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Use FlaxSEED, not Flax OIL!

F

Frackal

Guest
Why anyone uses flax oil other than being unaware of flaxseed I have no idea...


Flax oil:

-Tastes nasty

- Low in estrogen mimicking, testosterone boosting, cholesterol enhancing lignans

- No fiber

- No protein

Flax Seed:

- Tastes fine

- Rich in Lignans, in high enough dosages said to perhaps be as powerful in action as nolvadex

- Rich in fiber

- Complete protein source


No contest IMO.... In fact, I feel an optimal before bed meal consists of casein/cottage cheese, whey protein and flaxSeed.....I use this whether bulking or cutting, it will keep insulin response down ... according to some sources flaxseed is highly unlikely to be stored as bodyfat, contains FFA and fibers to enhance the slow digesting action of the protein...in fact, with this combination I NEVER feel 'flat' in the morning, my muscles are always full...


So (in my opinion) use FLAXSEED to grow, flax oil if you want to puke often.
 
you likey flax seed... you must no likey testosterone.. or maybe you likey prolactin..

study1. reduced estrogen and reduced test in men (prolactin not measured)

study2. reduced estrogen, no affect on testosterone, increased prolactin

the lack of affect on testosterone is not suprising in women, as most is adrenal..


what does this mean?

well.. not really clear.. but it could mean that the phytoestrogenic lignans are HPTA suppresive..

something that one may want to consider the japanese bodyfat storage pattern and consider the phytoestrogen rich diet they consume..

If you are on TEST there is probably less reason not to take the lignans..(tehy do have considerable "health values".. if you are off.. well there may be some negative impact on anabolism..

then again nothing conclusive here..

Urology 2001 Jul;58(1):47-52 Related Articles, Links


Pilot study of dietary fat restriction and flaxseed supplementation in men with prostate cancer before surgery: exploring the effects on hormonal levels, prostate-specific antigen, and histopathologic features.

Demark-Wahnefried W, Price DT, Polascik TJ, Robertson CN, Anderson EE, Paulson DF, Walther PJ, Gannon M, Vollmer RT.

Division of Urologic Surgery, Duke University Medical Center, Durham, North Carolina, USA.

OBJECTIVES: Dietary fat and fiber affect hormonal levels and may influence cancer progression. Flaxseed is a rich source of lignan and omega-3 fatty acids and may thwart prostate cancer. The potential effects of flaxseed may be enhanced with concomitant fat restriction. We undertook a pilot study to explore whether a flaxseed-supplemented, fat-restricted diet could affect the biomarkers of prostatic neoplasia. METHODS: Twenty-five patients with prostate cancer who were awaiting prostatectomy were instructed on a low-fat (20% of kilocalories or less), flaxseed-supplemented (30 g/day) diet. The baseline and follow-up levels of prostate-specific antigen, testosterone, free androgen index, and total serum cholesterol were determined. The tumors of diet-treated patients were compared with those of historic cases (matched by age, race, prostate-specific antigen level at diagnosis, and biopsy Gleason sum) with respect to apoptosis (terminal deoxynucleotidyl transferase [TdT]-mediated dUTP-biotin nick end-labeling [TUNEL]) and proliferation (MIB-1). RESULTS: The average duration on the diet was 34 days (range 21 to 77), during which time significant decreases were observed in total serum cholesterol (201 +/- 39 mg/dL to 174 +/- 42 mg/dL), total testosterone (422 +/- 122 ng/dL to 360 +/- 128 ng/dL), and free androgen index (36.3% +/- 18.9% to 29.3% +/- 16.8%) (all P <0.05). The baseline and follow-up levels of prostate-specific antigen were 8.1 +/- 5.2 ng/mL and 8.5 +/- 7.7 ng/mL, respectively, for the entire sample (P = 0.58); however, among men with Gleason sums of 6 or less (n = 19), the PSA values were 7.1 +/- 3.9 ng/mL and 6.4 +/- 4.1 ng/mL (P = 0.10). The mean proliferation index was 7.4 +/- 7.8 for the historic controls versus 5.0 +/- 4.9 for the diet-treated patients (P = 0.05). The distribution of the apoptotic indexes differed significantly (P = 0.01) between groups, with most historic controls exhibiting TUNEL categorical scores of 0; diet-treated patients largely exhibited scores of 1. Both the proliferation rate and apoptosis were significantly associated with the number of days on the diet (P = 0.049 and P = 0.017, respectively). CONCLUSIONS: These pilot data suggest that a flaxseed-supplemented, fat-restricted diet may affect prostate cancer biology and associated biomarkers. Further study is needed to determine the benefit of this dietary regimen as either a complementary or preventive therapy.


Nutr Cancer 2001;39(1):58-65 Related Articles, Links


Flaxseed consumption influences endogenous hormone concentrations in postmenopausal women.

Hutchins AM, Martini MC, Olson BA, Thomas W, Slavin JL.

Department of Food Science and Nutrition, University of Minnesota, St. Paul, MN 55108, USA.

Lignans, similar in structure to endogenous sex steroid hormones, may act in vivo to alter hormone metabolism and subsequent cancer risk. The objective of this study was to examine effects of dietary intake of a lignan-rich plant food (flaxseed) on serum concentrations of endogenous hormones and binding proteins (estrone, estrone sulfate, 17 beta-estradiol, sex hormone-binding globulin, progesterone, prolactin, dehydroepiandrosterone sulfate, dehydroepiandrosterone, androstenedione, testosterone, and free testosterone) in postmenopausal women. This randomized, crossover trial consisted of three seven-week feeding periods, during which 28 postmenopausal women, aged 52-82 yr, consumed their habitual diets plus 0, 5, or 10 g of ground flaxseed. Serum samples collected during the last week of each feeding period were analyzed for serum hormones using standard diagnostic kits. The flaxseed diets significantly reduced serum concentrations of 17 beta-estradiol by 3.26 pg/ml (12.06 pmol/l) and estrone sulfate by 0.09 ng/ml (0.42 nmol/l) and increased prolactin by 1.92 micrograms/l (0.05 IU/ml). Serum concentrations of androstenedione, estrone, sex hormone-binding globulin, progesterone, testosterone, free testosterone, dehydroepiandrosterone, and dehydroepiandrosterone sulfate were not altered with flaxseed feeding. In this group of postmenopausal women, consuming flaxseed in addition to their habitual diets influenced their endogenous hormone metabolism by decreasing serum 17 beta-estradiol and estrone sulfate and increasing serum prolactin concentrations.
 
Flax seeds are very hard to chew, and the oil is not that bad tasting. But I am a nut case, because I like to suck on my clomid tabs.
 
macrophage69alpha said:
you likey flax seed... you must no likey testosterone.. or maybe you likey prolactin..

study1. reduced estrogen and reduced test in men (prolactin not measured)

study2. reduced estrogen, no affect on testosterone, increased prolactin

the lack of affect on testosterone is not suprising in women, as most is adrenal..


what does this mean?

well.. not really clear.. but it could mean that the phytoestrogenic lignans are HPTA suppresive..

something that one may want to consider the japanese bodyfat storage pattern and consider the phytoestrogen rich diet they consume..

If you are on TEST there is probably less reason not to take the lignans..(tehy do have considerable "health values".. if you are off.. well there may be some negative impact on anabolism..

then again nothing conclusive here..

Urology 2001 Jul;58(1):47-52 Related Articles, Links


Pilot study of dietary fat restriction and flaxseed supplementation in men with prostate cancer before surgery: exploring the effects on hormonal levels, prostate-specific antigen, and histopathologic features.

Demark-Wahnefried W, Price DT, Polascik TJ, Robertson CN, Anderson EE, Paulson DF, Walther PJ, Gannon M, Vollmer RT.

Division of Urologic Surgery, Duke University Medical Center, Durham, North Carolina, USA.

OBJECTIVES: Dietary fat and fiber affect hormonal levels and may influence cancer progression. Flaxseed is a rich source of lignan and omega-3 fatty acids and may thwart prostate cancer. The potential effects of flaxseed may be enhanced with concomitant fat restriction. We undertook a pilot study to explore whether a flaxseed-supplemented, fat-restricted diet could affect the biomarkers of prostatic neoplasia. METHODS: Twenty-five patients with prostate cancer who were awaiting prostatectomy were instructed on a low-fat (20% of kilocalories or less), flaxseed-supplemented (30 g/day) diet. The baseline and follow-up levels of prostate-specific antigen, testosterone, free androgen index, and total serum cholesterol were determined. The tumors of diet-treated patients were compared with those of historic cases (matched by age, race, prostate-specific antigen level at diagnosis, and biopsy Gleason sum) with respect to apoptosis (terminal deoxynucleotidyl transferase [TdT]-mediated dUTP-biotin nick end-labeling [TUNEL]) and proliferation (MIB-1). RESULTS: The average duration on the diet was 34 days (range 21 to 77), during which time significant decreases were observed in total serum cholesterol (201 +/- 39 mg/dL to 174 +/- 42 mg/dL), total testosterone (422 +/- 122 ng/dL to 360 +/- 128 ng/dL), and free androgen index (36.3% +/- 18.9% to 29.3% +/- 16.8%) (all P <0.05). The baseline and follow-up levels of prostate-specific antigen were 8.1 +/- 5.2 ng/mL and 8.5 +/- 7.7 ng/mL, respectively, for the entire sample (P = 0.58); however, among men with Gleason sums of 6 or less (n = 19), the PSA values were 7.1 +/- 3.9 ng/mL and 6.4 +/- 4.1 ng/mL (P = 0.10). The mean proliferation index was 7.4 +/- 7.8 for the historic controls versus 5.0 +/- 4.9 for the diet-treated patients (P = 0.05). The distribution of the apoptotic indexes differed significantly (P = 0.01) between groups, with most historic controls exhibiting TUNEL categorical scores of 0; diet-treated patients largely exhibited scores of 1. Both the proliferation rate and apoptosis were significantly associated with the number of days on the diet (P = 0.049 and P = 0.017, respectively). CONCLUSIONS: These pilot data suggest that a flaxseed-supplemented, fat-restricted diet may affect prostate cancer biology and associated biomarkers. Further study is needed to determine the benefit of this dietary regimen as either a complementary or preventive therapy.


Nutr Cancer 2001;39(1):58-65 Related Articles, Links


Flaxseed consumption influences endogenous hormone concentrations in postmenopausal women.

Hutchins AM, Martini MC, Olson BA, Thomas W, Slavin JL.

Department of Food Science and Nutrition, University of Minnesota, St. Paul, MN 55108, USA.

Lignans, similar in structure to endogenous sex steroid hormones, may act in vivo to alter hormone metabolism and subsequent cancer risk. The objective of this study was to examine effects of dietary intake of a lignan-rich plant food (flaxseed) on serum concentrations of endogenous hormones and binding proteins (estrone, estrone sulfate, 17 beta-estradiol, sex hormone-binding globulin, progesterone, prolactin, dehydroepiandrosterone sulfate, dehydroepiandrosterone, androstenedione, testosterone, and free testosterone) in postmenopausal women. This randomized, crossover trial consisted of three seven-week feeding periods, during which 28 postmenopausal women, aged 52-82 yr, consumed their habitual diets plus 0, 5, or 10 g of ground flaxseed. Serum samples collected during the last week of each feeding period were analyzed for serum hormones using standard diagnostic kits. The flaxseed diets significantly reduced serum concentrations of 17 beta-estradiol by 3.26 pg/ml (12.06 pmol/l) and estrone sulfate by 0.09 ng/ml (0.42 nmol/l) and increased prolactin by 1.92 micrograms/l (0.05 IU/ml). Serum concentrations of androstenedione, estrone, sex hormone-binding globulin, progesterone, testosterone, free testosterone, dehydroepiandrosterone, and dehydroepiandrosterone sulfate were not altered with flaxseed feeding. In this group of postmenopausal women, consuming flaxseed in addition to their habitual diets influenced their endogenous hormone metabolism by decreasing serum 17 beta-estradiol and estrone sulfate and increasing serum prolactin concentrations.

this (or something similar) is what i read a long while back. so i stopped with my "high lignan" flax oil and switched to normal flax oil. i used to send my ex G/F to the health food store to get it for me, and we always debated how to say lignan....lol....lig-nan or lye-nan. i'd bitch slap her when she brought back regular flax....j/k....she always got me the right stuff. but i haven't used it since i read a few reports on the above mentioned side effects. that is ALL i need. jesus. used flax oil ever since.........like 6 years or more
 
Well that just sucks.... but why have I read a hundred times, even from Dan Duchaine (pretty sure it was awhile ago) saying flax et al was the most anabolic supplement, to numerous articles saying it boosts testosterone and inhibits estrogen similiar to nolvadex...

Here are a couple I found real quick:



"Will Brink:" The implication of the above study is obvious for women, but men reading this should see the clear potential benefits: flax seeds and high lignan flax oil may be a natural anti estrogen as powerful as Nolvadex and would explain why I have seen reductions in gyno in men taking high amounts of flax oil"

That's from this article: http://boards.elitefitness.com/forum/showthread.php?s=&threadid=159445&highlight=flaxseed+nolvadex


"In addition, the lignans in Flax Seed Oil have been shown to have potent anti-estrogenic effects. This is of special interest to bodybuilders who are always looking to increase their testosterone to estrogen ratio."

This is actually from the mass quantities website ...

I guess they were mistaken? That sucks, I like flaxseed, (well flaxMEAL to be specific to those of you who commented about the seed above)...

Oh well, back to Udo's choice oil :bawling: I guess I'll get more omega-6's anyhow
 
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I like minimal amounts of flax seed; good fiber content and not bad on protein.

As an omega-3 source flax in any form is shitty. Your body utilizes long-chain omega-3 fats; flax contains short-chain omega-3 fats. Short chains can be converted to long chains with great difficulty -- in the neighborhood of 1 to 5 percent.

What does this mean? It means that you're only getting 1 to 5 percent of the omega-3 content that flax oil contains.

Fish oil is a far superior choice for omega-3s.
 
worst thing is that you find differing opinions on this subject. but the negative reports outweigh the positive. so i just stay away from it (high lignans) just in case. udo's is the best, but i hate to go to the nutrition world to get it when i work at GNC and i get flax oil for like a dollar a small bottle (wholesale GNC product price).
 
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