You need to understand that steroids will add mass with increase in diet.
Dieting while on steroids to loose fat will only help you keep the muscle you already have.
There is no sence in using steroids to loose body fat and gain muscle in my opinion. This can be done but you are better off doing a cycle, getting big and then dieting down afterwards.
You are wasting your money trying to get big and lean at the same time.
Eating enough cals. The right amount or protein and clean cals will grow muscle. Doing this well on steroids will grow more muscle. Steroids also simulate fat loss in many different ways. Yes you can lose bodyfat and gain muscle well using steroids. Hell you can do it with out them but well on them it will happen even faster.
All androgenic steroids bind to the AR receptor at different levels, not only in muscles but fat cells too. When steroids bind to the AR receptor in the fat cells stronger they cause more subcutaneous fat loss.
Some steroids have a dramatic effect on fat lose in a number of ways. For now we will look at just one. Proviron.
1.Proviron strongly suppresses the forming of estrogen. Estrogen
causes fat storage. Hence why woman have more bf% then men.
2. Proviron has a High affinity to the Ar in fat cells. Androgen receptors are found in fat cells as well as muscle cells, and while they act on the AR in muscle cells to promote growth, they also act directly on the AR in fat cells to affect fat burning. The stronger the androgen binds to the A.R, the higher the lipolytic (fat burning) effect on adipose (fat)tissue. And if you add, for example, testosterone to the mix, it increases the number of AR's in muscle and fat
3. Proviron like almost all steroids steroids Decreases the levels of an enzyme called lipasel. This forms lipids in fat cells and may be upregulated by how strongly a steroid attaches to a AR receptor in the fat cells(of course Proviron being one of the best as its attaches to Ar's at a higher rate then almost all aas).
4. Proverion like all steroids upregulate the glucocortisoid receptor. By blocking this receptor they control coristol and is this is how people on steroids can workout more often, without as much worry about overtraining. Controlling cortisol should lead to fat loss because cortisol promotes fat gain and muscle loss. When you prevent cortisol from attaching to the glucocorticoid receptor, you lower lipasel. This is the same enzyme we mentioned earlier, that helps form lipid.
The Androgen Receptor
There are many mechanisms behind the ability of androgens to reduce body fat. However, one key determinant of the amount of adipose tissue reduced is that particular androgen's ability to bind to the AR.
I need to mention that most androgens interact with both AR and GR (Glucocorticoid Receptors). We'll touch on that later. For now, let me explain why it matters how well an androgen binds to the AR in terms of reducing adipose tissue. Most of you know that ARs are present in tissue such as muscle. This is one of the mechanisms behind their ability to induce muscular hypertrophy. Now what does this have to do with body fat? Simple, AR's are present in adipose tissue as well. (1)
What does this mean? Well, it's been shown that the higher the density of ARs, the more that lipid uptake is inhibited. (2) It's also been shown that androgens that bind avidly to the AR cause an increase or upregulation of AR in adipocytes. (1) I think the greater the androgen binds to the AR, the more upregulation of AR in adipocytes occurs. This would lead to a significant reduction in subcutaneous adipose tissue. (3)
Notice that I specifically mentioned subcutaneous adipose tissue (fat right beneath the skin) and not visceral adipose tissue (fat around the internal organs). Why did I bother to differentiate between the two? Simple. For the most part, we bodybuilders are concerned only with subcutaneous adipose tissue. Visceral fat doesn't have much of an effect on a person's appearance. For that reason, we're only concerning ourselves with subcutaneous adipose tissue.
Now, what other mechanisms of action can account for the effects seen with those steroids that bind tightly to the AR? Well, those that bind tightly to the AR will decrease LPL (Lipoprotein Lipase), which is an enzyme that causes lipid accumulation. (4) They may also decrease Acetyl-CoA Carboxylase and Fatty Acid Synthetase.(5)
Another interesting note is that androgens have been shown to increase adenyl cylclase as well. This is the enzyme which is responsible for the conversion of cytoplasmic ATP into cyclic AMP. Increasing its concentrations is a good thing, in other words.
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So well no steroid should be labeled as a (fat burner) like say clen or albuteral They do Have a dramatic effect on fat lose in many ways. Any way you slice the cake it still boils down to diet and training but this thing about steroids having no effect on fat is not true.
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