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Arimidex = less mass?

Joe Stenson said:
You'll gain less WATER, and as a result, you might not gain as much strength. Thus, indirectly you might not gain as much muscle, but directly they don't have an effect on muscle built.
not true. you will still gain water intramuscularly, which is the "cell volumisation" spoken about that leads to better contractility/workouts. the water you will miss out on carrying around is in the skin

The Shadow: it wont knock it out, itll just block its effects for a while until the body metabolises and gets rid of the existing estrogen

arimidex is a good thing to be on if you are using something that will cause increased estrogen levels. it doesnt hinder gains, but even if it did, it would be smarter to use arimidex and block estrogen related sides (esp gyno), look harder, stress your heart less etc rather than carrying around lots of muscle, water, and bitch tits.
 
knightwolf said:
Btw... Does femera help you to fight gyno (like a-dex(? Or does it only hold down the water?
arimidex, femara and aromasin do the same thing (with aromasin having a slightly different approach to knocking out the aromatase enzyme). Femara has - for whatever reason - a bad reputation for killing your libido, i had experienced that myself.

as for reducing gains: estrogens are anabolic by itself but its a complicated story since when t conversion is reduced more t remains available. i doubt that there exists any credible evidence like a sci. study to guide us.
 
GoldenDelicious said:
not true. you will still gain water intramuscularly, which is the "cell volumisation" spoken about that leads to better contractility/workouts. the water you will miss out on carrying around is in the skin

My bad, thanks for clearing that up.
 
1 mg is a health man reduces it 50%.

As men age, serum testosterone levels decrease, a factor that may contribute to some aspects of age-related physiological deterioration. Although androgen replacement has been shown to have beneficial effects in frankly hypogonadal men, its use in elderly men with borderline hypogonadism is controversial. Furthermore, current testosterone replacement methods have important limitations. We investigated the ability of the orally administered aromatase inhibitor, anastrozole, to increase endogenous testosterone production in 37 elderly men (aged 62-74 yr) with screening serum testosterone levels less than 350 ng/dl. Subjects were randomized in a double-blind fashion to the following 12-wk oral regimens: group 1: anastrozole 1 mg daily (n = 12); group 2: anastrozole 1 mg twice weekly (n = 11); and group 3: placebo daily (n = 14). Hormone levels, quality of life (MOS Short-Form Health Survey), sexual function (International Index of Erectile Function), benign prostate hyperplasia severity (American Urological Association Symptom Index Score), prostate-specific antigen, and measures of safety were compared among groups. Mean +/- SD bioavailable testosterone increased from 99 +/- 31 to 207 +/- 65 ng/dl in group 1 and from 115 +/- 37 to 178 +/- 55 ng/dl in group 2 (P < 0.001 vs. placebo for both groups and P = 0.054 group 1 vs. group 2). Total testosterone levels increased from 343 +/- 61 to 572 +/- 139 ng/dl in group 1 and from 397 +/- 106 to 520 +/- 91 ng/dl in group 2 (P < 0.001 vs. placebo for both groups and P = 0.012 group 1 vs. group 2). Serum estradiol levels decreased from 26 +/- 8 to 17 +/- 6 pg/ml in group 1 and from 27 +/- 8 to 17 +/- 5 pg/ml in group 2 (P < 0.001 vs. placebo for both groups and P = NS group 1 vs. group 2). Serum LH levels increased from 5.1 +/- 4.8 to 7.9 +/- 6.5 U/liter and from 4.1 +/- 1.6 to 7.2 +/- 2.8 U/liter in groups 1 and 2, respectively (P = 0.007 group 1 vs. placebo, P = 0.003 group 2 vs. placebo, and P = NS group 1 vs. group 2). Scores for hematocrit, MOS Short-Form Health Survey, International Index of Erectile Function, and American Urological Association Symptom Index Score did not change. Serum prostate-specific antigen levels increased in group 2 only (1.7 +/- 1.0 to 2.2 +/- 1.5 ng/ml, P = 0.031, compared with placebo). These data demonstrate that aromatase inhibition increases serum bioavailable and total testosterone levels to the youthful normal range in older men with mild hypogonadism. Serum estradiol levels decrease modestly but remain within the normal male range. The physiological consequences of these changes remain to be determined.


This shows that in an elderly man who have less testosterone then someone on cycle it blocks it 50%. Estrogen also leads to an increase in SHBG and a down regulation of the AR-Receptor in large doses. Your body overcompensates for the test by increasing aromatase. I believe that it won't hinder your gains, and its just hard to tell with the bloat related increasing in strength and the glucose utilization. I think if you were to compensate for the glucose utilization by eating more you would have the same effets, if not greater.
 
Adex, femera, and aromasin are all aromatase inhibitors. Aromasin is actually a suicide inhibitor, but all do the same thing. They prevent gear like test, dbol, etc, from aromatizing to estrogen. Nolvadex doesn't do this, it just competes with estrogen for the same receptors, meaning it will bind in place of estro, but it won't actually DO anything. For preventing bloat, the first 3 are better. For getting rid of gyno once it starts, nolva is better. Overall... combine them for best of both worlds. I like aromasin and nolva, best estrogen control and best lipid profile.
 
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