browncloud said:
Geo,
I am curious about your shock theory. It seems to me that i am losing hair even on the sides of my head where i have plenty. How long can this last? Maybe it is the placebo effect but i have been off the proviron for about 5 days now and it seems to be getting better already.
BC
It's not a theory, it's fact and it happened to me.
acute stresses (metabolic, endocrine, chemical, emotional) are well-documented to cause these types of loss.
there are 2 types of "shock fallout" (I think thats my phrase so don't search on it) telogen effluvium (TE) and anagen effluvium (AE). telogen and anagen are 2 phases in the life cycle of hair. anagen is growing phase, then bulbs off and transitions to Telogen phase which is the resting period. this telogen hair remains on head till next anagen phase starts, at which point the old telogen hair is pushed out, and the new hair then starts to grow tru. Normally this is staggered around head so that it is not noticable (iow some small percentage in telogen at any one time)
in a telogen effluvium, the outside stressor forces all or most hair into telogen phase. then after a delay (2 months or so?) the anagen phase starts and a diffuse all over your head shed happens. (but assumming the stressor removed you get most of it back)
in anagen effluvium a more immediate breaking off of growing hair at the scalp line occurs, and when stressor removed get regrowth (think chemo patient)
**when diffuse hair loss occurs outside of the normal male pattern baldness area's , TE or AE is likely culprit. ie hairloss at sides of head is probably NOT mere MPB.**
you can ID TE by white bulb at the end of most shed hairs. ID AE by lack of bulbs (since they just snapped off at scalpline).
The third variety is part accepted fact part conjecture. normal DHT levels cause MPB in the predisposed over many hair growth cycles with squezing off the follicle resulting in progressive miniaturization. the abnormally ultra-suprapysiological level of DHT in AAS (ab)users (and proviron is basically a pure DHT analogue if I'm not mistaken) I believe cause a modified TE of sorts. That is, an abnormally large number of hairs forced into telogen phase, BUT then instead of the shed happening a few months later when the anagen phase starts, the acute choking off of the hair by the huge DHT levels is sufficient to cause the fallout to happen quickly. The longer and higher the dose of DHT, the greater the shed. In my case, the shed was preceeded by almost a feeling of "pain" in the scalp, along with huge big pumpy muscles and oily skin. p.s. no roids invoolved at the time.
In my case, a week-long mega-mega dosing on avena sativa (don't ask) which free's up bound test caused a diffuse TE to occur within weeks (about 3). iow, lots of newly freed test to convert to DHT.
so the shed hairs had the white bulb characteristic of TE, but fell quickly without the delay (which would be characteristic of AE).
I still didn't go to sleep, so I forgot what the question was, but you get the idea.
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