lipolytic properties of both ephedrine and caffeine. Recent research seems to contradict this however. At a study at The University of London's Department of Nutrition and Dietetics, 40 women (20 non-obese and 20 obese) were given either ephedrine and caffeine (30 mg and 100 mg) or ephedrine, caffeine and aspirin (30 mg, 100 mg, and 300 mg) post prandially (1050 kJ liquid meal). Using indirect calorimetry, observations were made in all groups every 30 minutes for 160 minutes. There was no significant differences between the groups that received aspirin and the groups that did not. We conclude that aspirin does not potentiate the acute thermic effect of ephedrine and caffeine.6
In another study conducted at King's College in London, it was demonstrated that aspirin does not potentiate the thermogenic response to ephedrine in lean women and only slightly so in obese women.7 The increase seen in this study represented a 1.2 kcal per hour increase in metabolism for lean women and a 2.4 kcal per hour increase in metabolism in obese women over use of ephedrine alone, which over an entire week, would represent less than one ounce of bodyweight. We feel that this is insignificant.7
Based on these two studies, we feel that aspirin and its analogs do not play a substantial role in inducing or increasing thermogenesis and/or lipolysis but they do have the potential to induce SAEs when consumed over long periods of time (e.g. ulceration of the stomach and increases in bleeding times). Therefore, we feel that aspirin and it's analogs should not be used in any thermogenic/lipolytic stack."
6) Horton TJ, Geissler CA. Post-prandial Thermogenesis with Ephedrine, Caffeine and Aspirin in Lean, Predisposed Obese and Non-obese women. Int J Obes Relat Metab Disorder, 1996 Feb;20(2):91-95.
7) Horton TJ, Geissler CA. Aspirin Potentiates the Effect of Ephedrine on the Thermogenic Response to a Meal in Obese but not Lean Women. Int J Obes, 1991 May;15(5):359-366.
Most of the arguements that people make for aspirin state that aspirin extends the thermogenic effects of ephedrine by inhibiting the enzyme cyclooxygenase and therefore decreasing prostaglandin synthesis (why aspirin is an anti-inflammatory). This helps combat one of ephedrine's effects. Ephedrine stimulates the release of norepinephrine, which stimulates the release of adenosine and the synthesis of prostaglandins by the activated tissue. Since aspirin inhibits the synthesis of prostaglandins (1), it is believed it enhances the effect of norepinephrine (the reason why caffeine is included in the stack to combat adenosine). By increasing the amount of norepinephrine one would expect to increase lipolysis. Caffeine achieves this, however in actual studies aspirin as yet to prove the same. One can only hypothesize on why aspirin's effect on prostaglandins does not effect lipolysis.
There have been no studies that I am aware of that show that an ECA stack is more effective than an EC stack. The only studies that have been proven to work are when an ECA stack has been done in a study against a placebo. Couple this information with the effects aspirin has on the stomach (nausea, heartburn, and sometimes bleeding ulcers) one could conclude that the most effective stack would be just Ephedrine and Caffeine.
1. Rawson ES, Clarkson PA. Ephedrine as an ergogenic aid. Performance-enhancing Substances in Sport and Exercise. Ed Bahrke MS, Yesalis CE. Human Kinetics. 2002.
needtogetaas
New member
no I am using the ephedrin hcl.samoth said:
Mikus
New member
sawastea said:
good info, just in my experiences (i'm not obese though) i get a bit better cardio performance when combining yohimbine. I can maybe get an extra couple minutes of cardio out of just an ephedra stack, if that makes sense.
But i do agree, it's not needed and i definetly wouldn't recommend it to someone who was obese.
T
The Shadow
Guest
samoth said:
word.
and dont EVER use ANY EPHEDRA PRODUCTS WITH ORAL YOHIMBINE
EVER
EVER
Umm... Swastea... you're arguing a good point and all, but it's not what I was addressing: "Nothing to do with the whole 'synergism in ECA' aspect, mind you." I'm arguing more the effect of aspirin on the human orgainism, eg. 'thinning the blood' and such, and not specific mechanisms exclusive to ephedrine or caffeine. EC_A_ has always kind of been a rule of thumb in the weightlifting community, but I have not seen any compelling evidence for specific mechanistic synergism either. For what it's worth, I've been using ephedrine for 8-9 years, and have never noticed or "felt" any difference when combining with aspirin.
Yohimbine HCL does NOT agree with me. I don't know why. I tried it twice, over a year apart, and experienced the same effects. Like, umm... not sure how to explain it. Not sick per se, yet still nausious. More of a I-just-injested-a-poison-and-feel-like-shit kinda anxiety... it really screwed with my mentality or sense of well-being more than the physical I-feel-like-shit kinda thing.
My mind associates yohimbine HCL with rat poison. I won't go near it. I could never figure out why I had those effects from it, though.
My mind associates yohimbine HCL with rat poison. I won't go near it. I could never figure out why I had those effects from it, though.
Mikus
New member
samoth said:
I have a buddy who gets that way when taking yohimbine. It would be interesting to know why.
And shadow, why do you say no oral yohimbine products? There use to be a load of ECAY stacked products on the market...so i'm just curious why you say not to stack oral yohimbine (and for that matter, why would it matter oral vs transdermal)
T
The Shadow
Guest
Mikus said:
Key phrase is "used to be"......
Transdermal is preferable as you get a response form a much lower dose...the effective oral dose makes a lot of folks sick to their stomach and makes your skin feel like its literally crawling.
NYC stacks were "good" as well......again - they are off the market for a reason
I take Yohimbe extract at 500mg a capsule...
Yohimbe seemed to be the magic ingrediant in my eca stack. Really helped with the loss.
But it makes my girl get jittery, hot/cold, shakey, and somewhat nausea, i guess everyone is different.
But the best stack i have ever ever ever had had to be
Caffeine, Aspirin, Bioprene, Yohimbine HCL, Coleus Forskohili, and Citrus Aurantium
Melted Fat off me.
Yohimbe seemed to be the magic ingrediant in my eca stack. Really helped with the loss.
But it makes my girl get jittery, hot/cold, shakey, and somewhat nausea, i guess everyone is different.
But the best stack i have ever ever ever had had to be
Caffeine, Aspirin, Bioprene, Yohimbine HCL, Coleus Forskohili, and Citrus Aurantium
Melted Fat off me.
herdus33
New member
a good site ive been using for awhile is www.thatswholesale.com ......they have quite a few diff ephedra products......most are eca regulation stacks.....not bad prices either
TheMesonator
New member
Shadow,
Any ideas on where else to buy those bolt ephedrine pills? That site you listed can't deliver to california, where I'm located. I was really excited to see that, as I feel like Guaf messes me up.
Any ideas on where else to buy those bolt ephedrine pills? That site you listed can't deliver to california, where I'm located. I was really excited to see that, as I feel like Guaf messes me up.
needtogetaas
New member
it might be a little more expensive but alote of conveinience stores sell them.you must be over 18 and they will only sell you 2 bottles at a time.TheMesonator said:
TheMesonator
New member
needtogetas said:
Thanks need, I'll take a look. I always find myself coughing when I take Eph with Guaf. Very interested to see if I can manage to kick that cough since it never happened till I used ephedrine with guaf. Yuck!
offshore88
New member
Turanabol said:
I'm currently taking Lipodrene as well. How many do you take/day? Recommended dosage is 2/day, but I've read that 75mg ephedrine/day is better, which would be 3, any thoughts?
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