Tren raises pgf levels which burns fat, Fonz has some excellent theories on it, which I agree since I eat like a pig on it and dont put on much fat.
Have you seen this thread?
http://boards.elitefitness.com/foru...105661&perpage=20&highlight=fina&pagenumber=1
My theory on why fina burns fat... (post #1)
Huck:
I have not located scientific evidence to support this,but I think that fina(tren)may be a potent mediator of PROSTAGLANDINS,which are the most thermogenic hormones found in the human body.The reason I'm starting to associate the two is there are some similarities between fina and Lutalyse that are quite commonly reported.#1-A great deal of users report a feeling of warmth rush over them for a period of time following a fina injection.#2-Many users have what is referred to as a 'fina-cough' following a fina injection...An influx of prostaglandin molecules(or in this case a greatly hightened activation of them)leads to the lungs having to metabolize and break the substance down.Then of course is the fat burning that accompanies both agents.I believe that based on these striking similarities of the two compounds,that there may be a connection...Now,if we can only get the biologists to run a study for us...
HUCK
.....I think that fina only activates DORMANT prostaglandins that one already has,as opposed to flooding the system with exogenous PG's.But who knows,maybe it helps proliferate new ones?
FONZ
I guess its time to reveal another of Fonz's little secrets....
When injected, trenbolone cause a rise in BOTH PGF2A and
PGE2.
PGF2a is what is called the GOOD prostaglandin.
PGE2 is the BAD prostaglandin.
However, the rise of PGF2A > PGE2 by a small margin.
By taking Enteric-coated aspirin(500mg/day) REDUCES
both PGF2A and PGE2 because they are produced
in a 1:1 ratio.
By decreasing PGE2, you reduce the specific prostaglandin
to BASELINE levels(or less again), while PGF2A remains
elevated ABOVE normal levels.
The whole point of this, is that PGE2 levels can play havoc
on your endocronoligal system even though PGF2A
is still greater than PGE2(BUT, PGE2 is still above baseline
normal levels)
Fonz
HUCK
.....If my theory holds up,then yes,an individuals prostaglandin receptor(PGR) populations/physiological response to the tren molecule would play a large factor in this effect,that is,those with higher populations of PG's will burn more fat on tren than others
{Q? on fina cough vs PGF-2A cough}.....But the one question I will still pose is why are the LUNGS responding to the glue/fillers,if that's what's causing the cough?Normally,foreign substances are shuttled in blood straight to the liver or kidneys for detoxification/metabolization.Could it be this exaggerated effect is from the PG molecule actually carrying some of these impurities along with it to the lungs,and the lungs having to work that much harder to break MULTIPLE substances down,as opposed to one?Hmmm...We need to see if a university will test it for us,haha.