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Step one is to review how the body regulates the production of testosterone.
When the brain detects low levels of testosterone, low levels of GnRH, low levels of LH, the brain, (hypothalamus) will release GnRH which will then stimulate the release of LH from the ant. pituitary gland in the sella tursica of the skull, and this LH will then stimulate the production of testosterone in the balls. [There are of course, several other ways the body regulates testosterone in the body, such as its production of TeBG - and one should consider them in their over all strategy]
The brain, (hypothalamus), also monitors estrogen and progesterone levels in body. High levels will inhibit the production of GnRH --> LH --> Testosterone. With this in mind, a good strategy would be to PREVENT the formation estrogens during HRT in the first place.
Now as you well know, Clomid works by 'competing' with estrogen at various receptors in the body. Although it won't prevent the 'formation' of estrogens, it will block allot of estrogen receptors, including those at the breast and brain (hypothalamus). With less estrogen able to reach the hypothalamus of the brain, (as clomid is blocking it), the brain responds by producing more GnRH, and then LH, and finally, testosterone.
Academically speaking, the brain (hypothalamus) doesn't begin to 'think' about recovery until after it detects low GnRH, LH, and Testosterone. Clomid will artificially 'fool' the hypothalamus into thinking 'everything is alright' --- And so one can argue that true recovery cannot begin until after the clomid is discontinued.
Realistically however, there are several good arguments to suggest that 'in the long run', clomid will attribute to a faster recovery -- At least by virtue of its ability to compete with post cycle estrogen accumulation! And clomid usage will stimulate hypophysiotrophic neurons in the hypothalamus of the brain to release GnRH along its peptidergic pathways leading to the pituitary gland....which in turn will release LH, and thereby stimulate the production of testosterone.
Clinically, timing is the key to the successful application of clomid in this strategy. There are many things that will affect the timing in such a strategy, none the least of which is that the continuous release of GnRH will ultimately suppress the production of testosterone!
Within hours of continuous GnRH release, the pituitary gland will begin to stop releasing LH -- And naturally the production of testosterone will stop as well. With the continuous release of GnRH, such as sponsored by clomid usage, you will see that by about the 5th day, plasma LH levels will fall well under 5 ng/ml.
On the other hand, the pulsatile release of GnRH would stimulate a consistent release of LH, and ultimately -- testosterone.
So you can see there are allot of things to consider in attempting to use clomid to achieve the constant release of LH, and testosterone -- Such as striving to stimulate the production of GnRH intermittently -- Like once every 90 minutes. But how can you do that considering the pharmacokinetics of clomid, (and nolvadex), as it takes about 5 days just to excrete half of the ingested dose!
Im going to end the story abruptly here, and give you time to consider this material. Besides, PT Barnum taught us that any PR is good PR! I want the scandal to continue bending the minds of many into deep thought!
h19
When the brain detects low levels of testosterone, low levels of GnRH, low levels of LH, the brain, (hypothalamus) will release GnRH which will then stimulate the release of LH from the ant. pituitary gland in the sella tursica of the skull, and this LH will then stimulate the production of testosterone in the balls. [There are of course, several other ways the body regulates testosterone in the body, such as its production of TeBG - and one should consider them in their over all strategy]
The brain, (hypothalamus), also monitors estrogen and progesterone levels in body. High levels will inhibit the production of GnRH --> LH --> Testosterone. With this in mind, a good strategy would be to PREVENT the formation estrogens during HRT in the first place.
Now as you well know, Clomid works by 'competing' with estrogen at various receptors in the body. Although it won't prevent the 'formation' of estrogens, it will block allot of estrogen receptors, including those at the breast and brain (hypothalamus). With less estrogen able to reach the hypothalamus of the brain, (as clomid is blocking it), the brain responds by producing more GnRH, and then LH, and finally, testosterone.
Academically speaking, the brain (hypothalamus) doesn't begin to 'think' about recovery until after it detects low GnRH, LH, and Testosterone. Clomid will artificially 'fool' the hypothalamus into thinking 'everything is alright' --- And so one can argue that true recovery cannot begin until after the clomid is discontinued.
Realistically however, there are several good arguments to suggest that 'in the long run', clomid will attribute to a faster recovery -- At least by virtue of its ability to compete with post cycle estrogen accumulation! And clomid usage will stimulate hypophysiotrophic neurons in the hypothalamus of the brain to release GnRH along its peptidergic pathways leading to the pituitary gland....which in turn will release LH, and thereby stimulate the production of testosterone.
Clinically, timing is the key to the successful application of clomid in this strategy. There are many things that will affect the timing in such a strategy, none the least of which is that the continuous release of GnRH will ultimately suppress the production of testosterone!
Within hours of continuous GnRH release, the pituitary gland will begin to stop releasing LH -- And naturally the production of testosterone will stop as well. With the continuous release of GnRH, such as sponsored by clomid usage, you will see that by about the 5th day, plasma LH levels will fall well under 5 ng/ml.
On the other hand, the pulsatile release of GnRH would stimulate a consistent release of LH, and ultimately -- testosterone.
So you can see there are allot of things to consider in attempting to use clomid to achieve the constant release of LH, and testosterone -- Such as striving to stimulate the production of GnRH intermittently -- Like once every 90 minutes. But how can you do that considering the pharmacokinetics of clomid, (and nolvadex), as it takes about 5 days just to excrete half of the ingested dose!
Im going to end the story abruptly here, and give you time to consider this material. Besides, PT Barnum taught us that any PR is good PR! I want the scandal to continue bending the minds of many into deep thought!
h19
