Recent content by JS27

Nobody is asking for your propietary information. The science simply doesn't fit. And here's the conundrum: You claim to have been able to increase the transdermal delivery to roughly 30-40%, when pharmaceutical companies (Who have an R&D Budget bigger than your entire company*100000000000)...

You're out of your element. But it is christmas after all, so i won't delve too much into it. In any case, one person not responding to aromasin falls into the statistical variance for that given drug. No drug works for everybody. So, if that's your evidence, you better get more. :)

It's not exactly rocket science. But since you insist, I'll happily oblige. ADT has extremely poor bio-availibility(orally). (Methylating the compound makes it orally available). (i.e. Patented compounds) Yoir claim is this: That ADT can be absorbed transdermally via the skin at a rate...

Never had one problem with any of your products. (Save T-Rex for some reason). But do you realize how this whole thread sounds? Let's be completely candid here...there is no way this "New, un-tested, and un-tried" (Emphasis on un-tested) supplement is better than anastrozole. On paper...sure...

"Our new Aromatese Inhibitor is much more effective than Arimidex and possibly stronger than Aromasin." Now that's COMEDY. Talk about a totally irresponsible thing to say. You do realize that if somebody ends up with gyno due to this latter post, the blame is going to be laid right up to your...

^^^^^

Please realize that most people do not spend all day in front of the computer. In any case, ATD = androst-1,4,6-triene-3,17-dione It's basically a androstenedione analogue. A C-19 Steroid. Exemestane for example is methyleneandrost-4-ene-3,17-dione. The methylation increases it's oral...

Emphasis on "naturally occuring" :) Lets be completely candid here....it's potency is not even going to come close to that of Anastrozole. It's a bit irresponsible to claim that it's effects will be comparable to A-dex. This whole thing is starting to remind me of the whole "Chrysin" Debacle...

The IGF-1 will be useless if you mix it and you let it sit for a few hours. You have to inject right after mixing it. So in your case you would inject 40mcg AM and 40mcg PM.

I'm doing a 2 week cycle. 140mcgs/day for 14 days. I am using AAS(Test+Fina)(250mg/week + 150mg EOD). Most people complain of fatigue at higher doses, but I have yet to feel this(I'm at Day 7) Measuring 70mcgs on a 100IU Insulin pin is really quite a pain though. :) In any case, quite...

Nothing herbal is going to come close to anastrozole for reducing estrogen levels. Sorry to state the obvious, but that needs to be said. If you're worried about your lipid levels, then don't use an aromatase inhibitor (i.e. Arimidex and Femara). You can use exemestane instead. or if that...

When you filter the Trenbolone from the pellets, there is still a small residue left. The pellets are have Methyl-cellulose (A form of fibre) included in them in order to make them hold together(When they are compressed to form them). The cough is simply your lungs metabolizing the...

It is toxic if you don't take the proper precautions. But you can minimize accutane's side-effects by taking the proper precautions and not taking large doses. Prescription-style dosaging ranges anywhere from 80-120mgs/day This gives you EXTREME dry skin. I see no reason to ever go that...

A low dose of IGF-1 can easily let you keep your muscle mass during PCT. (40-50mcgs/day being a low dose). Since it has no impact on your HPTA, it is pretty much perfectly suited for your PCT needs. GH takes far too long to start working.