GD and other brainiacs...

[halfaclue]

I was reading an article about how your body releases certain hormones that can be converted to test or estrogen depending on the recptors (or smething like that). My question...you take anti-e's when on to avoid the extra estrogen build up...what about taking anti-e's all year round? Wouldn't this keep your natural production of estrogen lower and thus make it easier to stay leaner???

 

[Goodfellow]

I dont believe so because the endocrine system is based on a a loop. Your body would sence a lower levels of estrogen and in turn produce less testosterone in an effect to ballance it out. At least thats what I think anyway....

 

[thx9000]

I was reading an article about how your body releases certain hormones that can be converted to test or estrogen depending on the recptors (or smething like that). My question...you take anti-e's when on to avoid the extra estrogen build up...what about taking anti-e's all year round? Wouldn't this keep your natural production of estrogen lower and thus make it easier to stay leaner???

Not all fat is estrogenic and low estrogen levels is not, at all, a healty thing.

 

[halfaclue]

OK..it seemed like a good idea in my head but that is why I am not in the science fields

 

[DIVISION]

I was reading an article about how your body releases certain hormones that can be converted to test or estrogen depending on the recptors (or smething like that). My question...you take anti-e's when on to avoid the extra estrogen build up...what about taking anti-e's all year round? Wouldn't this keep your natural production of estrogen lower and thus make it easier to stay leaner???

Depends on what anti-estrogen you are taking year round. Is it an aromatase inhibitor like Arimidex or Femara or is it one that just blocks estrogen receptor sites like Nolvadex?

All anti-estrogens are not the same.

 

[halfaclue]

Depends on what anti-estrogen you are taking year round. Is it an aromatase inhibitor like Arimidex or Femara or is it one that just blocks estrogen receptor sites like Nolvadex?

All anti-estrogens are not the same.

I was thinking of nolvadex.

 

[GoldenDelicious]

brainiac. thats a new one.

anyway what you have to think of is whether or not this supposed increase in estrogens (if it occurs at all) is problematic. hormones are funny molecules...as soon as you think you know what they do, someone publishes a new study and you start scratching your head again and so there is an excellent chance that using an anti estrogen (ill lump them together for ease) will interrupt or unbalance some perfectly normal process, and give you sides which are totally disproportionate to the (questionable, if any) benefit of having low estrogen

i mean, it wasnt too long ago that we thought estrogens didnt do terribly much in the male adult, and then a few studies were published showing that estrogen was essential for libido

thinking of it as a bodybuilder, perhaps another thing to think about is what using various anti estrogens is going to do to your balls- initially there will be some stimulation, and an increase in testosterone etc, but after a while chances are there will be desensitisation- which sucks because your PCT options are completely blown away (what do you think youre going to use post cycle? tribulis? lol) and also, youre going to start upsetting your hpta

i mean, bottom line is that i think that there will be hardly any benefit estrogenically, if any at all, and then youre opening yourself up to a host of far worse events due to the drug use

for a more complete answer id have to see the info youre going off, but the trouble with those sorts of articles is that theyre not wholistic enough. thats the trouble with raw biochemistry, you get some biochemist getting up and saying blahblah enzyme increases blahblah substrate by 300%!!!!! therefore, its implicated in blahblahblah!!!! then some idiot marketer jumps on it and starts selling it with all these fabulous claims...but no one mentioned that blahblah enzyme is one of hundreds involved in the same cascade, and its significance is totally, totally overstated. imo of course.

 

[halfaclue]

The article was about aging men in their 30's fighting off fat gain and how your test levels are much higher vs estrogen when your 18 vs your test levels being lower and estrogen being higher (not higher then test but higher then when your 18) and thus allowing the fat to start building up. Thanks for the info though.

 

[juve]

The estrogen is not the problem
The problem is that 98-99% of all testosterone is bound to SHBG - inhibit SHBG and your body should adjust estrogenic response accordingly not to mention liberating plasma free test, but eliminating estrogen or precluding its conversion will wreack havoc on your endocrine, cardiovascular systems as well as nervous systems (estrogen is linked to many feedback mechanisms regulating neurotransmitter regulation and receptor accumulation).

 

[einstein1905]

The estrogen is not the problem
The problem is that 98-99% of all testosterone is bound to SHBG - inhibit SHBG and your body should adjust estrogenic response accordingly not to mention liberating plasma free test, but eliminating estrogen or precluding its conversion will wreack havoc on your endocrine, cardiovascular systems as well as nervous systems (estrogen is linked to many feedback mechanisms regulating neurotransmitter regulation and receptor accumulation).

The problem is that estrogen is very anabolic, but estrogen also increases SHBG. Androgens, on the other hand, reduce SHBG, thereby increasing the % of bioavailable AAS (or just test and DHT if you're natural). It's difficult to say which is the lesser of two evils; the higher estrogen at the expense of elevated SHBG, or the reduced estrogen but higher bioavailable androgens.
The anabolic properties of estrogen have been too far underplayed in the past IMO