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Elevated ALT Liver enzyme.

c gheller

New member
I have heard that this enzyme can be elevatede due to weight training and does not necessarily mean you have liver problems. Reading is 46u/l (<40). Is this true. Thanks.
 
Thanks but article on elevated ALT enzymes does not mention if weight training can have an effect.


With drug-induced liver enzyme abnormalities, the enzymes usually normalize weeks to months after stopping the medications.


Weight training does not affect your liver, gear can, but in actually does not,


Boozed is a culprit and so is Tylenol,

But as the quote above infers if liver enzymes are elevated do to certain medications it is temporary, this does not mean booze or gear.
 
solidspine said:
Weight training does not affect your liver, gear can, but in actually does not

Agreed for injects but what about 17aa orals?. . .definitely cause liver stress which elevates enzymes. . .
 
solidspine said:
Weight training does not affect your liver, gear can, but in actually does not,


Boozed is a culprit and so is Tylenol,

But as the quote above infers if liver enzymes are elevated do to certain medications it is temporary, this does not mean booze or gear.

Transaminase levels are not liver function tests. They are tests of inflamation. All muscle tissue will release transaminase if it is inflamed. This is why heavy weight training can raise ALT levels. It's not from the liver but from the muscle tissue that is releasing the transaminase.

If you have an ALT level that is 46 suck it up and stop being such a pussy. If you had liver problems they would be in the thousands. But with that said if you are always slightly elevated suspect hep C. Hep C raises alt but only slightly if it is in the chronic stage.
 
c gheller said:
I have heard that this enzyme can be elevatede due to weight training and does not necessarily mean you have liver problems. Reading is 46u/l (<40). Is this true. Thanks.


Hey, I just had some blood work done a little while ago.

ALT - measurement to see if the liver is damaged. low levels of ALT are normally found in the blood. When your liver becomes diseased, it releases more ALT into the blood stream, so this would make for a high reading in the blood stream. Most causes of elevated ALT are due to liver disease (ALT can also be found in the Kidney, Heart, muscles, and pancreas).

The Reference Range for ALT is 14-53 U/L

You're reading is on the high side, but still w/in the reference Range


The other readings that you want to look at to see if you have liver damage are:

AST (reference range 9-43 U/L)
Gamma GT (reference range 14-73 U/L)
T Bili (reference range 0.2 - 1.2mg/dl)
 
Last edited:
Full Lipid profile + ALT + AST + HbA1c

this is your liver profile picture you want. one piece doesn't describe the whole picture.

good luck
 
c gheller said:
I have heard that this enzyme can be elevatede due to weight training and does not necessarily mean you have liver problems. Reading is 46u/l (<40). Is this true. Thanks.


True.
 
I have heard many times that heavy training can raise alt.. Also, the gt or ggt (whatever the f.... its called) is the real indicator of liver damage.
 
digimon7068 said:
Agreed for injects but what about 17aa orals?. . .definitely cause liver stress which elevates enzymes. . .


yeah bro, but that is why we don't do 10 week cycles of 17aaS
 
Right GGT can only come from an organ that is stressed so you need to measure all three. If the GGT is not elevated then it's not coming from your liver. Print this and bring it to your doctor. :)

Clin J Sport Med. 1999 Jan;9(1):34-9.

Anabolic steroid-induced hepatotoxicity: is it overstated?

Dickerman RD, Pertusi RM, Zachariah NY, Dufour DR, McConathy WJ.

The Department of Biomedical Science, University of North Texas Health Science Center, Fort Worth 76107-2699, USA.

OBJECTIVE: There have been numerous reports of hepatic dysfunction secondary to anabolic steroid use based on elevated levels of serum aminotransferases. This study was conducted to distinguish between serum aminotransaminase elevations secondary to intense resistance training and anabolic steroid-induced hepatotoxicity in elite bodybuilders. DESIGN: This was a case-control study of serum chemistry profiles from bodybuilders using and not using anabolic steroids with comparisons to a cohort of medical students and patients with hepatitis. PARTICIPANTS: The participants were bodybuilders taking self-directed regimens of anabolic steroids (n = 15) and bodybuilders not taking steroids (n = 10). Blood chemistry profiles from patients with viral hepatitis (n = 49) and exercising and nonexercising medical students (592) were used as controls. MAIN OUTCOME MEASURES: The focus in blood chemistry profiles was aspartate aminotransferase (AST), alanine aminotransferase (ALT), gamma-glutamyltranspeptidase (GGT), and creatine kinase (CK) levels. RESULTS: In both groups of bodybuilders, CK, AST, and ALT were elevated, whereas GGT remained in the normal range. In contrast, patients with hepatitis had elevations of all three enzymes: ALT, AST, and GGT. Creatine kinase (CK) was elevated in all exercising groups. Patients with hepatitis were the only group in which a correlation was found between aminotransferases and GGT. CONCLUSION: Prior reports of anabolic steroid-induced hepatotoxicity based on elevated aminotransferase levels may have been overstated, because no exercising subjects, including steroid users, demonstrated hepatic dysfunction based on GGT levels. Such reports may have misled the medical community to emphasize steroid-induced hepatotoxicity when interpreting elevated aminotransferase levels and disregard muscle damage. For these reasons, when evaluating hepatic function in cases of anabolic steroid therapy or abuse, CK and GGT levels should be considered in addition to ALT and AST levels as essential elements of the assessment.
 
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