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DBol, anyone else
- Thread starter a creed
- Start date
CO B-man
New member
I never really thought about it but when I first started a cycle I could never understand why I could sleep so well at the beginning and not the end while on. I only noticed this because I think I was born an insomniac. I have drastically improved that (at least 65% with diet and excersize but not completely). I always use D-bol to kick off a bulker. Hmm. I am curious as to why now too.
needtogetaas
New member
its bin so long.
needtogetaas
New member
np any time good buddy.a creed said:thanks for the input bro............
macrophage69alpha
New member
1: Life Sci. 1996;58(7):573-83. Links
Chronic anabolic-androgenic steroid treatment affects brain GABA(A) receptor-gated chloride ion transport.Bitran D, Hilvers RJ, Frye CA, Erskine MS.
Department of Psychology, College of the Holy Cross, Worcester, MA 01610, USA.
Previous research in this laboratory has shown that chronic treatment of adult male rats with an anabolic-androgenic steroid (AAS) produced anxiolytic behavior and increased the functional response of cortical gamma-aminobutyric acid(A) (GABA(A)) receptors. The experiments reported here were aimed at further characterizing the effect of chronic AAS exposure on cerebral cortical GABA(A) receptors. Adult male rats were injected with dianabol (1,4-androstadien-17alpha-methyl-17beta-ol-3-one; 10 mg/kg/day, SC) for 4 weeks. A significant decrease in ventral prostate gland weight was found after 2 weeks of dianabol, and returned to control levels 3 and 10 days after steroid discontinuation. Testicular weights decreased throughout the treatment period but reached statistical significance only during the withdrawal period. Serum 3alpha-androstanediol level was marginally increased afer 2 weeks of dianabol injection, and was significantly decreased at 3 and 10 days after withdrawal. GABA-stimulated 36chloride (Cl-) influx in cortical synaptoneurosomes was increased in animals treated with dianabol for 2 and 4 weeks, and remained elevated 3 days after dianabol withdrawal, returning to control levels at withdrawal day 10. The increase in receptor efficacy was associated with a transient increase in receptor sensitivity (inverse of EC50), apparent after 2 weeks of AAS treatment and at withdrawal day 3. In a follow-up experiment, metabolites of dianabol were tested for the in vitro efficacy in potentiating GABA-stimulated Cl- transport. Only 3alpha-androstanedial and androsterone were found to have potent stimulatory effects. The 3beta-reduced metabolites were inactive, as were metabolites that contained a methyl group at the 17alpha position. These results point to significant facilitative effects of dianabol treatment on brain GABA(A) receptors via the metabolic formation of neuroactive steroids.
Chronic anabolic-androgenic steroid treatment affects brain GABA(A) receptor-gated chloride ion transport.Bitran D, Hilvers RJ, Frye CA, Erskine MS.
Department of Psychology, College of the Holy Cross, Worcester, MA 01610, USA.
Previous research in this laboratory has shown that chronic treatment of adult male rats with an anabolic-androgenic steroid (AAS) produced anxiolytic behavior and increased the functional response of cortical gamma-aminobutyric acid(A) (GABA(A)) receptors. The experiments reported here were aimed at further characterizing the effect of chronic AAS exposure on cerebral cortical GABA(A) receptors. Adult male rats were injected with dianabol (1,4-androstadien-17alpha-methyl-17beta-ol-3-one; 10 mg/kg/day, SC) for 4 weeks. A significant decrease in ventral prostate gland weight was found after 2 weeks of dianabol, and returned to control levels 3 and 10 days after steroid discontinuation. Testicular weights decreased throughout the treatment period but reached statistical significance only during the withdrawal period. Serum 3alpha-androstanediol level was marginally increased afer 2 weeks of dianabol injection, and was significantly decreased at 3 and 10 days after withdrawal. GABA-stimulated 36chloride (Cl-) influx in cortical synaptoneurosomes was increased in animals treated with dianabol for 2 and 4 weeks, and remained elevated 3 days after dianabol withdrawal, returning to control levels at withdrawal day 10. The increase in receptor efficacy was associated with a transient increase in receptor sensitivity (inverse of EC50), apparent after 2 weeks of AAS treatment and at withdrawal day 3. In a follow-up experiment, metabolites of dianabol were tested for the in vitro efficacy in potentiating GABA-stimulated Cl- transport. Only 3alpha-androstanedial and androsterone were found to have potent stimulatory effects. The 3beta-reduced metabolites were inactive, as were metabolites that contained a methyl group at the 17alpha position. These results point to significant facilitative effects of dianabol treatment on brain GABA(A) receptors via the metabolic formation of neuroactive steroids.
macrophage69alpha
New member
basically dianabol upregulates GABA transmission, it is anxiolytic. though assuredly as with most things you will find people with the opposite reaction.
macrophage69alpha
New member
that is not surprising...
A
ALIN
Guest
a creed said:
I never have aproblem sleeping anymore. DBOL always gives me a energetic feeling.
bgdaddy123
New member
I just started my dbol yesterday, so I will see if I sleep any better> I doubt it I have a 6 month old 
BOOEY
Banned
ooh how i love dbol. im running it 8 weeks this time around (currently in week 5) @ 80 mgs/day (currently stacking with EQ and Test). i get really really tired throughout the day when im on dbol. and come to think of it, i do sleep pretty well although i do have to get up in the middle of the night to piss (probably cus im drinking a gallon of water/day). yes, i love dbol.
