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Quickest dose of HcG to plump my balls

wait a second, I heard more than 500iu a week could give you gyno and has estrogen side effects

Yes.. you can totally rebound from that, as well as desensitizing your leydig cells...

Too much or two little is a waste..
 
You guys are saying that 500iu once a week towards the end of the cycle is enough to fully recover them from atrophy? I thought you were supposed to do either 500iu ED for 10 days or 1500iu E3D for 2-3 weeks if you weren't running it straight through
 
500iu one a week is not enough...500iu every 3 days is better
 
wait a second, I heard more than 500iu a week could give you gyno and has estrogen side effects

Large doses cause a spike in test which means more E due to aromatization. HCG directly stimulates the testes to produce test, it only takes 3x a week injections at 250iu to maintain normal test production WHILE on a cycle. Thats your normal test production ON TOP OF the AAS your using.


Triptorelin appears to have promise BUT, just like HCG mimics LH and therefor decreases pituitary secretion so does Triptorelin.
 
Triptorelin, like hcg, does stimulate lh, but unlike hcg, it also stimulates , FSH. I believe the best approach would be low dose hcg on cycle, with a single 100mcg dose of triptorelin at the start of pct(as soon as esters have cleared).
 
Triptorelin, like hcg, does stimulate lh, but unlike hcg, it also stimulates , FSH. I believe the best approach would be low dose hcg on cycle, with a single 100mcg dose of triptorelin at the start of pct(as soon as esters have cleared).

HCG doesnt stimulate LH, it MIMICKS LH thereby causing the anterior pituitary gland to STOP producing LH while HCG is in the system.


Triptorelin, according to j_p's post, does the following.....

By causing constant stimulation of the pituitary, it decreases pituitary secretion of gonadotropins luteinizing hormone (LH) and follicle stimulating hormone (FSH).

The general definition of stimulate generally means to excite to activity or arouse so the cut and paste he provided from wiki is contradictory because it says it stimulates it AND DECREASES LH and FSH.

Im not saying its a bad product at all, probably awesome in fact, but shit is just starting to become popular and already the description of the product makes no sense. We need to round up some good studies that highlights what it does so folks can better understand it and we can show everyone some solid research behind it. Thats what moves product in my book.
 
HCG doesnt stimulate LH, it MIMICKS LH thereby causing the anterior pituitary gland to STOP producing LH while HCG is in the system.


Triptorelin, according to j_p's post, does the following.....



The general definition of stimulate generally means to excite to activity or arouse so the cut and paste he provided from wiki is contradictory because it says it stimulates it AND DECREASES LH and FSH.

Im not saying its a bad product at all, probably awesome in fact, but shit is just starting to become popular and already the description of the product makes no sense. We need to round up some good studies that highlights what it does so folks can better understand it and we can show everyone some solid research behind it. Thats what moves product in my book.

as far as i can tell thats why it is reccomended not to be used for long only in a dose or few.
but i will find more well rounded studys I have recently read on it and post them up. i need to go right now though, will do tomorrow though. :-)
 
Im think there are plenty of studies out there look up Trelstar by Watson for one. [FONT=&quot]Triptorelin[/FONT] is marketed under numerous brand names.

Yep, there are plenty of studies out there and what they are telling me is the primary use for triptorelin is to SHUT DOWN test production to slow down or stop the growth of prostate cancer cells.

Following the first administration, there is a transient surge in circulating levels of luteinizing hormone (LH), follicle-stimulating hormone (FSH), testosterone, and estradiol . After chronic and continuous administration, usually 2 to 4 weeks after initiation of therapy, a sustained decrease in LH and FSH secretion and marked reduction of testicular steroidogenesis are observed. A reduction of serum testosterone concentration to a level typically seen in surgically castrated men is obtained. Consequently, the result is that tissues and functions that depend on these hormones for maintenance become quiescent. These effects are usually reversible after cessation of therapy.

On the surface the initial surge of testosterone (around 3 days after injection) sounds like a good thing, BUT after that it nearly completely suppresses the bodies release of LH and FSH for 3-4 weeks, which in turn means no test production. Certainly not something I want to do post cycle.
 
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