All You Need To Know About--> Arimidex - Arimason - HCG

[gymrat surfer]

Amazing how many repetitive threads there are when you are searching on these drugs. To keep things simple and not ask the same questions again.

Can you please post the best link from a previous thread on Arimidex - Arimason - HCG

Please have these threads be informative covering all facets on each drug from A-Z.

Looking forward to these links..

Let's put an end to asking the same questions over and over again. Yikes.

 

[tical]

Aromasin is a considered a "suicide" inhibitor which means that it kills the enzyme responsible for the production of estrogen. This means that your body has to regenerate this enzyme before estrogen can be effectively produced again.

Arimidex works by blocking the enzyme from producing estrogen, it does not kill it.

I believe one can achieve greater *control* of estrogen by using an inhibitor like arimidex. There are many factors involved with the regeneration and sensitivity of the aromatose enzyme and using aromasin to actually kill these enzymes could leave you in the hole for a while. This varies from person to person. Remember, you don't want to ELIMINATE all estrogen. You just want it to be at an optimal level.

HCG acts on the leydig cells in the testicles by mimicking LH (leutenizing hormone). LH is the signal produced by your pituitary gland (at the base of your brain) that travels to your testicles to stimulate testosterone production.

When you're using exogenous hormones (steroids), the increased levels of testosterone is interpreted by the brain to the effect of "oh, there's plenty of testosterone here, time to stop producing." LH levels will drop to 0 and at this point there are no longer any signals being sent to your testicles to make them "work". This is why your balls shrink when you're on a cycle. We use HCG to mimic LH so that the testicles do not atrophy (like a muscle would if it is no longer being used).

Low dose HCG should be used throughout any cycle up to the last week of usage. If you've been supporting your testicles throughout your cycle with low dose HCG (250iu 2-3 times a week) you should not require any huge doses of HCG the last week or two before you start SERM therapy (clomid/nolva).

Using HCG during PCT is not helpful, as a matter of fact, it just gets in the way. If you increase your testosterone via HCG, your estrogen will increase and this combination will too will blunt the release of pituitary LH, similar to being on a cycle ("oh, estrogen is way up there, there must be too much testosterone, let's hold off").

For a successful PCT, you want your testicles to produce testosterone due to brain signaling, not HCG. This is achieved by using SERMs which selectively bind to / block estrogen receptors, making the brain think that there is a decreased amount of estrogen ("oh, there's no estrogen, this must mean there is no testosterone, let's start making some") - this increases LH production, in turn signaling your balls to get rolling again. As you taper off the SERM, your natural HPTA "balance" can return to support itself.

 

[gymrat surfer]

Great educational response, how about dosage schedule on each one

 

[tical]

Great educational response, how about dosage schedule on each one

As far as estrogen control, it's difficult to comment on that because everyone reacts different to these types of compounds (anti E's and AAS).

The only way to really know is through symptomatic experience and labwork.

A typical TRT protocol is around 100mg of testosterone cyp, HCG 250iu twice a week, and .25 to .5mg of arimidex twice a week.

So for example:

Mon - HCG 250iu
Tue - HCG 250iu
Wed - T Cyp 100mg, .5mg Arimidex
Thur - Nothing
Fri - Nothing
Sat - .5mg Arimidex
Sun - Nothing

This was my protocol and it kept my estrogen at reasonable levels.

As I've stated in other PCT threads, 12.5mg of clomid alone is enough to induce a huge spike in LH production from the pituitary. More isn't better or even necessary.

Unnecessarily high doses of SERMs for PCT are likely the cause for all of the hatred towards them. "I feel suicidal. I feel like a cry baby. It killed my libido. Fuck clomid, etc."

A SERM selectively acts on estrogen receptors. How that translates to each person is different. Too much will obviously give some nasty side effects, so the goal is to use as little as possible and then taper off.

Also, when using a SERM, be prepared to have a lower libido. Consider it a bonus if you maintain a strong libido throughout PCT.

For those that don't have a libido while on a SERM think about why this is the case:

#1 you're using a drug that is acting on your estrogen receptors
#2 you're experiencing an increase in testosterone which will also increase estrogen
#3 estrogen imbalances can create emotional, libido, and erectile issues

Remember, it is only temporary and it is just one of the sacrifices you make when playing the juicing game. Not a big deal if you're psychologically prepared for it and don't freak out because you suddenly don't have morning wood everyday like you did when you were on.

My personal PCT would be around 6-8 weeks:

Week 1, 12.5mg clomid ED
Week 2, 12.5mg clomid ED
Week 3, 12.5mg clomid ED
Week 4, 12.5mg clomid ED (get blood work to asses T/LH levels)
Week 5, 6.25mg clomid ED
Week 6, 6.25mg clomid ED
Week 7, 6.25mg clomid EOD
Week 8, 6.25mg clomid EOD

Week 13, get blood work to assess the end result

 

[gymrat surfer]

hmmm, would like some other opinions.

 

[endojuicedr]

You can find a lot of info on all 3 with a simple search. I wouldn't even worry about adex when aromasin is available. Hcg you will get different opinions but you can either run it through cycle or at the end.