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Deca cycle
- Thread starter Magick69
- Start date
juicer99
New member
Try a low dose test, like 200mg/wk and see if that hurts the hairline. If not you could keep upping it until about 500mg/wk. I would run no less than 300mg of NPP/wk imo, 300-600 seems to be the standard for deca. I'm not that well versed on NPP though so someone else may be able to help you better in that department.
Magick69
New member
ok lets give new impulse to the thread; still i am thinking and unsure between deca only cycle or equipose (10 weeks -400 mg) only cycle for the hairloss issue.....
so today i found this
===================================================
"Keep More Hair AAS, but HPTA Function is Nice Too Protocol
by Author L. Rea
As most are aware, many AAS (Anabolic-Androgenic Steroids) aromatize or convert to estrogens to some degree and estrogens shut down male androgen production by way of the HPTA. HPTA refers to the Hypothalamus-Pituitary-Testes-Axis, which is our body’s androgen regulatory system.
The use of nandrolone decanoate alone can shut-down HPTA (Hypothalamus-Pituitary-Testes Axis) function even though it aromatizes at a reasonably low rate. It simply requires a longer time of activity in the system to do so when compared to most other aromatizing AAS.
Interesting is that after a maximum of 28 days of nandrolone decanoate administration, at a dosage of 200-400mg/w, there is about a 40-50% average decrease in HPTA activity and its markers (FSH, LH and LHRH). But when testosterone cypionate is employed alone, 28 days of administration results in near total HPTA shut-down.
The inclusion of methandrostenolone in an AAS protocol, regardless of the daily administration pattern, has severe HPTA suppressive potential. Alone and administered once daily does still result in a slightly limited degree of HPTA inhibition...though still significant.
So the issue becomes what drug has been effectively used as the base and could another be added. When it comes to hair loss some men feel that, though natural, it is near comparable to castration, while others seem to care not at all.
Each of us carries a genetic encoding for hair loss. Our hormone profiles dictate the rate and degree of loss. The three primary hormones having the greatest effect upon hair loss are DHT, estrogen and IGF-1. Each plays a role and must act in symphony to trigger an increased rate of drain clogging hair suicide.
When DHT is blocked, but estrogen and IGF-1 are present, hair expenditure is slowed...but not stopped. If IGF-1 is blocked, or estrogen, the same is true again.
*An interesting fact is that I have noted that many who use 500mg of Cytadren and 20mg of Tamoxifen daily in divided doses tend to retain hair better than those who opt for other off-cycle estrogen/cortisol suppression protocols. The answer may be in that Tamoxifen inhibits IGF-1 activity while it blocks estrogen receptors and Cytadren inhibits cortisol formation and acts as an estrogen biosynthesis inhibitor. The key may be additional control of stress hormones as well as one of the evil three.
So the basis for a reasonably successful "Keep More Hair AAS, but HPTA Function is Nice Too Protocol" has been a 28 day structure alternating nandrolone decanoate and methenolone enanthate.
The intent being to decrease DHT and estrogen activity while exiting at a point of reasonable HPTA function.
The aromatization product of nandrolone is nor-17b-estradiol, which is much weaker than the aromatization by-product of testosterone (17b-estradiol). This means less of an HPTA negative feed-back loop and less negative hair follicle activity.
Methenolone is a DHT derivative but the drug itself is less active upon hair follicles than DHT itself. This oddly enough has a "short term" anti-DHT effect. Since DHT derivatives are not affected by the 5-alpha-reductase or aromatase enzymes there is no resulting increase in DHT itself or estrogen.
Alternating these drugs appears to allow for the best of both worlds with a decrease in potential hair loss. The addition of 12.5-25mg 2xd of Oxandrolone has also been noted to result in some very high quality lean tissue growth.
Warning: This is an example intended for discussion purposes only. The use of any drug must be under the guidance of a licensed health care professional only.
"Keep More Hair AAS, but HPTA Function is Nice Too Protocol Example"
Day
1. Nandrolone Decanoate 400mg
2.
3.
4.
5.
6.
7.
8. Methenolone Enanthate 200mg
9.
10.
11.
12. Methenolone Enanthate 200mg
13.
14.
15.
16. Nandrolone Decanoate 400mg
17.
18.
19.
20.
21.
22.
23. Methenolone Enanthate 200mg
24.
25.
26.
27. Methenolone Enanthate 200mg
28.
*Any of the many HPTA stimulation protocols I have written in the past would layer well over this example…obviously.
=========================================================
now my question is if to primobolan i substitute anavar or turinabol will have the same effect of Rea's cycle ?
so today i found this
===================================================
"Keep More Hair AAS, but HPTA Function is Nice Too Protocol
by Author L. Rea
As most are aware, many AAS (Anabolic-Androgenic Steroids) aromatize or convert to estrogens to some degree and estrogens shut down male androgen production by way of the HPTA. HPTA refers to the Hypothalamus-Pituitary-Testes-Axis, which is our body’s androgen regulatory system.
The use of nandrolone decanoate alone can shut-down HPTA (Hypothalamus-Pituitary-Testes Axis) function even though it aromatizes at a reasonably low rate. It simply requires a longer time of activity in the system to do so when compared to most other aromatizing AAS.
Interesting is that after a maximum of 28 days of nandrolone decanoate administration, at a dosage of 200-400mg/w, there is about a 40-50% average decrease in HPTA activity and its markers (FSH, LH and LHRH). But when testosterone cypionate is employed alone, 28 days of administration results in near total HPTA shut-down.
The inclusion of methandrostenolone in an AAS protocol, regardless of the daily administration pattern, has severe HPTA suppressive potential. Alone and administered once daily does still result in a slightly limited degree of HPTA inhibition...though still significant.
So the issue becomes what drug has been effectively used as the base and could another be added. When it comes to hair loss some men feel that, though natural, it is near comparable to castration, while others seem to care not at all.
Each of us carries a genetic encoding for hair loss. Our hormone profiles dictate the rate and degree of loss. The three primary hormones having the greatest effect upon hair loss are DHT, estrogen and IGF-1. Each plays a role and must act in symphony to trigger an increased rate of drain clogging hair suicide.
When DHT is blocked, but estrogen and IGF-1 are present, hair expenditure is slowed...but not stopped. If IGF-1 is blocked, or estrogen, the same is true again.
*An interesting fact is that I have noted that many who use 500mg of Cytadren and 20mg of Tamoxifen daily in divided doses tend to retain hair better than those who opt for other off-cycle estrogen/cortisol suppression protocols. The answer may be in that Tamoxifen inhibits IGF-1 activity while it blocks estrogen receptors and Cytadren inhibits cortisol formation and acts as an estrogen biosynthesis inhibitor. The key may be additional control of stress hormones as well as one of the evil three.
So the basis for a reasonably successful "Keep More Hair AAS, but HPTA Function is Nice Too Protocol" has been a 28 day structure alternating nandrolone decanoate and methenolone enanthate.
The intent being to decrease DHT and estrogen activity while exiting at a point of reasonable HPTA function.
The aromatization product of nandrolone is nor-17b-estradiol, which is much weaker than the aromatization by-product of testosterone (17b-estradiol). This means less of an HPTA negative feed-back loop and less negative hair follicle activity.
Methenolone is a DHT derivative but the drug itself is less active upon hair follicles than DHT itself. This oddly enough has a "short term" anti-DHT effect. Since DHT derivatives are not affected by the 5-alpha-reductase or aromatase enzymes there is no resulting increase in DHT itself or estrogen.
Alternating these drugs appears to allow for the best of both worlds with a decrease in potential hair loss. The addition of 12.5-25mg 2xd of Oxandrolone has also been noted to result in some very high quality lean tissue growth.
Warning: This is an example intended for discussion purposes only. The use of any drug must be under the guidance of a licensed health care professional only.
"Keep More Hair AAS, but HPTA Function is Nice Too Protocol Example"
Day
1. Nandrolone Decanoate 400mg
2.
3.
4.
5.
6.
7.
8. Methenolone Enanthate 200mg
9.
10.
11.
12. Methenolone Enanthate 200mg
13.
14.
15.
16. Nandrolone Decanoate 400mg
17.
18.
19.
20.
21.
22.
23. Methenolone Enanthate 200mg
24.
25.
26.
27. Methenolone Enanthate 200mg
28.
*Any of the many HPTA stimulation protocols I have written in the past would layer well over this example…obviously.
=========================================================
now my question is if to primobolan i substitute anavar or turinabol will have the same effect of Rea's cycle ?
