Ok he said it was something to do with the" hemodynamic response", whatever that is. In fact heres the summary of the study from June 2010. If you've got any more recent and credible research to prove that what Im feeling while 'on' is all just make believe please post. In the end only i know how my body feels while on, I wish it didn't have this effect on me but it does.
Through multiple cycles it has done this every time regardless of the compound (with the partial exception a S4) So I set about looking for reasons why? Perhaps this will help lesser experienced guys make a a better choice than I did in assuming AAS would improve my ability to run and swim long distances.
Since I cant post the link here is the study:
Acute cardiovascular response in anabolic androgenic steroid users performing maximal treadmill exercise testing
Journal of Strength & Conditioning Research:
June 2010 - Volume 24 - Issue 6 - pp 1688-1695
doi: 10.1519/JSC.0b013e3181dc46c9
Original Research
Maior, AS, Simão, R, Salles, BF, Alexander, JL, Rhea, M, and Nascimento, JHM. Acute cardiovascular response in anabolic androgenic steroid users performing maximal treadmill exercise testing. J Strength Cond Res 24(6): 1688-1695, 2010-The purpose of this study was to investigate the cardiovascular effects of anabolic androgenic steroid (AAS) use, specifically the hemodynamic response, during maximal treadmill exercise testing by comparing the exercise response between users of AAS (U-AAS) and non-AAS users (N-AAS). Twenty-four men (n = 12; 29 ± 3.4 years and n = 12; 29.5 ± 8.2 years for the U-AAS and N-AAS groups, respectively) with regular participation in both resistance (mean = 6 d·wk−1) and aerobic exercise (mean = 2 d·wk−1) volunteered for the study. Both groups of subjects completed a ramp-protocol maximal treadmill exercise test to volitional fatigue. Several hemodynamic and metabolic measures were obtained before, during, and after testing. The results demonstrate for the first time that chronic administration of high doses of AAS (355.4 ± 59.47 mg·wk−1) lead to hemodynamic and metabolic response impairment. In conclusion, the chronotropic significant incompetence in the current study was reflected by an exaggerated hemodynamic response to exercise. Furthermore, the findings suggest that nonusers of AAS showed increases in V̇o2max when compared to the AAS group. Therefore, this study provides a contraindication to AAS use, especially in those at increased risk of cardiovascular events.