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hair loss stack?

kiwee

Banned
ive started to thin a little on top so im now implementing this regimine, please critique, gd bad or otherwise, all and any advice is appreciated...btw i do not use aas, so thinning isnt due to that...

finasteride ED

nioxin/nizoral EOD rotation

rogaine upon waking and bedtime

saw palmetto (DHT inhibition)

green tea, carnitine, biotin (hair regrowth)

dostinex EOD (inhibit prolactin from GHB usage, and orgasm...prolactin causes hair loss)

herbal supp from dermatologist (supposedly very similar to propecia as per dr recomendation...when a dr tells u not to use propecia and sells you an herbal supp to replace the propecia, which means he loses money, its most likely a gd recommendation)

nolvadex ED (i use this since, supposedly, for men who are natural, it can provide mild anabolic effects...i have so much i figured id give it a go)

can you guys think of anythin i can add to this regimine? and what are your thoughts of it as it currently is...thanks much in advance!
 
almost forgot, im going to add toco8 and growth hormone to this as well...supposedly 3iu gh ed can have a mild regrowth effect on hair
 
I've stated before that this thinning and eventual baldness can be stalled a bit, but not stopped. Some guys and women get it, some don't. I got it a lot.

Rogain will grow some fuzz and you can color it to make it thicker.

Fin is powerful. Read the sides.

I think you'll slow it down a lot and would just add the color comment.
 
Looks like a good stack.

Why Nolvadex? For increasing test levels? Nolvadex can cause hairloss. It can also have a re-bound effect once you stop it. Do you need Nolvadex or any SERM (Selective Estrogen Receptor Modulator)? What does your blood test show your total and free testosterone levels?

Over use of Nolvadex can possibly reduce IGF (Insulin-like-Growth-Factor) levels, which are important for muscle growth.

I just don't see the need for Nolvadex other then for PCT or gyno related issues.
 
Because if you inhibit de DHT conversion, you're "killing" the effects of the steroids.

If you use 5 mg you inhibit more. If you use 1 mg you inhibit less. And directly the % of DHT that you inhibit are the % of steroid effect that you're losing. If you want use it... use.

Read about 5-alpha, and about DHT (3x more potent than test) and you see that.
 
Finasteride (marketed as Proscar, Propecia, Fincar, Finpecia, Finax, Finast, Finara, Finalo, Prosteride, Gefina, Appecia, Finasterid IVAX, Finasterid Alternova) is a synthetic antiandrogen which acts by inhibiting type II 5-alpha reductase, the enzyme that converts testosterone to dihydrotestosterone (DHT).

Dihydrotestosterone (DHT) Full name: 5α-Dihydrotestosterone, abbreviating to 5α-DHT; is a biologically active metabolite of the hormonetestosterone , formed primarily in the prostate gland, testes, hair follicles, and adrenal glands by the enzyme 5α-reductase by means of reducing the 4,5 double-bond. Dihydrotestosterone belongs to the class of compounds called androgens, also commonly called androgenic hormones or testoids. Androgens are part of the biology of gender by stimulating and controlling the development and maintenance of masculine characteristics. DHT is 3 times more potent than testosterone; testosterone is 5-10 times more potent than adrenal androgens.

If you inhibit DHT...

PS: can you link me the BigCat post about use finas?
 
I'd think that theoretically any DHT blocker would negatively impact strength gains more than hypertrophy gains. Would seem to hurt the intended effects of an androgenic stack substantially more than a primarly anabolic stack.
 
im not using any anabolics, the 5mg dose is good for when your on, but since im not on, i lowered the dose to approx 1mg ED, which, essentially will decrease dht with minor sides
 
I'd think that theoretically any DHT blocker would negatively impact strength gains more than hypertrophy gains. Would seem to hurt the intended effects of an androgenic stack substantially more than a primarly anabolic stack.

You think that using finasteride (inhibitin only type I) the gains (hypertrophy) are the same or similar?
 
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