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What is estrogen responsible for in men?
- Thread starter sk*
- Start date
I know there are high estrogen levels in the brain and it is required for healthy brain function.
It is important for good cholesterol profiles.
It is important for bone density (think post menopausal women getting osteoperosis from low estrogen levels).
Contrary to what some here believe, it is NOT important for labido. Estradiol has nothing whatsoever to do with sex drive. That is the job of testosterone (and DHT).
It is important for good cholesterol profiles.
It is important for bone density (think post menopausal women getting osteoperosis from low estrogen levels).
Contrary to what some here believe, it is NOT important for labido. Estradiol has nothing whatsoever to do with sex drive. That is the job of testosterone (and DHT).
--Bone growth and health (http://www.medscape.com/viewarticle/410890_1)
--Fusion of bone plates (ie: needed to tell male body it's time to stop growing).
--glucose metabolism
--HPTA regulation (http://www.endotext.org/male/male17/male17.htm)
"...When given testosterone alone, these subjects revealed a significant decrease in mean basal LH and FSH levels as well as LH pulse amplitude, demonstrating a direct suppressive effect on the pituitary of testosterone and its metabolites. Mean LH levels and LH frequency were suppressed to a greater extent in normal control subjects during testosterone administration suggesting also a hypothalamic site of action of testosterone in suppressing GnRH secretion. In order to discriminate the impact of testosterone as opposed to its aromatized products, both groups of subjects were administered with testosterone plus testolactone. The addition of the aromatase inhibitor completely prevented the suppression of gonadotropin secretion by testosterone in both normal and GnRH deficient men: in fact the mean LH levels increased significantly in both groups. The increase in mean LH levels was greater in the normal men who received testolactone alone compared to normal men who received testosterone plus testolactone, thus revealing also a direct effect of androgens in normal men.
...." This article gets even more interesting, but I have to read each paragraph like 2x to understand it...
--Hair and skin health.
--Cholesterol esp HDL levels.
--Mood?
--Fusion of bone plates (ie: needed to tell male body it's time to stop growing).
--glucose metabolism
--HPTA regulation (http://www.endotext.org/male/male17/male17.htm)
"...When given testosterone alone, these subjects revealed a significant decrease in mean basal LH and FSH levels as well as LH pulse amplitude, demonstrating a direct suppressive effect on the pituitary of testosterone and its metabolites. Mean LH levels and LH frequency were suppressed to a greater extent in normal control subjects during testosterone administration suggesting also a hypothalamic site of action of testosterone in suppressing GnRH secretion. In order to discriminate the impact of testosterone as opposed to its aromatized products, both groups of subjects were administered with testosterone plus testolactone. The addition of the aromatase inhibitor completely prevented the suppression of gonadotropin secretion by testosterone in both normal and GnRH deficient men: in fact the mean LH levels increased significantly in both groups. The increase in mean LH levels was greater in the normal men who received testolactone alone compared to normal men who received testosterone plus testolactone, thus revealing also a direct effect of androgens in normal men.
...." This article gets even more interesting, but I have to read each paragraph like 2x to understand it...
--Hair and skin health.
--Cholesterol esp HDL levels.
--Mood?
b1ewsw32
New member
Estrogen is a very important constituent in the male system, and is responsible for a variety of functions and preservations.
Too litle estrogen can be detrimental for cognitive abilities, and can totally diminish your libido.Too much can create femenizing characteristics of increased water retention(bloat),gynecomastia,fat distribution in abdominal regions,irratibility, headaches(probably from increased BP).As mentioned previously,it is essential for healthy skin and hair,bone mass preservation,ideal lipid levels.It has been used for alzheimer's disease with great success, as it keeps beta-amyloid levels at bay,which diminishes the formation of plaques and tangles. When I've used arim's at too high of a dose,I felt retarded and my libido suffered,after decreasing the dose, things returned to normal.At times when my test dose was too high without incorporating an anti-e,I had headaches,and became very moody,irritable and depressed.
You must strive to keep the same level as you produce naturally,before cycle>this can be obviously achieved by serum e-levels before and during cycle.
Another important reason for adequate e levels is the nessesity of this hormone to produce adequate IGF-levels.
I've dug up some evidence to support e's role in brain efficiency and male libido for my bro's
http://apu.sfn.org/content/Publications/BrainBriefings/estrogen.html
1: Clin Endocrinol (Oxf). 1999 Oct;51(4):517-24. Related Articles, Links
Role of oestrogen in male sexual behaviour: insights from the natural model of aromatase deficiency.
Carani C, Rochira V, Faustini-Fustini M, Balestrieri A, Granata AR.
Chair of Endocrinology, Department of Internal Medicine, University of Modena, Italy.
OBJECTIVE: In order to evaluate the role of oestrogens on human male sexual behaviour, the gender-identity, psychosexual orientation and sexual activity of a man with a congenital lack of oestradiol resulting from an inactivating mutation of the aromatase P450 gene was investigated. The psychosexual and sexual behavioural evaluations were performed before and during testosterone treatment and before oestradiol treatment, during three phases of different dosages of oestradiol treatment. DESIGN: The study was performed before (phase A) and during (phase B) testosterone enanthate treatment (250 mg i.m. every 10 days, for 6 months), during testosterone withdrawal (phase C), and during each of the following transdermal oestradiol treatments: 50 microg twice a week for 6 months (phase D); 25 microg twice a week for 9 months (phase E), and 12.5 microg twice a week for 9 months (phase F). MEASUREMENTS: Sexual behaviour was investigated by a sexological interview and by a 2-month self-reported daily diary performed during each phase of the protocol study. Furthermore, during each oestradiol treatment (phase C, D, E and F), a study of depression, anxiety trait and sexual behaviour was performed by the Beck Depression Inventory (BDI), the Spielberger Trait Anxiety Inventory (STAI) and the Golombok-Rust Inventory of Sexual Satisfaction (GRISS), respectively. Sexual orientation and gender-identity were evaluated by the BEM Sex Role Inventory (BSRI). Serum testosterone and oestradiol were measured during each phase of the study. RESULTS: Before oestradiol treatment (phase C), serum oestradiol was undetectable, while it rose to 356.1, 88.1 and 55.1 pmol/l during phases D, E and F, respectively. Before any oestradiol treatment, during phase D, phase E and phase F serum testosterone was 18.13, 0.72, 14.3 and 18.51 nmol/l, respectively. The patient's gender-identity as assessed by BSRI and by the sexological interview was clearly male. The psychosexual orientation evaluated by BSRI, by the sexological interview and by the analysis of the self-filled diary was heterosexual. Relevant modification of the patient's sexual behaviour occurred only during oestrogen treatment. This was more evident during both phase E and phase F, and concerned the behavioural parameters with an increase of libido, frequency of sexual intercourse, masturbation and erotic fantasies. A reduction of BDI and STAI scores was detected during the oestrogen phases. CONCLUSIONS: The study of the sexual behaviour in this man with aromatase deficiency suggests that oestrogens in humans do not affect gender-identity and sexual orientation but could have a role in male sexual activity.
Too litle estrogen can be detrimental for cognitive abilities, and can totally diminish your libido.Too much can create femenizing characteristics of increased water retention(bloat),gynecomastia,fat distribution in abdominal regions,irratibility, headaches(probably from increased BP).As mentioned previously,it is essential for healthy skin and hair,bone mass preservation,ideal lipid levels.It has been used for alzheimer's disease with great success, as it keeps beta-amyloid levels at bay,which diminishes the formation of plaques and tangles. When I've used arim's at too high of a dose,I felt retarded and my libido suffered,after decreasing the dose, things returned to normal.At times when my test dose was too high without incorporating an anti-e,I had headaches,and became very moody,irritable and depressed.
You must strive to keep the same level as you produce naturally,before cycle>this can be obviously achieved by serum e-levels before and during cycle.
Another important reason for adequate e levels is the nessesity of this hormone to produce adequate IGF-levels.
I've dug up some evidence to support e's role in brain efficiency and male libido for my bro's
http://apu.sfn.org/content/Publications/BrainBriefings/estrogen.html
1: Clin Endocrinol (Oxf). 1999 Oct;51(4):517-24. Related Articles, Links
Role of oestrogen in male sexual behaviour: insights from the natural model of aromatase deficiency.
Carani C, Rochira V, Faustini-Fustini M, Balestrieri A, Granata AR.
Chair of Endocrinology, Department of Internal Medicine, University of Modena, Italy.
OBJECTIVE: In order to evaluate the role of oestrogens on human male sexual behaviour, the gender-identity, psychosexual orientation and sexual activity of a man with a congenital lack of oestradiol resulting from an inactivating mutation of the aromatase P450 gene was investigated. The psychosexual and sexual behavioural evaluations were performed before and during testosterone treatment and before oestradiol treatment, during three phases of different dosages of oestradiol treatment. DESIGN: The study was performed before (phase A) and during (phase B) testosterone enanthate treatment (250 mg i.m. every 10 days, for 6 months), during testosterone withdrawal (phase C), and during each of the following transdermal oestradiol treatments: 50 microg twice a week for 6 months (phase D); 25 microg twice a week for 9 months (phase E), and 12.5 microg twice a week for 9 months (phase F). MEASUREMENTS: Sexual behaviour was investigated by a sexological interview and by a 2-month self-reported daily diary performed during each phase of the protocol study. Furthermore, during each oestradiol treatment (phase C, D, E and F), a study of depression, anxiety trait and sexual behaviour was performed by the Beck Depression Inventory (BDI), the Spielberger Trait Anxiety Inventory (STAI) and the Golombok-Rust Inventory of Sexual Satisfaction (GRISS), respectively. Sexual orientation and gender-identity were evaluated by the BEM Sex Role Inventory (BSRI). Serum testosterone and oestradiol were measured during each phase of the study. RESULTS: Before oestradiol treatment (phase C), serum oestradiol was undetectable, while it rose to 356.1, 88.1 and 55.1 pmol/l during phases D, E and F, respectively. Before any oestradiol treatment, during phase D, phase E and phase F serum testosterone was 18.13, 0.72, 14.3 and 18.51 nmol/l, respectively. The patient's gender-identity as assessed by BSRI and by the sexological interview was clearly male. The psychosexual orientation evaluated by BSRI, by the sexological interview and by the analysis of the self-filled diary was heterosexual. Relevant modification of the patient's sexual behaviour occurred only during oestrogen treatment. This was more evident during both phase E and phase F, and concerned the behavioural parameters with an increase of libido, frequency of sexual intercourse, masturbation and erotic fantasies. A reduction of BDI and STAI scores was detected during the oestrogen phases. CONCLUSIONS: The study of the sexual behaviour in this man with aromatase deficiency suggests that oestrogens in humans do not affect gender-identity and sexual orientation but could have a role in male sexual activity.
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