pursuit said:
Hey dude,
It is actually estrogen that stops people growing in their late teens. People that have a genetic defect that cause them not have any estrogen (ie aromatase defiecency), actually grow to be very very tall and growth plates usually close in the late 20s or never.
So I would suggest trying an aromatase inhibitor (AIFM, Arimidex, letroloze).
This may help you to grow a bit more. You will not grow anymore if you're growth plates have closed. Similarly, it wouldn't have much effect if they are about to close - as you are 18 this probably the case.
Hi, just thought I'd back up my comments with this study...
ORIGINAL ARTICLE
Treatment with the aromatase inhibitor letrozole during adolescence increases near-final height in boys with constitutional delay of puberty
Matti Hero*, Sanna Wickman*and Leo Dunkel†
Summary
Objective We investigated whether inhibition of oestrogen biosynthesis with the aromatase inhibitor, letrozole, during adolescence improves near-final height in boys with constitutional delay of puberty.
Patients and methods Seventeen boys with constitutional delay of puberty were randomized to receive testosterone (T) enanthate (1 mg/kg i.m.) every 4 weeks for 6 months in combination with placebo (Pl, n = 8), or the aromatase inhibitor letrozole (Lz, 2·5 mg/day orally) (n = 9), for 12 months. After treatment, patients were followed up until near-final height. Height discrepancy was calculated as near-final height minus mid-parental target height.
Measurements The primary end point was the difference in near-final height between the groups treated either with T + Pl or T + Lz. Secondarily, height discrepancy and gain in height standard deviation score (SDS) were analysed in both groups.
Results Boys treated with T + Lz reached a higher mean near-final height than did boys on T + Pl (175·8 vs. 169·1 cm, respectively, P = 0·04). In T + Lz-treated boys, mean near-final height did not differ from their mid-parental target height (175·8 vs. 177·1 cm, P = 0·38), whereas in T + Pl-treated boys, mean near-final height was lower than mid-parental target height (169·1 vs. 173·9 cm, P = 0·007). T + Lz-treated boys had a greater increment in height SDS over the pretreatment height SDS than T + Pl-treated boys (+1·4 SDS vs.+0·8 SDS, P = 0·03).
Conclusions Our findings indicate that in adolescent boys an increase in adult height can be attained by use of aromatase inhibitors.