CB chimed in with this when I said it was nonsense "50% better"
Well i AM taking sides. Smalls is right and any idiot with minimal amout of IQ should be able to figure that out. Its simple..our body preceives steroids as TOXINS that must be secreted out of our body...just like any other drug. Liver is the the organ responsible for excreting toxins out of our body. Now...anyone doubt that?? Another fact you should know? when u drink something it must pass thru your liver first before it will enter ur blood stream (excluding fat, which will get into blood via lymphatic system). Another fact, winny is WATER BASED, not oil.
So when you drink winny...it must pass thru liver BEFORE it gets into your blood stream and out to muscles. Yes i know it is a 17-AA drug, however, 17AA does not mean that your liver will NOT excrete it...its just makes it harder, so while your liver is excreting some of the drug out of ur body, some will get thru. IF liver was not able to excrete 17AA drugs out of our body then we would not need to take drugs over and over again. If you can comprahend the fact that liver excretes drugs out of body it won't take a genius to figure out that to get the most out of the drug, you must minimize the times it gets thru to ur liver.
Now, when winny is injected, it stays in your muscle until it gets carried away with blood flow..from where it goes around your body with the blood until the blood goes thru the liver again. Thus this way your muscles get the first exposure to ALL of the drug injected, rather than say 50% of the drug taken orally.
This is the study that smalls posted for his theory
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Anyway, until very recently, I thought, that there's no difference between oral and injectable form of Stanozolol's administration.
Well, the study, I found really brought some confusion into this matter. Unfortunatelly, it doesn't explain why, it's just states the facts.
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The effect of stanozolol on 15nitrogen retention in the dog.
Can J Vet Res 2000 Oct;64(4):246-8 (ISSN: 0830-9000)
Olson ME; Morck DW; Quinn KB [Find other articles with these Authors]
Animal Health Unit and Gastrointestinal Sciences, University of Calgary, Alberta.
[email protected].
The objective of the study was to determine the influence of either oral or intramuscular administration of stanozolol on nitrogen retention in dogs by using a non-invasive 15N-amino acid tracer technique. Ten healthy, intact, adult male sled dogs received either stanozolol tablets, 2 mg/dog PO, q12h, for 25 days (Group 1, n = 5) or an intramuscular injection of 25 mg of stanozolol on Days 7, 14, 21, and 28 (Group 2, n = 5). A 15N amino acid (5.27 mmol) was infused intravenously into each dog on Day 0 (before stanozolol treatment) and on Day 31 (after stanozolol treatment). Urine was collected by catheterization from each animal 3 times daily for 3 consecutive days. The 15N-urea enrichment in urine was determined by high-resolution mass spectrometry and the total amount of urea in the urine was determined. Both oral and injectable stanozolol resulted in significant (P < 0.05) increases in amino acid nitrogen retention compared to pretreatment values. Oral stanozolol increased nitrogen retention from 29.2 +/- 8.2% to 50.3 +/- 9.2%, while stanozolol injection increased nitrogen retention from 26.6 +/- 9.9% to 67.0 +/- 7.5%. The response to intramuscular administration was significantly greater than the response to the oral dosing regime. Stanozolol increases amino acid nitrogen retention in dogs, as has been previously observed in rats. This action of stanozolol may be beneficial in dogs under stress of surgical trauma and chronic disease.
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Oral dosage was 28mg/week
Injections - 25mg/week
Effectiveness of injections over oral was more then 50%
Dogs were healthy, male and very fit. Dogs metabolism is very close to one in humans, unlike rats, were exceptions sometimes happens.
Of course, it's not the end, and we still should be looking for answers, but I, personally, found this study extremely interesting.
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