wgarrett said:
No medication other than 50mg t3 daily and 160mcg clen 2weeks on/off, those are medications I guess. Should I continue my cycle and just add the proviron and HCG, or come off completly and start pct now. thanks for all your help.
Your estrogen and SHBG is high no doubt.You used cyp which wont show diminishing estrogen despite cessatation of use for at least 4 wks.
Your ultimate solution would be a combonation of HCG,aromasin and proviron, and dostinex or other dopamine agonists ie.selegeline.
Regardless of no evidence of increased prolactin levels DA's, particularly dostinex will decrease ejaculatory latencies,serotonin will increase them.
I agree with ulter re:dht for ejaculation as well as freeing up your testosterone and slightly decreasing your circulating estrogen.
The regulation of male sexual behavior by the sex hormones
[Article in Russian]
Gladkova AI.
UkrNII of Pharmacotherapy of Endocrine Diseases, Kharkov.
The article provides a review of the clinical and experimental data, both published and the author's own observations. They demonstrate that the hormone regulation of sex behaviour takes place at the central and the peripheral levels. The major role is played by androgenes, of which testosterone is the main regulator of sex appeal (libido, courtship behaviour in animals),
while the most important regulator of ejaculation, in particular, of the time of ejaculation, is the non-aromatized androgene dihydrotestosterone (DHT). There is no rigid correlation between the level of sex activity and the level of androgenes, if the latter are within the normal individual and physiological range. However, both in case of hypo- and hyperendrogeny, the observations indicate depression of sexuality, though the mechanism of the depression are different. The author established the optimal levels of androgenes, which maintain the individual elements of the sexual act. The role of estrogene in regulation of the male sex appeal is not yet clear. As a rule, use of estrogenes results in depression of the male sexuality. At the same time, combination of DHT and estradiole (in laboratory and productive animals) leads to normalisation of erection and ejaculation disorders. These result is, probably, due to the central effect of estrogene, which prolongs the DHT effect and censures its accelerated penetration into cellular neuronal structures, as well as to the peripheral effect of DHT.
The clenbuterol may or may not have been a factor,best to avoid....
Effect of clenbuterol on sexual behavior in male rats.
Benelli A, Zanoli P, Bertolini A.
Institute of Pharmacology, University of Modena, Italy.
The intraperitoneal injection of clenbuterol, a beta-2 adrenoceptor agonist which readily penetrates into the brain, modified the copulatory pattern of male rats. In sexually vigorous rats, the acute administration of clenbuterol at the dose of 0.1 or 1 mg/kg reduced the number of mounts and/or intromissions and increased the postejaculation interval, while the repeated administration (7 days) of a dose of 0.1 mg/kg/day had no effect. In sexually sluggish rats, both the acute (0.01 or 0.1 mg/kg) and repeated (0.1 mg/kg/day x 7 days) administration of clenbuterol increased the percent of animals achieving ejaculation and succeeding in initiating a new copulatory series within the respective cutoff times, and reduced mount, intromission and ejaculation latencies, as well as the postejaculation interval. These data show that clenbuterol negatively affects the copulatory behavior of sexually vigorous male rats, but improves that of sexually sluggish ones, and provide further evidence that central beta-receptor activation produces behavioral effects.
I myself have noticed a constrictive effect with clenbuterol and increased orgasmic latencies.
[email protected]. eod with HCG 2000iu/wk-titrate as needed,
[email protected] every 3rd day and definitely proviron as reccomended by Ulter, as it will work quickly to free-up and enhance DHT.
Good Luck..B32