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bicepts101
New member
Oiltanker said:
from reports of users..masteron seems to dry you out better and without the joint pains of winny. winny is horrible for lipids and masteron is not. winny is horrible for the liver. masteron is not. anotherr plus of masteron is the injections dont leave you limping like a retard all cycle. masteron seems to give better strengh gains cause of the higher aggresion it may bring you. (im not exaclty sure on that one)....ive also heard that winstrol has some fat burning effects. ill dig up something on that.
FONZ....where do u stand on the theories that winstrol accually raises body temp, helping to burn fat? ive never heard you speak of it...
bicepts101 said:
Winstrol doesn't raise body temperature. No AAS raises body temperature. They burn fat through other means like PGF2A increases(Tren). AAS also burn fat through enhanced glucose clearance, therfore lowering insulin levels and leading to more FFA's to be burned for fuel. Like Anadrol and Nandralone.
What winstrol does(And what it was manufactured for) was to reduce edema in dogs. Edema = accumulation of water. Therefore, winstrol acts as a sort off water shedder. It'll dry you out.
bicepts101
New member
Fonz said:
ok bro...heres what shredded has to say:
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First off, I am mystified about the skepticism surrounding the assertion that winstrol is pyrogenic. Pyrogenic simply means 'fever producing', and it is a well known side effect of this steroid, even listed in common medical journals as a side effect. Keep in mind that this occurs at doses as low as 2mg/day. I think Nigel Verger, in one of his articles, provides a table from a medical journal that lists this and other effects. Anyway...
The primary use of stanozolol in humans is to treat hereditary angioedema. This condition causes swelling of the face, hands, feet, neck, throat, and other areas, as a result of an improper distribution of water in the organism, not connected (necessarily) to sodium levels. Ok, so this drug reduces the swelling, again, in doses as low as 2mg/day.
You see where I am going with this, I am sure. We can conclude from the very use of this drug that it has a water-redistributing effect on the organism. In fact, anyone who has used it for any period of time can attest to the effects resulting from internal dehydration, such as constipation, cramps, headaches, dry mouth, blurred vision, etc. Now, this is not to say that the subcutaneous water retention is not possible on this drug, because that particular effect is a seperate condition from the effect of water redistribution around the organs and musculature. I will leave the differential explanation to someone more knowledgable, but I guarantee that winstrol will remove water from specific areas, including many organs. This is dangerous if prolonged indefinitely.
About this tendon issue...I think I have that solved as well. Consider the recent study (I will get it) that showed that winstrol increased collagen synthesis in small doses, and that the increase seems to be dose related. Well, let's think about that. If the receptors are not evenly distributed throughout the tendon, then the accelerated growth would not occur evenly. Rather, it would occur more rapidly directly around the areas that were more densely populated with receptors, which could conceivably result in a nodule of growth in the tendon. This would result in a tendon that is not uniform in its tensile strength, and would be susceptible to rupture at the area directly above or below the nodule. This is speculation, but it is a theory that makes sense to me.
Lastly. Winstrol has a unique anticatabolic action. For a great description of this, you should read Dr. Scruggs little paragraph about stanozolol. I will try and find it. Anyway, it turns out that winstrol has little or no effect on GH and IGF-1 levels, and therefore does not effectively recruit and impact sattelite cells. So the anabolic of effect of winstrol is minimal, at best. So why do we get stronger? Well, it does increase nitrogen retention, and we know how important that is for muscle growth. But, more importantly, it preserves the muslce we already have, meaning that any new growth will not be 'cancelled out' by muscle degeneration. Testoserone is superior in terms of being anabolic/androgenic, but testosterone also creates protien degradation at a much more accelerated rate than does winstrol. So, yoiu don't build as much muscle, but you keep what you get. Also, winstrol increases bone density. Now, it is interesting to note that winstrol is second only to trenbelone in terms of its anticatabolic property, and in fact, I think it is superior on a mg for mg basis, but I cannot yet support this claim with the appropriate science. Call it a hunch for now. Of course, my hypothesis that winstrol acted on a unique receptor found in the skin was a hunch for about a year, and later turned out to be proven in a study that was posted on AF, and that receptor is as of yet unidentified. Lets just say I had my reasons. Moving on...
Here is what we are left with. A superior anticatabolic compound that increases body temperature. Well, shit! What more do you need for cutting? Moreover, our strength is greatly enhanced because we are able to build on top of the matured muscle rather than burn it up. Ok. This little overview might help to clarify some of the statements that were, unfortunately, clumsily written in the initial post.
Koivu_11 said:
No. Changing the first functional group from 2-alpha-Methyl(Masteron) and reducing it to 1-alpha-methyl, changes the anabolic activity of the compound considerably. The androgenicity stays the same, but the anabolic variant goes way down. As you already know proviron is an strong androgen but a poor anabolic.
i.e. Masteron can be reduced to Proviron at the expense of its anabolic properties.
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