Anavar for Bridging?

[nydj66]

I have read a couple references on the web that claim that Anavar does not inhibit your natural tesosterone levels like other steroids; possibly because it does not aromatize to estrogen at all. That would make it the perfect (although expensive) choice for bridging.

However, I have read posts here (possibly from a doctor) that say bridging is not possible period. So who is right? Anyone tried bridging with Anavar?

 

[alltraps]

i bridged after my last cycle with 15mg anavar, got tested 11 weeks after cycle and my levele were WAY below average. and i did all the regular PCT

 

[nydj66]

Thanks alltraps. Kama to you, bro.

 

[Cypptanon]

20mg/day Var bridge for four weeks following a 16 weeker - my blood test was at week six, and I'll have the results on Monday afternoon. I'll let you know more on Tuesday.

Cyp

 

[junk]

20mg/day Var bridge for four weeks following a 16 weeker - my blood test was at week six, and I'll have the results on Monday afternoon. I'll let you know more on Tuesday.

Cyp

Post your blood test results when they are in.

 

[needsize]

i bridged after my last cycle with 15mg anavar, got tested 11 weeks after cycle and my levele were WAY below average. and i did all the regular PCT

thats likely your normal test reading, the doc just didnt realize you normally have the test levels of a 10 year old girl....

 

[The Terminator]

I have bridged with 10 mg Dbol in the AM, and very recently with 20 mg of Anavar in the AM, and BOTH times my sex drive took a plummet...
I would advise against it

 

[Ttokkyo Jack]

Yeah i don't think that it would be a good idea to use it for bridging. However, I do believe that it is possible to use anavar without suppressing your natural testosterone production, by taking a small amount only in the morning. I just dont think that this would work post cycle because your hpta is already suppressed.

 

[Whacked]

ANY androgen (even at the small doses mentioned above) WILL impact HPTA recovery and prevent full recovery. To WHAT degree depends entirely on the individual.

I advise against it as well.

Creatine/lrIGF-1/P-GH/GH/Phosphaditly Serine (anti-cortisol) are all non-androgenic post cycle options.

 

[majutsu]

These questions are answered on the package label. Which I would read before taking. Under adult males, adverse reactions, testosterone suppression.

Also as little as 2.5ed in teens caused significant suppression. With their weights, this would be like 5-10ed in adult males. And there is certainly no guarantee that going as low as 2.5 ed would even avoid this HTPA suppression.

Below is the study, valued for clarity, hourly measurement of test and lh, and following the people up to a year!!!

Effect of low dose oxandrolone and testosterone treatment on the pituitary-testicular and GH axes in boys with constitutional delay of growth and puberty.

Crowne EC, Wallace WH, Moore C, Mitchell R, Robertson WH, Holly JM, Shalet SM.

Department of Endocrinology, Christie Hospital Trust, Manchester, UK.

OBJECTIVE: To investigate the effect of low dose oxandrolone and testosterone on the pituitary-testicular and GH-IGF-I axes. DESIGN: Prospective double-blind placebo-controlled trial. PATIENTS: Sixteen boys with constitutional delay of growth and puberty (CDGP) with testicular volumes 4-6 ml were randomized to 3 months treatment: Group 1 (n = 5), daily placebo: Group 2 (n = 5), 2.5 mg oxandrolone daily or Group 3 (n = 6), 50 mg testosterone monthly intramuscular injections with assessment (growth, pubertal development and overnight hormone profiles) at 0, 3, 6 and 12 months. MAIN OUTCOME MEASURES: LH and GH profiles (15-minute samples) were analysed by peak detection (Pulsar), Fourier transformation and autocorrelation. Testosterone levels were measured hourly and insulin, SHBG, IGF-I, and IGFBP-3 levels at 0800 h. Statistical analysis was by multivariate analysis of variance for repeated measures. RESULTS: LH and testosterone parameters increased significantly with time in all 16 (LH AUC, P < 0.001; peak amplitude, P = 0.02; number of peaks, P = 0.02; testosterone AUC, P = 0.02; morning testosterone, P = 0.002). In Group 2 (anavar), however, LH and testosterone parameters decreased at 3 months followed by a rebound increase at 6 and 12 months. SHBG levels were markedly reduced at 3 months (P = 0.006) and a wider range of dominant GH frequencies was present although GH AUC was not increased until 6 months, with an increase in GH pulse frequency but not amplitude. IGF-I levels were increased at both 3 and 12 months. In Group 3, pituitary-testicular suppression was not apparent, but GH levels increased with an increase in GH amplitude at 3 and 12 months. CONCLUSION: Oxandrolone transiently suppressed the pituitary-testicular axis and altered GH pulsatility. Testosterone increased GH via amplitude modulation.

Also below is the package label for oxandrin: Oxandrin Package Inset

However, these are two different questions, 1)whether anavar bridges have any utility and 2)whether anavar causes testosterone suppression . . .