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the aids controversy

asteroth

New member
Did you know?

the "aids hypothesis"has been attacked by nearly 300 scientists with certifiable credentials such as;

Dr Peter Duesberg-professor of mulecular biology university of berkely;

Dr walter Gilbert Professor of Mulecular biology,1980 Nobel prize for chemistry;

Dr Roger cunningham,immunologist,microbiologist,and director of the center for immunology at the state university of NY.

log on to www. aidsmyth.com,or virusmyth.com to learn ,first hand,on the numerous reaserch papers,demonstrating without a shadow of a doubt,that HIV does not cause aids...but drugs do!!!!!!!!!
Drugs(not refering to steroids here) destroy your immune system and leave you defenseless...
Stop using AZT and it's derivatives...
keep yourself informed,and beware of the CDC and its politically inspired "scientific facts".

p.s
I have taken the liberty to copy some off the articles posted at www.virusmyth.com...while responding to some of the people who posted a reply to the above Aids controversy comment...
If you're HIV positive,I think you owe it to yourself to be aquainted with the dissenting opinion p r i o r to choosing the "right' treatment
Be Aware...
discuss this articles with your doctor and challenge him to provide you with a prompt convincing response prior to determining a course of action
It's your health...your Life




"If there is evidence that HIV causes AIDS, there should be scientific documents which either singly or collectively demonstrate that fact, at least with a high probability. There is no such document."

Kary Mullis, Biochemist, 1993 Nobel Prize for Chemistry

It's time to re-evaluate the HIV-AIDS hypothesis:

Is HIV really the cause of AIDS ?

A growing group of bio-medical scientists claim the cause of AIDS is still unknown. These heretics do not believe in the lethal AIDS virus called HIV. They claim that the virus is indeed harmless. Most of them think AIDS is also not sexually transmitted; it probably has toxic causes. People die because they are poisoned to death by toxic antiviral drugs. Part of the AIDS dissidents even question the existence of a virus entity. These HIV skeptics say that the AIDS virus has never really been isolated, and the AIDS tests are worthless...

This website tells you their story.

This website contains more than 1200 web pages with over 850 articles. If you are new make a short TOUR first, then you can best go to the INTRODUCTION page for a small selection of articles to start with. Listen to some Real AUDIO files, or watch some VIDEO documentaries first. See the NEWS page for the latest developments and articles.




CONTROVERSY MISSING VIRUS THE GROUP
WHISTLE BLOWERS CENSORSHIP
BOOKSHELF FRONT NEWS
FORUMS FIND

See also:
AidsMyth.com AidsMag.com

AIDSMAG
JUNE 2001. AIDSReTHINK.

THE RETHINKER
INTERVIEW
Dr. Mohammed Al-Bayati



interviewed by
Steven D. Keller
June, 2001

1. Steven: Thank you for giving this interview. First of all I would like to establish with the readers who you are, your credentials and what led you to take a serious look at AIDS and its causes?

I am a pathologist and a toxicologist with a Ph.D. in comparative pathology from the University of California Davis and a dual board certified toxicologist (DABT & DABVT). I have over twenty years experience in research, teaching, diagnostic, and doing consulting work in the fields of toxicology and pathology. I have also served as an Expert Witness on several cases involving the exposure of people to chemicals in the workplace, exposure of people to wrong therapeutic agents, and reaction of people to the side effects of therapeutic agents. In these cases, I identified the cause(s) of illnesses and provided the treating physicians and attorneys with reports describing my findings. In these reports, I also presented to the treating physicians my recommendation for monitoring and treating these illnesses.

In 1997, I established my toxicology consulting firm (Toxi-health International) in Dixon California (10 miles west of the University of California Davis) and my website contains a description of my company's wide range of services (http://www.toxi-health.com). In October of 1997, I evaluated the medical record and the case history of a 60 year-old-white male who was suffering from pulmonary fibrosis. He was treated with immunosupprassant medications (Azathioprine and prednisone). He consulted with me to find out if his exposure to chemicals in his workplace such as Jet Fuel and/or his medications has initiated or contributed to his illness.

My review of his medical record revealed that this man was suffering from AIDS.
He developed AIDS following his treatment with two months course of prednisone (60 mg per day) and two weeks course of azathioprine (50-100 mg per day) for lung fibrosis. His blood analysis revealed a CD4+ T cells count of 255/µL (normal range 542 to 1595/µL) and a CD4+T cells /CD8+ T cells ratio of 0.6 (normal range 1.0-3.5). He was also suffering from a severe lymphocytopenia (peripheral blood lymphocytes count was 483/µL). Prior to his treatment with prednisone, his lymphocytes count were normal (1513/µL). In addition, he suffered from pneumonia and developed a severe fungal infection of the skin at various regions of his body and very painful sores in his mouth (gum and cheeks which looked like white cottage cheese). He was not infected with the Human Immune Deficiency Virus (HIV) as determined by three tests at various clinics.


My communication with his treating physician, lead to the termination of his treatment with prednisone and Azathioprine. On May 19, 1998, 22 days after the last dose of prednisone, his CD4+ T cells and CD8+ T cells counts were 657 cells/µL and 659 cells/µL, respectively. These counts were 247% and 153% of the values during the tapering of prednisone for the CD4+ T cells and the CD8+ T cells, respectively. In addition, his fungal infection and pneumonia were resolved following treatment with short course of antibiotic (Doxycycline 200mg per day for 2 weeks) and topical antifungal agent (Loprox).

This case led me to evaluate the medical literature on AIDS worldwide to find out if there were other individuals with AIDS, who were HIV-negative, and to investigate the causes of AIDS. Prior to this time, my belief was that HIV caused AIDS as we have been told by the United States Center for Disease Control and Prevention (CDC) and the AIDS establishment since 1984. My investigation of the causes of AIDS worldwide took about two years and I presented my findings in my book " Get All The Facts: HIV Does not Cause AIDS" and in my articles. The first twenty pages of my book and the articles are posted on my website (http://www.toxi-health.com) and http://www.news-gap.com. My findings show clearly that HIV is not the cause of AIDS.

2. Steven: I've read your book, "Get All the Facts: HIV Does Not Cause AIDS" and I was really impressed by the information you found in your research. What can a toxicologist/pathologist offer to the current sea of information without getting bogged down by the populous information that we already have?

We can play a very important role in evaluating the medical information presented by physicians, scientists, and researchers and to provide correct interpretations of the information to get to the correct cause(s) of illness. For example, the CDC and the AIDS establishment stated that the epidemiology of AIDS indicates that AIDS is caused by virus called HIV, but my evaluation of the epidemiology of AIDS revealed that HIV is not the cause of AIDS.

The correct approach that should be taken to solve AIDS, or any other complicated chronic medical problems, is by evaluating all medical evidence concerning each risk group, namely, a differential diagnosis. I used this approach in this case to figure out the causes of AIDS in each risk group. I have found that the epidemiology and other medical evidence indicate clearly that HIV is not the cause of AIDS and that AIDS is caused by the use of immunosuppressive medications that have been used to treat wide range of illnesses caused by the use of drugs and alcohol. In Africa, AIDS is caused by severe malnutrition and the release of endogenous cortisol. Any individual suffering from severe malnutrition has AIDS regardless if he or she is HIV-positive or HIV-negative. In addition, AIDS in people suffering from malnutrition can be reversed by giving proper nutrition and supportive medical care. I gave many examples in my book to illustrate these points.

3. Steven: Given what has happened to Dr. Peter Duesberg and having his funding scaled back because of his viewpoints and public questioning of HIV and AIDS, are you not concerned for your job security? Are you not stepping on the toes of the mainstream view that a virus is what actually causes AIDS?

Prior to November of 1997, I believed that HIV was the cause of AIDS and I did not have any intention to investigate this issue. However, I discovered in November of 1997 that AIDS can be caused by other agents, and that HIV is a harmless virus. I also realized that AZT and the antiviral drugs are killing people, which changed my direction. It became my duty as a scientist to investigate this issue, to find out the truth, and to present my findings to our government and to the public. I have been spending a tremendous amount of time and money on this issue for the last four years without any financial help from any source. This has been extremely hard on my family, but what keeps me going is the reward of saving lives and our vital resources.

My findings were evaluated by professor Otto G. Raabe, a toxicologist from the University of California Davis, as well as other scientists and physicians who have been using my findings to save lives. In August of 1999, I sent many letters with a copies of my book "Get All The Facts: HIV does not cause AIDS" to President Clinton and other government officials and to Governor Gray Davis asking them to have my findings evaluated by experts but unfortunately no action has been taking yet. In spring of 2000, I sent similar letters with copies of my books to President Mbeki and the Embassy of South Africa in Washington D.C. My book was submitted to President Mbeki's Expert AIDS Panel where the medical evidence was evaluated and used. The panel report is posted on http://www.harmsen.net.

I do not understand why our government is ignoring this huge medical evidence that shows HIV does not cause AIDS while basing their entire AIDS program on unsupported hypothesis. Robert Gallo stated that HIV enters CD4+ T cells because they have special receptors for HIV and that HIV kills CD4+T cells selectively. I have found no truth for this hypothesis. Most individuals infected with HIV show hyperplasia of all cell components of lymph nodes (It has more cells than normal). In addition, HIV is present in all cells in the lymph nodes. Our government's decision of basing the entire AIDS program on the HIV-hypothesis is a very dangerous and costly decision. This faulty decision has been resulting in the exposure of millions of people to very toxic antiviral drugs worldwide unnecessarily and wasting billions of dollars.

My stand on this issue has cost me a lot of my personal time, plenty of money and business opportunities. However, I will continue to present the medical evidence to physicians, scientists and to the people of the world to save lives and vital resources. I cannot stand by and watch the mass killing of people or the killing of the unborn with AZT and other antiviral drugs. I am asking people to read the medical evidence that I have presented on this issue and to request that our government evaluate the medical evidence presented that clearly shows that HIV is not the cause of AIDS. The tragic poisoning of people by AZT and other anti-viral drugs has to be stopped. Now, we know what causes AIDS worldwide and we know how to cure AIDS. The medical evidence, which proves my point, is presented in Anthony Fauci's publications, and in my book and my articles. I will be happy to discuss my findings with our government and with public.

4. Steven: Within your report, which is very detailed, it is your opinion that people suffering from AIDS is a direct result from chronic drug use, both illicit and prescription. Can you talk a little about this and how you came up with these findings?

I evaluated the medical evidence and determined the causes of AIDS worldwide by performing differential diagnosis. In the USA, the total cases of AIDS in adults was 573,800 as of January 1, 1997 and about 90% of these cases were male homosexuals and heterosexuals, and homosexual drug users. The appearance of AIDS in the United States and Europe in drug users and homosexuals occurred in the early 1980's and coincided with the synergistic actions of several events. Briefly, these include the spread of illicit drug use, especially smoking crack cocaine and heroin in 1970's, the approval of glucocorticoids aerosol by the United States Federal Drug Agency 1976, the wide use of the glucocorticoid inhalers to treat chronic respiratory illnesses resulting from inhaling cocaine and heroin, the wide use of alkyl nitrites by homosexuals to facilitate anal sex in 1970's, and the wide use of steroids to treat chronic gastrointestinal tract illness in homosexuals. The approval of antiviral drugs (AZT and protease inhibitors) and the steroids by the U.S. FDA to treat patients with AIDS and asymptomatic patients infected with HIV has been exacerbating the problem.

The regular use of alcohol, heroin, cocaine, amphetamines, and alkyl nitrite cause chronic health problems of the nervous system, respiratory system, cardiovascular system, kidneys and other tissues in these individuals. The majority of these health problems are usually treated with high doses of glucocorticoids and/or cytotoxic drugs. In addition, some homosexual men use rectal glucocorticoids to treat inflammation. For example, the treatment of a patient with prednisone at 60 mg per day for about three months can actually cause AIDS. This treatment and doses are often given to patients suffering from lung fibrosis, thrombocytopenia, and other chemically induced chronic illnesses. I listed in Tables 12 and 14 of my book more than 30 illnesses in risk groups that are treated with prednisone and/or other immunosuppressant medications. For example, Anthony Fauci in his book entitled "Principles of Internal Medicine" published by McGraw-Hill in 1998, 14th edition (p. 1463) described the treatment for patient with lung fibrosis as follows: "A trial of oral prednisone is begun at a dose of 1 mg/kg daily and continued for about 8 weeks. Should the disease not respond or be progressive, additional immunosuppression with cyclophosphamide should be considered. The objective is to reduce the white blood cell count to approximately half the normal baseline value, causing a distinct drop in the total lymphocyte count. However, a minimum count of 1000 PMNs/µL should be maintained". At this dose levels, the CD4+T cells count in the peripheral blood of the treated individual is expected to be <300/µL which meets the definition for AIDS set by the US Center For Diseases Control and Prevention (CDC).

The following are two examples of HIV-negative woman who used drugs and a homosexual man who developed AIDS-defining illnesses (Tuberculosis or Kaposi's sarcoma) following the use of glucocorticoids to treat chronic illnesses.

… A 33-year-old HIV-negative previously healthy female nurse developed acute bilateral pulmonary infiltrates after 18 hours of intense rock cocaine (crack) smoking. Open lung biopsy showed a chronic interstitial pneumonia with extensive accumulation of free silica within histocytes associated with pulmonary fibrosis. The patient was treated with supplemental O2 and antibiotics and released. Ten months later she returned with progressive dyspnea due to diffuse reticular nodular interstitial and alveolar processes. Transbronchial lung biopsy revealed histocytic interstitial infiltrates with polarizable foreign material. The patient was unsuccessfully treated with high doses of prednisone (1 mg/kg/day for eight weeks) followed by a trial of cyclophosphamide. She died due to respiratory failure with a superimposed mycobacterial infection. She was HIV-negative. The time from her first admission to the hospital with interstitial pneumonia and the appearance of tuberculosis and her death was about 21 months. [Chest 100(4): 1155-7, 1991].

… A 61-year-old HIV-negative homosexual white male patient in whom Kaposi's sarcoma developed while being treated with prednisone for Henoch-Schonlein purpura, had the Kaposi's sarcoma clinically resolved completely after 18 months with discontinuation of the steroids. He was treated with prednisone for six months at dose levels of 80-15 mg daily. The CD4+T cell/ CD8 T cells ratio obtained after his acute illness was 1.0 [Am J Med 1987; 82 (2): 313-7].

Furthermore, the reversal of CD4+ T cells depletion in the peripheral blood was also reported in HIV+ homosexual men after the termination of their treatment with glucocorticoids. Sharpstone et al., 1996 reported that eight HIV+ males with inflammatory bowel disease who used rectal steroid preparation had a decline in their CD4+ T cells at a rate of 85 cells/µL per year. Four of them underwent colonectomy that eliminated the need for the steroid and their CD4+ T cells increased 4 cells/µL per year. Eight case-matched controls that did not have surgery continued to have a decline of 47 cells/µL per year as the result of the use of rectal steroid [Eur. J. Gastroentrol. Hepatic 8(6): 575-8, 1996]. In addition, investigators from George Washington University and the National Institutes of Health reported a case of HIV-positive homosexual man with ulcerative colitis. Approximately 3 weeks prior to surgery for ulcerative colitis that was unresponsive to corticosteroids, the patient's CD4+ T cell count was 930 cells/ml of blood and the count fell to 313 cells//ml within 10 days of treatment with corticosteroids. Five days postoperatively, the patient become asymptomatic and was discharged on tapering prednisone without the use of antiretroviral agents. After surgery, the patient's CD4 T cell count progressively rose. The CD4 T cell counts were 622 cells/ml and 843 cells/ml at 3 and 6 weeks following the operation, respectively [Journal of Human Virology 2(1) 52-7, 1999].

The patient was still HIV-positive after operation. The result of this case clearly demonstrates that the reduction of CD4+T cells resulted from the use of corticosteroids treatment and that HIV is a harmless virus.

5. Steven: It has been well established that both the ELISA and the Western Blot tests are non-standardized between countries and even labs, which has led to many false positives. These false positives along with the "hit early, hit hard" theory, in turn, have led many doctors to prescribed AZT, protease inhibitors and even prednisone to patients who were not showing any signs of illnesses related to AIDS. What is your understanding of these two misleading tests and because they are non-standardized, do they not falsely implicate people to have a "dangerous illness" when in fact they may not?

My investigation was focused on finding the causes of AIDS and the link between HIV and AIDS. When I found that HIV is not the cause of AIDS, then the issue of the HIV test became unimportant. In fact, I have found that the majority of people who participated in the major four AZT clinical trials that were conducted in the USA between 1986-1992 were HIV-negative prior to their treatment with AZT and their diagnoses were based only on clinical symptoms. The four published clinical trials are (1) Fischl et al., The New England Journal of Medicine 317 (4): 185-191 (1987); (2) Fischl et al., The New England Journal of Medicine 323 (15): 1009-1014 (1990); (3) Volberding et al., The New England Journal of Medicine 322 (14): 941-949 (1990); and (4) Hamilton et al., The New England Journal of Medicine 326(7): 437-443 (1992). Briefly, a total of 2,482 patients participated in these studies, and only 22% were HIV-positive prior to their treatment with AZT and the rest of the subjects were HIV-negative (62%) and untested (16%).

6. Steven: In your report you state, "damage to the immune system is rapidly reversible after removal of the true insulting agent or treatment of the true causes." Could you give us some examples of what you mean by "insulting agents" and "treatment of the true causes"?

In my answer to Q4 I gave several examples of the reversal of Kaposi's sarcoma in an HIV-negative homosexual man following the cessation of treatment with prednisone, as well as the reversal of the reduction in CD4+ T cells counts in HIV-positive homosexual men following the cessation of their treatment with corticosteroids. In these patients the insulting agent that caused AIDS-defining illnesses (Kaposi's sarcoma and severe reduction in CD4+ T cells counts) was corticosteroids. With the cessation of treatment, there was a reversal of these illnesses.

Below are two more examples of patients who developed Kaposi's sarcoma. Their tumors were reversed following the cessation of the use of prednisone. A 66-year old man with a severe bronchial asthma developed KS following treatment with prednisone 10 to 50 mg daily or on alternate days for about five years [Arch Dermatol 166 (11): 1280-2].

The second patient developed generalized Kaposi's sarcoma (extensive skin and stomach lesions) 24 months after renal transplantation while on cyclosporin (CyA) and prednisolone. His Kaposi's sarcoma disappeared completely upon withdrawal of CYA. CYA was introduced following an episode of acute rejection. Within 8 weeks, Kaposi's sarcoma reappeared on the skin at the same sites as the previously healed lesions. The tumor completely disappeared again upon withdrawal of CYA. Azathioprine was then introduced and Kaposi's sarcoma lesions reappeared 6 month later [Am J Nephrol 1992; 12(5): 384-6].

Furthermore, severe malnutrition has been known to cause immune dysfunction and other serious health effects. This should be considered in the differential diagnosis in HIV infected patients with AIDS, who are suffering from severe malnutrition, before implicating HIV as the cause of AIDS in Africa. Actually the finding of atrophy of lymphoid tissue in people suffering from malnutrition was observed as early as 1925. For example, Jackson's review on this topic in 1925 noted that many investigators had found a pronounced tendency of atrophy of lymphoid tissue in all conditions of malnutrition. Thymus weight was exquisitely sensitive to malnutrition and was earlier designated as the "barometer of nutrition" [Woodruff, 1972 Lancet 1(7741): 92-3)].

The functions of the immune system, especially the cellular immunity, are impaired in malnutrition cases. The severity of the impairment is dependent on the degree of malnutrition in both human and animals. I presented the results of studies of 345 malnourished children and two experimental animal models in my book that shows the impact of food deprivation on the size of the thymus and the lymphoid organs (Al-Bayati, 1999 Get All The Facts: HIV does not Cause AIDS). For example, the size of the thymus of 42 malnourished children was reduced by 90% as compared with a case-match normal control (Parent et al. Am. J. Clin. Nutr. 60(2): 274-8, 1994). In a second study involving 110 malnourished children, the thymic area was found to be 20% of the size in healthy children and the size of the thymus increased from 20% of normal in a malnourished child to 107% of normal following 9 weeks of proper feeding children (Chevalier et al., J. Trop Perdiatr 44(5): 304-7, 1998).

The reversal of the reduction in CD4+T cell count was also reported in HIV+ pregnant women following proper feeding [Fawzi et al., The Lancet 351:1447-1482, 1998). Briefly, the influence of diet on T cells counts in peripheral blood in 1,075 HIV-infected pregnant women who had poor nutritional status were studied. The CD4+ T cell counts of the women who received multivitamin increased from 424/µL to 596/µL during six months of proper feeding.

7. Steven: You mentioned in your report that corticosteroids and glucocorticoids are the "major causative agent in the U.S. AIDS epidemic." Could you explain what these two drugs are, how in your opinion they became major players in the AIDS epidemic and which medications use corticosteroids and glucocorticoids?

Corticosteroids and glucocorticoids are different names of hormones released from the cortex of the adrenal glands. In humans, this hormone is called cortisol and in animals it is called cortisone. There are many commercial names for glucocorticoids such as prednisone, asthmacort, flonase, and others.

Cortisol has many functions and some of these functions are the inhibition of protein synthesis and the enhancement of glucose synthesis in liver during food deprivation. In starvation cortisol is released from the adrenal glands to convert protein and lipid to glucose in liver. Glucose is the primary fuel for heart and brain. Therefore, people who are suffering from malnutrition have high levels of cortisol in blood and urine and the cortisol levels usually go down following feeding a balance diet for certain duration. For example, the lymphocyte function of 30 black children with severe malnutrition as assessed by the delayed hypersensitivity reaction and morphology of lymphocyte transformation was found to be impaired and serum cortisol level was elevated. The function of lymphocyte and cortisol level returned to normal after 30 days of feeding [Zeng et al., Hua His. I Ko. Ta. Hsueh Pao 22(3): 337-9, 1991].

We learned about the anti-inflammatory function of cortisol in the 1940s. It was approved by the US FDA to be used as immunosuppressant agent to be given to patients who needed organ transplants. Corticosteroids usually depress the functions and the size of the immune system; therefore, reducing the chance of rejecting the new organ by the host. People who are treated with high doses of corticosteroids for periods of months usually suffer from low T cells counts and AIDS-defining illness such as tuberculosis, Kaposi's sarcoma, and other illnesses as described in Fauci's vast number of publications.

The use of glucocorticoid compounds has been increasing with time and in the USA, more than 10 million Americans use glucocorticoid as anti-inflammatory and anti-allergy drug per year. The effect of these compounds on the immune system depends upon the amount and the duration of use. However, the damage is reversible in most cases.

In the USA, the total cases of AIDS in adults was 573,800 as of January 1, 1997 with about 90% of these cases being male homosexuals, and heterosexual and homosexual drug users (Fauci, et al., 1998. Harrison's Principles of Internal Medicine. McGraw-Hill Companies, Inc. New York USA, ed. 14). Inhaling cocaine and heroin use causes many respiratory illnesses that require long-term treatment with glucocorticoids. In addition, there are many other drug-induced illnesses such as thrombocytopenia, peripheral neuropathy, and chronic kidney problems that are treated with high doses of glucocorticoids. In my book, I list more than 30 conditions in risk groups that are treated with high doses of glucocorticoids (Tables 12-15) and the doses used to treat each of these condition can cause AIDS. I gave many examples in my answer to Q4, which shows that treatment of individuals with prednisone for lung fibrosis and other illnesses induced by drugs, cause AIDS. In my answer to Q4, I also gave examples for the use of prednisone by homosexuals to treat chronic inflammation of the colon that resulted in the reduction of CD4+ T cells counts. The HIV-positive patients who selected to have surgery to remove the inflamed portion of their colon and eliminate the use of prednisone, the reduction of CD4+ T cells was reversed following the termination of the glucocorticoids.

Hemophiliac patients are treated with high doses of glucocorticoids and other immunosuppressant agents. These facts are explained in Fauci's book, Harrison's Principles of Internal Medicine [McGraw-Hill Companies, Inc. New York USA, ed. 14].
The hemophiliac patients are treated with immunosuppressive agents (cyclophosphamide and glucocorticoids) to prevent the development of antibodies against factors VIII and IX. Patients with severe hemophilia also have serious chronic joint problems resulting from bleeding inside the joints. The chronic joints problems are treated with glucocorticoids.

8. Steven: Could you talk a little bit about how AZT and most currently, protease inhibitors, destroy the bone marrow leading to the loss of bone marrow cells and eventually to the many illnesses attributed to the HIV virus and AIDS?

AIDS patients have been treated with antiviral medications based on the assumption that HIV is the cause of AIDS. This approach has been a failure and it is not based on sound medical facts. It is killing people and must be stopped immediately. In my answer to Q5, I stated that a total of 2,482 patients participated in four major AZT clinical trials conducted in USA between 1986 to 1992. Only 22% were HIV-positive prior to their treatment with AZT and the rest of the subjects were HIV-negative (62%) and untested (16%). This information indicates that the designs and the conclusions of these studies are invalid. Below is a brief summary of the design and the result of

Fischl et al., 1990 study [The New England Journal of Medicine 323 (15): 1009-1014 (1990)] that shows the wide range of AZT toxicity and the problem with the design of this study. 1) This study did not include a placebo group, and only 39% of patients were HIV-positive at baseline based on the virology data presented. The authors were aware of this fact. They stated "thirteen subjects of 146 tested who were negative for HIV antigen before treatment later had detectable levels of antigen during the 128 weeks of treatment". The data presented below clearly shows that AZT is very toxic as indicated by the new opportunistic diseases developed, bone marrow toxicity, and mortality rates. There is a linear relationship between total AZT ingested (g) by the subject and the mortality rates in both treatment groups (Table 1). The equations for the lines for both groups are presented below.

Y = 0.0896x - 25.27 R2 = 0.9998 for standard dose ŠŠ.(1)
Y = 0.2147 x - 33.47 R2 = 0.9828 for low dose ŠŠŠŠ..(2)

Where y = Mortality rate and X= AZT ingested (g) per individual
Based on equations # 1 and 2 a 100% mortality rates will be reached at 31
and 33 months of treatment for the standard dose (1.5 g AZT per day) and
low dose, respectively.

Table 1. Total AZT ingested (g) per individual
and mortality rates per treatment group
----------------------------------------------------------
Treatment Low dose* Standard dose*
Duration AZT % AZT %
Months Ingested(g) Mortality Ingested(g) Mortality
----------------------------------------------------------
12 236 19 548 24
18 345 37 822 48
24 455 66 1095 73
----------------------------------------------------------
*Number of subjects in each group at baseline was 262 and 524 per the study. Number of HIV-positive patients at baseline was 205 (39% of total).

Design: They conducted a randomized controlled trial in 524 subjects who had a first episode of Pneumocystis carinii pneumonia and only 39% of these individuals had detectable serum levels of HIV antigen before treatment and the 61% were HIV-negative or untested. These subjects were assigned to receive zidovudine in either a dose of 250 mg taken orally every four hours (the standard-treatment group, n=262) or a dose of 200 mg taken orally every four hours for four weeks with 100 mg taken every four hours thereafter. (The low-dose group, n=262). The median length of follow-up was 25.6 months. The total amount of AZT ingested (g) per individual during the study period is presented in Table 1.

Results: A new AIDS-defining opportunistic infections developed in 429 subjects (82%) in the AZT treated groups. The hemoglobin level declined to less than 80 g per liter (baseline= 121 g per liter) in 101 subjects in the standard-treatment group and in 77 subjects in the low-dose group. The neutrophil counts declined to less than 750 per ul (baseline = 2200 per ul) in 134 subjects in the standard-treatment group and in 96 in the low-dose group. One hundred eighty-three subjects (35%) were withdrawn from zidovudine therapy because of toxic reactions such as sever anemia and severe neutropnea. One hundred thirty-four subjects (26 percent) received red-cell transfusions. The mortality rates were 66% and 73% in the low and standard AZT doses, respectively, at 24 months of treatment (Table 1).

Comments: The authors of the above study stated that AZT showed a therapeutic value by reducing the severity of illness in AIDS patients and low-dose therapy was associated with a better survival rate. It is very obvious that the design and the conclusions of this study are not valid based on the facts presented above. The result of the study show that there is a linear relationship between total AZT ingested (g) by the subject and the mortality rates in both treatment groups.

Protease inhibitors usually cause severe damage in kidney, liver, and other organs. The severity of the damage depends upon the amount of the drugs taken and the duration of use. Some studies show that the CD4+ T cell counts were increased after treatment with AZT and/or protease inhibitors. This information was interpreted as a good response to the medications. On the contrary, the elevation of T cells is not a good response in these conditions, but rather, it indicates severe tissue damage and infection because elevation of CD4+ T cells counts also occur due to inflammation in tissues. This explains the injury and death of the patients following treatment with these drugs. For example, the CD4+ T cell counts were increased following the treatment of HIV negative nurses with AZT, who took AZT as a prophylactic. They developed severe symptoms following 3 weeks of treatment with AZT (Get All The Facts: HIV does not cause AIDS, Table 24). In addition to the failure of the antiviral drugs, AIDS patients suffering from immune deficiency are also treated with glucocorticoids. This practice is not supported by any known mechanism of action. The antiviral medications and the glucocorticoids not only fail to cure AIDS, but they cause severe damage to sick people with AIDS. Prescribing these medications to AIDS patient is just like putting gasoline on a fire. The proponents of the HIV hypothesis failed to anticipate this disaster and they claimed that AZT prolonged lives.

9. Steven: It is astonishing when you explain how AZT works and when one realizes that this is the drug of choice for pregnant women who test HIV+ and their babies whether they test positive or not. At the same time, AZT is also the drug of choice that is being pushed upon malnourished people in South Africa. How can this important information make a difference within these two groups of people, and have you been contributing any of this information to President Thabo Mbeki and/or the United States government? If so, what has been their response?

As I stated in my answers to the previous questions, the designs and the conclusions of the AZT clinical trials are not valid. AZT is very toxic to bone marrow and tissues with high cell division rates such as embryonic tissue. Treatment of pregnant women with AZT will definitely cause very serous damage to the embryo and fetus.

The cause of AIDS in infant and children in the industrialized countries are the use of cocaine, heroin, and alcohol by mothers during their pregnancy. The use of cocaine during pregnancy was usually associated with a high prevalence of premature births and low birth weights. Drug exposed infants usually had immature lung profiles and other serious health problems. These health problems are usually treated with glucocorticoids. As of January 1, 1997, the number of infants and children in USA diagnosed with AIDS was 6,891. Ninety percent of these cases had mothers who were drug users.

In Africa, the cause of AIDS in infants and children is malnutrition. The study of Fawzi et al. [Fawzi et al., The Lancet 351:1447-1482, 1998] clearly demonstrated that HIV is a harmless virus and the impairment of the immune system in a mother (HIV-positive) who suffers from malnutrition can be reversed by feeding the mother proper nutrition. This measure also improved the outcome of pregnancy. For example, in Tanzania, 1,075 HIV-infected pregnant women between 12 and 27 weeks' gestation received vitamin A (n=269), multivitamins excluding vitamin A (n=269), multivitamins including vitamin A (n=270), or a placebo (n=267). In this study, multinutrition supplementation decreased the risk of low birth weight (<2500 g) by 44%, severe preterm birth (<34 weeks of gestation) by 39%, and small size for gestational age at birth by 43%. During pregnancy, all T-cells subsets (CD4+, CD8+, and CD3+) increased in all groups between baseline (mean 18 week's gestation weeks' gestation and 6 weeks postpartum). There was a significantly larger increase in the CD4+ T cell counts and percentage of CD4+ T cells among women assigned multivitamins. The mean increases between baseline and 6 weeks postpartum were 167 cells/µL and 112 cells/µL among women on multivitamins and those on no vitamins, respectively.

As I stated earlier (Q3), in August of 1999, I sent letters with copies of my book, Get All The Facts: HIV does not cause AIDS, to President Clinton and other government officials including Governor Gray Davis, asking them to have my findings evaluated by experts. Unfortunately no action has been taking yet. In spring of 2000, I sent similar letters with copies of my books to President Mbeki and the Embassy of South Africa in Washington D.C. My book was submitted to President Mbeki's Expert AIDS Panel and the medical evidence was evaluated. The panel report is posted on http://www.harmsen.net.

The result of the large study in Tanzania presented above clearly shows that HIV is a harmless virus and that the depression of the immune system in pregnant women suffering from malnutrition can be reversed by nutrition. This treatment also improved the outcome of pregnancy. On the contrary, Fischl et al., 1990 study [The New England Journal of Medicine 323 (15): 1009-1014] described above shows a linear relationship between the amount of AZT ingested by the individual and mortality rate. It is very sad and frustrating to know that the CDC and the AIDS establishment are giving AZT to pregnant women even with studies that show the depression in the immune system can be reversed by nutrition. I hope that our government will pay attention to the medical evidence presented in my book and my articles that show HIV is not the cause of AIDS, and that AZT and protease inhibitors are killing people. I hope that our government will take the proper actions soon to end the suffering of people who are unnecessarily ingesting these toxic medications.

10. Steven: You mention a powerful antioxidant, Alpha Lipoic Acid in your report. What is this chemical and how can it help to rebuild one's immune system that has been destroyed by AZT, protease inhibitors and Prednisone? What other antioxidants or products have you found in your research that may work to help rebuild damaged cells, bone marrow and immune systems?

Alpha lipoic Acid (ALA) is a very powerful antioxidant that has been used to prevent injuries caused by chemicals in human and animal studies. It has also been given to diabetic patients for the last two decades to prevent tissue damage, has been used in Europe to reverse peripheral neuropathy in diabetic patients, and has been shown to be effective and safe in several clinical trials. This drug is very effective in preventing and reversing injuries resulting from metabolic changes and/or exposure to chemicals that induce lipid peroxidation. It is sold without prescription and has no side effects at the therapeutic doses (Up to 600 mg per day) for a period of 3 months or more in most people. In Chapter 14 of my book, I presented a brief description of the results of nine studies to show the efficacy and safety of Alpha lipoic acid. Vitamins E, C and trace elements such as zinc and selenium are also very important in the healing process of damage caused by metabolic changes and/or the exposure to chemicals such as AZT and other medications.

11. Steven: Do you have any trial studies going on? What are they and how can someone become involved with them?

Our clinical study is posted on http://www.aliveandwell.org. We have also started to evaluate cases. For more details please check the site below. I do differential diagnosis to identify the cause(s) of illness and I send my report to the physician(s). My report usually contains a description of the cause(s) of the problem and my recommendations for clinical tests and treatment.


Resolution of AIDS in HIV Positive Patients:
A Clinical Study of Non-HIV Causes and Treatments for AIDS Illnesses
Mohammed A. Al-Bayati, PhD, DABT
Juan Jose Flores, MD, PhD
Lisa M. Hosbein, MD, FACOG
Christine Maggiore, American Foundation for AIDS Alternatives
http://www.aliveandwell.org/index.php?page=study

I have evaluated the case histories and the medical records of many HIV-positive AIDS cases and I have found that the causes of illness in these people are the use of the antiviral medications and the glucocorticoid medications. For example, I evaluated the medical record of an HIV-positive individual who is suffering from thrombocytopenia with low CD4+ T cell count. In the last 10 years, this individual has been treated with 24 prescription medications and 12 of these medications cause thrombocytopenia, five drugs cause damage in the immune system. However, this individual has been treated with AZT and glucocorticoids that cause thrombocytopenia and AIDS on the assumption that HIV is the cause of these health problems in this individual.

I receive many letters, emails and telephone calls per month from people who are desperate, and in need answers and help. I usually spend about 20 hours per case to evaluate the medical records and case history of patients who are suffering from AIDS. As I mentioned earlier, most of these patients do not have the money and unfortunately, we don't have any funding to handle the load. Currently, there is no money allowed or given by the United States Government to any AIDS project not dealing with the HIV as the cause of AIDS. AIDS and HIV are treated by our government as political issues, not as health issues and this policy is, unfortunately, causing human tragedy worldwide.

We have submitted our proposal to private sources. We are hoping to get funding to save people's lives and to provide more medical evidence showing that the depression in the immune system, in HIV-positive AIDS patients, is reversible without the use of toxic antiviral medications as I presented in many examples above, in my book and articles.

12. Steven: Although your ideas are much easier to comprehend and seem more logical compared to the mainstream ideas of what AIDS is, what has the reaction been from your comrades and fellow physicians? Do you work with or see a growing number of physicians in the San Francisco, Los Angeles, San Diego area or even nationwide or internationally, who share your concerns and ideas?

I spent about two years to evaluate the causes and pathology of AIDS worldwide prior to releasing my findings to the public. In my book, I identified the causes of AIDS and described the symptoms and pathology of AIDS in all risk groups. My book is divided into fifteen chapters. There is a specific chapter designated for each risk group and other chapters to explain the causes and pathology of the AIDS-defining illnesses. For example, chapter 10 contains the description of the causes of AIDS in infants and children, while chapter 11 describes the cause of AIDS in hemophiliacs. The entire index is posted on http://www.toxi-health.com. I wrote this book as a medical book so physicians can use it to diagnose cases of AIDS accurately and order proper clinical tests to treat the patient with AIDS successfully. It is very easy to follow and understand.

My conclusion that HIV is not the cause of AIDS is based on published medical evidence and not on hypothesis; therefore, any physician or scientist who reads and examines the information in my book will come to the same conclusion. Fore example, Professor Otto G. Raabe is a toxicologist from the University of California Davis and has read my book two times, evaluated the medical evidence carefully and came to the conclusion that HIV is not the cause of AIDS. He wrote the foreword for my book, which is posted on http://www.toxi-health.com. Below is one paragraph of his report.

"Dr. Al-Bayati's detailed evaluation of the world-wide AIDS epidemic approaches the literature head-on and lets the chips fall where they may. Because of his objective use of differential diagnosis and his sensitivity and understanding of both pathological and toxicological factors, he is able to convincingly demonstrate that the convergence of several factors other than HIV represent the true causes of AIDS. This book deserves careful attention, especially from physicians who must decide the course of medical treatment for their various patients."

Furthermore, Professor Juan Jose Flores, MD, PhD, Dr. Lisa M. Hosbein, MD, FACOG, and other physicians and scientists, who have read my book, have come to the same conclusion that the HIV is not the cause of AIDS. In addition, individuals who have read my book, reach the same conclusion. There are many articles written about my findings and I have been on many radio and TV shows. In the last 20 months, I have not received any disagreement from any physician or a scientist about my findings. Some of these articles and our clinical studies are posted on the internet.

Physicians usually depend upon the recommendations from our government and the pharmaceutical companies about the treatment of AIDS patients and the usage of medications. Most of the time they have no way of knowing nor understanding the implications involving these recommendations or whether they are valid or not. I informed the US government and many State governments in August of 1999 and requested that my findings to be evaluated, unfortunately, no action has been taken yet. It is the responsibility of our government to take the right step by evaluating the evidence and to protect the public from taking highly toxic drugs such as AZT and other antiviral drugs, which are currently used to treat patients with AIDS.

13. Steven: You really strip away the intricate layers of Anthony Fauci's report, "Clinical Categories of HIV infection" and "Harrison's Principle of Internal Medicine" to reveal some pretty astonishing facts that were either covered up, left out or pushed aside. Of all the information you uncovered, what would be the most important for our readers to know about when it comes to A. Fauci et al. work?

As I stated earlier in Q1, prior to October of 1997, I believed that HIV was the cause of AIDS, as has been reported by the CDC and the AIDS establishment. When I found that the treatment of a patient with prednisone (60 mg per day for 10 week) and Azathioprine for lung fibrosis could cause AIDS and that the damage to the immune system is reversible upon termination of this treatment, I became very interested in the issue of AIDS. I pursued my plan to evaluate the medical literature to find out if there were other cases who developed AIDS as result of treatment with immunosuppressant medications, and to evaluated the link between HIV and AIDS.

My research of the various literature lead me to the vast numbers of publications by Anthony Fauci that dealt with the effects of glucocorticoids on the functions of the immune system. Dr. Fauci has been studying the effects of glucocorticoids and other immunosuppressant medications on the function of B cells and T cells since the 1970s. I was very surprised to find that Fauci described the symptoms of AIDS in 1976 in people using high doses of corticosteroids over a period of a few months, as stated below, and that there are more than 40 medical conditions in risk groups such as pulmonary fibrosis, thrombocytopenia, peripherial neuropathy, etc that have been treated with high doses of glucocorticoids and other immunosuppressant medications for long time. A. Fauci has not considered these factors at all when stating that HIV is the cause of AIDS.

For example, Fauci et al., 1976 stated that, "we have reviewed many aspects of the host defenses that are altered by corticosteroids, and the combined effects of these changes must be considered in trying to understand the relation between corticosteroids and infections. Since the defect with corticosteroids is broad, it is not surprising that many types of infections seem to occur more often in patients treated with corticosteroids. Of the bacterial infections, staphylocococcal and Gram-negative infections, as well as tuberculosis and listeriosis infections, probably occur most often. Certain types of viral, fungal, and parasitic infections also occur often. Patients with lupus erythematosus, rheumatoid arthritis, and renal transplants have more infection with steroid administration. Study of bronchial aerosols showed that with higher doses of steroid in the aerosol, Candida infections of the larynx and pharynx occurred more often" [Annals of Internal Medicine 84: 304-15, 1976.].

Furthermore, I reviewed Fauci's book entitled "Principles of Internal Medicine" published by McGraw-Hill in 1998, 14th edition. This book contains detailed descriptions of the impact of illicit drugs, glucocorticoids, and malnutrition on health and the functions of the immune system, but yet Fauci has not considered any of the information presented in his book when dealing with the AIDS issue. For example, the opportunistic diseases described on page 1,792 in Fauci's book under the name of Clinical Categories of HIV infection, are the same opportunistic diseases that are described in the same book in organ transplant patients treated with high doses of immunosuppressive medications. It is very difficult for me to imagine that A. Fauci, the Director of the AIDS program at the NIH, spent billions of dollars to find a cure for AIDS, yet the true answers are presented in his publications.

Fauci and his colleagues conducted a study in 1995 and found that the lymph nodes of some patients infected with HIV had hyperplasia (It has more cells then normal). This observation contradicts the HIV-hypothesis that HIV kills infected cells [J. Immunol 154 (10): 5555-66, 1995]. Also in this study, some lymph nodes showed atrophy and necrosis in T cells and B cells. These changes were independent of the viral load or the duration of the infection. Hence, they did not question the HIV-hypothesis but, rather, they modified the hypothesis to fit their new findings. Briefly, they examined 29 HIV positive lymph nodes and found twelve of these lymph nodes with follicular hyperplasia and extensive germinal centers, five with follicular hyperplasia mixed with follicular involution, twelve lymph nodes with a mixture of follicular involution and lymphocyte depletion, and five lymph nodes with lymphocyte depletion. They stated that, "apoptosis was not restricted only to CD4+ T cells; both B cells and CD8+ T cells were found to undergo apoptosis. They stated that the increased intensity of the apoptotic phenomenon in HIV infection is caused by the general state of immune activation, and is independent of the progression of HIV disease and the levels of viral load".

In 1992, scientists and physicians reported to the CDC cases with AIDS and/or low CD4+ T cells, yet these cases were HIV-negative. However, Fauci and the CDC did not investigate these cases. Instead they explained the problem by giving a new name for this disease: "Idiopathic CD4+ T cells lymphocytopenia (ICL)". Fauci stated in his book, cited above on page 1845, that the immunologic abnormalities in ICL are somewhat different from those of HIV infection. ICL patients often have decreases in CD8+ T cells and in B cells. However, on page 1,809-11 of the same book, Fauci stated that during the late stage of HIV infection, there is a significant reduction in the numbers of CD8+ T cells and the absolute number of circulating B cells may be depressed in primary infection.

In addition, Fauci's approach for solving the AIDS crisis is by defining conditions as they show up. The classic definition is the "long-term nonprogressors" term applied to people previously infected with HIV approximately 10 years or more, who have not yet showed any health problems. The number of these people as of January 1, 1997 was about 28,690. It only took one clinical case for me to see that the HIV-hypothesis has serious flaws and is incorrect. Yet A. Fauci, with billions dollars to spend, ignores thousands of cases (HIV-positive and HIV-negative) that obviously demonstrated the inconsistencies of the HIV hypothesis.

In 1981, the symptoms of patients diagnosed with AIDS are purely symptoms of chronic glucocorticoids use. Now the symptoms of patients with AIDS include bone marrow damage and damages of many organs (liver, kidneys, etc) as a result of the use of a wide range of powerful antiviral drugs and more steroids. In addition, Fauci and the CDC made the picture of AIDS even more complicated by adding cervical dysplasia and cervical cancer in 1993 to the cumulative list of AIDS-defining illnesses. These illnesses are also occurring in women with normal immune system functions, which has nothing to do with the hypothesis that HIV kills T cells. By taking this arbitrary step, AIDS has become a disease affecting the whole community; not only homosexuals and drug using men. This action has increased the fear in the community and also has increased the funding to the NIH as well as the sale of antiviral drugs.

The medical evidence presented in my book, along with the information presented in the Fauci's publications, indicate that A. Fauci, the Director of the AIDS program, and the CDC overlooked essential medical evidence, which indicates that HIV is not the cause of AIDS, antiviral medications do not work, and that drugs and malnutrition play important parts in the pathogenesis of AIDS. Furthermore, they have been changing their hypothesis and the stated incubation period for HIV, to make them fit new findings. AIDS in 1989 was much more complicated than the AIDS in 1984, prior to the approval of AZT. AIDS in 2001 has become more complicated than the AIDS in 1989, prior to the approval of steroid and protease inhibitors in the treatment of AIDS. As I stated earlier, treating an AIDS patient with corticosteroids and AZT is just like putting gasoline on a fire. AZT kills the stem cells in bone marrow that produce T cells and the glucocorticoids inhibit the functions of T cells; therefore, reducing their numbers in both thymus and blood circulation.

Today, we have about 2,000 active ingredients in our prescription and nonprescription drugs and thousands of chemicals in use in our workplace. Yet, toxicology is excluded from our health care industry. Physicians need to consult with toxicologists who have the expertise and the resources to evaluate toxic effects of chemicals used in workplace and side effects of medications. This approach can save lives and resources. This problem needs to be addressed urgently by our health care industry, among scientists, and in our government. We need to devote resources to solve current problems and not to create more problems. The war on AIDS is a classic example of mismanagement of great resources. Imagine if a toxicologist and a pathologist, with expertise in differential diagnosis, had been consulted about the few cases of AIDS that appeared in 1981Šmany lives and billion of dollars would have been saved! We must learn from this tragic and expensive lesson because we cannot afford to continue ignoring or repeating it.

14. Steven: How can people, who are interested in your book or have further questions, contact you?

They can check my website (http://www.toxi-health.com). My phone number is (707) 678-4484. Fax number is (707) 678-8505. My email: [email protected]
My company address: Toxi-health International, 150 Bloom Dr., Dixon, CA 95620
My book is also sold on Amazon.com.

15. Steven: Dr. Al-Bayati, I would like to thank you for your time and commitment to this article and more important to this issue of what AIDS is and is not. It has been truly wonderful working with you and I wish you all the success.

Thank you very much for asking me to do this very important interview. I hope that other reporters will take the same courageous action that you have taken to find the truth about what causes AIDS and by providing this vital information to the public. Your action certainly will save lives and vital resources. God bless you!






THE RETHINKER
 
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I agree

A buddy of mine was diagnosed with HIV 12 years ago after doing a prostitute. Well to this day 12 years later he is in awesome shape and he has been juicin for 8 of those years and man you would never tell he has HIV.
Oh yeah and he has never ever taken any of those harse medications for HIV either so there is proof right there!!!!!
 
HIV+ = AIDS

Please..., a word of caution to those that doubt that HIV does not cause AIDS. I have seen the effects of HIV drugs on those who are HIV+, and it is an overwhelming conclusion that they truly benefit the patient. Looks can be very deceiving.

I took the author's advice and checked the Aidsmyth website. It is to inform people about the myths surrounding the disease and NOT the "myth" that AIDS is caused by HIV. I did not find the article where MOLECULAR doctors supported such a ridiculous hypothesis, but I found many articles on how the drugs are of benefit and how they can best be distributed.

I too, have been HIV+ since 1989 and do not look sick. I am just starting to take serostim, and am hoping to build both my immune system and body. Those who are HIV+ are well advised to seek a doctor who is willing to combine conventional HIV drugs, steroids, and growth hormones.

The proof is in the numbers ie., CD4 COUNTS and VIRAL LOADS, and not in looks.
 
please listen to bbwannab....AIDS is caused by the HIV retrovirus...u can see it under the electron microscope in tissue specimens...study the writings and leadership of Dr. Jerome Groopman, Boston, one of the great pioneers in caring and i mean caring for AIDS and HIV+ patients..CD4 counts and viral load testing is very necessary...I've seen guys that were on death's door and wasted a few years ago who are now working and enjoying life....yes, the drugs have side effects but can be monitored or rescheduled as to the individual patient and steroids can do wonders and gh...the AIDS virus attacks the CD4 lymphocytes and upsets the immune system and later aggressive CD8 cells appear which really wipe out the immune system...this was known long before the new drugs were instigated...AZT is a drug of the past as a sole Rx...
 
There are many documented cases where people with HIV don't exhibit any noticeable signs of the AIDS virus, but I gotta go with the other guys here; HIV is a direct link to contracting AIDS -hundreds of thousand s of HIV+ patients have died from AIDS-but I haven't seen any evidence/data where someone has died from AIDS without it.
 
To all supporters of the "Aids Hypothesis":
You are hereby invited to read the following articles,published by the"Hiv/Aids chief dissenter Dr Peter Duesberg:
1."Results fall short for HIV Theory"(www.virusmyth.com/aids/data/pdinsight.htm)
2"Hiv is not the cause of Aids"(www.duesberg.com/ch2.html)
3."Is the Aids virus a science fiction/immunosuppressive behaviour,not HIV ,may be the cause of Aids(www.virusmyth.com/aids/data/pdpolicy.htm)
4."A challenge to the Aids establishment"(www.virusmyth.com/aids/data/pdbiotech87.htm)
5."Aids epidemiology:inconsistencies with human immunodeficiency virus and with infectious disease(www.virusmyth.com/aids/data/pdpnas91.htm)
6."The role of drugs in the origin of Aids"(www.virusmyth.com/aids/data/pdbiopharm.htm)

After reading /studying above articles,please repost your original responses with the necessary correction...
If you encounter any data that you feel or consider scientifically incorrect,please enlighten us with a point by point refutation...otherwise your comments are simply arbitrary and bear no scientific value...

The long term efficacy of the drug cocktails reffered by the proponents of the "Aids/HIV hypothesis,is questionable(see"protease inhibitors have no unique effect on cd4 cell counts"www.virusmyth.com/aids/new/reuthaartfail2.htm,Reuters 24 July 2000)if anything,their side effect tax the recipient immune system even further;Nearly 50% of cocktail users do not survive and ultimately die...In any event,none of the proponents of the accepted hypothesis claims that the protease inhibitors constitue a cure for Aids...

The purpose of the virusmyth.com site is to inform of the dissenting opinion which states clearly that"HIV DOES NOT cause Aids"Here's a quote from "Aids myth buster"
Myth #2:'HIV virus causes Aids"
15 years of research has failed to prove the mechanism.In fact,HIV may be an effect of disease...not a cause"The holy grail of Aids science is to eventually discover the means by which an unproven HIV virus causes the 30 different diseases labelled as Aids"....

Obviously,one of the respondents to the original aids controversy post,managed to reflect his own subjective thoughts right onto the computer screen....

Frankly, words such as "ridiculous hypothesis",simply indicate a level of indignation,reflected towards the dissenting opinion,which the CDC and the money hungry pharmaceutical companies wish to choke,instead of refute point by point...

"There is very little direct evidence for the presence of the HIV virus in persons with aids.There is not a single report of virus titer from an aids patient.Indeed,virus can only be isolated from 50% of symptomatic and asymptomatic carriers;and then O N LY by techniques originally designed to activate latent viruses..viral genomes have only been detected in about 15% of persons who test positive for HIV antibody.In these persons about 1 proviral genome is found in 100-1000 suceptible lymphocytes.In other words,85% of antibody positive persons carry either less provirus than this or NONE at all...even in those patients who are HIV infected the virus is usually totally dormant,once immune defciency is aquired and Aids appear...The active,infectious form of the virus CANNOT be issolated from the blood or other tissues of most HIV positive aids patients.Even the dormant form of HIV,resting quietly inside infected cells,can be found in only one of every 1000 T cells in the patient"
In light of the above F A C T S the "proof is the numbers"claimed by one of the respondents is laughable....thus, low CD4 counts indicate patogenic viral activity,NOT OF HIV which can hardly be traced,thanks to their "positive"hiv antibodies,but rather to that of the specific infectious viruses that cause any of the 30 diseases that comprise the syndrom known as "Aids'...same applies to "viral load"...the load that is measured is not that of the HIV virus,but that of the specific virus that causes any of the ailments that are refered as aids..

Finally...please read and respond to above articles...you owe it to yourselves...whether you become a dissenter or not,makes no difference...but your decision as far as your own ideal treatment,will be based upon thourough understanding of BOTH hypothesis...

may you all live long an healthy lives
 
This is year 2001

Please update your sources. The one was from 1988, over 13 years old.

Today in 2001, we know we can slow the progression of symptomatic HIV or "full blown AIDS", by nutrition, exercise, HIV drug therapy, growth hormones, and steroids. There is no cure!

Also you may want to include sources written by authors other than the same professor Dr. Duesberg.
 
BBwannab is clearly referring to"HIV is not the cause of Aids"(dated July 29nth 1988)...
However...relative "antiquity"of a scientific article is not by itself,sufficient to dispute its validity and accuracy TODAY...Science does not find fault with previous discoveries and findings just because they are "old"...A fact is a fact,regardless of its age...

Duesberg,along with 300 other top notch scientists,still attest to the scientific validity and accuracy of their original hypothesis reflected in the 1988 article(which mainly discusses "causation of Aids"and not newly advanced drug treatments.).
No new data is available as of yet to discredit or disqualify the dissenting group explanation to Aids...
The CDC has been challenged on numerous occasions to rebut Duesberg's conclusions,in a point by point format,but its response was muted,faint and consisted mainly of smear campaign,and threats against any dissent from the "accepted"HIV/hypothesis

BBwannab(and all ),are urged to read and reread the 1988 article on its entierty(and not just the "title" and "date")and point out any information they deem scientifically in error..
Once more...If you don't challenge the facts presented, with counter scientific data, to indicate otherwise,you're expressing an arbitrary opinion lacking any scientific validity...

I agree that " full blown Aids"can be slowed by proper nutrition,exercise growth hormones and steroids...
I disagree with the assertion that "hiv drug therapy"can slow the progression of Aids from a "long term"perspective(see,"protease inhibitors have no unique effect on CD4 cell counts"www.virusmyth.com/aids/new/reuthaartfail2.htm)

there's a plethora of other scientists,aside of Duesberg,that published numerous articles to refute,and rebut the currently,widely held HIV/Aids Hypothesis...
Some of their names are provided below...use your search engines to find additional information about their writings...

1.Dr Walter Gilbert,and Dr Roger Cunningham were mentioned in my 1st post
2.Dr Richard Strohman-a biologist for 35 years,and Professor Emeritus of Cell Biology at the University of California at Berkeley
3.Dr.Alfred Hassig-Emeritus Professor in immunology at the University of Bern,former Director Swiss Red Cross blood banks.
4.Dr.Henk Loman Professor of Biophysical Chemistry at the Free University in Amsterdam..
5.Dr Harry Rubin-Professor of Molecular and Cell Biology,University of California at Berkeley...
6.Dr Fabio Franci-Specialist in Preventive Medicine and infectious Diseases Trieste Italy.
7.Dr.Gordon Stewart-Emiritus Professor of Epidemiology University of Glasgow..
8.Dr Michael Lange-Head of Auds Programme St.Lukes New York
9.Dr Stefan Lanka-Virologist Germany
10.Dr.Joseph Sonnabend

It's your health on the line here,not mine...I just think you owe it to yourself to know and learn both opinions
before you decide what course of action to take....
Be Blessed



THE GROUP




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The Group for the Scientific Reappraisal of the HIV-AIDS Hypothesis came into existence as a group of signatories of an open letter to the scientific community. The letter (dated June 6, 1991) has been submitted to the editors of Nature, Science, The Lancet and The New England Journal of Medicine. All have refused to publish it. In 1995 The Group was able to get a letter published in Science.

You can also sign the statement below online.

To the editor:

It is widely believed by the general public that a retrovirus called HIV causes the group diseases called AIDS. Many biochemical scientists now question this hypothesis. We propose that a thorough reappraisal of the existing evidence for and against this hypothesis be conducted by a suitable independent group. We further propose that critical epidemiological studies be devised and undertaken.


Signatories:


Charles A. Thomas, Jr. Ph.D. (Mol. Biologist, Pres. Helicon Fnd., San Diego, CA)
Harvey Bialy, Ph.D. (Editor Bio/Technology, New York, NY)
Harry Rubin, D.V.M. (Prof. Cell Biology, Univ. Cal. Berkeley, CA)
Richard C. Strohman, Ph.D. (Prof. Cell Biology, Univ. Cal. Berkeley, CA)
Phillip E. Johnson (Prof. Law, Univ. Cal. Berkeley, CA)
Gordon J. Edlin, Ph.D. (Prof. Biochem. & Physics, Univ. Hawaii, HI)
Beverly E. Griffin, Ph.D. (Dir. Dept. Virology, Royal Postgrad. Med. School, London, UK)
Robert S. Root-Bernstein (Prof. Physiology, Michigan State Univ., East Lansing, MI)
Gordon Stewart, M.D. (Emeritus Prof. Public Health, Epidemiologist, Isle of Wight, UK)
Carlos Sonnenschein, M.D. (Tufts Univ., Medicine, Boston, MA)
Richard L. Pitter, Ph.D. (Dessert Research Inst., Univ. Nevada System, Reno NV)
Nathaniel S. Lehrman, M.D. (Psychiatrist, Roslyn, NY)
John Lauritsen (Author 'Poison by Prescription', New York, NY)
William Holub, Ph.D. (Biochemist, Live Sciences Inst. New York, NY)
Claudia Holub, Ph.D. (Biochemist, Live Sciences Inst. New York, NY)
Frank R. Buianouckas Ph.D. (Prof. Mathematics, Cuny, Bronx, NY)
Philip Rosen, Ph.D. (Prof. Physics, Univ. Mass. Amherst, MA)
Steven Jonas, M.D. (Prof. Preventive Medicine, Suny Stony Brook, NY)
Bernard K. Forscher, Ph.D (Ret. Editor Proc. Nat. Acad. Sci., Santa Fe, NM)
Kary B. Mullis, Ph.D. (Biochemist, PCR inventor, Consultant, La Jolla, CA.)
Jeffrey A. Fisher, M.D. (Pathologist, Mendham, NJ)
Hansueli Albonico, M.D. (General Practitioner, Langnau, Switzerland)
Robert Hoffman, Ph.D. (Prof. Dept. Pediatrics Univ. Cal. Med. School, San Diego, CA)
Timothy H. Hand, Ph.D. (Dept. Psychology, Oglethorpe Univ. Atlanta, GA)
Eleni Eleopulos, M.D. (Royal Perth Hospital, Perth, West Australia)
Robert W. Maver, F.S.A., M.A.A. (Dir. Research, Mutual Benefit Life, Kansas City, MO)
Ken N. Matsumura, M.D. (Chairman Alin Foundation & Research Inst., Berkeley, CA.)
David T. Berner, M.D. (Condon, MT)
Theodor Wieland, Ph.D. (Max Planck Institut, Heidelberg, Germany)
Joan Shenton, M.A. (Meditel, London, UK)
John Anthony Morris, Ph.D. (Biochemist, Bell of Atari College Park, MD)
Sungchul Ji, Ph.D. (Prof. Pharmacology & Toxicology, Rutgers Univ., Piscataway, NJ)
 
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CONTROVERSY



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Dr. Kary Mullis, Biochemist, 1993 Nobel prize for chemistry:
"If there is evidence that HIV causes AIDS, there should be scientific documents which either singly or collectively demonstrate that fact, at least with a high probability. There is no such document." (Sunday Times (London) 28 nov. 1993)


Dr. Serge Lang, Professor of Mathematics, Yale University:
"I do not regard the causal relationship between HIV and any disease as settled. I have seen considerable evidence that highly improper statistics concerning HIV and AIDS have been passed off as science, and that top members of the scientific establishment have carelessly, if not irresponsible, joined the media in spreading misinformation about the nature of AIDS." (Yale Scientific, Fall 1994)


Dr. Harry Rubin, Professor of Molecular and Cell Biology, University of California at Berkeley:
"It is not proven that AIDS is caused by HIV infection, nor is it proven that it plays no role whatever in the syndrome." (Sunday Times (London) 3 April 1994)


Dr. Richard Strohman, Emeritus Professor of Cell Biology at the University of California at Berkeley:
"In the old days it was required that a scientist address the possibilities of proving his hypothesis wrong as well as right. Now there's none of that in standard HIV-AIDS program with all its billions of dollars." (Penthouse April 1994)


Dr. Steven Jonas, Professor of Preventive Medicine, Suny Stony Brook, NY:
"Evidence is rapidly accumulating that the original theory of HIV is not correct." (Sunday Times (London) 3 April 1994)


Dr. Harvey Bialy, Molecular Biologist, editor of Bio/Technology:
"HIV is an ordinary retrovirus. There is nothing about this virus that is unique. Everything that is discovered about HIV has an analogue in other retroviruses that don't cause AIDS. HIV only contains a very small piece of genetic information. There's no way it can do all these elaborate things they say it does." (Spin June 1992)


Dr. Roger Cunningham, Immunologist, Microbiologist and Director of the Centre for Immunology at the State University of New York at Buffalo:
"Unfortunately, an AIDS 'establishment' seems to have formed that intends to discourage challenges to the dogma on one side and often insists on following discredited ideas on the other." (Sunday Times (London) 3 April 1994)


Dr. Gordon Stewart, Emeritus Professor of Public Health, University of Glasgow:
"AIDS is a behavioural disease. It is multifactorial, brought on by several simultaneous strains on the immune system - drugs, pharmaceutical and recreational, sexually transmitted diseases, multiple viral infections." (Spin June 1992)


Dr. Alfred Hässig, (1921-1999), former Emeritus Professor of Immunology at the University of Bern, and director Swiss Red Cross blood banks:
"The sentence of death accompanying the medical diagnosis of AIDS should be abolished." (Sunday Times (London) 3 April 1994)


Dr. Charles Thomas, former Professor of Biochemistry, Harvard and John Hopkins Universities:
"The HIV-causes-AIDS dogma represents the grandest and perhaps the most morally destructive fraud that has ever been perpetrated on young men and women of the Western world." (Sunday Times (London) 3 April 1994)


Dr. Joseph Sonnabend, New York Physician, founder of the American Foundation for AIDS Research (AmFAR):
"The marketing of HIV, through press releases and statements, as a killer virus causing AIDS without the need for any other factors, has so distorted research and treatment that it may have caused thousands of people to suffer and die." (Sunday times (London) 17 May 1992)


Dr. Andrew Herxheimer, Emeritus Professor of Pharmacology, UK Cochrane Centre, Oxford:
"I think zidovudine [AZT] was never really evaluated properly and that its efficacy has never been proved, but it's toxicity certainly is important. And I think it has killed a lot of people. Especially at the high doses. I personally think it not worth using alone or in combination at all." (Continuum Oct. 2000)


Dr. Etienne de Harven, Emeritus Professor of Pathology, at the University of Toronto:
"Dominated by the media, by special pressure groups and by the interests of several pharmaceutical companies, the AIDS establishment efforts to control the disease lost contact with open-minded, peer-reviewed medical science since the unproven HIV/AIDS hypothesis received 100% of the research funds while all other hypotheses were ignored." (Reappraising AIDS Nov./Dec. 1998)


Dr. Bernard Forscher, former editor of the U.S. Proceeding of the National Academy of Sciences:
"The HIV hypothesis ranks with the 'bad air' theory for malaria and the 'bacterial infection' theory of beriberi and pellagra [caused by nutritional deficiencies]. It is a hoax that became a scam." (Sunday Times (London) 3 April 1994)




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Well, I guess that all of those people in africa dying of aids should stop taking all of those highly expensive aids drugs too.

Hey did you know that if you jerk off you go blind?
 
My personal belief is that HIV affects each victim differently and that contracting the virus usually but not always leads to the recipient displaying AIDS symptoms-but it IS always present in full-blown AIDS cases.If you are putting forward a conspiracy theory that falsely blames HIV for the AIDS epidemic, then you must have some identity of the real culprit causing AIDS in millions around the world. The most popular seems to be government bio-chemical testing gone bad.And rather than slathering us with another ream of documents, just simply state your theory scenario.
 
Champ...
"Aids"is a collective name for nearlly 30 different diseases that are immune deficiency related... of which some,agreeably induced or caused by malnutrition and poor sanitary conditions...

With the discovery of hiv,and in the presence of hiv antibodies,all previous diseases that were previously and correctly attributed to the causes above are now blamed on a harmless virus..for the purpose of satiating the greed and ever growing and expanding pockets of the money hungry pharmaceutical companies...
All retroviruses are harmless...why should hiv be any different

I can't stress enough the importance of reading the articles mentioned in my earlier posts...and if you find anything there that is worth joking about,I'll laugh along with you...
Be well
 
Proviron
Wrong again...
HIV can hardly be detected in full blown aids patients due to the fact that their blood already contain the hiv antibodies that elimante its presence to a virtual zero.
your statement therefore that:"HIV IS always present in full-blown AIDS cases".is scientifically in error....

I wrote it before and I'll copy it for you again:

"There is very little direct evidence for the presence of the HIV virus in persons with aids.There is not a single report of virus titer from an aids patient.Indeed,virus can only be isolated from 50% of symptomatic and asymptomatic carriers;and then O N LY by techniques originally designed to activate latent viruses..viral genomes have only been detected in about 15% of persons who test positive for HIV antibody.In these persons about 1 proviral genome is found in 100-1000 suceptible lymphocytes.In other words,85% of antibody positive persons carry either less provirus than this or NONE at all...even in those patients who are HIV infected the virus is usually totally dormant,once immune defciency is aquired and Aids appear...The active,infectious form of the virus CANNOT be issolated from the blood or other tissues of most HIV positive aids patients.Even the dormant form of HIV,resting quietly inside infected cells,can be found in only one of every 1000 T cells in the patient"

If you have any scientific data that contradicts the above quotation,please enlighten us all and post here..

No one is claiming "a conspiracy theory that falsely blames HIV for the AIDS epidemic"..there is no conspiracy here ,governmental,or otherwise,just a false
diagnosis of the cause,and a failure of an hypothesis to provide biochemical proof for the havoc hiv is being accused of...
The dissidents' hypothesis suggests prolong usage of drugs as the culprit for aids.
See:
1.The role of drugs in the origin of Aids"(www.virusmyth.com/aids/data/pdbiopharm.htm)

2.Is the Aids virus a science fiction/immunosuppressive behaviour,not HIV ,may be the cause of Aids(www.virusmyth.com/aids/data/pdpolicy.htm)
Do yourself a favour...
read the articles!!!!!!!!!!!!!!!!!!!
 
asteroth ..knows what hes talking about and I mentioned this along time ago as well...HIV DOES NOT CAUSE AND HAS NEVER BEEN SHOWN TO CAUSE AIDS...THIS IS FACT , AS WELL AS AIDS NOW IS A 8 BILLION DOLLAR A YEAR INDUSTRY FOR THE MDS....HIV WAS A ROUND LONG BEFORE FORMALLY KNOW AS HLTV3...ROBERT GALLO CHANGED THE NAME IN THE EARLY 80S.....THIS WAS A LOW LIFE RETROVIROLOGIST WHO TRIED TO BLAME CANCER ON A VIRUS IN THE 60S, AZT WAS ADMINISTERED TO THJESE PATIENTS WHO LATER DIED OF GRANULOCYTOPENIA..ECT..AIDS AS YOU CALL IT TODAY....AIDS IS A COMBINATION OR 1 OF MANY ALREADY KNOW DISEASES....QUESTION FOR THE IGNORANTS???????
PATIENT 1)..DRUG USER , HOMELESS, POOR NUTRITION..IS HIV -, WHAT DID HE DIE FROM? TB
PATIENT 2).. DRUG USER, HOMELESS, POOR NUTRITION,..IS HIV +...WHAT DID HE DIE FROM???AIDS

THEY DIED OF THE SAME THING TB..BUT LEAVE IT TO THE MDS TO CREATE THEN DESTROY..
AIDS....WHAT CAUSES YOUR IMMUNE SYSTEM TO BREAKDOWN
1)DRUGS
2)POOR NUTRITION
3)CHEMOTHERPY(AZT)
4 ) RADITION
5)NEGATIVE ATTITIUDE
MDS ARE INVOLVED IN ALL THE ABOVE...
BRO YOU 100% RIGHT..i COULD GO ON....i SPOKE WITH ROBERT WILNER BEFORE HE DIED..HE WAS ON THE VERGE OF THE SECRET, BUT WAS MURDERED BY THE PHARMACUETICAL COMPANIES...FACT
 
OK I read the articles and I totally disagree with them. Duesberg was proven wrong by Blattner. And Duesberg didnt post Blattners response to his last response to save face...i mean it is his website.
The only good point that Duesberg makes

"I wonder whether there is truly any disease that, in the presence of antibody to HIV, would not be called AIDS."

this was in his response to blattner after blattner totally proved him wrong.

I mean listen to this guys idiotic response to blattner

"They claim that "interruption of HIV infection almost completely prevented the further appearance of blood-transfusion-associated AIDS." However, according to the CDC, transfusion-associated AIDS cases in adults have doubled to 752 cases and pediatric cases tripled to 68 in the year ending May 1988 compared to the previous year (1). This happened 3 years after antibody-positive transfusions were reduced 40-fold with the AIDS test (9). The steep increase in transfusion AIDS cases despite the great reduction of HIV-contaminated transfusions argues directly against HIV as the cause of AIDS."

Is this guy really a doctor.....the AIDS stats increased beucase it takes years to develop AIDS from HIV...3 years after...what is that...You will have to wait about 10 years (with current medicine) before the rate begins to decline.

I would love to pick apart everything but i must be going
 
Wow...someone is finally reading Duesberg's articles...hopefully the trend will continue...

Anyway, to claim,as Champ does, that"Duesberg didnt post Blattners response to his last response to save face"...is an outright lie!!!

Not that Champ is deliberately lying...but if he is not misinformed,he is certainly blessed with selective reading...
Unless Champs is reffering to some hidden from sight,yet to be published article that he his the sole person that knows of its existence...and somehow
simply forgot to inform us where he saw it....

For his , and anybody else's convenience I have copied the articles ,including the "accidentally ommited,or missing"
"Duesberg's Response to Blattner and Colleagues"
It is Duesberg's response to Blattner that remained unanswered and not the other way around...

Note the pompous arrogance with which Duesberg's articles are being reffered to...
"listen to this guys idiotic response"..." Is this guy really a doctor....."
almost reminicent of the Catholic church approach to Gallileo and Copernicus for suggesting that the earth circles the sun,and not the other way around...

Ridicule,and mockery ,instead of point by point refutation of the dissenting hypothesis,are a clear reflection of the hiv camps' frastration for their inability to produce a shred,and iota, of a biochemical evidence,which will demonstrate unequivocally that hiv causes the immunodeficiency effects,attributed to it,or their utter failure to produce a cure inspite of the billions of dollars in research money at its disposal...

Duesberg was never proven wrong by Blattner or by anyone else, for that matter...even blattner admits that"Knowledge of the cause of a disease (etiology) is important for control of that disease and gives a basis for understanding the pathology of the disease. However, knowing the cause of a disease does not mean that there is complete understanding of its pathology. Discovering the pathogenetic mechanisms of HIV in AIDS is a major focus for research. ..."

In any event when champs writes:"this was his response to blattner after blattner totally proved him wrong"he expresses his own arbitrary ,opinion...nothing more..the dissenting hypothesis by Duesberg and others lives on cause it was never scientifically disproven
and tha is an undisputable fact...

The response that Champs is quoting, makes perfect sense,but one has to know and understand what their reading...
Let me rephrase that particular quotation:
"since the CDC took measures to control through HIV testing the nations blood supply so as to prevent further hiv infection (by blood transfusion)...How is it possible than that inspite of those measures"transfusion-associated AIDS cases in adults have doubled to 752 cases and pediatric cases tripled to 68 in the year ending May 1988 compared to the previous year (1). This happened 3 years after antibody-positive transfusions were reduced 40-fold with the AIDS test (9). The steep increase in transfusion AIDS cases despite the great reduction of HIV-contaminated transfusions argues directly against HIV as the cause of AIDS."
To this Champs responds:"Is this guy really a doctor.....the AIDS stats increased beucase it takes years to develop AIDS from HIV...3 years after...what is that...
Champ is wrong...wrong on all counts...
Aids doesn't necessarily take "years" to develop...
Here is a quote from:"Aids the Facts"by John Langone Page 11:
"But while the time that elapses between infection and presence of antibodies may be charted,the lenght of time that an infected individual will remain free of symptoms seems to v a r y w i l d l y from DAYS,or WEEKS, to many years DEPENDING on how the infection WAS AQCUIRED"
Note that the above reference by Duesberg, is to transfusion assosiated contraction of Aids, and not to other ways of infection,and bearing in mind that the time of appearance of Aids symptoms seems to vary it is,odd,how inspite of the cautionary measures the cdc takes with our blood supply....t r a n s f u s i o n related Aids is still up..
Obviously Duesberg knows exactly what is talking about
He makes perfect sense...
Blattner has evaded and avoided Duesberg sharp tongue for quite some time...Let him come out of the closet!!!!!!



HIV Is Not the Cause of AIDS By Peter H.Duesberg

Science, Vol. 241, pp. 514-517, July 29, 1988. Human immunodeficiency virus (HIV) is not the cause of AIDS because it fails to meet the postulates of Koch and Henle, as well as six cardinal rules of virology. 1) HIV is in violation of Koch's first postulate because it is not possible to detect free virus (1, 2), provirus (3-5), or viral RNA (4, 6, 7) in all cases of AIDS. Indeed, the Centers for Disease Control (CDC) has established guidelines to diagnose AIDS when all laboratory evidence for HIV is negative (8). 2) In violation of Koch's second postulate, HIV cannot be isolated from 20 to 50% of AIDS cases (1, 9-11). Moreover, "isolation" is very indirect. It depends on activating dormant provirus in millions of susceptible cells propagated in vitro away from the suppressive immune system of the host. 3) In violation of Koch's third postulate, pure HIV does not reproduce AIDS when inoculated into chimpanzees or accidentally into healthy humans (9, 12, 13). 4) In contrast to all pathogenic viruses that cause degenerative diseases, HIV is not biochemically active in the disease syndrome it is named for (14). It actively infects only 1 in 104 to > 105 T cells (4, 6, 7, 15). Under these conditions, HIV cannot account for the loss of T cells, the hallmark of AIDS, even if all infected cells died. This is because during the 2 days it takes HIV to replicate, the body regenerates about 5% of its T cells (16), more than enough to compensate for losses due to HIV. 5) It is paradoxical that HIV is said to cause AIDS only after the onset of antiviral immunity, detected by a positive "AIDS test," because all other viruses are most pathogenic before immunity. The immunity against HIV is so effective that free virus is undetectable (see point 1), which is why HIV is so hard to transmit (9, 12, 13). The virus would be a plausible cause of AIDS if it were reactivated after an asymptomatic latency, like herpes viruses. However, HIV remains inactive during AIDS. Thus the "AIDS test" identifies effective natural vaccination, the ultimate protection against viral disease. 6) The long and highly variable intervals between the onset of antiviral immunity and AIDS, averaging 8 years, are bizarre for a virus that replicates within 1 to 2 days in tissue culture and induces antiviral immunity within 1 to 2 months after an acute infection (9, 17). Since all genes of HIV are active during replication, AIDS should occur early when HIV is active, not later when it is dormant. Indeed, HIV can cause a mononucleosis-like disease during the acute infection, perhaps its only pathogenic potential (9, 17). 7) Retroviruses are typically not cytocidal. On the contrary, they often promote cell growth. Therefore, they were long considered the most plausible viral carcinogens (9). Yet HIV, a retrovirus, is said to behave like a cytocidal virus, causing degenerative disease killing billions of T cells (15, 18). This is said even though T cells grown in culture, which produce much more virus than has ever been observed in AIDS patients, continue to divide (9, 10, 18). 8) It is paradoxical for a virus to have a country-specific host range and a risk group-specific pathology. In the United States, 92% of AIDS patients are male (19), but in Africa AIDS is equally distributed between the sexes, although the virus is thought to have existed in Africa not much longer than in the United States (20). In the United States, the virus is said to cause Kaposi's sarcoma only in homosexuals, mostly Pneumocystis pneumonia in hemophiliacs, and frequently cytomegalovirus disease in children (21). In Africa the same virus is thought to cause slim disease, fever, and diarrhea almost exclusively (22, 23). 9) It is now claimed that at least two viruses, HIV-1 and HIV-2, are capable of causing AIDS, which allegedly first appeared on this planet only a few years ago (20). HIV-1 and HIV-2 differ about 60% in their nucleic acid sequences (24). Since viruses are products of gradual evolution, the proposition that within a few years two viruses capable of causing AIDS could have evolved is highly improbable

Blattner and Colleagues Respond to Duesberg

Biology is an experimental science, and new biological phenomena are continually being discovered. For example, recently some RNA molecules were shown to act as enzymes, ribozymes, even though biochemistry text books state that all enzymes are proteins. Thus, one cannot conclude that HIV-1 does or does not cause AIDS from Duesberg's "cardinal rules" of virology. In fact, the Henle-Koch postulates of 1840 and 1890 were formulated before the discovery of viruses. They are a useful historical reference point, but were not regarded as rigid criteria by Koch himself and should not be so regarded today (1). Duesberg's description of the properties of viruses is in error and provides no distinction between knowing the cause of a disease, that is, its etiology, and understanding the pathogenesis of this disease. Duesberg is noted for his discoveries about the viral oncogene src. There is no question that the expression of this gene in chicken fibroblasts results in sarcomas. However, no one can yet explain how the expression by the src oncogene of an altered tyrosine protein kinase results in a cell becoming neoplastic. Similarly, there are many unanswered questions about the pathogenesis of AIDS, but they are not relevant to the conclusion that HIV causes AIDS. Duesberg presents six (or nine) cardinal rules for viruses. Most are not relevant to the question of etiology and are misleading or wrong about viruses in general and HIV in particular. 1-2) It was formerly true that evidence for the presence of HIV-1 could not be found in all AIDS patients. But the overwhelming seroepidemiologic evidence pointing toward HIV as the cause of AIDS spurred research to improve the sensitivity of the detection methods. Better methods of virus isolation now show that HIV infection is present in essentially all AIDS patients (2). The CDC definition of AIDS has been revised several times as new knowledge has become available and will undoubtedly be revised again. The 1981 CDC definition of AIDS did not mention HIV, since no strain of HIV was known until 1983. Many cases of AIDS are diagnosed on clinical grounds alone because of the lack of availability or expense of HIV-1 antibody testing or because HIV testing is discouraged in some communities. Thus, rates of confirmation of AIDS cases by HIV testing in the United States vary geographically as reflected in CDC surveillance statistics. 3) It is true that HIV does not cause AIDS in chimpanzees. Most viruses are species-specific in host range and in capacity to produce disease. For example, herpes B virus, yellow fever virus, and dengue virus cause serious diseases in humans, but produce no disease symptoms during infection in many species of monkeys (3). Moreover, a virus closely related to HIV, simian immunodeficiency disease virus or SIV, causes an AIDS-like disease in rhesus macaques, but seldom, if ever, causes immunodeficiency in African Green monkeys (4, 5). HIV-1 does indeed cause AIDS when inoculated into humans with no underlying medical condition. Accidental needlestick injuries with HIV-contaminated needles have resulted in HIV seroconversion and then clinical AIDS (6). 4) It is true that HIV infects only a small fraction of T cells. However, about 15% of the macrophage and monocyte cells from AIDS patients are positive for a viral protein, p24 (7), and the high concentration of this protein in the blood of AIDS patients indicates virus activity (8). The exact mechanism of CD4 cell depletion in AIDS patients is not known, but several indirect mechanisms are known by which HIV can cause CD4 cell depletion in laboratory studies and could operate in vivo. 5-6) Many viruses are highly pathogenic after evidence of immunity appears. For example, reactivated herpes zoster virus causes shingles, and reactivated herpes simplex virus causes local lesions as well as lethal necrotizing encephalitis; moreover, hepatitis B virus causes chronic active hepatitis, equine infectious anemia virus causes anemia, and visna virus causes central nervous system degeneration after the appearance of specific neutralizing antibodies (3, 9). (The last two viruses are lentiretroviruses as is HIV.) These diseases also can have long and variable latent periods. 7) It is true that some retroviruses, in particular, the highly oncogenic retroviruses of the kind that Duesberg has worked with, are not cytocidal and promote cell growth. Most retroviruses have no effect on cell growth (9, 10). However, Rous-associated virus-2, spleen necrosis virus, visna virus, and HIV kill infected cells in culture and can establish a chronic stage of infection in which surviving infected cells continue to divide (11). 8) It was apparently "paradoxical for a virus to have a country-specific host range and a risk group-specific pathology." The properties of HIV resolved this paradox because the distribution of AIDS was found to mirror the distribution of HIV. The nature of the spread of the virus and the type of the AIDS-related clinical syndrome depend on social and environmental factors. Sexually active gay men and parenteral drug abusers were the first conduit for spread of HIV in the United States, whereas in some developing countries of Africa, young heterosexually active men and women were the major focus of spread. It is common for life-style to be a major determinant for the spread of an infectious agent. For example, until a vaccine became available, hepatitis B virus was clustered among the same U.S. populations that are now infected by HIV. The underlying pathology in AIDS is immune deficiency. The nature of the opportunistic agents that invade the susceptible host is a function of which agents are most prominent in a particular population. For example, in the United States Pneumocystis is most prominent in affluent gay men, while human mycobacterial infections and toxoplasmosis are more frequent in socially disadvantaged Caribbean immigrants. Other agents, such as Cryptococcus, are more prominent in developing countries. 9) It is true that there are two viruses that cause human AIDS, HIV-1 and HIV-2. The origin of these HIVs is an interesting scientific question that is not relevant to whether or not HIV causes AIDS. Since a primate lentiretrovirus also causes an AIDS-like disease in rhesus monkeys, just as a cat lentiretrovirus, feline immunodeficiency virus, causes an AIDS-like disease in cats (12), one can suggest either that there is strong selection among retroviruses for this kind of pathology (13) or that the virus ancestor to HIV already had this property. In favor of the first hypothesis is the existence of feline, murine, and primate AIDS caused by retroviruses in a different subfamily from the lentiretroviruses (14). In summary, although many questions remain about HIV and AIDS, a huge and continuously growing body of scientific evidence shows that HIV causes AIDS. References and Notes: A.S. Evans, Yale J. Biol. Med. 49, 175 (1976). References 1 and 2 from Duesberg report isolation of HIV-1 from 100% of AIDS patients; I.S.Y. Chen (UCLA) reports isolation of HIV-1 from 100% of AIDS patients (personal communication); R.C. Gallo, M. Popovic, S. Z. Salahuddin, S. Gardner, and co-workers now isolate HIV-1 from more than 90% of AIDS patients. Duesberg's references 5 and 7 do not report on AIDS patients at all. B.N. Fields et al., Eds. Virology (Raven, New York, 1985); F. Fenner, B.R. McAuslan, C.A. Mims, J. Sambrook, D.O. White, The Biology of Animal Viruses (Academic Press, New York, ed. 2, 1974). N.L. Letvin et al., Science 230, 71 (1985). Duesberg's reference 13 deals only with HIV-1 transmission, not disease occurrence. AIDS Program, Hospital Infections Branch, CDC, Morbid. Mortal. Weekly Rep. 37, 229 (1988). This pattern of AIDS development following HIV-1 seroconversion is the same as that seen for pediatric and adult blood transfusion cases and mother-to-child transmission, and in a multitude of prospective studies of gay men, hemophiliacs, and other populations in developed and developing countries. S. Crowe, J. Mills, I. Kirihara, P. Lakas, M. McGrath, Abstracts of the Fourth International Conference on AIDS, Stockholm (1988). G.G. Jackson et al., Ann. Int. Med. 108, 175 (1988). Macrophages and monocytes and not T cells appear to be the major reservoir of HIV infection in humans. N. Teich, I. Wyke, T. Mak, A. Bernstein, W. Hardy, in RNA Tumor Viruses, Molecular Biology of Tumor Viruses, R. Weiss, N. Teich, H. Varmus, J. Coffin, Eds. (Cold Spring Harbor Laboratory, Cold Spring Harbor, NY, ed. 2, 1982), pp. 785-998. H.M. Temin, Rev. Infect. Dis. 10, 399 (1988). --. and V.K. Kassner, J. Virol. 13, 291 (1974); J. Gen. Virol. 27, 267 (1975). N.C. Pedersen et al., Science 235, 790 (1987). H.M. Temin, in Concepts in Viral Pathogenesis, A.L. Notkins, M.B.A. Oldstone, Eds. (Springer-Verlag, New York, 1988), vol. 3. D.M. Mosier, Immunol. Invest. 15, 233 (1986). HIV Causes AIDS W. Blattner, R. C. Gallo, H. M. Temin AIDS, a new disease, was first recognized in 1981, clustered in male homosexuals, intravenous drug abusers, and hemophiliacs in the United States and among sexually active heterosexuals in some countries of equatorial Africa. Human immunodeficiency virus (HIV) was first discovered in 1983 and was definitively linked in 1984 to AIDS patients and to groups whose members were at high risk for developing AIDS. The serological test for antibodies to HIV was developed at this same time and showed that HIV infection in the United States was concentrated in those populations at highest risk for AIDS, namely, male homosexuals, intravenous drug abusers, and hemophiliacs (1). The strongest evidence that HIV causes AIDS comes from prospective epidemiological studies that document the absolute requirement for HIV infection for the development of AIDS. It has been shown for every population group studied in the United States and elsewhere that, in the years following the introduction of HIV and subsequent seroconversion of members of that population, the features characteristic of progressive immunodeficiency emerge in a predictable sequence resulting in clinical AIDS (2-4). Furthermore, other epidemiological data show that AIDS and HIV infection are clustered in the same population groups and in specific geographic locations and in time. Numerous studies have shown that in countries with no persons with HIV antibodies there is no AIDS and in countries with many persons with HIV antibodies there is much AIDS (3). Additionally, the time of occurrence of AIDS in each country is correlated with the time of introduction of HIV into that country; first HIV is introduced, then AIDS appears. It is also noteworthy that HIV infection, and not infection with any other infectious agent, is linked to blood transfusion-associated AIDS (5). Similarly, in HIV-infected pregnant women, mother-to-child perinatal transmission of HIV occurs approximately 50% of the time, and over 95% of HIV-infected infants develop AIDS by 6 years, while their uninfected siblings never develop AIDS (3, 6). Support for the linkage of HIV infection and AIDS comes as well from the results of public health interventions where interruption of HIV infection almost completely prevented the further appearance of AIDS in blood transfusion recipients (4). After the introduction of the HIV antibody screening test in the United States, the transmission of HIV in the blood supply in the United States was reduced from as high as 1 in 1,000 infected units in some high risk areas to less than an estimated 1 in 40,000 units countrywide (7). (The recently recognized cases of virus transmission by blood transfusion are due to donors being missed by current antibody screening tests during the window of seroconversion. There is a period of about 4 to 8 weeks in which newly HIV-infected persons are capable of transmitting HIV, but have not yet developed antibodies.) As a result of the decrease in blood transfusion-associated transmission of HIV, the incidence of blood transfusion-associated AIDS among U.S. newborns showed a decline (4). Thirteen of the cases of blood transfusion-associated seroconversion identified since the start of blood bank screening were recently investigated (7). In one of these cases, the recipient of one unit of blood was one of a pair of fraternal twins. This baby seroconverted and developed AIDS without any other risk factor. Her twin and her mother received no blood products, developed no HIV antibodies, and remained healthy. The blood donor became HIV seropositive and developed AIDS. Scientists conclude that a virus causes a disease if the virus is consistently associated with the disease and if disruption of transmission of the virus prevents occurrence of the disease. HIV can be detected by culture in most AIDS patients and by culture or polymerase chain reaction in most HIV seropositive individuals (8, 9). Epidemiological data show that transmission of HIV results in AIDS and blocking HIV transmission prevents the occurrence of AIDS. Thus, we conclude that there is overwhelming evidence that HIV causes AIDS. Knowledge of the cause of a disease (etiology) is important for control of that disease and gives a basis for understanding the pathology of the disease. However, knowing the cause of a disease does not mean that there is complete understanding of its pathology. Discovering the pathogenetic mechanisms of HIV in AIDS is a major focus for research. Duesberg's Response to Blattner and Colleagues

Blattner, Gallo, and Temin defend the hypothesis that HIV causes AIDS only with epidemiology and anecdotal clinical cases in which AIDS is correlated with antibody to HIV, but not with active virus. I submit that this is insufficient because such evidence cannot distinguish between HIV and other causes, unless there is also evidence for biochemical activity of HIV in AIDS. 1 ) My opponents say that "following introduction of HIV in a population ... immunodeficiency emerges in a predictable sequence." Instead, epidemiological surveys show that the annual incidence of AIDS among persons with antibody to HIV varies from almost 0 to over 10%, depending on factors defined by lifestyle, health, gender, and country of residence (see point 8 of my preceding statement). Among antibody-positive Americans the avenge conversion rate is 1% [10,000 to 20,000 (1) of 1 to 2 million (2, 3)] but that of certain hemophiliacs (4) or male homosexuals (5) is 10% or higher. These discrepancies between the epidemiologies of HIV antibody and AIDS indicate that neither HIV nor antibody to it is sufficient to cause AIDS. 2 ) The argument that HIV, "not ... any other infectious agent," is linked to AIDS in blood transfusion recipients and in congenitally infected children is presumptuous for several reasons. Blood transfusion does not distinguish between HIV and "any other infectious agent" or blood-borne toxin. Further, it is presumed that the recipient had no risk factors other than HIV during the average of 8 years between HIV transfusion and AIDS symptoms. The transfusion evidence would be more convincing if AIDS appeared soon after a singular transfusion in generally healthy recipients. Transfusion AIDS cases, however, only occur very late after infection and mostly in persons with health risks, such as hemophilia, that are not representative of healthy individuals. Likewise, it is presumptuous to assume that HIV was the cause of AIDS in antibody-positive children, of whom 96% had other health risks, such as mothers who are prostitutes or addicted to intravenously administered drugs or blood transfusions for the treatment of hemophilia or other diseases (1, 6). The references to these cases would have been more convincing if antibody-negative controls had been included, having none of "the broad range of clinical diseases ... and the diversity of signs and symptoms of patients infected with HIV" (6). 3) According to authoritative sources, the primary defect of AIDS is a T cell deficiency induced by HIV infection (3, 7, 8). Therefore, it comes as a surprise that the primary clinical symptom of the children with AIDS was a B cell, not a T cell, deficiency (6). In fact, one of these same sources reports that "to fit observations from children into definitions for adult patients is unwise" (3). I wonder whether there is truly any disease that, in the presence of antibody to HIV, would not be called AIDS. 4) They claim that "interruption of HIV infection almost completely prevented the further appearance of blood-transfusion-associated AIDS." However, according to the CDC, transfusion-associated AIDS cases in adults have doubled to 752 cases and pediatric cases tripled to 68 in the year ending May 1988 compared to the previous year (1). This happened 3 years after antibody-positive transfusions were reduced 40-fold with the AIDS test (9). The steep increase in transfusion AIDS cases despite the great reduction of HIV-contaminated transfusions argues directly against HIV as the cause of AIDS. 5) In addition to the correlation that "in countries with many persons with HIV antibodies there is much AIDS," it is necessary to demonstrate some HIV-specific biochemical activity at the onset of AIDS to prove that HIV causes AIDS. All other viruses and microbes are very active when they cause fatal, degenerative diseases similar to AIDS. There is also abundant generic evidence that this activity is necessary for pathogenicity. Antibodies are evidence for the absence of an active virus, not a prognosis for future disease or death. Prior claims for etiology without genetic or molecular evidence for activity proved to be some of the most spectacular misdiagnoses in virology: (i) Based on epidemiological evidence, "scientists concluded" that Epstein-Barr virus was the cause of Burkitt's lymphoma-until the first virus-free lymphomas were found (10). (ii) On epidemiological grounds, human and bovine retroviruses were believed to cause leukemia after bizarre latent periods of up to 40 years in humans (11)-but finding these viruses in billions of normal cells of millions of asymptomatic carriers has cast doubt on this view (12). It is scarcely surprising that these leukemias arose from virus-infected cells. Consistent with this view, these "viral" leukemias are clonal and not contagious, behaving like virus-negative leukemias, and the associated "leukemia" viruses are not biochemically active (12). (iii) "Slow viruses" were accepted as causes of Alzheimer's, kuru, and Creutzfeldt-Jakob disease (13) on the basis of the same kind of epidemiology and transmission evidence used here for HIV-but these viruses have never materialized. These examples illustrate that correlations without evidence for biochemical activity are not sufficient to prove "etiology." 6) I fully support the view that "knowledge of the cause of a disease (etiology) is important for control." Since the cause of AIDS is debatable, the control of AIDS may not be achieved by controlling HIV. This is particularly true for the highly toxic "control" (preventive or therapeutic) of AIDS with azidothymidine (AZT)-AZT is designed to inhibit viral DNA synthesis in persons who have antibodies to a virus that is not synthesizing DNA (14
 
From the mouth of a Nobel prize winner

Who will stand up to the challenge?!

Kary Mullis, Nobel Prize winner in Chemistry

The following was written by Kary Mullis for the introduction to the book "Inventing the AIDS Virus" by Peter H. Duesberg (Regnery Publishing, INC; Washington DC, 1996):


In 1988 I was working as a consultant at Specialty Labs in Santa Monica, CA, setting up analytic routines for the Human Immunodeficiency Virus (HIV). I knew a lot about setting up analytic routines for anything with nucleic acids in it because I invented the Polymerase Chain Reaction. That's why they hired me.

Acquired Immune Deficiency Syndrome (AIDS), on the other hand, was something I did not know a lot about. Thus, when I found myself writing a report on our progress and goals for the project, sponsored by the National Institutes of Health, I recognized that I did not know the scientific reference to support a statement I had just written: "HIV is the probable cause of AIDS."

So I turned to the virologist at the next desk, a reliable and competent fellow, and asked him for the reference. He said I didn't need one. I disagreed. While it's true that certain scientific discoveries or techniques are so well established that their sources are no longer referenced in the contemporary literature, that didn't seem to be the case with the HIV/AIDS connection. It was totally remarkable to me that the individual who had discovered the cause of a deadly and as-yet-uncured disease would not be continually referenced in the scientific papers until that disease was cured and forgotten. But as I would soon learn, the name of that individual - who would surely be Nobel material - was on the tip of no one's tongue.

Of course, this simple reference had to be out there somewhere. Otherwise, tens of thousands of public servants and esteemed scientists of many callings, trying to solve the tragic deaths of a large number of homosexual and/or intravenous (IV) drug-using men between the ages of twenty-five and forty, would not have allowed their research to settle into one narrow channel of investigation. Everyone wouldn't fish in the same pond unless it was well established that all the other ponds were empty. There had to be a published paper, or perhaps several of them, which taken together indicated that HIV was the probable cause of AIDS. There just had to be.

I did computer searches, but came up with nothing. Of course, you can miss something important in computer searches by not putting in just the right key words. To be certain about a scientific issue, it's best to ask other scientists directly. That's one thing that scientific conferences in faraway places with nice beaches are for.

I was going to a lot of meetings and conferences as part of my job. I got in the habit of approaching anyone who gave a talk about AIDS and asking him or her what reference I should quote for that increasingly problematic statement, "HIV is the probable cause of AIDS."

After ten or fifteen meetings over a couple years, I was getting pretty upset when no one could cite the reference. I didn't like the ugly conclusion that was forming in my mind: The entire campaign against a disease increasingly regarded as a twentieth century Black Plague was based on a hypothesis whose origins no one could recall. That defied both scientific and common sense.

Finally, I had an opportunity to question one of the giants in HIV and AIDS research, DL Luc Montagnier of the Pasteur Institute, when he gave a talk in San Diego. It would be the last time I would be able to ask my little question without showing anger, and I figured Montagnier would know the answer. So I asked him.

With a look of condescending puzzlement, Montagnier said, "Why don't you quote the report from the Centers for Disease Control? "

I replied, "It doesn't really address the issue of whether or not HIV is the probable cause of AIDS, does it?"

"No," he admitted, no doubt wondering when I would just go away. He looked for support to the little circle of people around him, but they were all awaiting a more definitive response, like I was.

"Why don't you quote the work on SIV [Simian Immunodeficiency Virus]?" the good doctor offered.

"I read that too, DL Montagnier," I responded. "What happened to those monkeys didn't remind me of AIDS. Besides, that paper was just published only a couple of months ago. I'm looking for the original paper where somebody showed that HIV caused AIDS.

This time, DL Montagnier's response was to walk quickly away to greet an acquaintance across the room.

Cut to the scene inside my car just a few years ago. I was driving from Mendocino to San Diego. Like everyone else by now, I knew a lot more about AIDS than I wanted to. But I still didn't know who had determined that it was caused by HIV. Getting sleepy as I came over the San Bernardino Mountains, I switched on the radio and tuned in a guy who was talking about AIDS. His name was Peter Duesberg, and he was a prominent virologist at Berkeley. I'd heard of him, but had never read his papers or heard him speak. But I listened, now wide awake, while he explained exactly why I was having so much trouble finding the references that linked HIV to AIDS. There weren't any. No one had ever proved that HIV causes AIDS. When I got home, I invited Duesberg down to San Diego to present his ideas to a meeting of the American Association for Chemistry. Mostly skeptical at first, the audience stayed for the lecture, and then an hour of questions, and then stayed talking to each other until requested to clear the room. Everyone left with more questions than they had brought.

I like and respect Peter Duesberg. I don't think he knows necessarily what causes AIDS; we have disagreements about that. But we're both certain about what doesn't cause AIDS.

We have not been able to discover any good reasons why most of the people on earth believe that AIDS is a disease caused by a virus called HIV. There is simply no scientific evidence demonstrating that this is true.

We have also not been able to discover why doctors prescribe a toxic drug called AZT (Zidovudine) to people who have no other complaint other than the fact that they have the presence of antibodies to HIV in their blood. In fact, we cannot understand why humans would take this drug for any reason.

We cannot understand how all this madness came about, and having both lived in Berkeley, we've seen some strange things indeed. We know that to err is human, but the HIV/AIDS hypothesis is one hell of a mistake.

I say this rather strongly as a warning. Duesberg has been saying it for a long time.



Top of Page
 
I just figured that there was another response becuase It was written in 1988. Sorry for the speculative assumption. I do not however still pompously and arrogantly disagree:)

Duesberg points to cases of "AIDS" in which the HIV virus is not present. Where HIV is present, he argues that it is merely a "marker" of other factors(drug abuse or "foreign proteins.")
This is becuase Duesberg defines AIDS himself as the presence of one of 30 "opportunistic" diseases in combination with an at least slightly suppressed immune system.
The CDC disagrees becuase they say HIV is necessary for AIDS. However there are a group that shows "one of thirty oppurtunistic diseases" and also have "a depressed immune system". The disease that causes this is called "ICL," for idiopathic CD4+ lymphocytopenia. People who degenerate from this disease would "according to Duesberg" have AIDS. But the CDC doesnt consider them as AIDS patients becuase they are not infected with HIV.

Duesberg "thinks" that some retroviruses causes disease. Only thoses with the onc-genes have the capacity to cause disease.

"Firstly, there are lots of retroviruses without oncogenes that are generally thought to cause disease: Moloney murine leukemia virus, feline leukemia virus, equine infectious anemis virus, visna, bovine leukemia virus, caprine arthritis-encephalitis virus, etc, just to name a few, and secondly, alot of the molecular biology of how some of these viruses cause disease has been determined. "http://www.unsw.edu.au/clients/microbiology/jon/cause."

It must be asked what Duesberg is really saying by suggesting that the statement
HIV causes AIDS is wrong.
It can be read
"HIV always causes AIDS"; or
"AIDS is always caused by HIV"; or
"HIV sometimes causes AIDS".
Duesberg is using philosophy, not science to prove the "HIV causes Aids" theory wrong
If he was using science he would show

".. that a substantial proportion of African AIDS sufferers do not have 'evidence of HIV infection' ". Such refutation would be a severe blow to our hypothesis.

".. that prevention of 'transmission of HIV', for example perinatal prevention, does not prevents AIDS in children". By demonstrating this, our virusmyth scientists will have a convincing argument, not only against the importance of HIV, but also against perinatal use of anti-retrovirals!

".. that markers of immune suppression, evidence of opportunistic infections, and mortality rates are not changed in patients who are compliant with state-of-the-art anti-retroviral therapy and have evidence of substantial suppression of 'HIV replication' " (using, for example, Dr Mullis' PCR technology to document suppression of 'viral load' in blood). By demonstrating this, Duesberg et. al. would destroy the rationale for anti-retroviral therapy"

asteroth
Aids doesn't necessarily take "years" to develop...
Here is a quote from:"Aids the Facts"by John Langone Page 11:
"But while the time that elapses between infection and presence of antibodies may be charted,the lenght of time that an infected individual will remain free of symptoms seems to v a r y w i l d l y from DAYS,or WEEKS, to many years DEPENDING on how the infection WAS AQCUIRED"
asteroth....that is saying that depending on the how you got HIV, the time it takes to aquire AIDS symptoms varies. Hence, HIV is a cause of AIDS. I am sorry I thought you were defending Deusberg.

asteroth
t r a n s f u s i o n related Aids is still up..

from 1985-88 or now. It would take at least half the average "life left" to start to show a decline(if no improvements in medicine). And I wont even account for the fact that new medications have decreased the amount of people disgnosed with AIDS in proportion to the hiv population so you must be talking about 12 years ago.

By the way, before I read this I knew nothing about HIV/AIDS ,but even I knew that Hiv leads to AIDS. I was curious to find out if I had mis informed, but I dont believe i have been mislead.
 
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Response to Champ part 1

While reading Champs last reply to my post,it became quite evidently apparent,that further studying of the dissenting hypothesis is necessary on his behalf...
Duesberg,for example,responds to all of Champs comments and questions,in the articles mentioned earlier,I shall attempt to point out a few,for evryones benefit...

Prior to that I should mention,that I have no accademic medical background,and that my interest in the Aids controversy stems from utmost quiriosity and a befuddled amazement at the face of notable scientists battling each other over what is or is not a "scientific fact".

I thought that such a disputes had no place in a research lab,but rather in the lofty worlds of philosophy and religion...further more,some may get the impression by Champ,or maybe even by myself,that the Aids controversy is a one man crusade(Duesberg)...

It is absolutely not!!

Hundreds of scientists jumped at the dissenting bandwagon,and voiced their disapproval,as soon as it became indisputably clear,that no biochemical evidence is available which would support classification of HIV as "cause" of Aids...
in light of that,and in the presence of Noble prize winners in microbiology,attacking the Aids hypothesis,how is a layman to determine who is right,and who is wrong?!

reviewing Champs repsonse concerning aids contraction through blood transfusion, and other issues raised by him ;I can not help but notice an attempt to try and project ,or force thoughts into an explanation you vehemently disagree with ,thus totally reinventing it,turning it upside down,untill it becomes irecognisable even to its author...

imagine,for the sake of argument,an atheist,quoting biblical verses in order to stress to a bliever,that the bible teaches anything but moral conduct...Is resorting to biblical quotations(by the atheist) to make a point,a right reserved exclusivley to rabbis and monks....Can't an atheist point out biblical discrepancies without being accused of believing in God..Should he be labeled a "believer" just for knowing ,and reciting from his opponents sacred book...

Similarly, knowing what the aids hypothesis claims about the duration of the manifestation of symptoms in aids patients,and quoting it to champ,does not mean I agree with whatever the hypothesis suggests...the quotation only serve to highlight the apparent contradiction,with regards to blood transfusion contraction of Aids...

If,tainted blood transfusion causes Aids ,than how come taking measures to reduce the risk of contaminated blood did not result in less blood transfusion associated aids following those measures,but rather an even higher rate of contraction?

champ's answer was "it takes years to develop AIDS from HIV...3 years after...what is that" ...

in response I quoted "Aids Facts"by John Langone showing that Champs reply stands in contrast to the Hiv/Aids hypothesis he so dearly accepts without question...consequently and in light of that contradiction,another reason,that is not related to HIV contaminated blood must be found to explain the above dilema...Duesberg has always maintained that HIV is not,and has never been the cause of immuno deficiency,in one of his articles he suggests the following concerning transfusion related Aids:"The thesis that Hiv transfusions cause Aids in other patients is also entirely uncontrolled.Indeed,a controlled study might be difficult because 50% of American patients(other than hemophiliacs)die within 1 year,and 60% within three 3 years a f t e r t r a n s f u s i o n-long before the average 10 years HIV is said(by the establishment) to require for pathogenicity have elapsed.the pathogenic conditions that necessitated the transfusions are obviously deadlier than the h y p o t h e t i c a l pathogen HIV"

Champs continuously and forcibly rapes Duesberg's words,insisting on impregnating him with his o w n HIV/aids hypothesis seed...
He asks for example "what Duesberg is really saying by suggesting that the statement HIV causes Aids is Wrong?",and accusing him of "using philosophy"...

amazing and facinating as it may seem,anyone aquainted with the dissenting camp assumption in general,and Duesberg's articles in particular,knows fully well that the A B C of the Drug/Aids hypothesis is that"HIV DOES NOT CAUSE AIDS"(Drugs do!)

No if's or but's...

Following are several quotations:
Human immunodeficiency virus(HIV)is N O T THE CAUSE OF AIDS because it fails to meet the postulates of Koch and Henle,as well as six cardinals rules of virology....HIV is in violation of Koch's postulate because it is n o t possible to detect f r e e virus,provirus,or viral RNA in ALL cases of Aids....HIV can not be isolated from 20% to 50% of Aids cases,moreover isolation is very indirect...In contrast to A L L pathogenic viruses,that cause degenerative diseases,HIV is N O T biochemically active, in the disease syndrom it is named for...It actively infects only 1 in 104 to >105 T cells.Undr these conditions HIV can not account for the loss of T cells,the hallmark of Aids,even if all infected cells died;this is because during the 2 days it takes hiv to replicate,the body regenerates,about 5% of its T cells,more than enough to compensate for the losses due to HIV.It is p a r a d o x i c a l that HIV is said to cause aids only A F T E R the onset of a n t i viral i m m u n i t y detected by a positive "Aids test",because A L L other viruses are most pathogenic b e f o r e immunity.The immunity against HIV is so effective that free viruses is undetectable,which is why HIV is hard to transmit...HIV remains INACTIVE during Aids,thus the "Aids test"identifies effective natural vaccination,the ULTIMATE PROTECTION against viral disease...

In light of the above it seems that Champ and the HIV/Aids establishment are the ones engaged in "philosophy"instead of responding to the quoted scientific F A C T S(in contrast to an "hypothesis")in a point by point refutation...

To this day Blattner and his associates failed to explain how is it possible that a virus that bearly infects 1 in a 104 T cells, is responsible for any immune deficiency...

The assumption that HIV is a harmless virus,which does not cause Aids,is so unbelievably,startling and disturbing to Champ(in this case),that he even fails to accepts and acknowledge simple written English sentences...he hears the music but refuses to listen to the tunes...he still can not believe that when duesberg's writes that "Hiv does not cause Aids"he really means it!

relentlessly he twists and misquotes :"Duesberg defines Aids Himself"he says "as the presence of of 30 opportunistic diseases ..."But Duesberg makes no such definition what so ever the CDC does,he simply uses their own definition :"Aids is a syndrom of 25 parasitic neoplastic and noninfectious diseases,including in the united states 53% pneumonia,19%wasting disease,13%candidiasis,11%Kaposi sarcoma,6%dementia,3%hlymphoma,and 2% tuberculosis"(Centers for Disease Control(1990)HIV/Aids Surveillance Jan issue)

Duesberg points of the existence of 4621 documented cases of complete immune deficiency with no trace of HIV exposure,and in contrast to Koch's postulate that the virus must be found in affected tissue in ALL CASES OF THE DISEASE;the medical facts,which Blattner has not yet disputed are:that no HIV at all can be isolated from at least 10 to 20 % of Aids patients,no direct trace of HIV could be found in the majority of Aids cases ,and HIV can not be isolated from the cells of in the lesions of Kaposi's sarcoma,nor from nerve cells of patients with Aids dementiaa."

Inspite of the above the CDC "disagrees" because"HIV is necessary for Aids"but yet provides no explanation as to "W H Y "HIV is necessary for Aids particularly in light of the fact that P R I OR CLAIMS for etiology without genetic or mulecular evidence for activity proved to be some of the most spectacular misdiagnoses in virology(based on epidemiological evidence" scientists concluded" that Epstein Barr virus was the cause of Burkitt's lymphoma until the first virus free lymphomas were found...On epidemiological grounds,human and bovine retroviruses were believed to cause leukemia after bizarre latent periods of up to 40 years in humans,but finding these viruses in billions of normal cells,of millions of asymptomatic carriers has cast doubt on this view)...in response to the CDC unscientifically based insistence ' Duesberg responds:"that the HIV/Aids,hypothesis is the ONLY link that hold together the 30 heterogenous Aids diseases.Aids is define as a syndrom that occurs only in the presence of HIV.for example,if tuberculosis occurs in the presence of antibodies to HIV it is Aids.In the absence of those antibodies,it is tuberculosis.Given this definition the link between Aids and HIV is unfalsifiable....however,to date,the HIV Aids hypothesis remains just that an UNPROVEN HYPOTHESIS.It is supported O N L Y by circumstantial evidence,primarily by the claim that all Aids patients carry antibodies against HIV.But this correlation is biased by the practice of excluding from Aids statistics those patients with AIDS DEFINING diseases in whom no trace of HIV can be found,the disease of such a patient will be diagnosed either by its old name,for example,pnumonia or Kaposi sarcoma,or will be called idiopathic CD-4 Lymphcytopenia"

This kind of scientific verbal arm twisting, and linguistic acrobatics propagated by the CDC, instead of providing hard proof in the form of Biochemical evidence to explain once an for all WHY is HIV necessary for Aids when it is scientifically proven to bearly infect any T cells at all brings to mind Bill clinton's twisting of the word "is"...

How long will the CDC and its Money hungry pharmaceutical cronies will be able to mislead the American and world public?!

Duesberg does not "think""that some retro viruses cause disease...but this point will be delt with in the 2nd part of this post
good night
Asteroth
 
While reading Champs last reply to my post,it became quite evidently apparent,that further studying of the dissenting hypothesis is necessary on his behalf...

:)

reviewing Champs repsonse concerning aids contraction through blood transfusion, and other issues raised by him ;I can not help but notice an attempt to try and project ,or force thoughts into an explanation you vehemently disagree with ,thus totally reinventing it,turning it upside down,untill it becomes irecognisable even to its author...

Thank you:)....you are pretty good at it yourself.


imagine,for the sake of argument,an atheist,quoting biblical verses in order to stress to a bliever,that the bible teaches anything but moral conduct...Is resorting to biblical quotations(by the atheist) to make a point,a right reserved exclusivley to rabbis and monks....Can't an atheist point out biblical discrepancies without being accused of believing in God..Should he be labeled a "believer" just for knowing ,and reciting from his opponents sacred book...

OK Plato ,for the sake of arguement,
You still(as atheist) said that GOD existed,... to try to prove that GOD didnt exist. It is ok if an atheist uses quotes from the bible, but if he tries to prove that the catholic god exists so that buddist god doesnt, the atheist has lost his whole religion, and just wants to argue with someone.

But we are talking science here and not philosophy and theology


Duesberg has always maintained that HIV is not,and has never been the cause of immuno deficiency,in one of his articles he suggests the following concerning transfusion related Aids:"The thesis that Hiv transfusions cause Aids in other patients is also entirely uncontrolled.Indeed,a controlled study might be difficult because 50% of American patients(other than hemophiliacs)die within 1 year,and 60% within three 3 years a f t e r t r a n s f u s i o n-long before the average 10 years HIV is said(by the establishment) to require for pathogenicity have elapsed.the pathogenic conditions that necessitated the transfusions are obviously deadlier than the h y p o t h e t i c a l pathogen HIV" [/quote]

OK, so there is no way to "be sure" if HIV passed in blood transfusions cause AIDS becuase there cannot be a controlled study. That doesnt mean that HIV is not the cause of immune deficiency. All it means is that you cannot have a controlled study with blood transfusion recipients....what is your point?

another reason,that is not related to HIV contaminated blood must be found to explain the above dilema

....dilema..being rise in AIds, while decreasing #of contaminated transfusions...(for those who are not following)
as I said that study was in 1986-88. Show me a recent study.

To this day Blattner and his associates failed to explain how is it possible that a virus that bearly infects 1 in a 104 T cells, is responsible for any immune deficiency...

The assumption that HIV is a harmless virus,which does not cause Aids,is so unbelievably,startling and disturbing to Champ(in this case),that he even fails to accepts and acknowledge simple written English sentences...

To this day Blattner and his associates failed to explain how is it possible that a virus that bearly infects 1 in a 104 T cells, is responsible for any immune deficiency...


How about these sentences.......
"The answer to this paradox was provided in January 1995. Two reports in Nature demonstrated that the infected CD4+ cells were being destroyed as quickly as they were infected, so the measured CD4+ population in the blood of an HIV sufferer at any one time was always free of infection (see Living Marxism, December 1995). In an accompanying editorial John Maddox conceded that Duesberg was 'right to have argued all along that the usually slow decline of CD4+ cells is not consistent with what one would expect from a specific cytotoxic viral mechanism' (Nature, 373: 189, 1995). "

looks like you are the one who should do some "further studying"

Human immunodeficiency virus(HIV)is N O T THE CAUSE OF AIDS because it fails to meet the postulates of Koch and Henle,as well as six cardinals rules of virology....HIV is in violation of Koch's postulate because it is n o t possible to detect f r e e virus,provirus,or viral RNA in ALL cases of Aids....HIV can not be isolated from 20% to 50% of Aids cases,moreover isolation is very indirect...In contrast to A L L pathogenic viruses,that cause degenerative diseases,HIV is N O T biochemically active, in the disease syndrom it is named for...It actively infects only 1 in 104 to >105 T cells.Undr these conditions HIV can not account for the loss of T cells,the hallmark of Aids,even if all infected cells died;this is because during the 2 days it takes hiv to replicate,the body regenerates,about 5% of its T cells,more than enough to compensate for the losses due to HIV.It is p a r a d o x i c a l that HIV is said to cause aids only A F T E R the onset of a n t i viral i m m u n i t y detected by a positive "Aids test",because A L L other viruses are most pathogenic b e f o r e immunity.The immunity against HIV is so effective that free viruses is undetectable,which is why HIV is hard to transmit...HIV remains INACTIVE during Aids,thus the "Aids test"identifies effective natural vaccination,the ULTIMATE PROTECTION against viral disease...



blattner....proven Duesberg wrong in 1988 in response

"the Henle-Koch postulates of 1840 and 1890 were formulated before the discovery of viruses. They are a useful historical reference point, but were not regarded as rigid criteria by Koch himself and should not be so regarded today"

But those postulates were not the basis of Duesbergs later arguements. This Koch postulate was just to further his arguement in 1988.

I would suggest reading
http://www.informinc.co.uk/LM/LM95/LM95_Futures.html
it is a bit more up to date than what you are currently referring to.

:)
 
Hi, I got a couple questions for you, Iam trying to make up my mind if hiv leads to aids. Why is their so many symptoms associated with hiv that only get worse when you reach full blown aids. I mean people with hiv and aids complain of haveing fevers everyday, hows that possible with a virus that isint suppose to do anything, then there is thrush, why is it that so many people with hiv have thrush when this virus is supposed to be harmless...
 
Response to Champ part 2

The butchery of words and misquotations of Duesberg's continues unabated..."He thinks",says Champ,that some retro viruses cause disease;O N L Y those with the onc-genes have the capacity to cause disease",but alas,Duesberg has never written in any of his articles published todate,that "only" those with the onc-gene have the capacity...."below is the related quotation :

"Based on animal and human models, retroviruses depend almost exclusively on perinatal transmission for survival. They are very difficult to transmit horizontally by immune competent animals and humans, because they are chronically suppressed, first by maternal antibody and then by the baby's own (76, 77), and possibly also by cellular suppressors (34). Even retroviruses with s a r c o m a g e n i c or leukemogenic oncogenes have n e v e r s p r e a d horizontally in b r e e d i n g colonies (29, 85)."

Therefore, specific strains of mice, chickens, or humans from geographically distinct regions are often marked for generations by distinct strains of perinatally transmitted latent retroviruses (85, 86). For example, HTLV is endemic in certain islands of Japan and marks specific ethnic groups among mixed populations in the Caribbean (86). Wild animals (29, 85, 86) or humans (42, 43, 86) with an acute retrovirus infection are virtually never observed.

Acute retrovirus infections r e s u l t from e x p e r i m e n t a l infection or horizontal infections among mass-bred animals, typically prior to immune competence with virus strains not covered by maternal antibodies (76, 77, 85). Since perinatal transmission of HIV is at least 50% efficient (18, 20, 34, 62), and sexual transmission is <0.2% efficient, it appears that HIV, like o t h e r retroviruses, d e p e n d s on perinatal transmission for survival.
Therefore, it c a n n o t be f a t l l y p a t h o g e n i c in most infections within 2-10 years, as postulated by the virus-AlDS hypothesis.

This provides the only plausible explanation for the random distribution of HIV in even as few as 0.03% of 17- to l9-year-old healthy Americans (53) and in about 10% of Africans of all ages (31, 34, 49, 51).
This explains why no more than 2456 AIDS cases have been recorded among about 75 million Americans under the age of 19 in the last 9 years (1), although at least 0.03%, or 25,000, can be estimated to be perinatally infected (53). It appears that >90% of perinatally infected Americans are asymptomatic for at least 19 years.

Other quotations concerning retro viruses:

The hallmark of all retroviruses is that they do not kill cells that they infect. HIV is the only retrovirus that is asserted to kill its host cell. Several researchers, including HIV discoverer Luc Montagnier, have found that HIV does not kill its host cell in laboratory tests.

Retroviruses are typically not cytocidal. On the contrary, they often promote cell growth. Therefore, they were long considered the most plausible viral carcinogens (9). Yet HIV, a retrovirus, is said to behave like a cytocidal virus, causing degenerative disease killing billions of T cells (15, 18). This is said even though T cells grown in culture, which produce much more virus than has ever been observed in AIDS patients, continue to divide (9, 10, 18). 8) It is paradoxical

A fourth paradox of the HIV hypothesis has been noted by several virologists. HIV belongs to a class of viruses known as the retroviruses, which are very simple in structure and contain much less genetic information than most other viruses. Most types of viruses are lytic, meaning that they kill the cells they infect and thereby cause disease. Retroviruses, on the other hand, do not generally kill cells. Upon infecting cells, they copy their genetic information into the DNA of their new host cells. From that point forward, retroviruses depend on allowing their host cells to continue living, while they slowly produce new virus particles that are ejected from the cell. Retroviruses are therefore poor candidates to blame serious diseases on, particularly fatal conditions involving the deaths of huge numbers of cells, such as AIDS. Indeed, some 50 to 100 latent retroviruses have been found to reside in the DNA of all humans, passed along to each successive generation for as long as human beings have existed. Past research by Harry Rubin has shown that retroviruses cannot infect any cells that do not divide. Neurons in the human brain do not divide after the first year of life, so HIV cannot possibly infect those cells. This would explain why HIV has not been isolated from these cells, and confirms the difficulty it would also face in causing dementia. Harvey Bialy, research editor of the professional journal Bio/Technology, argues that the simple genetic structure of HIV does not differ sufficiently from other retroviruses to account for its supposedly different behavior. The genetic information carried by HIV is not unusual for retroviruses; it contains no gene different enough from the genes of other retroviruses to be a possible "AIDS gene." In addition, HIV uses all of its genetic information when it first infects, rather than saving some to be used years later. In other words, there is no conceivable reason HIV should causes AIDS 10 years after infection, rather than early on when it is unchecked by the immune system. Bialy also points out the

The virus-AIDS hypothesis also totally fails to explain how the virus depletes T-helper cells, and why it takes at least 3-5 years to do so. Unlike all other animal viruses, retroviruses need mitosis to initiate infection. Moreover, no HIV gene remains inactive during replication, which takes about 1-2 days, as with all other retroviruses. Thus HIV would be expected to kill T cells and cause AIDS when it first infects an organism and not 5 years later when it is biochemically inactive and suppressed by antiviral immunity. The 5-year latency presents proponents of the hypothesis with two bizarre options: either old T cells die 5 years after infection, or the offspring must die in the 50th generation, given a one-month generation time for the average T cell. Nevertheless, killing of T cells within weeks, not years, after infection has been observed in cell culture-in apparent agreement with the claim that the virus kills T cells. But this type of killing is fundamentally different from the unconditional cell lysis achieved by true cytocidal viruses. It involves cell fusion mediated via HIV antigens on the surface of infected cells, and is conditional on the cells and virus isolates used. Further, it does not occur in chronically infected human T-cell lines that grow indefinitely in culture yet produce more virus than any other system, nor has it ever been observed in blood taken from an AIDS patient. In fact, limited cell killing by fusion is a common feature of retroviruses, none of which have as yet been claimed to cause AIDS. It seems clear from the foregoing that the virus-AIDS hypothes

When HIV first infects a person, it can reach moderately high concentrations in the blood - yet AIDS never shows up at that time and T cell levels remain normal. Within days or weeks, the immune system makes antibodies against HIV, and the virus quickly disappears, from the blood. Years later, if AIDS shows up at all, the virus rarely comes back to life and multiplies again. In other words, AIDS never strikes a patient until years after the active virus has been permanently eliminated from the body. This strongly suggests that AIDS is caused by something else. As an alternative to the HIV

Attempting to show "that a substantial proportion of African Aids sufferers do not have evidence of HIV infection"is virtual impossibility due to the CDC'S own definition of Aids which necessitates the presence of HIV,inspite of the fact that numerous cases exist(as previously discussed)where patients exhibit total immune deficiency,in an E X A C T S A M E M A N N ER as Aids patiens,but WITHOUT HIV antibodies presence...
The CDC "redefines"those Aids cases rather than admit to the fallacy of its HIV/Aids hypothesis,and its apparent contradiction to all of Koch's postulates...

What Duesberg,therefore suggests to further discount the plausability of the wrongly accepted HIV/aids view,with regards to African Aids is the following:

The situation in Africa is even more puzzling and casts further doubt on the HIV hypothesis. Most of the media publicity in America on AIDS in Africa is based on the large extent of HIV infection, not on the extent of AIDS cases themselves. Nonetheless, although HIV infection appears to be extremely widespread, present in many areas in 10 to 15 percent of the population, the total number of AIDS cases so far reported in the entire continent of Africa amounts to merely 41,000. Proponents of the HIV hypothesis often try to argue that this low figure is the result of under reporting of AIDS cases. Even in Uganda, however, which has a reputation for conscientious reporting, 800,000 people are HIV positive, but only 10,000 are reported to have died of AIDS. A paper and accompanying editorial in the July 25, 1987, issue of the British medical journal "The Lancet" argued that AIDS in Africa is actually not a major epidemic; the paper was written by a doctor from Cromwell Hospital in London, Felix Konotey-Ahulu, who had just returned from an extensive investigative tour of the areas of Africa with the most AIDS cases. The story in Haiti is similar. Only 2,3000 AIDS cases have been reported during the past decade in a country where HIV infection is thought to be rampant. Even if this number is underreported, the prevalence of AIDS is much lower than would be predicted by the HIV hypotheses.

AIDS Risks of HIV-infected Persons Differ 10- to 65-Fold Depending on Their Country. If AIDS is caused by HIV, the ratio of infected to diseased carriers should be similar in different countries. However, in the United States about 10% (or 100,000) (1) of 1 million HIV-positives (16,18, 49, 50) have developed AIDS since 1985, but in Uganda only 0.8% (or 8000) of 1 million (51), and in Zaire only 0.15% (4636) (52) of 3 million HIV-positives (34). Although AIDS surveillance by some African countries has been questioned, surveillance by Uganda is reported as "highly successful," providing "the highest number . . . of cases . . . in Africa" (51). Since the HIV epidemics of both the United States and Africa are said to be new and to have an African origin (17, 20, 36, 45), but the AIDS risks of infected Americans are 10- to 65-fold higher than those of Africans, country-specific pathogens are necessary for AIDS. Moreover, if AIDS equaled opportunistic infections resulting from immunodeficiency, more, instead of less, AIDS per HIV carrier would be expected in Africa than in the United States.

Similarly,the request to show that"prevention of transmission of HIV for example,prenatal prevention does not prevent Aids in children....."falls in the same category of "un falseability"...i.e how can one show or substantiate a "cause" for a particular disease,when one already provides a plethora of scientific evidence that clearly and undisputably indicates on a biochemical level,that the assumed cause is N O T really the cause....In effect the establishment demands of the dissenting camp to prove a negative...The onus of proof lays with those that make extraordinary claims...that's a number 1 epistemological rule...

nevertheless Duesberg provides an explanation as follows:

The question naturally arises as to why people outside these behavioral health-risk groups, including hemophiliacs and children, would develop some of the AIDS diseases. The answers lie in the risk factors too rarely reported to the public. Hemophilia has always been a fatal condition. This has only been partly alleviated by recent medical advances. Not only are blood transfusions still frequently needed, but blood clotting factors used by hemophiliacs today are somewhat immunosuppressive themselves. Interestingly, the controlled epidemiological study of hemophiliacs, cited above, found evidence to support the idea that hemophilia may be an inherently immune-deficient condition on its own. In the case of Ryan White, now often cited as an example of an AIDS death, the Hemophilia Foundation of Indiana has confirmed that his death was due to such complications as liver failure and internal bleeding, conditions that typically result from hemophilia itself. Indeed, White already had a severe case of hemophilia, ultimately requiring clotting factor therapy every day. He also underwent daily AZT therapy, the dangers of which are reviewed below. Infants diagnosed as having AIDS have developed their conditions due to combinations of most of the above risk factors. Published CDC data shows that some 95 percent of these babies are born to mothers who are confirmed drug addicts and/or sexual partners of IV drug users (frequently a code word for prostitutes), or the babies are themselves hemophiliacs or recipients of blood transfusions. The risk behavior of many of their mothers has reached these victims, but their conditions are renamed AIDS when in the presence of antibodies against HIV. Finally, those few AIDS cases in which no risk factors exist are due to the clinical definition of AIDS. Having contracted, for whatever reason, one or more diseases on the AIDS list in the presence of antibodies against HIV, these people are diagnosed as having this syndrome. In many instances, this means the patients are not given sufficient conventional therapies for the conventional disease, but are instead treated with the drug AZT

conclusion:
It is apparent to any impartial observer that Champs claim of reading Duesberg's articles looks more like a grand misreading of almost every sentence...His reading resembels that of a rabbi's reviewing of the New Tastament,for the purpose of finding flaws contradictions and refutations ,rather than gain a general understanding of what christianity is all about...

Be blessed
Asteroth
 
blattner...."proven" Duesberg wrong in 1988 in response

"the Henle-Koch postulates of 1840 and 1890 were formulated before the discovery of viruses. They are a useful historical reference point, but were not regarded as rigid criteria by Koch himself and should not be so regarded today"

This is not "proof" but an "opinion"...Is Champ aware of the difference between "evidence of a truth or a fact"and a person's own belief or formal statement!?

Even the CDC contends that:
"Among many criteria used over the years to prove the link between putative pathogenic (disease-causing) agents and disease, perhaps the most-cited are Koch's postulates, developed in the late 19th century. Koch's postulates have been variously interpreted by many scientists, and modifications have been suggested to accommodate new technologies, particularly with regard to viruses (Harden. Pubbl Stn Zool Napoli [II] 1992;14:249; O'Brien, Goedert. Curr Opin Immunol 1996;8:613). However, the basic tenets remain the same, and for more than a century Koch's postulates, as listed below, have s e r v e d as the l i t m u s test for determining the cause of A N Y epidemic disease:

Thus even by the CDC own account Koch's postulates have been applied and utilized since their inception "a century ago"to determine causality of "ALL epidemic diseases"
The only virus that seems to "transcend' this "historical reference"(as per Blattner)is HIV
Why?
What makes HIV so special so as not to be subbmited like any other retrovirus to the rigors of the "l i t m u s test for determining the cause of A N Y epidemic disease"
Blattner response to Duesberg in this point is clearly an excuse a justification of a f a u l t and a d e f e c t in the HIV/AIDS hypothesis...n o t proof!!
Anyway...
With such so called "proofs" provided above by Champ
Who needs science?!



champ says:
How about these sentences.......
"The answer to this paradox was provided in January 1995. Two reports in Nature demonstrated that the infected CD4+ cells were being destroyed as quickly as they were infected, so the measured CD4+ population in the blood of an HIV sufferer at any one time was always free of infection (see Living Marxism, December 1995). In an accompanying editorial John Maddox conceded that Duesberg was 'right to have argued all along that the usually slow decline of CD4+ cells is not consistent with what one would expect from a specific cytotoxic viral mechanism' (Nature, 373: 189, 1995). "

looks like you are the one who should do some "further studying"


This is not an answer to the Paradox...if it is... try and explain the alleged "answer" in your own words,rather than quote an explanation you might misunderstand, and demonstrate how it reconciles with the following
statement:
"It(HIV) actively infects only 1 in 104 to >105 T cells.Undr these conditions HIV can not account for the loss of T cells,the hallmark of Aids,even if all infected cells died;this is because during the 2 days it takes hiv to replicate,the body r e g e n e r a t e s,about 5% of its T cells,more than enough to compensate for the losses due to HIV.It is p a r a d o x i c

Champs says:
Thank you....you are pretty good at it yourself.

It would be great if you provided us with at least one example of misquotation,as you did so abundantly with Dusbergs articles...

Champ says:
OK Plato ,for the sake of arguement,
You still(as atheist) said that GOD existed,... to try to prove that GOD didnt exist. It is ok if an atheist uses quotes from the bible, but if he tries to prove that the catholic god exists so that buddist god doesnt, the atheist has lost his whole religion, and just wants to argue with someone.

But I never said that "God Exist to try and prove that he didn't"...
I was merely suggesting that your 3 year explanation iscontradictory and inconsistent with your o w n hypothesis...It's that simple

Champ says:
OK, so there is no way to "be sure" if HIV passed in blood transfusions cause AIDS becuase there cannot be a controlled study. That doesnt mean that HIV is not the cause of immune deficiency. All it means is that you cannot have a controlled study with blood transfusion recipients....what is your point?

The point is that there is NO PROOF or evidence to suggests "as a matter of fact" that HIV passes in blood transfusions...Just an assumption...hypothesis..
As far as the AIDS establishment is concerned "not being
sure"is enough to warrant any HIV related issue the aura of "a fact"...that's not science but pseudo science
 
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Soda writes:

Hi, I got a couple questions for you, Iam trying to make up my mind if hiv leads to aids. Why is their so many symptoms associated with hiv that only get worse when you reach full blown aids. I mean people with hiv and aids complain of haveing fevers everyday, hows that possible with a virus that isint suppose to do anything, then there is thrush, why is it that so many people with hiv have thrush when this virus is supposed to be harmless...

1.Read all above mentioned articles by Duesberg and make up your own mind who is more believable...

2.The symptoms associated with AIDS are there as a result of o t h e r pathogenic viruses that attack a body unable to put up a "T Cell' fight due to its immune deficiency...According to the dissenting opinion the immune deficiency in most Aids patients is aresult of prolong drug usage,exposure to AZT,and frequent STD's

ACTIVE virus Has been rarely detected todate in most Aids cases...HIV positive people carry ANTIBODIES of the HIV virus,not necessarily the virus itself....

Do yourself a favour...read the articles and log on to the www.virusmyth.com

Be well
ASteroth
 
So your saying that drug use like coke,weed,extasy leads to aids, alot of my freands use drugs on a regular basis and none of them have aids. I read all the articles your posting but its so hard to belive that hiv does not lead to aids. Like you said your self hiv gets into the dna, is it not possible that after a while all the new t cells being produced are already infected with the hiv virus, and when to many t cells get infected it progresses to aids.
 
Soda...
How many people do you know that exhibit lung cancer symptoms after being smokers for one or two years?!

nicotine and other drugs have a d e s t r u c t i v e comulative effect on the body which materializes after y e a r s of regular use...

The amount of years prior to the on set of various lung cancers in smokers,or aids related symptoms in HIV positive individuals vary from one person to another...

Other HIV positive persons who have been infected for nearly 20 years,but kept a healthy lifestyle,free of recreational drugs,and other poisions,D O N T seem to exhibit any Aids symptoms at all ,and probably never will

Why...If the HIV virus is responsible for "immune deficiency"and an overall distruction of CD4+ T cells,how come it has no ability to reawaken an wreck havock in people that have been HIV positive for that long...

Remember Koch postulates state clearly that"The germ must cause the sickness in A L L hosts"

How Does the CDC account for those(above mentioned) HIV positive persons who still haven't exhibit Aids related symptoms.."It's a mystery" they say,and they are right...the HIV/AIDS hypothesis is a mystery on its e n t i r e t y

In my opinion,any individual discovering for the first time that they are HIV positive should immediately stop all drug related usage,and focus their attention on boosting
their immune sytem through adaptation of a healthy life style,and proper supplamantation...Beware of AZT which seems to further deteriorate the immune system in the l o n g r u n rather than cure anything


You write:"is it not possible that after a while all the new t cells being produced are already infected with the hiv virus, and when to many t cells get infected it progresses to aids."

As of yet...the CDC has not indisputably proven that "new T cells being produced are all infected".On the contrary,current evidence suggests that newly produced T cells,are HIV free..
 
Thank you for editing some of your personal statements about me as they did not reflect well on you.


Asteroth said..
Asteroth said..
It is apparent to any impartial observer that Champs claim of reading Duesberg's articles looks more like a grand misreading of almost every sentence


Perhaps then you could restate the quotes after writing them explaining their context, and perhaps citing the source. This might help people(including me) understand what you are trying to say. No pun intended here.

His reading resembels that of a rabbi's reviewing of the New Tastament,for the purpose of finding flaws contradictions and refutations ,rather than gain a general understanding of what christianity is all about...


I can't believe you are comparing religion to science again. I am providing a point by point refutation of your agruement and if you have a problem with that then please pm me. Duesberg did the same thing to the hiv/aids hypothesis. Both sides have flaws which is why there is still debate about it. Some questions previously asked and debated about have been answered. Others have brought new questions.

Champ wrote....."the Henle-Koch postulates of 1840 and 1890 were formulated before the discovery of viruses. They are a useful historical reference point, but were not regarded as rigid criteria by Koch himself and should not be so regarded today"

Asteroth wrote....This is not "proof" but an "opinion"...Is Champ aware of the difference between "evidence of a truth or a fact"and a person's own belief or formal statement!?

As a matter of fact asteroth I do know what evidence of truth and fact are.
Why didnt Duesberg mention koch in his response to blattner. Because he used them as support for his theory, his theory was not based on them. He was wrong to use a postulate to try to prove somthing and blattner pointed that out to him.
Do you know what the definition of a postulate is....
"Something assumed without proof as being self-evident or generally accepted, especially when used as a basis for an argument"
It was koch himself who said that is was not rigid fact.
Blattner did admit that it did not follow the Koch "presumption".


champ wrote....
How about these sentences.......
"The answer to this paradox was provided in January 1995. Two reports in Nature demonstrated that the infected CD4+ cells were being destroyed as quickly as they were infected, so the measured CD4+ population in the blood of an HIV sufferer at any one time was always free of infection (see Living Marxism, December 1995)

Asteroth wrote........
This is not an answer to the Paradox...if it is... try and explain the alleged "answer" in your own words,rather than quote an explanation you might misunderstand, and demonstrate how it reconciles with the following
statement:
"It(HIV) actively infects only 1 in 104 to >105 T cells.Undr these conditions HIV can not account for the loss of T cells,the hallmark of Aids,even if all infected cells died;this is because during the 2 days it takes hiv to replicate,the body r e g e n e r a t e s,about 5% of its T cells,more than enough to compensate for the losses due to HIV.It is p a r a d o x i c


Maybe this article will help you. It was written in 1998...10years after your 1 in 104 by Duesberg(1988). It is also from the same school that Duesberg taught at.


New AIDS data explains T-cell puzzle and suggests new treatments, according to UC researchers



by Kathleen Scalise BERKELEY -- Besides preventing HIV from killing T-cells, anti-retroviral treatments may increase the body's ability to generate new cells in some patients, according to recent data from a new diagnostic technique developed by UC Berkeley and UC San Francisco researchers. The technique allows human cells, including T-cells, to be directly traced from the time they are created until they die.

T-cell death versus production is an ongoing debate in the AIDS community. Strategies for preventing or reversing immune deficiency depend on knowing what makes T-cell count fall.

Previously many researchers believed the underlying cause of AIDS, which is immune deficiency, could best be explained by abnormally high rates of cell death that forced the immune system to battle back with high rates of new cell generation. But the availability of the new technique has revealed an important role for the T-cell production systems as well. It found T-cell production rates were higher in patients on potent protease inhibitor treatments.

"Our research shows that T-cell count in AIDS patients on protease inhibitor treatments is closely related to the number of cells their body can produce each day," said Marc Hellerstein, M.D./Ph.D., associate professor of nutritional sciences at UC Berkeley and associate professor of medicine at UC San Francisco. "T-cells do have a shorter life-span in AIDS than in healthy people, but the production of new cells is also an important factor in determining T-cell counts."

Hellerstein will present the data Tuesday, February 3 at 10:45 a.m. Central Standard Time (8:45 a.m. Pacific Standard Time) at the 5th Conference on Retroviruses and Opportunistic Infections in Chicago. A description of the new technique was published in the Jan. 20 issue of the Proceedings of the National Academy of Sciences. Joseph M. McCune, M.D./Ph.D., of the Gladstone Institute of Virology and Immunology at UC San Francisco collaborated in the studies, conducted with AIDS patients at San Francisco General Hospital.

AIDS is caused by the retrovirus HIV. Like many other viruses, HIV enters human cells in order to reproduce, since viruses cannot replicate on their own. It attacks and gains entry to cells that have CD4 receptors on their surface. The most significant of these are helper T lymphocytes, or T-cells, which respond to foreign substances and perform critical functions in warding off infections.

From the time the virus is first detectable, it begins to replicate in T-cells. This replication eventually destroys the cell. However, in early stages of the infection T-cell levels generally remain near normal. This may be because the immune system is still able to churn out new cells to replace those that are destroyed. This period of the disease is the so-called dormant stage since few symptoms are apparent and it can last years.

Eventually, however, T-cells are destroyed more rapidly than the immune system can produce them, causing a drastic reduction in their number. The result is a serious impairment of the immune system and increased susceptibility to opportunistic infections, a hallmark of full-blown AIDS.

The body's T-cell stores in AIDS have been compared to a sink with the drain wide open and the faucet on full force. "The big question has been, is there a reason to turn up the faucet, if the drain is still wide open?" said Hellerstein. "Well, now we have reasons to believe that the faucet is involved, as well."

Hellerstein and McCune came to this conclusion by tagging T-cells using deuterium, or "heavy" hydrogen, to label DNA in T-cells as they are produced. Deuterium is a non-radioactive, and thus safe, hydrogen isotope double the weight of regular hydrogen. It can only be detected in newly generated DNA by mass spectrometry and affords a very reliable count of the number of new cells generated.

In the past, cell generation could not be directly measured because techniques involved radioactive or toxic chemicals unsuitable for human consumption. The availability of a safe, reliable method for measuring the rate of cell generation in humans is expected to be useful in a number of diseases.

Use of this technique in AIDS patients revealed tremendous individual variability, said Hellerstein. Some patients have an impaired T cell regeneration system, while other patients make extremely high numbers of T cells. Also, some patients responded to protease inhibitor therapy with high T-cell production rates and improvement of T-cell counts, he said, while other patients did not get a boost in their T-cell counts, despite suppressing the virus. These latter individuals were unable to increase T-cell production rates. Still other patients developed resistance of the virus to protease inhibitors, but maintained high T-cell counts. They were found to be producing tremendous numbers of T-cells each day. These different response patterns might represent clinically useful sub-groups of patients, says Hellerstein.

"Identification of patients who have good virologic control but cannot generate more T-cells may suggest the need for immunostimulatory therapies in addition to antiviral drugs, for example," he said. Such therapies might include transplanting immune stem cells or thymic tissue, or administering agents such as interleukin-2.

UC Berkeley has applied for a patent on the new technique and SpectruMedix Corporation of State College, Pennsylvania, has acquired rights to the technique. SpectruMedix is a U.S. company producing medical and scientific technologies and associated instrumentation.

The technique is expected to be useful for treatment of a wide variety of diseases, including AIDS, cancer, heart problems and osteoporosis. It allows physicians to safely and rapidly measure the efficacy of particular drug therapies in individual patients and gives physicians the ability to tailor treatment based on individual response to the drug. The test is not yet commercially available to physicians.


http://www.berkeley.edu/news/media/releases/98legacy/02_05_98b.html

notice the source post:)

try and explain the alleged "answer" in your own words,rather than quote an explanation you might misunderstand

OMG...LMAO....I cant believe you said that......do bible comparisons count as own words to you:) sorry asteroth to flame you but i just cant beleive you said that.

Champs says:
Thank you....you are pretty good at it yourself.

Asteroth says:
It would be great if you provided us with at least one example of misquotation,as you did so abundantly with Dusbergs articles...



Didnt mean to insult you...unless you were trying to insult me? Misquotations are hard to find when you do not source post. Or is that all of your origional work:)JK

Asteroth wrote..........

Duesberg has always maintained that HIV is not,and has never been the cause of immuno deficiency,in one of his articles he suggests the following concerning transfusion related Aids:"The thesis that Hiv transfusions cause Aids in other patients is also entirely uncontrolled.Indeed,a controlled study might be difficult because 50% of American patients(other than hemophiliacs)die within 1 year,and 60% within three 3 years a f t e r t r a n s f u s i o n-long before the average 10 years HIV is said(by the establishment) to require for pathogenicity have elapsed.the pathogenic conditions that necessitated the transfusions are obviously deadlier than the h y p o t h e t i c a l pathogen HIV"

Champ wrote......
OK, so there is no way to "be sure" if HIV passed in blood transfusions cause AIDS becuase there cannot be a controlled study. That doesnt mean that HIV is not the cause of immune deficiency. All it means is that you cannot have a controlled study with blood transfusion recipients....what is your point?

Asteroth wrote......
The point is that there is NO PROOF or evidence to suggests "as a matter of fact" that HIV passes in blood transfusions...Just an assumption...hypothesis..
As far as the AIDS establishment is concerned "not being
sure"is enough to warrant any HIV related issue the aura of "a fact"...that's not science but pseudo science


LOL There IS proof that HIV passes in blood transfusions, Your quote was saying that you cannot track how "AIDS develops from HIV" in blood transfusion recipients becuase more than half die off in 3 years, hence there cannot be a study done that will hold water.

And you are talking to me about not understand articles?
 
Anyone reading my recent posts,and Champ's counter posts,may come to conclude that the "Aids Controversy" is nothing but a duel between two individuals with opposing points of view.

Alas,this would certainly be an undesirable impression.

I started this thread not for the purpose of commenting on someone's intellectual ability,but rather for the purpose of bringing to notice,(particularly among HIV positive persons)the dissenting view to the HIV/AIDS hypothesis.

I am almost certain that the honorable Champ,is HIV negative,(as I am),and consequently,to the two of us,this may be just a debate(albeit,heated at times)whereas to HIV positive persons,the information presented thus far,may be a matter of grave concern... .
My responses to Champ,therefore should be viewed in the context of gathering as much information as possible,prior to determining a course of treatment which may be tantamount to life or death ...

I would prefer to think that my antagonist is as concerned as I am,regarding the "correct and proper"treatment for HIV positive individuals,and that his counter responses are not for the sake of saving face,nor for the purpose of salvaging some impenetrable "pompous arrogance"bubble,panctuated with a yellow smiley...

I apologize in advance if I may seem to some as judgemental and accusatory...it is simply a reaction and a subjective impression to what I perceive as misquotations,omissions,and total misrepresentation of the dissenting opinion.

I am pleased to read that:
"both sides have flaws which is why there is still debate about it"
simply because the"flawless" CDC in its incesant arrogance and omniscient attitude towards the dissenting opinion,will never admit to any "flaw" in the HIV/AIDS hypothesis,much less to any "debate"about its validity and alleged scientific soundness...the only debate the CDC might confirm,exists,is the one that takes place amongst the dissenters,on how to further perpetuate the "lie" that HIV does not cause AIDS...

Or maybe,Champs ment something entirely different,in which case I would ask...what "flaws"is he referring to when evoking the words"both sides have flaws"and when was the last time any CDC representative debated a dissenter in a public forum...

Henceforth,I should make every attempt to evaluate champs responses,without characterising or presuming his intentions...

Champ wrote:

"LOL There IS proof that HIV passes in blood transfusions, Your quote was saying that you cannot track how "AIDS develops from HIV" in blood transfusion recipients becuase more than half die off in 3 years, hence there cannot be a study done that will hold water".

I say:
Of course there is proof that "HIV passes in blood transfusion"

But the correct reading of my quote is:
"the point is that there is NO PROOF or evidence to suggest "as a matter of fact"that HIV that passes in blood transfusion cause AIDS,because there can not be a controlled study"

The "no proof" portion of the sentence is NOT denying passaage of HIV through transfusion,but rather the assumption that the HIV aquired through transfusion causes AIDS...
True...I didn't quote the e n t i r e sentence...but the 3 dots indicated just that..i.e that it wasn't quoted in its entierty.

Champ says..
"Why didnt Duesberg mention koch in his response to blattner. Because he used them as support for his theory, his theory was not based on them. He was wrong to use a postulate to try to prove somthing and blattner pointed that out to him.
Do you know what the definition of a postulate is....
"Something assumed without proof as being self-evident or generally accepted, especially when used as a basis for an argument"
It was koch himself who said that is was not rigid fact.
Blattner did admit that it did not follow the Koch "presumption.


response:
Since Blattner was expressing his own personal opinion and not some scientific evidence,Duesberg saw no point in arguing over a point of view..as indicated before, Blattner's point of view stands in contrast to the CDC own evaluation of Koch's postulate:

"Among many criteria used over the years to prove the link between putative pathogenic (disease-causing) agents and disease, perhaps the most-cited are Koch's postulates, developed in the late 19th century. Koch's postulates have been variously interpreted by many scientists, and modifications have been suggested to accommodate new technologies, particularly with regard to viruses (Harden. Pubbl Stn Zool Napoli [II] 1992;14:249; O'Brien, Goedert. Curr Opin Immunol 1996;8:613). However, the basic tenets remain the same, and for more than a century Koch's postulates, as listed below, have s e r v e d as the l i t m u s test for determining the cause of A N Y epidemic disease"


True...postulate is "something assumed without proof as being self evident or generally accepted,especially when used as a basis for an argument"
but ,regardless of the definition,or maybe inspite of it, Koch's postulates have been used(including by Blattner himself) in the last 100 years as a litmus test to determine causality of "a n y epidemic" disease for every virus known in existence,
Duesberg is justified in his demand that the one and the same litmus test mentioned above, be applied to determine causality of immune deficiency by HIV.

Furthermore,Duesberg does not claim that the HIV failure to pass Koch's postulates is by itself "scientific proof",but he rather implies that the HIV virus "got an F"in the CDC approved litmus test .

Unlike blattner Duesberg insists on a molecular biochemical evidence to indicate causality of disease,due to previous misdiagnosis by the scientific community:
"Based on epidemiological evidence, "scientists concluded" that Epstein-Barr virus was the cause of Burkitt's lymphoma-until the first virus-free lymphomas were found (10). (ii) On epidemiological grounds, human and bovine retroviruses were believed to cause leukemia after bizarre latent periods of up to 40 years in humans (11)-but finding these viruses in billions of normal cells of millions of asymptomatic carriers has cast doubt on this view (12). It is scarcely surprising that these leukemias arose from virus-infected cells. Consistent with this view, these "viral" leukemias are clonal and not contagious, behaving like virus-negative leukemias, and the associated "leukemia" viruses are not biochemically active (12). (iii) "Slow viruses" were accepted as causes of Alzheimer's, kuru, and Creutzfeldt-Jakob disease (13) on the basis of the same kind of epidemiology and transmission evidence used here for HIV-but these viruses have never materialized. These examples illustrate that correlations without evidence for biochemical activity are not sufficient to prove etiology"

Ignoring the above request,will not change the plain ,self evident fact,that scientists,are not infallible, and they do, sometimes, err...

Since HIV is the only virus known in existence to fail the Koch's postulates litmus test,Blattner is confronted with a dilemma,a contradiction,an outright paradox....his only way to get out of the hole he dug himself into is to cast doubt, all of a sudden, and degrade the scientifically accepted " litmus test" as nothing more than an "historical reference"which is not to adhere to "rigidly".. and therefore rely conclusively on epidemiological corrolaton(as "proof" or "evidence") that may be circumstantial...

If this is indeed blattner's true approach to Koch's Postulate; one should ask ..why did he, adhere to it so rigidly,prior to the discovery of HIV..

Champ writes:

OMG...LMAO....I cant believe you said that......do bible comparisons count as own words to you... sorry asteroth to flame you but i just cant beleive you said that. "

"maybe this article will help you. It was written in 1998...10years after your 1 in 104 by Duesberg(1988). It is also from the same school that Duesberg taught at.
New AIDS data explains T-cell puzzle and suggests new treatments, according to UC researchers "

Requesting a law professor,or a scientist,(or Champ for that matter), to paraphrase out side sources,and quotations they use in their research papers,or while lecturing in class,does not cast an iota of doubt on their intelectual capacity,or comprehension ability.Paraphrasing is utilized for the purpose of simplification and further clarification of any complex issue,and it is routinely adaptaed as a common practice.

Since in my opinion ,champ's quotation of "Kathleen Scalise's article does not solve the above mentioned T cell paradox,I invite him to further clarify the article and explain or demonstrate H O W ,in his opinion,this particular research he quoted solves the still pending discrepancies..

To begin with..
the purpose of the work quoted is to explore the hypothesis that :anti-retroviral treatments m a y increase the body's ability to generate new cells in s o m e patients"

Thus, it says absolutely nothing ,nor does it elaborate or attempts to resolve any paradoxes which it does not acknowledge as purpose of the research...

However

the problems as explained by Duesberg are:

In contrast to A L L pathogenic viruses,that cause degenerative diseases,HIV is N O T biochemically active, in the disease syndrom it is named for...It actively infects only 1 in 104 to >105 T cells.Undr these conditions HIV can not account for the loss of T cells,the hallmark of Aids,even if all infected cells died;this is because during the 2 days it takes hiv to replicate,the body regenerates,about 5% of its T cells,more than enough to compensate for the losses due to HIV.It is p a r a d o x i c a l that HIV is said to cause aids only A F T E R the onset of a n t i viral i m m u n i t y detected by a positive "Aids test",because A L L other viruses are most pathogenic b e f o r e immunity.

Thus:

The quoted research did not prove or demonstrated that HIV is biochemically active in the disease syndrom it is named for..

Did not prove that HIV infects more than just 1 in 104>105 T cells,exactly as it did 10 years ago...

Did not demonstrate how under the above mentioned conditions,HIV can account for the loss of T-cells,the hallmark of Aids.

Did not disprove the fact that "during the 2 days it takes hiv to replicate,the body regenerates,about 5% of its T cells,more than enough to compensate for the losses due to HIV."

Did not elaborate on the paradox of h o w is it possible that HIV causes AIDS A F T E R the on set of antiviral immunity (detected by a positive "aids test" )when virtually A L L other viruses are most pathogenic b e f o r e immunity"

In Short the article quoted by Champ,in an attempt to cast new and "current" light (by the same school that Duesberg taught at)on the problems stated above is irrelevant on its entirety...

Moreover..

The article itself presents us with a plethora of unresolved and disputed comments,"as though they were facts"

Consider this:

Previously many researchers believed the underlying cause of AIDS, which is immune deficiency, could best be explained by abnormally high rates of cell death that forced the immune system to battle back with high rates of new cell generation. But the availability of the new technique has revealed an important role for the T-cell production systems as well. It found T-cell production rates were higher in patients on potent protease inhibitor treatments.

question:

Why was T-Cell production higher?

Response by

Dr. Mohammed Al-Bayati (see my first post for the full interview with DR Al Bayati)

Protease inhibitors usually cause severe damage in kidney, liver, and other organs. The severity of the damage depends upon the amount of the drugs taken and the duration of use. Some studies show that the CD4+ T cell counts were increased after treatment with AZT and/or protease inhibitors. This information was interpreted as a good response to the medications. On the contrary, the elevation of T cells is not a good response in these conditions, but rather, it indicates severe tissue damage and infection because elevation of CD4+ T cells counts also occur due to inflammation in tissues. This explains the injury and death of the patients following treatment with these drugs. For example, the CD4+ T cell counts were increased following the treatment of HIV negative nurses with AZT, who took AZT as a prophylactic. They developed severe symptoms following 3 weeks of treatment with AZT (Get All The Facts: HIV does not cause AIDS, Table 24). In addition to the failure of the antiviral drugs, AIDS patients suffering from immune deficiency are also treated with glucocorticoids. This practice is not supported by any known mechanism of action. The antiviral medications and the glucocorticoids not only fail to cure AIDS, but they cause severe damage to sick people with AIDS. Prescribing these medications to AIDS patient is just like putting gasoline on a fire. "

furthermore,

See "Protease inhibitors have no unique effect on CD4 cell counts"as published by Reuters July 24th 2000
(Champs Article is dated:NEWS RELEASE, 02/05/98)
"PROTEASE INHIBITORS HAVE NO UNIQUE EFFECT ON CD4 CELL COUNTS
Reuters 24 July 2000

Westport -- Triple therapy for HIV-infected patients, including regimens containing a protease inhibitor, do not have any unique effects on CD4 cell counts independent of reductions in plasma viral load, according to findings reported in the July issue of AIDS.
The data appear to contrast with recent evidence suggesting that such regimens are able to maintain an immunologic benefit even after plasma viral rebound, Dr R. Harrigan, of St Paul's Hospital in Vancouver, British Columbia, and other investigators for the AVANTI and INCAS studies explain.
The team examined the correlation between CD4 cell counts and plasma viral load over 52 weeks using data from 3 randomized clinical trials: AVANTI-2, AVANTI-3, and INCAS. The studies compared dual nucleoside therapy with triple combination therapy that included a protease inhibitor, with or without a nonnucleoside reverse transcriptase inhibitor.
"The data presented in these randomized double-blinded trials suggest that the specific antiretroviral regimen used neither increases nor decreases the strength of the correlation between the change in CD4 cell count and the change in plasma viral load," Dr Harrigan and colleagues say. The strength of the correlation was similar among all groups of patients in the studies, regardless of the type of treatment they received.
Discordant increases or decreases in plasma load and CD4 cell counts did occur in some patients, but they were not linked to any particular therapeutic regimen and were regarded as "natural variation" by the authors.
"The data provide no evidence for a CD4 cell count benefit unique to protease inhibitors," the investigators conclude, "at least in first-line regimens up to 1 year in patients with moderate baseline CD4 cell count."
AIDS. 2000;14:1383-1388.

Evaluate this "facts" for example:

"AIDS is caused by the retrovirus HIV. Like many other viruses, HIV enters human cells in order to reproduce, since viruses cannot replicate on their own. It attacks and gains entry to cells that have CD4 receptors on their surface. The most significant of these are helper T lymphocytes, or T-cells, which respond to foreign substances and perform critical functions in warding off infections.

From the time the virus is first detectable, it begins to replicate in T-cells. This replication eventually destroys the cell. However, in early stages of the infection T-cell levels generally remain near normal. This may be because the immune system is still able to churn out new cells to replace those that are destroyed. This period of the disease is the so-called dormant stage since few symptoms are apparent and it can last years.

Response:
In my previous posts,I have outlined all that is currently known about retroviruses,in response to one of Champs comments regarding retroviruses,however,since he chose not to reply to that information,I shall present it again:

"The hallmark of all retroviruses is that they do not kill cells that they infect. HIV is the only retrovirus that is asserted to kill its host cell. Several researchers, including HIV discoverer Luc Montagnier, have found that HIV does not kill its host cell in laboratory tests. "

"Retroviruses are typically not cytocidal. On the contrary, they often promote cell growth. Therefore, they were long considered the most plausible viral carcinogens (9). Yet HIV, a retrovirus, is said to behave like a cytocidal virus, causing degenerative disease killing billions of T cells (15, 18). This is said even though T cells grown in culture, which produce much more virus than has ever been observed in AIDS patients, continue to divide (9, 10, 18). 8) It is paradoxical "

and the most startling comment concerning the correct
understanding of "killing of cells"by HIV

"A fourth paradox of the HIV hypothesis has been noted by several virologists. HIV belongs to a class of viruses known as the retroviruses, which are very simple in structure and contain much less genetic information than most other viruses. Most types of viruses are lytic, meaning that they kill the cells they infect and thereby cause disease. Retroviruses, on the other hand, do not generally kill cells. Upon infecting cells, they copy their genetic information into the DNA of their new host cells. From that point forward, retroviruses depend on allowing their host cells to continue living, while they slowly produce new virus particles that are ejected from the cell. Retroviruses are therefore poor candidates to blame serious diseases on, particularly fatal conditions involving the deaths of huge numbers of cells, such as AIDS. Indeed, some 50 to 100 latent retroviruses have been found to reside in the DNA of all humans, passed along to each successive generation for as long as human beings have existed. Past research by Harry Rubin has shown that retroviruses cannot infect any cells that do not divide. Neurons in the human brain do not divide after the first year of life, so HIV cannot possibly infect those cells. This would explain why HIV has not been isolated from these cells, and confirms the difficulty it would also face in causing dementia. Harvey Bialy, research editor of the professional journal Bio/Technology, argues that the simple genetic structure of HIV does not differ sufficiently from other retroviruses to account for its supposedly different behavior. The genetic information carried by HIV is not unusual for retroviruses; it contains no gene different enough from the genes of other retroviruses to be a possible "AIDS gene." In addition, HIV uses all of its genetic information when it first infects, rather than saving some to be used years later. In other words, there is no conceivable reason HIV should causes AIDS 10 years after infection, rather than early on when it is unchecked by the immune system.

The virus-AIDS hypothesis also totally fails to explain how the virus depletes T-helper cells, and why it takes at least 3-5 years to do so. Unlike all other animal viruses, retroviruses need mitosis to initiate infection. Moreover, no HIV gene remains inactive during replication, which takes about 1-2 days, as with all other retroviruses. Thus HIV would be expected to kill T cells and cause AIDS when it first infects an organism and not 5 years later when it is biochemically inactive and suppressed by antiviral immunity. The 5-year latency presents proponents of the hypothesis with two bizarre options: either old T cells die 5 years after infection, or the offspring must die in the 50th generation, given a one-month generation time for the average T cell. Nevertheless, killing of T cells within weeks, not years, after infection has been observed in cell culture-in apparent agreement with the claim that the virus kills T cells. But this type of killing is fundamentally different from the unconditional cell lysis achieved by true cytocidal viruses. It involves cell fusion mediated via HIV antigens on the surface of infected cells, and is conditional on the cells and virus isolates used. Further, it does not occur in chronically infected human T-cell lines that grow indefinitely in culture yet produce more virus than any other system, nor has it ever been observed in blood taken from an AIDS patient. In fact, limited cell killing by fusion is a common feature of retroviruses, none of which have as yet been claimed to cause AIDS.

Response by :Dr. Mohammed Al-Bayati (see my first post for the full interview with DR Al Bayati)

"Robert Gallo stated that HIV enters CD4+ T cells because they have special receptors for HIV and that HIV kills CD4+T cells selectively. I have found no truth for this hypothesis. Most individuals infected with HIV show hyperplasia of all cell components of lymph nodes (It has more cells than normal). In addition, HIV is present in all cells in the lymph nodes. Our government's decision of basing the entire AIDS program on the HIV-hypothesis is a very dangerous and costly decision. This faulty decision has been resulting in the exposure of millions of people to very toxic antiviral drugs worldwide unnecessarily and wasting billions of dollars. "



and finally in response to the following statement:

"Eventually, however, T-cells are destroyed more rapidly than the immune system can produce them, causing a drastic reduction in their number. The result is a serious impairment of the immune system and increased
susceptibility to opportunistic infections, a hallmark of full-blown AIDS"

Answer:

How is possible...what kind of biochemical proof does Kathleen Scalise's possesses that would allow such bogus statements,to be presented as though they were virtual facts..

Read the following:

When HIV first infects a person, it can reach moderately high concentrations in the blood - yet AIDS never shows up at that time and T cell levels remain normal. Within days or weeks, the immune system makes antibodies against HIV, and the virus quickly disappears, from the blood. Years later, if AIDS shows up at all, the virus rarely comes back to life and multiplies again. In other words, AIDS never strikes a patient until years after the active virus has been permanently eliminated from the body. This strongly suggests that AIDS is caused by something else.

"The immunity against HIV is so effective that free viruses is undetectable,which is why HIV is hard to transmit...HIV remains INACTIVE during Aids,thus the "Aids test"identifies effective natural vaccination,the ULTIMATE PROTECTION against viral disease...

conclusion:
I could challenge Champ,who has no medical scientific credentials, to question himself whether he truly believes he knows better than Dr Walter Gilbert(Professor in molecular biology,1980 Nobel prize for chemistry),or Dr Kary Mullis(Biochemist,winner of Nobel prize for Chemistry)who discard outright the alleged scientific validity of the HIV/AIDS hypothesis,but Champ will retort right back at me(and he be correct in doing so)with the exact same argument,since I am not a certified scientist my self...

who am I then,to challenge Blattner...

A dilemma indeed!
whom are we to believe...?!

At first,and prior to studying and investigating the dissenting opinion,I brushed it aside,as a crock pot phenomenon...than,upon closer look,I was alarmed to discover that the dissenting camp includes some of the brightest minds in medical science,and that the list was ever so growing...

Furthermore,it became clear to me,that the CDC and the pharmaceutical companies,are engaged in a vicious campaign to silence Duesberg and collegues,rather than allocate even a fraction of the vast amount of billions of dollars( in AIDS research money)to explore the dissenting Drugs/AIDS hypothesis.

Indeed,rather than,propel the scientific investigative spirit,allow it to prevail,and award the necessary funds for the dissenting camp,the CDC and its retinue of Yes sayers have assumed the role of the "grand inquisitor",suffocating opposition with blatant threats,dismissals,loss of jobs,and withrawal of funds from Dr's and researchers who dare exhibit,or cast doubt on the HIV/AIDS hypothesis.

Truly,the CDC's behaviour suggests,it probably has something to hide,and in fear of losing it's credibility, it would rather reinvent Aids over and over again,than admit to the p o s s i b i l i t y that the HIV/AIDS hypothesis might be flawed.

("they have been changing their hypothesis and the stated incubation period for HIV, to make them fit new findings. AIDS in 1989 was much more complicated than the AIDS in 1984, prior to the approval of AZT. AIDS in 2001 has become more complicated than the AIDS in 1989, prior to the approval of steroid and protease inhibitors in the treatment of AIDS"from Mohamed Al bayati..see 1st post)

The profiteering money greedy pharmaceutical companies,while shedding crocodile tears,must ensure a hefty return on their 20 year investment,regardless of the possibility that the medications they drug asymptomatic hiv positive persons with,may eventually cause the immune deficiency syndrom they're supposed to prevent.

Their loyalty will forever lie with the mighty dollar God,and the ever pressing share holders...the ailing patients seem to be a nuisance,they have no other choice but contend with...

This mixture of science, money,politics greed , false arrogance and self importance... ultimately lead to an established inquisition,proliferating bogus and unsubstantiated "scientific facts''.

Be blessed.











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the plain Truth aboutHaemophiliacs


Haemophiliacs did not die of HIV contracted from blood factor products, but principally from drug side-effects, an international group of scientists has claimed in a new sumission to the Lindsay Tribunal, Thursday 21st June.

The AidsMyth voluntary group based in Ireland, incorporates five Aids-rethinker scientists of international repute appointed to South African President Mbeki's Advisory Panel on HIV/AIDS.

Their submission says side-effects of steroid drugs prescribed to haemophiliacs are the same as the list of medical conditions from AIDS. Similarly, they claim that side-effects of the anti-HIV drugs prescribed to haemophiliacs cause symptoms attributed to AIDS.

Haemophilliacs can be false-positive on HIV tests according to the scientists. They provide evidence to the Tribunal that blood products could not have transmitted infectious HI virus to haemophiliacs. And they say the US Centres for Disease Control has made statements consistent with their findings.

They assert that the principal causes of observed morbidity and mortality in haemophiliac patients included the immune-suppressing effects of blood factor therapy itself; side-effects of prescribed cortico-steroids or pneumonia medication; and fatal side-effects of anti-HIV drugs erroneously prescribed as a result of incorrect HIV/AIDS diagnosis.MORE


The plain truth about haemophiliacs
 
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