Ok, found it.
This "theory" comes from a Bruce Kneller/Brock Strasser article from Testosterone.net and is based on the infamous faulty study by Minto et. al.
Kneller/Stasser.......same person.......is generally disregarded as a steroid authority because he is wrong so often.
Here are some breakdowns of this report by some pretty smart people:
My take on this: (Bchemist)
"This study concluded that gluteal injections yielded far superior plasma levels as opposed to injections in the deltoid.
Of all the locations that nandrolone injections were given in this study (100 mg/ml x 1 ml in the glutes, 25 mg/ml x 4 ml in the glutes and 100 mg/ml x 1 ml in the deltoid), the deltoid injections yielded the lowest plasma levels of nandrolone by a huge factor, with peak concentrations being 50% lower than the 100 mg/ml gluteal injection."
The way I am reading this is that this applies to a single injection vs. 4 smaller injections. Does this same principle apply to an 8 week cycle of Deca? I doubt that the study is applicable to the way we use deca for long periods of time. The key here is the idea of "wasted gear". My question is this, "Is the Deca actually wasted, or just much more slowly absorbed into the bloodstream via the muscle showing a lower peak concentration?"
"Also interesting: short-chain esters (steroids of shorter half life) yield a much higher plasma concentration of steroid than steroids of longer side chain esters. In this study, a single 100 mg/ml x 1 ml intragluteal injection of nandrolone phenylpropionate caused a peak plasma concentration of almost double that of the 100 mg/ml x 1 ml intragluteal injection of nandrolone decanoate."
Of course this will be true. That is the point of short chain esters...they cleave the ester from the nandrolone molecule at much more rapid rate, giving a greater peak concentration on a mg to mg basis. Plus an shorter ester is lighter, thus giving you an automatically higher amount of nandrolone per 100mg....not double, but higher.
"And this: A single injection of only 100 mg of nandrolone phenylpropionate caused almost complete suppression of endogenous Testosterone by day three and lasted until around day eight."
Because it is out of the bloodstream quicker and therefore does less "damage".
"Endogenous levels of Testosterone didn’t return to baseline levels for almost fifteen days, while the same type of injection with nandrolone decanoate caused almost complete inhibition of endogenous by day four. Endogenous levels of Testosterone didn’t return to baseline levels for greater than twenty days! All this from a single, 100-mg injection of nandrolone"
Again, this will be apparent as the duration of deca is longer than the lighter ester and therefore will cause greater suppression over a longer time period.
And this: I believe Animal already posted Minto study,on wich Kneller(or whoever he is) based his article.
I'll reposted here, so we can look again at original research:
Pharmacokinetics and pharmacodynamics of nandrolone esters in oil vehicle: effects of ester, injection site and injection volume.
J Pharmacol Exp Ther 1997 Apr;281(1):93-102 (ISSN: 0022-3565)
Minto CF; Howe C; Wishart S; Conway AJ; Handelsman DJ [Find other articles with these Authors]
Department of Anaesthesia and Pain Management, Royal North Shore Hospital, University of Sydney, Australia.
We studied healthy men who underwent blood sampling for plasma nandrolone, testosterone and inhibin measurements before and for 32 days after a single i.m. injection of 100 mg of nandrolone ester in arachis oil. Twenty-three men were randomized into groups receiving nandrolone phenylpropionate (group 1, n = 7) or nandrolone decanoate (group 2, n = 6) injected into the gluteal muscle in 4 ml of arachis oil vehicle or nandrolone decanoate in 1 ml of arachis oil vehicle injected into either the gluteal (group 3, n = 5) or deltoid (group 4, n = 5) muscles. Plasma nandrolone, testosterone and inhibin concentrations were analyzed by a mixed-effects indirect response model. Plasma nandrolone concentrations were influenced (P < .001) by different esters and injection sites, with higher and earlier peaks with the phenylpropionate ester, compared with the decanoate ester. After nandrolone decanoate injection, the highest bioavailability and peak nandrolone levels were observed with the 1-ml gluteal injection. Plasma testosterone concentrations were also influenced (P < .001) by the ester and injection site, with the most rapid, but briefest, suppression being due to the phenylpropionate ester, whereas the most sustained suppression was achieved with the 1-ml gluteal injection. Plasma inhibin concentrations were also significantly influenced by injection volume and site, with the lowest nadir occurring after the nandrolone decanoate 1-ml gluteal injection. Thus, the bioavailability and physiological effects of a nandrolone ester in an oil vehicle are greatest when the ester is injected in a small (1 ml vs. 4 ml) volume and into the gluteal vs. deltoid muscle. We conclude that the side-chain ester and the injection site and volume influence the pharmacokinetics and pharmacodynamics of nandrolone esters in an oil vehicle in men.
I don't think, it's a news for us, that 4 weekly injections of 1ml is much better then 1 injection of 4ml.
And this: (E2)
Whoever wrote this is an idiot.
That's all i have to say.
Just by the way it's written it screams, "i'm a fool!"
Ironmaster:
In short, the theory doesn't hold water because the study is stupid...........he used different volumes and frequencies of injections in the delts vs the glutes.....so we learn nothing here.
Most everything you read at T-Mag should be taken with a grain of salt.
NO STUDY SUPPORTS THE IDEA THAT GLUTE INJECTS ARE SUPERIOR
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