From their website:
"Pentabosol™ is a proprietary blend containing specific synergistic ratios of the active ingredients carnitine, hydroxycitric acid, chromium, aspartic acid, and biotin in a pleasant tasting, convenient powder to be taken twice daily on an empty stomach, preferably on first arising and just before retiring. The synergistic actions of these safe and natural nutrient ingredients increase the rate of fat loss while dieting both by enhancing the delivery of fat to the mitochondria for burning and by uncoupling the oxidative-phosphorylative pathway by several mechanisms.
Pentabosol™ appears to work via three mechanistic steps with the potential for interactive synergism:
Pentabosol™ turns on the fat burning process in a natural fashion, not by promoting lysis of adipose tissue with an increase of FFA (free fatty acid) acting as the driving force behind increased fat oxidation, but rather by activating the rate limiting step in mitochondrial fat burning pathways through stimulation of carnitine synthase and the carnitine fatty acid shuttle.
Pentabosol™ works in synergy with low-carbohydrate-diet-induced hormonal changes (specifically lowered levels of circulating insulin, an improved insulin-to-glucagon ratio, and lower blood glucose) to maximally disinhibit fatty acid oxidation. This generates high-energy electrons at a higher rate than hepatic ADP is generated and, hence, uncouples hepatic fatty acid oxidation from hepatic energy requirements. This ketogenic process is thermogenic and partially uncouples the pathway of fat oxidation and respiratory chain ATP synthesis. The increased fatty acid oxidation drives the activation of pyruvate carboxylase and facilitates the intramitochondrial transport of aspartate and the subsequent metabolism of aspartate to oxaloacetate (OAA) and on to malate. Malate carries the glut of the high energy electrons being generated into a futile electron transport cycle (which is also thermogenic) and this drives the gluconeogenic process which consumes energy (as ATP), thus further uncoupling oxidative-phosphorylation pathways and acting to increase hepatic thermogenesis. By increasing gluconeogenesis, hepatic glycogen stores are expanded and this decreases appetite.
By administration of biotin, glycolitic tone is increased. Because fat oxidation drives gluconeogenic pathways in the liver simultaneously, this creates a number of futile carbohydrate metabolic cycles that consume ATP and further uncouple oxidative-phosphorylation pathways thus acting in a thermogenic fashion, with calories lost as heat.
Since the three steps in the liver a) increased fat burning, b) increased electron shuttle, and c) increased gluconeogensis and futile carbohydrate cycles drive each other, the following metabolic effects occur: 1) lowered respiratory quotient (RQ), an indicator of increased fat burning, 2) increased thermogenesis, and 3) increased hepatic glycogen stores to decrease appetite and increase energy. "
This stuff is garbage, get yourself some ala and eca and you'll see better results.
Unfortunately, no drug is going to do the work for you =/. A starve diet would be stupid as you said. Work out a balanced diet and multiply your weight x 12 to get your BMR, or if you have the discipline, try out one of Mr. X's ckd diets as it will lead to the most rapid fatloss.
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