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Mini Viper
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Mainly for newbies who don't know what PCT is:
Recovery of natural testosterone post cycle:
A brief run down of the body’s mechanism of producing testosterone:
Hypothalmus:
Using current and recent hormone levels this part of the brain releases LHRH – Luteinising Releasing Hormone, and
GNRH – Gonadotrophin Releasing Hormone
In turn these then act on a part of the brain called the:
Pituitary:
Here LHRH stimulates the Pit. To release LH
GHRH stimulates the Pit. To release FSH.
LH and FSH then act in the..
The Hypothalmus and Pituitary are otherwise known as the HPTA
Testes:
LH stimulates the leydig cells in the Testes to produce Testosterone
FSH stimulates the testes to produce Sperm.
Inhibition
Inhibition acts on all 3 levels of production,
Hypothalmus sensing high androgens releases less LHRH and GNRH
Pituitary both from reduced LHRH and GNRH, and also excessive estrogen from steroid use and
also insufficient estrogen from using too much anti-aromatose (femera and arimidex) as well as purely non aromatisable steroids which drive estrogen down.
This has the effect of down regulating the sensitivity of the Pituitary to LHRH so it is a double sword of inhibition.
Testes:
Without sufficient LH produced by the pituitary the Testes shut down and atrophy and no natural testorone is the end result.
PREVENTING INHIBITION:
A) Minimising the effect on the HPTA
You cannot prevent inhibition at the HPTA but you can limit the effect somewhat by keeping estrogen under control.
If using large amounts of aromtising steroids, test, dianabol etc, using moderate amounts of anti-aromatose to keep estrogen in the normal range is wise. How do you know this – by blood tests.
Usually 0.25mg of Arimidex ED per 500mg of aromatising steroid is sufficient here.
So for example, 750mg of testosterone/week and 30mg of Dianabol ED = approx 1000mg of aromatising steroid so a dose of 0.5mg ED of Arimidex would be used
B) Minimising the effect on the Testes during the cycle
When the testes atrophy especially for long periods it means post cycle there is substantial lag in picking up hormone production. It is far better to prevent rather cure Testicle atrophy.
A drug called HCG is used to do so. This mimics the effects of LH on the Testes, meaning despite using steroids, the Testes continue to produce testosterone, and donot atrophy, meaning post cycle they are up and running for a much better and fuller recovery.
To do this a regime of 500iu of HCG used twice each week for the duration of the cycle, ceasing its use 10 days before starting Clomid.
This is so that the boost in Testosterone Hcg causes can subside allowing recovery of the HPTA whilst being used as long as possible to prevent atrophy.
C) Clomid Use to Restore the HPTA
The HPTA – the brain is the starting point for testosterone production, and without getting this crucial part back online you will not recover as fully or quickly.
As well as Testosterone and other ‘male’ steroids, the HPTA also uses levels of estrogen to regulate Testosterone production…
Estrogen is produced in the male by the aromatisation of testosterone to estrogen through the aromatose enzyme.
The HPTA sensing high estrogen assumes levels of Testosterone must be too high and ceases or reduces LH production.
To our advantage when the body senses low estrogen it ups the production of LH in the brain.
To achieve this effect anti estrogens, clomid and nolvadex are used
These have the effect of blocking the reception of estrogen in the HPTA so the brain is tricked into thinking LOW estrogen therefore BOOST LH and consequently Testosterone.
Peace
Recovery of natural testosterone post cycle:
A brief run down of the body’s mechanism of producing testosterone:
Hypothalmus:
Using current and recent hormone levels this part of the brain releases LHRH – Luteinising Releasing Hormone, and
GNRH – Gonadotrophin Releasing Hormone
In turn these then act on a part of the brain called the:
Pituitary:
Here LHRH stimulates the Pit. To release LH
GHRH stimulates the Pit. To release FSH.
LH and FSH then act in the..
The Hypothalmus and Pituitary are otherwise known as the HPTA
Testes:
LH stimulates the leydig cells in the Testes to produce Testosterone
FSH stimulates the testes to produce Sperm.
Inhibition
Inhibition acts on all 3 levels of production,
Hypothalmus sensing high androgens releases less LHRH and GNRH
Pituitary both from reduced LHRH and GNRH, and also excessive estrogen from steroid use and
also insufficient estrogen from using too much anti-aromatose (femera and arimidex) as well as purely non aromatisable steroids which drive estrogen down.
This has the effect of down regulating the sensitivity of the Pituitary to LHRH so it is a double sword of inhibition.
Testes:
Without sufficient LH produced by the pituitary the Testes shut down and atrophy and no natural testorone is the end result.
PREVENTING INHIBITION:
A) Minimising the effect on the HPTA
You cannot prevent inhibition at the HPTA but you can limit the effect somewhat by keeping estrogen under control.
If using large amounts of aromtising steroids, test, dianabol etc, using moderate amounts of anti-aromatose to keep estrogen in the normal range is wise. How do you know this – by blood tests.
Usually 0.25mg of Arimidex ED per 500mg of aromatising steroid is sufficient here.
So for example, 750mg of testosterone/week and 30mg of Dianabol ED = approx 1000mg of aromatising steroid so a dose of 0.5mg ED of Arimidex would be used
B) Minimising the effect on the Testes during the cycle
When the testes atrophy especially for long periods it means post cycle there is substantial lag in picking up hormone production. It is far better to prevent rather cure Testicle atrophy.
A drug called HCG is used to do so. This mimics the effects of LH on the Testes, meaning despite using steroids, the Testes continue to produce testosterone, and donot atrophy, meaning post cycle they are up and running for a much better and fuller recovery.
To do this a regime of 500iu of HCG used twice each week for the duration of the cycle, ceasing its use 10 days before starting Clomid.
This is so that the boost in Testosterone Hcg causes can subside allowing recovery of the HPTA whilst being used as long as possible to prevent atrophy.
C) Clomid Use to Restore the HPTA
The HPTA – the brain is the starting point for testosterone production, and without getting this crucial part back online you will not recover as fully or quickly.
As well as Testosterone and other ‘male’ steroids, the HPTA also uses levels of estrogen to regulate Testosterone production…
Estrogen is produced in the male by the aromatisation of testosterone to estrogen through the aromatose enzyme.
The HPTA sensing high estrogen assumes levels of Testosterone must be too high and ceases or reduces LH production.
To our advantage when the body senses low estrogen it ups the production of LH in the brain.
To achieve this effect anti estrogens, clomid and nolvadex are used
These have the effect of blocking the reception of estrogen in the HPTA so the brain is tricked into thinking LOW estrogen therefore BOOST LH and consequently Testosterone.
Peace
