but that doesnt explain how mdrol is oral masteron. as you know, two compounds can have very similar structures, but completely different effects.
needto compared it to dianabol, who's effects are nothing like those of masteron.
no, you misunderstood needto.
dbol is basically oral EQ, but we all know dbol is way more powerful than EQ.
He was equating m-drol compared with masteron to dbol and it's relationship to EQ.
Here's anadrol for example:
2-hydroxymethylene-17B-hydroxy-17α-methyl-3-androstan-one
you can see that drol is also DHT derived and is barely different from either masteron or superdrol.
minus the 5alpha and the addition of hydroxymethylene, it is equal.
It has it's differences, but they are few. on a molecular level, a few differences can make a drastic change in the bahavior of the molecule
Anyways, here is a good explanation of it:
Superdrol has a chemical stucture of 2a,17a-dimethyl-17b-hydroxy-5a-androstan-3-one.
2a,17a-dimethyl-17b-hydroxy-5a-androstan-3-one is a derivative of Drostanolone (also called Dromostanolone), which is also known as Masteron (in the commercial AAS). Drostanolon has a chemical structure of 2a-methyl-17b-hydroxy-5a-androstan-3-one. Drostanolon is the 2-methylated form of DHT, so it has typically been used for reducing body-fat and water retention, while increasing muscle hardness and density, as it cannot form estrogen.
Now, 2a,17a-dimethyl-17b-hydroxy-5a-androstan-3-one is a derivative of Drostanolon, so it is structurally similar, except it is a di-methyl instead of (just) a methyl. It has one methyl in the 17th position, just like M-1-t and M-Dien. But it has a 2nd methyl in the 2nd position, which is un-like the other Methyls which we are familiar with.
So this is how the derivative differs from standard Drostanolon, in that it has another methyl in the 17th position (as standard Drostanolon has one in the 2nd position, and not the 17th like M-1-t).
Also, it is a saturated modified form of anadrol, so you get the best of both worlds without the progesterone, estrogen, bloat, hypertension problems with anadrol.
I think Sldge has one study abouts its positioning effects for estrogen reduction. Zero estrogen conversion with this one, because it's 5-reduced and A-ring alkylated on top of that. Also, the parent compound (masteron) is used exclusively as an anti-neoplastic for metastatic breast cancer, so it's a strong anti-e.
Sldge has named the chem. methasteron and the product Supderdrol.
2a,17a-dimethyl-17b-hydroxy-5a-androstan-3-one has an extremely favorable Q (anabolic/androgenic) ratio. Q ratio = 20 (m-1-t is between 5-16, depending on assay). 20% as androgenic as 17a-MT oral (the reference standard); (m-1-t is 100-220% as androgenic as 17a-MT, for comparison). Superdrol is 400-800% as anabolic as the reference standard (17a-MT); (m-1-t is 910-1600% comparatively). i.e. It looks very, very favorable on paper, and the initial tester reports (logs on AM) back this up so far.
Make more sense? the position of the methylation in masteron does not make it bioavailable when taken orally. The additional methylation in a different location on the chemical structure chain in superdrol makes it bioavailable orally.
Easier to understand?
Hit me up with some K for this one bros.
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