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My First Cycle

unclematty

New member
Hey,

Im about 5'10 175 and i like my size. im not new to lifting a dieting but i havent been doing either for about 6 months. I have retained some stucture but would like mainly to just keep my size, maybe gain like 5 to 10 pounds but be ripped.

This is goin to be my first cycle. Any suggestions, thanks?

I was thinking...
Winnie, or Eq, or both...but i need a little help.

And even after i get either one, how do i know how much to do, and how often?
 
Do more resarch on fina, Equipiose, tren, winstrol, winny, and trenbolone acetate.
 
At your size/stats I would go:
Winny at 25-50mgs/day for 8 weeks
Eq at 300-400mgs/week for 10 weeks

Clomid therapy at week 13 to restore your endogenous testosterone production.

Take alpha-lipoic acid to keep your liver safe(600-800mgs/day).Keep your protein intake sky-high from start to finish,with carb and fat intake moderate.You should see a very nice alteration in body composition/strength and maintain them if all the above factors are in place.
 
This cycle is not only used for beginners, but experts as well. This cycle is great for adding size and strength, while keeping a good portion of it. The sustanon and dbol add the size and contribute to the strength. The deca not only adds strength but solidifies the gains from the sustanon and dbol.



Week
Sustanon mg/week
Dbol mg/day - Twice a day
Deca mg/week

1
250
2 * 20
100

2
250
2 * 30
200

3
250
2 * 30
300

4
250
2 * 20
400

5
250
2 * 10
400

6
250
-
300

7
250
-
200

8
250
-
100


Juice needed: 8 vials of Sustanon 250 mg/vial
400 tabs of Dbol
10 amps of Deca durabolin 200 mg/amp


You may want to add HCG in the last month half way in your cycle 5,000ui per week every(7days) sufficient. This will insure you max out your gains. As most in cycles gains are attributed in the begining of every cycle and therefore dimish afterwards..

You may also want to wait 3 weeks after your cycle and take one tab of clomid every day for 2 1/2 weeks.. in order to jump start the suppresion of your hormone levels due to your cycle...

supplements are optional ,, proteins and others.
 
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One tab of clomid will do nothing except waste it and also do a search and you will find the right way to come off the gear.
 
ok so whats stronger than!

a 200mg cyclofenil tab or a 50mg clomid tab???

since you said, one tab of clomid won't do crap!

by the way cyclofenil is similar if not the same as clomid ,,, inotherwords they do the same thing....
 
I'm not saying clomid is not the right one to use. You should start with lets say 300mg the first day and then 100 mg for about one week and then 50 mg for one week. Also 5000iu of hcg a week will also do nothing this should be taken 2500iu to 5000ius every four to five days for no more then four weeks and if you want to use HCG you can also add it half way through your cycle so the coming off is not as harsh. What will one 50 mg clomid do after a cycle like the one you posted 50mg will do nothing against Deca and sust and d-bol.
 
Not to knock you off your chair man! but, yeah sound a bit funny you say 300mg of clomid??? are you for real !!! your dosages are way to high, I see your eager to advise but, if you haven't been doing this for say more than 10 years like me ,i suggest you not give that guy your advice ... i like your post love reading them but your missinforming the guy...

now as for the hcg... bb's mix in the hcg in the middle of their cycles in order to max out their gains .. by adding hcg they ensure that the receptor cites widen, thus binding the remaining anabolics running freely in the blood! inturn more muscle gains... get the point !

the test will bind quicker and fast to an opening thats bigger than it originaly was before start...... i need not explain anymore.
 
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LOL,widen the receptor?WTF?Bro,human chorionic gonadotropin is effective mid cycle because it temporarily boosts L/H & endogenous T levels that are completely shut down by the A/S you are taking.It does not do anything to 'widen your receptors'.
 
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Ok, you possed the challenge ! now prove hcg does not open your receptor sites....

show me your proff..............

show me where it says, hcg does not open your receptor sites !!
 
Arnie:

I 'm glad to see that you love my posts but I'm not saying I know everything but its shows by your post that you do not either. 300mg for one day is not to high and I think most people would say I'm correct with what I posted. Why are you so argumentative with everyone? We are all learing and it is not your place to say that because you have in your mind 10 years that you are right, cause its shows by Huckleberry's post you are not. So lighten up and relax you do not have to be right all the time. Later, by the way you to can learn a thing or too if you continue to read.
 
bigman1 said:
Arnie:

I 'm glad to see that you love my posts but I'm not saying I know everything but its shows by your post that you do not either. 300mg for one day is not to high and I think most people would say I'm correct with what I posted. Why are you so argumentative with everyone? We are all learing and it is not your place to say that because you have in your mind 10 years that you are right, cause its shows by Huckleberry's post you are not. So lighten up and relax you do not have to be right all the time. Later, by the way you to can learn a thing or too if you continue to read.
[/QUOTE

pay no mind to him. like he says, he's been around this for over 10 years. lmfao!! who gives a shit how long you have been in this shit for. :rolleyes:
 
bigman1 said:
Arnie:

I 'm glad to see that you love my posts but I'm not saying I know everything but its shows by your post that you do not either. 300mg for one day is not to high and I think most people would say I'm correct with what I posted. Why are you so argumentative with everyone? We are all learing and it is not your place to say that because you have in your mind 10 years that you are right, cause its shows by Huckleberry's post you are not. So lighten up and relax you do not have to be right all the time. Later, by the way you to can learn a thing or too if you continue to read.

not argumentative bro,, no worries .... ok !!! everythings kewl.. i myself don't know everything thats just impossible..however i do know juice and ..


and huck has not proven me wrong as of yet ! and he knows i'm right thats why he hasn't posted anything in-return.! see
 
Mike P.T.:

Your right but have you read any of his other posts? Is this guy for real? I think he needs to prove to Huckleberry where it says it opens your receptors, don't you? This guy is a joke I think he meant to say he is 10 years old and he just got his first computer, and just picked up the lastest copy of Anabolic Review 96' ha ha!!!!
 
bigman1 said:
Mike P.T.:

Your right but have you read any of his other posts? Is this guy for real? I think he needs to prove to Huckleberry where it says it opens your receptors, don't you? This guy is a joke I think he meant to say he is 10 years old and he just got his first computer, and just picked up the lastest copy of Anabolic Review 96' ha ha!!!!


hmmm now in another post he says site injections are purely a myth. ummm okie dokie :p :p :p
 
HUCKLEBERRY FINNaplex said:
Arnie,do you even know what a receptor is?Let's hear your definition.


FIRST YOU PROVE ME WRONG SMART GUY! ABOUT HCG AND ITS EFFECTS ON MUSCLE RECEPTORS...


YOU SAY IT DOES NOT OPEN MUSCLE RECPTORS .. I SAY IT DOES..


WELL, YOU GONNA PROVE ME WRONG OR SIT THERE ALL NIGHT WITH A HUNG MOUTH!
 
HUCKLEBERRY FINNaplex said:
That's what I thought,lol.You are embarrassing yourself more than you know with each consecutive post.Quit while you're ahead my friend.
LMAO!!! So this is what I have missed..Huck don't even bother with these guys...Your wasting your effort LOL He is just making a food of himself as he was never ahead to begin with LOL! ;)
Everyone knows Huck is one of the smartest guys around here or any board for that matter and it's people like this Arnie guy that try to bring the board down with there senceless posts

M18
 
HUCKLEBERRY FINNaplex said:
That's what I thought,lol.You are embarrassing yourself more than you know with each consecutive post.Quit while you're ahead my friend.

YOU STILL HAVEN'T PROVEN ANYTHING AND YOU FULL OF IT! HERES A BIT OF INFO... THATS ALL I AM GONA PROVIDE .

Anabolic steroids work by binding with the cytoplasmic (free within the cell) androgen receptor. Like all steroids, the steroid-receptor complex has a strong affinity for the nucleus. The complex is translocated into the nucleus and binds to DNA. It is also possible that the steroid and receptor dissociate in the nucleus and act on DNA separately. The rate limiting factor in this process appears to be the cytoplasmic concentration of the receptor, rather than the concentration of the steroid or translocation of the complex. Testosterone also appears to inhibit the catabolic (protein degrading) pathway associated with glucocorticoids, but it is unclear whether this is related to an interaction between testosterone and glucocorticoid or the nuclear behavior of the steroid-receptor complex.

Once in the nucleus, the steroid appears to enhance transcription of specific genes. The resulting mRNA is processed and sent out of the nucleus, resulting in increased protein synthesis.

Catabolic inhibition may also occur in the nucleus if the complex inhibits the transcription of catabolic enzymes.

The presence of the androgen receptor indicates a tissue is androgen sensitive, and it's concentration gives an indication of how sensitive. The receptor is present in a number of organs, including skeletal muscle. Skeletal muscle typically contains 0.5-3 femto (1E-12) moles per milligram of protein, while other androgen sensitive organs, like the prostate gland, may have up to 25 times more receptors.
 
arnieswarzennega said:


YOU STILL HAVEN'T PROVEN ANYTHING AND YOU FULL OF IT! HERES A BIT OF INFO... THATS ALL I AM GONA PROVIDE .

Anabolic steroids work by binding with the cytoplasmic (free within the cell) androgen receptor. Like all steroids, the steroid-receptor complex has a strong affinity for the nucleus. The complex is translocated into the nucleus and binds to DNA. It is also possible that the steroid and receptor dissociate in the nucleus and act on DNA separately. The rate limiting factor in this process appears to be the cytoplasmic concentration of the receptor, rather than the concentration of the steroid or translocation of the complex. Testosterone also appears to inhibit the catabolic (protein degrading) pathway associated with glucocorticoids, but it is unclear whether this is related to an interaction between testosterone and glucocorticoid or the nuclear behavior of the steroid-receptor complex.

Once in the nucleus, the steroid appears to enhance transcription of specific genes. The resulting mRNA is processed and sent out of the nucleus, resulting in increased protein synthesis.

Catabolic inhibition may also occur in the nucleus if the complex inhibits the transcription of catabolic enzymes.

The presence of the androgen receptor indicates a tissue is androgen sensitive, and it's concentration gives an indication of how sensitive. The receptor is present in a number of organs, including skeletal muscle. Skeletal muscle typically contains 0.5-3 femto (1E-12) moles per milligram of protein, while other androgen sensitive organs, like the prostate gland, may have up to 25 times more receptors.

Nice cut and paste bud ;)

M18
 
arnieswarzennega said:


YOU STILL HAVEN'T PROVEN ANYTHING AND YOU FULL OF IT! HERES A BIT OF INFO... THATS ALL I AM GONA PROVIDE .

Anabolic steroids work by binding with the cytoplasmic (free within the cell) androgen receptor. Like all steroids, the steroid-receptor complex has a strong affinity for the nucleus. The complex is translocated into the nucleus and binds to DNA. It is also possible that the steroid and receptor dissociate in the nucleus and act on DNA separately. The rate limiting factor in this process appears to be the cytoplasmic concentration of the receptor, rather than the concentration of the steroid or translocation of the complex. Testosterone also appears to inhibit the catabolic (protein degrading) pathway associated with glucocorticoids, but it is unclear whether this is related to an interaction between testosterone and glucocorticoid or the nuclear behavior of the steroid-receptor complex.

so mr. smart guy why don't u explain that in laymans terms. BWAHAHAHA!!!!

Once in the nucleus, the steroid appears to enhance transcription of specific genes. The resulting mRNA is processed and sent out of the nucleus, resulting in increased protein synthesis.

Catabolic inhibition may also occur in the nucleus if the complex inhibits the transcription of catabolic enzymes.

The presence of the androgen receptor indicates a tissue is androgen sensitive, and it's concentration gives an indication of how sensitive. The receptor is present in a number of organs, including skeletal muscle. Skeletal muscle typically contains 0.5-3 femto (1E-12) moles per milligram of protein, while other androgen sensitive organs, like the prostate gland, may have up to 25 times more receptors.
 
ooops my post didnt show up. fuck anyway on the previous post i wrote, "so mr. smart guy explain this to us in laymans terms. BWAHAHAHA!!!"
 
Mustang and Mike please don't hold your breath waiting for a response from the jag off all he's been doing is cutting and pasteing just like in the insulin post. As I said before this guy is a JOKE!!!!!!
 
Mike P.T. said:
ooops my post didnt show up. fuck anyway on the previous post i wrote, "so mr. smart guy explain this to us in laymans terms. BWAHAHAHA!!!"


your so smart you translate it ! man this is funny good one !
 
arnieswarzennega said:



your so smart you translate it ! man this is funny good one !

hmm lemmi see fuckface. never said i could. im familiar with enough of the terms but i wouldnt say i know it all. hmm arent you the one that posted it. good one dickhead. u love making yourself look bad dont you. :p :p :p :p :p :p :p :p
 
Arine why is when someone ask YOU something you either cut and paste or you just sit there and say 2nd grade shit why don't you go to bed cause I'm sure your mommy and daddy are wondering why your up past your bedtime. Get off the board would you? we will then respect you much more.
 
HUCKLEBERRY FINNaplex said:
LOL,widen the receptor?WTF?Bro,human chorionic gonadotropin is effective mid cycle because it temporarily boosts L/H & endogenous T levels that are completely shut down by the A/S you are taking.It does not do anything to 'widen your receptors'.


hey smart guy! bwaahahaha... heres your answerrrr see you couldn't answer it .... after all i don't work in a lab for nothing ...

bwahahahahah !!!!

Although the uterus is a target tissue for LH and its homologue hCG the second messenger system responding to LH/hCG in myometrial cells is not established. In this study we investigated the involvement of protein kinase A and protein kinase C in the action of hCG on porcine myometrial smooth muscle cells in vitro. Myometrium was obtained from ovariectomized gilts given 2.5 mg oestradiol benzoate plus 50 mg progesterone for five consecutive days. Myometrial cells were cultured for 48 h and different doses of hCG were then added. Increasing doses of hCG stimulated concentration-dependent increases in [(3) H]inositol phosphates (IPs) accumulation in incubations lasting 24 h. The highest dose of hCG (1000 mU/ml) increased turnover of IPs by 2.4-fold as reflected in elevations in IP(1) , IP(2) and IP(3) , and similar effects were observed with noradrenaline. The time- and concentration-dependent effects of hCG on IPs accumulation occurred between 16 and 24 h of incubation. Incubation of myocytes with the lowest doses of hCG (0.1 and 1 mU/ml) caused a significant increase in cAMP accumulation but the highest doses (10-1000 mU/ml) had no effect on cAMP concentrations. This is the first demonstration that LH/hCG receptor signalling leads to increased inositol phosphate turnover in myometrial cells as well as cAMP generation and it leads to the conclusion that both protein kinase A and protein kinase C signalling mechanisms are involved in gonadotrophin action in porcine myometrial smooth muscle cells.



BUDDY YOU GET THE DAM POINT YET ! THIS IS WHAT HCG DOESSSSSSSSSSS YOU HUCKY MAN!

The highest dose of hCG (1000 mU/ml) increased turnover of IPs by 2.4-fold as reflected in elevations in IP(1) , IP(2) and IP(3) , and similar effects were observed with noradrenaline.

BY THE WAY FOR THE UNINFORMED IN THIS STUDY NORANDRENALINE IS A STEROIDS ! SUGGESTING IP MAN IS IN THE CLAUSE!

BWAAHAHAHAH ! SO HUCK WHERES YOUR PROFF ! NO MAD!
 
arnieswarzennega said:





Although the uterus is a target tissue for LH and its homologue hCG the second messenger system responding to LH/hCG in myometrial cells is not established. In this study we investigated the involvement of protein kinase A and protein kinase C in the action of hCG on porcine myometrial smooth muscle cells in vitro. Myometrium was obtained from ovariectomized gilts given 2.5 mg oestradiol benzoate plus 50 mg progesterone for five consecutive days. Myometrial cells were cultured for 48 h and different doses of hCG were then added. Increasing doses of hCG stimulated concentration-dependent increases in [(3) H]inositol phosphates (IPs) accumulation in incubations lasting 24 h. The highest dose of hCG (1000 mU/ml) increased turnover of IPs by 2.4-fold as reflected in elevations in IP(1) , IP(2) and IP(3) , and similar effects were observed with noradrenaline. The time- and concentration-dependent effects of hCG on IPs accumulation occurred between 16 and 24 h of incubation. Incubation of myocytes with the lowest doses of hCG (0.1 and 1 mU/ml) caused a significant increase in cAMP accumulation but the highest doses (10-1000 mU/ml) had no effect on cAMP concentrations. This is the first demonstration that LH/hCG receptor signalling leads to increased inositol phosphate turnover in myometrial cells as well as cAMP generation and it leads to the conclusion that both protein kinase A and protein kinase C signalling mechanisms are involved in gonadotrophin action in porcine myometrial smooth muscle cells.



BUDDY YOU GET THE DAM POINT YET ! THIS IS WHAT HCG DOESSSSSSSSSSS YOU HUCKY MAN!

The highest dose of hCG (1000 mU/ml) increased turnover of IPs by 2.4-fold as reflected in elevations in IP(1) , IP(2) and IP(3) , and similar effects were observed with noradrenaline.

Again, nice cut a paste :rolleyes:

M18
 
LMAO!!!Inositol phosphates?!?!?In the UTERUS?!?BWAAAAAHAHAHAHAHA!!!What on God's green earth does that have to do with WIDENING THE ANDROGEN RECEPTOR(as you stated)?As I said before bro,you're making your self look worse and worse with each consecutive 'cut and paste' and ignorant statement....Yawn
 
HUCKLEBERRY FINNaplex said:
LMAO!!!Inositol phosphates?!?!?In the UTERUS?!?BWAAAAAHAHAHAHAHA!!!What on God's green earth does that have to do with WIDENING THE ANDROGEN RECEPTOR(as you stated)?As I said before bro,you're making your self look worse and worse with each consecutive 'cut and paste' and ignorant statement....Yawn

Did somebody mention inositol?
 
GaryWary said:


Did somebody mention inositol?

He was somehow trying to connect the increase of inositol phosphates(IP's)in a pig's uterus with the 'widening' of A/R's within striated muscle fibers.Scary ignorance coming from someone who was telling other folks "not to argue with him because he had 10 years of vast knowledge to his credit."
 
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