Information i found 1-AD vs. 1-TU

[WannaBPowerful]

1-Tu l 1-AD

Converts to 1-Test l Converts to 1-Test
doesnt aromatize l Doesnt aromatize
DHT sides unknown l Very possible DHT
Bypasses liver completly l Doesnt bypass liver
Puts on lean mass l Puts on lean mass
Longer half life than any l About 2-4 hour half life
prohormone
50mg pills, 60/bottle l 100mg pills, 60/bottle
6 weeks on 2 off l 6 weeks on 4 off

Thats the things i felt most important. From the info i found, 1-TU LOOKS to be the better product but its new so theres not too much feedback yet. We will have to see.

 

[Testoman]

i am not a prohormone expert but 1-TU is actual 1-test, as are many other 1-testosterone products whereas 1-AD is a precursor to 1-test.

Testoman

 

[Flash_75]

How does it bypass the liver? LOL!!

That is some serious hype. maybe it passes through the liver unchanged(during the first pass), but NOTHING gets past the liver.

 

[SlapItHard]

Anything can look good on paper, it's all about what works for you. Give both a shot, see which works better. Newer better products will always be coming out.

 

[pa1ad]

1-Tu l 1-AD

DHT sides unknown l Very possible DHT
Bypasses liver completly l Doesnt bypass liver
Puts on lean mass l Puts on lean mass
Longer half life than any l About 2-4 hour half life
prohormone
50mg pills, 60/bottle l 100mg pills, 60/bottle
6 weeks on 2 off l 6 weeks on 4 off

Thats the things i felt most important. From the info i found, 1-TU LOOKS to be the better product but its new so theres not too much feedback yet. We will have to see.

Some real bullshit here. I suggest you ask them to substantiate their claims with FACTS, rather than being a gullible sucker with blind faith

Some facts:

1) All 1-test products and precursors WILL convert to DHT and metabolites.

2) If the small amount of 1-TU that enters the lymphatic system (and only a minor fraction of these sort of undecanoate esters absorb lymphatically) then where is the rest going? If he is right, and it completely bypasses the liver then he must be saying that you poop out the majority of your 1-TU dose

3) What IS the half life of his product? What does he make this claim based on?

4) ACtually, there is only 32.4 mg of 1-testosterone in the 1-TU product. The rest of the 50mg is undecanoic acid (a worthless fatty acid). OTOH, 1-AD contains 100mg of 1-androstenediol (bound to nothing)

5) What does he base these cycle recommendations on? Give us a break

 

[curt2go]

POOP it out. LOL...LOL

 

[pa1ad]

Re: Re: Information i found 1-AD vs. 1-TU

Some real bullshit here. I suggest you ask them to substantiate their claims with FACTS, rather than being a gullible sucker with blind faith

Some facts:

1) All 1-test products and precursors WILL convert to DHT and metabolites.

2) If the small amount of 1-TU that enters the lymphatic system (and only a minor fraction of these sort of undecanoate esters absorb lymphatically) then where is the rest going? If he is right, and it completely bypasses the liver then he must be saying that you poop out the majority of your 1-TU dose

3) What IS the half life of his product? What does he make this claim based on?

4) ACtually, there is only 32.4 mg of 1-testosterone in the 1-TU product. The rest of the 50mg is undecanoic acid (a worthless fatty acid). OTOH, 1-AD contains 100mg of 1-androstenediol (bound to nothing)

5) What does he base these cycle recommendations on? Give us a break

To whom it may concern,

I did not have any problem with 1-TU product until this bozo decided to trash 1-AD as a means to promote the 1-TU

PA don't like those sort of shitty little games. PA bites back y'know.

 

[Sigmund Roid]

PA1AD, I have a question for you.

You claim that the double bond in the 1-2 carbon position is increasing the survivability of 1-AD. You base this on methenolone. Yes, methenolone is orally effective to some degree, but this is according to me only because of the 1alfa methylation, NOT the double bond.

And, although not entirely comparable, boldenone is NOT orally effective, which also has a 1-2 double bond.

Mesterolone is also orally effective, which proves even more that the 1a-methyl group is responsible for that.

 

[pa1ad]

PA1AD, I have a question for you.

You claim that the double bond in the 1-2 carbon position is increasing the survivability of 1-AD. You base this on methenolone. Yes, methenolone is orally effective to some degree, but this is according to me only because of the 1alfa methylation, NOT the double bond.

And, although not entirely comparable, boldenone is NOT orally effective, which also has a 1-2 double bond.

Mesterolone is also orally effective, which proves even more that the 1a-methyl group is responsible for that.

You are incorrect. Both the 1methyl and the 1,2 double bond contribute to shifting the 17beta OH equilibrium. You obviously never looked up the literature that i referenced. You really should make it a practice to actually read the literature before drawing your conclusions.


Galletti and Gardi, “Metabolism of 1-Dehydroandrostanes in Man”, J Steroid Biochem, 3 (1972), 933-936

Langecker, “Beziehungen Zwischen Substitution im Ring A und Abbau im Stoffwechsel bei Verwandten des Testosterons”, Acta Endocrin, 41 (1962), 494-506



If you want to discuss this further than read the aforementioned articles and get back to me. The second one is in German, however it has an abstract in english

 

[WannaBPowerful]

OK, so whats your conclusion on the 1-TU? Could it produce lean and solid mass? Will it cause side effects or water retention? Is it just plain crap?